Cardiac Failure Flashcards

1
Q

What is the prognosis?

A

50% of those diagnosed with HF die within 4 years

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2
Q

What percent of HF cases also have HTN?

A

75%

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3
Q

Increased length and decreased contractility lead to

A

decreased cardiac output

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4
Q

What activates the renin-angiotensin system:

A

increased sympathetic activity (increased HR and contractility)

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5
Q

What are the results of renin-angiotensin activation?

A

increased blood volume

LV hypertrophy

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6
Q

What happens to the frank-starling curve in HF?

A

it plateaus

unable to further compensate

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7
Q

Describe systolic HF:

A

“beach ball”
1. contractile dysfxn
2. inability of myocardium 3. to adequately contract to eject blood from LV
characterized by EF < 40%

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8
Q

Describe diastolic HF:

A

“raquetball”

  1. compliance dsyfxn
  2. decreased ability of myocardium to fill during diastole
  3. leads to decreased SV (and decreased CO)
  4. EF within normal range
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9
Q

After initial myocardial damage, what are the processes that cause altered mechanics?

A
  1. weak area unable to withstand increased pressure
  2. dilation occurs
  3. scaring occurs
  4. causes LV to change chape
  5. ventricular mass/volume increase : slowly negatively impacts LV fxn
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10
Q

What are the causes of diastolic HF?

A
  1. chronic HTN with LV hypertrophy
  2. hypertrophic CM
  3. Aortic stenosis with normal LVEF
  4. CAD
  5. restrictive cardiomyopathy
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11
Q

R HF can be due to pulmonary disease because:

A

chronic resistance in the lungs (pulmonary arterial system) means RV has to work harder

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12
Q

With chronic increase in RV end diastolic pressure, what happens?

A
  1. increased RA pressure
  2. JVD
  3. liver engorgement
  4. ascites
  5. peripheral edema
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13
Q

Class I

A

no limitation

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14
Q

Class II

A

light physical activity limitation

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15
Q

Class III

A

marked limitation of physical activity, comfortable at rest

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16
Q

Class IV

A

symptoms even at rest

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17
Q

Stage A

A

high risk for CHF development

no structural issues

18
Q

Stage B

A

structural disorder of the heart

no symptoms

19
Q

Stage C

A

past/current CHF symptoms associated with underlying heart disease

20
Q

Stage D

A

end-stage disease

require specialized treatment strategies

21
Q

Acute “uncompensated” HF:

A
  1. rapid failure of the heart
  2. causes rapid shift of blood from cardiac to pulmonary systems
  3. acute symptoms of dyspnea, edema, decreased activity
22
Q

Chronic “compensated” HF:

A
  1. changes to RAAS: Na and H20 retention, inc. contractility
  2. sympathetic stimulation
  3. inc. peripheral 02 extraction
  4. cardiac dilation to inc. SV
23
Q

What are the results of sympathetic stimulation?

A

inc. HR
inc. contractility
inc. rate of ventricular relaxation
arterial/venous constriction

24
Q

Auscultate for pulmonary edema. Expected sounds?

A

crackles

diminished breath sounds possible

25
Describe the pathway of back flow:
1. LV 2. LA 3. Pulmonary system 4. RV 5. RA 6. Venous system in body
26
Muscle changes in HF causes:
1. dec. blood flow 2. poor muscle nutrition/absorption 3. insulin resistance 4. inc. tumor necrosis factor (systemic inflammation) 5. abnormal skeletal muscle metabolism
27
Results of muscle changes in HF:
1. dec. muscle mass 2. inc. muscle atrophy (type II fibers) 3. myopathy
28
HF can lead to increased metabolic rate up to ___%.
70%
29
Depression is present in ___% of HF patients.
43%
30
B12 insufficiency is associated with:
neuropathy
31
PT consideration for patients with hx of ventricular arrhythmias:
monitor with telemetry for first treatment during activity
32
T or F: PT consideration should consider cognitive changes.
True. | Deficits are greater than that expected with normal aging in those patients with HF (25% to 74%)
33
Signs of worsening HF:
``` Increased fatigue Increased dyspnea Reduced exercise tolerance Increased edema or ascites Weight gain Loss of appetite Orthopnea Dry cough Cyanosis Jugular vein distension Paroxysmal nocturnal dyspnea (PND) S3 heart sounds ```
34
What is orthopnea?
inability to breathe well when lying flat; pt requires inc. # of pillows when sleeping
35
What is paroxysmal nocturnal dyspnea (PND)?
sudden onset SOB when supine
36
Pt needs to be in ______ HF to engage in exercise.
compensated
37
Systemic effects of chronic HF contribute to ______.
myopathy | consider resisted exercise program
38
3 main treatment options for HF:
meds lifestyle mods surgery
39
HF surgical options:
``` Coronary Artery Bypass Graft Angioplasty Valve Replacement Defibrillator Implantation Myectomy and/or ventriculotomy Gene/Stem Cell Therapy Ventricular Assist Device (VAD) Heart Transplant ```
40
HF drug options:
``` ACE inhibitor β-Blocker Electrolyte replacement: K+, Mg+ Diuretics Vasodilators Inotropes Vasopressors ```
41
HF lifestyle changes:
``` Low-saturated-fat, low-sodium diet Exercise Smoking cessation Losing weight Avoiding alcohol  Avoiding or limiting caffeine Reducing stress Regular check ups with the doctor ```
42
What data is usually monitored with telemonitoring?
1. BP, HR, SpO2 2. Weight 3. Blood sugar, HgA1C 4. Some systems allow for patient to enter subjective symptoms