Cardiac Failure Flashcards
What is the prognosis?
50% of those diagnosed with HF die within 4 years
What percent of HF cases also have HTN?
75%
Increased length and decreased contractility lead to
decreased cardiac output
What activates the renin-angiotensin system:
increased sympathetic activity (increased HR and contractility)
What are the results of renin-angiotensin activation?
increased blood volume
LV hypertrophy
What happens to the frank-starling curve in HF?
it plateaus
unable to further compensate
Describe systolic HF:
“beach ball”
1. contractile dysfxn
2. inability of myocardium 3. to adequately contract to eject blood from LV
characterized by EF < 40%
Describe diastolic HF:
“raquetball”
- compliance dsyfxn
- decreased ability of myocardium to fill during diastole
- leads to decreased SV (and decreased CO)
- EF within normal range
After initial myocardial damage, what are the processes that cause altered mechanics?
- weak area unable to withstand increased pressure
- dilation occurs
- scaring occurs
- causes LV to change chape
- ventricular mass/volume increase : slowly negatively impacts LV fxn
What are the causes of diastolic HF?
- chronic HTN with LV hypertrophy
- hypertrophic CM
- Aortic stenosis with normal LVEF
- CAD
- restrictive cardiomyopathy
R HF can be due to pulmonary disease because:
chronic resistance in the lungs (pulmonary arterial system) means RV has to work harder
With chronic increase in RV end diastolic pressure, what happens?
- increased RA pressure
- JVD
- liver engorgement
- ascites
- peripheral edema
Class I
no limitation
Class II
light physical activity limitation
Class III
marked limitation of physical activity, comfortable at rest
Class IV
symptoms even at rest
Stage A
high risk for CHF development
no structural issues
Stage B
structural disorder of the heart
no symptoms
Stage C
past/current CHF symptoms associated with underlying heart disease
Stage D
end-stage disease
require specialized treatment strategies
Acute “uncompensated” HF:
- rapid failure of the heart
- causes rapid shift of blood from cardiac to pulmonary systems
- acute symptoms of dyspnea, edema, decreased activity
Chronic “compensated” HF:
- changes to RAAS: Na and H20 retention, inc. contractility
- sympathetic stimulation
- inc. peripheral 02 extraction
- cardiac dilation to inc. SV
What are the results of sympathetic stimulation?
inc. HR
inc. contractility
inc. rate of ventricular relaxation
arterial/venous constriction
Auscultate for pulmonary edema. Expected sounds?
crackles
diminished breath sounds possible