Cardiac Rhythms Flashcards

1
Q

An irregularity or disturbance of the normal electrical activity of the heart
abnormal rhythm

A

dysrhythmia

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2
Q

taking a breath causes what type of pressure

A

negative

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3
Q

steps in the cardiac conduction system

A

1) SA node initiates rate
2) AV node relays atrial impulse to ventricles
3) bundles of His
4) Purkinje fibers- ventricular myocardium

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4
Q

one square on EKG paper is how many seconds

A

0.04 sec and 1 mm

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5
Q

needed for the diagnosis of heart muscle damage, enlargement of a cardiac chamber, and interruption of some portions of the cardiac conduction system

A

12 Lead EKG

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6
Q

describe a 12 lead EKG

A

looks at heart from 12 different angles

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7
Q

a print-out of a single lead or view

A

rhythm strip

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8
Q

single lead usually is sufficient for identifying basic

A

dysrhythmias

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9
Q

used for constant 24-hour monitoring in intensive care situations

A

single leads

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10
Q

how do you calculate the rate on an EKG

A

count R complex in 6 sec strip and multiply by 10

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11
Q

easy way to remember telemetry

A

White on Right, Clouds over Grass; Smoke over Fire and Christmas Tree on the bottom

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12
Q

every 15 large blocks is how many seconds

A

3 sec

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13
Q

simple way to estimate rate

A

from one R to the next how far apart? Each large square

300, 150, 75, 60, 50, 43

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14
Q

are small pauses in heart rate normal

A

yes

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15
Q

Dubin’s method can only be used if

A

the rhythm is regular

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16
Q

depolarization of the chambers _____ their contraction

A

provokes

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17
Q

When the heart is resting between “beats”, the EKG machine draws a straight line on the paper

A

isoelectric line

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18
Q

first deflection in the normal cardiac cycle

A

p wave

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19
Q

what does the p wave represent

A

depolarization of the atria

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20
Q

why does the depolarization of the SA node appear on EKG

A

does not generate enough voltage to be seen on the EKG, we assume it occurred to initiate the cycle

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21
Q

After the p wave…

A

there is a brief return to the isoelectric line, then the QRS complex

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22
Q

what does QRS complex represent

A

depolarization of ventricles

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23
Q

does the QRS normally point upwards or downwards

A

could be either, but all the QRS complexes should be pointed in the same direction

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24
Q

what is the duration of the QRS complex

A

0.04- 0.12 sec or about 1-3 blocks

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25
what does the T wave represent
reploarization of ventricles
26
is atrial repolarization seen on an EKG
no because the voltage is so low, however it occurd during the QRS complex
27
what occurs during the PR interval
atria contract/depolarization | (0.12-0.20) or about 3-5 blocks
28
what organs are nourished by blood first in circulation
heart and brain
29
what three things do you look at when diagnosing heart dysrthythmias
rate, rhythm, complex
30
a small increase in what electrolyte will cause the heart to stop
potassium (K)
31
what are some typical causes of dysrhythmias
Hypoxia Ischemia Sympathetic stimulation- stress, trauma, CHF, hyperthyroidism Medications-antiarrhythmics, bronchodilators, cardiac glycosides, etc. Electrolyte disturbances- K, Ca and Mg Stretch or hypertrophy
32
describe a normal sinus rhythm
rate: 60-100 or about 75 rhythm: regular PR: upright (0.16) QRS: 0.06
33
describe sinus bradycardia
``` SLOW rate: 23 or < 60 rhythm: regular PR: 0.24 QRS: 0.12 ```
34
how do you treat sinus bradycardia
atropine or pacemaker
35
describe sinus tachycardia
``` FAST, poor fill time rate: 150 rhythm: regular PR: 0.16 QRS: 0.04 ```
36
describe premature atrial contraction (PACs)
not super concerning, just keep an eye on *they are usually evident in atrial dysrthymias P wave comes early rhythm is irregular PR: 0.16 QRS: 0.06
37
describe atrial tachycardia
eventually if left untreated, lungs will fill with fluid fast rate, cells are overriding the AV node rate: 150-300 *P wave: differs in shape* PR: short, < 0.12
38
treatment for atrial tachycardia
beta blockers and calcium channel blockers
39
describe supraventricular tachycardia (SVT)
rate: rapid about 160 cause SOB, chest pain coming from atria
40
what is SVT treated with
Metoprolol or *adenosine (slows heart)
41
describe atrial flutter
hypertrophy of the heart *all blood is not emptied out of heart atrial rate: 250-300 *P waves look like FLUTTERS or SAW TOOTH
42
what is used as treatment for many dysrhthmias if other measures don't work
cardioversion therapy
43
describe atrial fibrillation
``` *most common dysrhythmia* low blood pressure, SOB "quivering", not kicking heart beat no discernible Ps rhythm: always IRREGULAR rate: about 70 wavy baseline ```
44
initial drug treatment for atrial fibrillation
Amiodarone | Dilitiazem
45
broadly describe what heart block is
longer period from SV to AV | ALL types are slow (brady)
46
describe first degree AV block
don't do anything, just monitor PR interval is prolonged (0.36) rate is regular bradycardic: 60
47
another name for second degree Heart Block Mobitz type 1
Wenkebach
48
describe 2nd degree Wenkebach
rate is slow, progressive* rhythm is irregular *PR gets longer then drops a beat* (0.2 then 0.24 then 0.26 then drops beat)
49
describe 2nd degree mobitz type 2
bundle branch delay | *drops every other beat*
50
treatment for 2nd degree heart block
atropine: to speed up heart and trigger nerves | ONLY give 3 doses max
51
treatment for dysrhythmias if pt has pulse
MEDICATION
52
treatment for dysrhythmias if pt has NO pulse
*dead | compressions (30:2) THEN defib.
53
describe 3rd degree heart block
* NO coordination between atria and ventricles (INDEPENDENT of eachother) rhythm: 40-60 * BAD: SOB, chest pain, dizziness
54
treatment for 3rd degree heart block
pacemaker or atropine(sometimes)
55
describe premature ventricular contraction (PVC)
``` no P wave preceding PVC feels like a skipped beat not very concerning unless couplet weird looking beat occur due to pain, fever or low O2 ```
56
describe couplet PVC
PVC occurring in pairs due to inadequate CO2 | this is a problem when they occur in pairs
57
describe ventricular tachycardia
rapid rate: 150 no Ps or QRS cardiac output is decreased heart is irritated
58
treatment for ventricular tachycardia
if there is a pulse, then amiodarone
59
what do you always do first BEFORE treatment of dysrhythmias
assess patient
60
describe polymorphic
no P waves associated with QRS poor cardiac output QRS complexes occuring from different foci
61
what is the procedure for V-fib if no pulse
1)epi 2) compressions 3) amiodarone 4) compressions
62
describe V-fib
ALWAYS SHOCK- defib no contraction of heart muscle only twitching no cardiac output/sudden death
63
describe idioventricular
slow rhythm no P or QRS waves poor output