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Pharmacology > Cardio: Antidysrhythmic Drugs > Flashcards

Cardio: Antidysrhythmic Drugs Flashcards

1
Q

How does Atropine increase HR?

What receptors does it act on?

A
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2
Q

What are the Class III drugs?

A
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3
Q

Give the Clinical Application of Verapamil

A

Note: More useful than digoxin for rate control in atrial fibrillation due to the Ca+2 channel blockade in AV node persisting during sympathetic stimulation.

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4
Q

Give the PKs and ADRs for Propranolol

A
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5
Q

What is the general mechanism of Class Ib drugs?

What happens to phase 3, APD, and QT?

What conditions are they useful in treating?

A
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6
Q

What are the Class IV Agents?

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7
Q

What is the Class II Agent?

A

Propranolol

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8
Q

What are the Class Ia Agents?

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9
Q

Describe the PKs of Procainamide

A
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10
Q

What is Ventricular Re-entry, and what kind of Disorders of Impulse is it?

A

Disorder of Impulse Conduction

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11
Q

What are the ADRs Quinidine?

A
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12
Q

Label each action potential as either

  • Atrium
  • Ventricle
  • SA node
  • AV Node
  • Purkinje Fibers
A
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13
Q

What are the Pharmacokinetics for Digoxin?

A
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14
Q

Give each of the following the title as

  • ST segment
  • P Wave
  • QT Interval
  • QRS Complex
  • T Wave
  • PR Interval
A
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15
Q

What are the General mechanisms for Class II drugs?

A
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16
Q

Concerning AV Nodal Blocks, describe the difference in 1st, 2nd and third degree. What kind of Disorders of Impulse are they?

A
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17
Q
  • What is the mechanism for Lidocaine?
  • What is Lidocaine used to treat?
  • What does Lidocaine do to the ERP and APD?
A
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18
Q

Concerning Class Ic drugs

  • What happens to QRS, APD, QT, and PR?
  • What happens to phase 0?
  • What is special about the mechanism kinetics?
A
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19
Q

What are the ADRs for Amiodarone?

A
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20
Q

What is the general mechanism for all Class I Antidysrhythmic drugs?

A

Block voltage-sensitive Na+ channels

21
Q

Concerning Quinidine

  • What Clinical Applications does it have?
  • What does it do to the QRS and QT?
A
22
Q

What are the main Ventricular (alliterations at the ventricle muscle mass) Dysrhythmias?

A
23
Q

What are the ADRs for Verapamil?

A
24
Q

What does the Cardiac Action Potential look like for the Atria, Purkinje fibers, and Ventricles?

What about the SA and AV nodes?

A
25
Q

What are the PKs of Lidocaine?

A
26
Q

Describe the general mechanism of Verapamil

  • What does it do to HR, SA automaticity, PR, AV conduction, CO, and ventricular contractility?
A
27
Q

Describe what Flecainide does to cardiac conduction, and the Clinical Applications for it

A
28
Q

Give the PKs for Verapamil

A
29
Q

What are the three major Consequences of Dysrhythmia?

A
30
Q
  • What is the general mechanism for Class Ia Antidysrhythmic drugs?
  • What do they do phase 0? phase 2? phase 3? How?
  • What happens to QRS and QT?
A
31
Q

What are the ADRs for Digoxin?

A
32
Q

Concerning the Causative Mechanisms for Dysrhythmia Generation, describe Disorders of Impulse Formation.

A
33
Q

What are the ADRs of Procainamide?

A
34
Q

Concerning Ibutilide

  • What is different about its mechanism compared to other drugs of the same class?
  • What effect does it have on MAP, HR, QRS, and PR?
  • What is notable about its PKs?
A
35
Q

What is Sotalol used to treat and channel does it effect that is unique among other NS Beta Blockers?

A
36
Q

Concerning Class III Agents

  • Give the general mechanism
  • What they do to phase 3 and QT
  • What they are generally useful for
A
37
Q

How do you manage Sinus Tachycardia?

A
38
Q

What kind of Disorders of Impulse are DADs or Delayed After- Depolarizations?

A
39
Q

What are the Clinical Applications for Digoxin?

  • What does it do to Na+/K+-ATPase?
  • What happens to ERP?
  • What happens to P-wave depolarizations?
A
40
Q

What are the main Supraventricular (alliterations at or above the level of the AV NODE) Dysrhythmias?

A
41
Q

Give the general effects and Clinical Applications for Propranolol

A
42
Q

What condition is Diltiazem useful for?

A
43
Q

Give the PKs for Amiodarone

A
44
Q

Concerning Amiodarone

  • What is the general mechanism
    • What does it do to ERP and APD
  • What the Clinical Applications
A
45
Q

Concerning Class IV Agents

  • Give the general mechanism
  • What it does to the SA Node and AV Node, and ventricular
    contractility
A
46
Q

What are the Class 5 drugs?

A
47
Q

What is Digoxin?

A

Class 5 Antidysrhythmic

Cardiac Glycoside

48
Q

Compare Verapamil and digoxin for use in controlling A-Fib.

A

Pharmacology (7 decks)

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