cardiac arrhythmias Flashcards
supraventricular arrhythmias
AF
Atrial flutter
ectopic atrial tachycardia
sinus bradycardia
ventricular arrhythmias
premature ventricular complexes
ventricular tachycardia
ventricular fibrillation
automaticity abnormalities
arise from a single cell
abnormalities of conduction
abnormal interaction between cells
bundle branch conduction delay on ECG
slightly widening of QRS
incomplete bundle branch block
right bundle branch block
produced late activation of right ventricle
deep S waves (leads I and V6), tall late R wave (lead V1)
left bundle branch block
deep S wave (V1) and tall late R wave (leads I and V6)
also abnormal QRS
left anterior hemiblock
occurs when a cardiac impulse spreads first through the left posterior fascicle, causing a delay in activation of the anterior and upper parts of the LV
clinical features of heart block
usually asymptomatic
RBBB- wide but physiological splitting of the second heart sound
LBBB- reverse splitting of the second sound
sinus tachycardia
P wave morphology similar to sinus rhythm
AVNRT
No visibly P wave, or inverted P wave immediately before or after the QRS
most common cause of palpation in patients with normal heart
AVRT
P wave visible between QRS and T wave
AF clinical findings
palpations chest pain dyspnoea deterioration of exercise capacity dizzy/faint 'irregularly irregular' pulse
AF finding on ECG
no P waves
irregularly irregular rate
f waves
Wolff-Parkinson White syndrome
caused by a congenital accessory conducting pathway between the atria and ventricles leading to a atrioventricular re-entry tachycardia (AVRT)