biopsychology Flashcards

1
Q

Structure + Function of Sensory, Relay + Motor Neurons: neurones (A01)

A

100 billion neurones in human nervous system 80% of which are located in brain- transmitting signals electrically + chemically these neurones provide nervous system w/primary means of communication

Neurones vary in size from less than millimetre to up to metre long depending on their function but all share same basic structure

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2
Q

Structure + Function of Sensory, Relay + Motor Neurones: myelin sheath (A01)

A

If myelin sheath was continuous this would have reverse effect + slow down electrical impulse- myelin sheath is segmented by gaps called nodes of Ranvier

speeds up transmission of impulse by forcing it to ‘jump’ across gaps along axon

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3
Q

Structure + Function of Sensory, Relay + Motor Neurones: synapse (A01)

A

end of axon are terminal buttons that communicate w/next neurone in chain across gap

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4
Q

Structure + Function of Sensory, Relay + Motor Neurones: nucleus + dendrites (A01)

A

cell body includes nucleus which contains genetic material of cell Branch-like structures called dendrites protrude from cell body receive signals from other neurones or from sensory receptors

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5
Q

Structure + Function of Sensory, Relay + Motor Neurones: axon (A01)

A

axon carries impulses away from cell body down length of neurone axon is covered in fatty layer of myelin sheath that protects axon + speeds up electrical transmission of impulse

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6
Q

sensory neurons (unipolar) (A01)

A

found in receptors such as eyes ears, tongue + skin- carry nerve impulses from PNS to spinal cord + brain (CNS) these nerve impulses reach brain they are translated into ‘sensations’ such as vision, hearing, taste + touch

not all sensory neurones reach brain as some neurones stop at the spinal cord allowing for quick reflex actions- have long dendrites + short axons

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7
Q

relay neurons (multipolar) (A01)

A

most common type of neurone

only found in brain + spinal cord (CNS)- connect sensory neurones to motor or other relay neurones

have short dendrites + short axons

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8
Q

motor neurons (multipolar) (A01)

A

neurones carry messages from CNS + control muscle movements

stimulated these neurones release neurotransmitters that bind to receptors on muscle + triggers response which leads to muscle movement

Muscle relaxation is caused by inhibition of motor neurone
have short dendrites + long axons

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9
Q

how neutrons function together (A01)

A

stimulus is presented

Sensory neurones send message through peripheral nervous system

message reaches spinal cord where it is passed to relay neurone
found throughout CNS

message is then either passed to motor neurone or sent to brain for further processing

motor neurone carries message to an effector such as muscle or gland motor neurone sends messages to muscles located in arm which contract - pulling hand away from candle

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10
Q

synaptic transmission (A01)

A

Signals w/in neurones are transmitted electrically but signals between neurones are transmitted chemically across synapse

electrical impulse reaches end of the neurone it triggers release of neurotransmitters from tiny sacs -synaptic vesicles

Neurotransmitters are chemicals that diffuse across synapse to next neurone in chain

neurotransmitter crosses gap it is taken up by postsynaptic receptor site – dendrites of next neurone-chemical message is converted back to an electrical impulse + process of transmission begins again in this other neurone effects of this last until neurotransmitters travel back where they came from to be taken up again by presynaptic neurone-‘reuptake’ which allows neurotransmitters to be stored again + made available for later use

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11
Q

neurotransmitters (A01)

A

Dozens of neurotransmitters have been identified in brain

Each neurotransmitter has its own specific molecular structure that fits perfectly into post-synaptic site like lock + key

Neurotransmitters also have specialist functions eg. dopamine affects nervous system in several ways including emotional arousal, pleasure + voluntary movement

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12
Q

Summation (A01)

A

neurone can receive both excitatory + inhibitory neurotransmitters at same time

likelihood of cell firing is therefore determined by adding up excitatory + inhibitory synaptic input- summation if net effect on post-synaptic neurone is inhibitory neurone will be less likely to fire

if net effect is excitatory neurone will be more likely to fire

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13
Q

Excitation + inhibition (A01)

A

Neurotransmitters have either an excitatory or inhibitory effect on neighbouring neurone

serotonin causes inhibition in receiving neurone resulting in neurone becoming more negatively charged + less likely to fire
message is likely to be stopped at post-synaptic neurone inhibitory synapse

dopamine causes excitation of post-synaptic neurone by increasing its positive charge + making it more likely to fire
synapse is more likely to cause post-synaptic neurone to fire- excitatory synapse

excitatory potential is like accelerator + an inhibitory potential is like brake

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14
Q

central nervous system (CNS): brain (A01)

A

involved in psychological processes + is centre of all conscious awareness

brain’s outer layer cerebral cortex is highly developed in humans + is what distinguishes our higher mental functions from those of animals

brain is divided to 2 hemispheres

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15
Q

central nervous system (CNS): spinal cord (A01)

A

extension of brain

responsible for reflex actions pulling your hand away from hot plate

responsible for relaying information between brain + rest of body

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16
Q

peripheral nervous system (PNS) (A01)

A

PNS transmits messages via millions of neurones too + from CNS

nerves outside brain + spinal cord function of PNS is to relay nerve impulses from CNS to rest of body + from body back to CNS

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17
Q

peripheral nervous system (PNS): Somatic nervous system (SNS) (A01)

A

controls voluntary actions

achieved by receiving info from senses + carrying sensory + motor info to + from CNS

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18
Q

peripheral nervous system (PNS): Automatic nervous system (ANS) (A01)

A

governs vital functions in body such as breathing, heart rate, digestion, sexual arousal + stress responses

ANS controls involuntary actions eg. breathing, heart rate + digestion via internal organs + glands in body

ANS carries only motor info to + from CNS

Primarily involved in responses that help us deal w/emergencies

Neurones travel to virtually every organ + gland w/in body preparing body for rapid action

eg. sympathetic nervous system causes body to release stored energy pupils to dilate + hair to stand on end

slows down Inhibits less important bodily processes eg. digestion + urination

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19
Q

endocrine system: glands (A01)

A

Organs in body that produce + secrete hormones to regulate many bodily functions

major endocrine gland is pituitary gland located in brain

called ‘master gland’ bc it controls release of hormones from all other endocrine glands in body

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20
Q

Automatic nervous system (ANS): Parasympathetic nervous system (A01)

A

Primarily involved in returning to body to rest state once emergency has passed

sympathetic branch causes heart rate to become faster peripheral branch slows heartbeat down

bodily processes that are inhibited by sympathetic branch are returned to normal

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21
Q

Automatic nervous system (ANS): sympathetic nervous system (A01)

A

involved in responses that prepare body for fight or flight
Impulses travel from SNS to organs in body to help us prepare for action when we are faced w/dangerous situation

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22
Q

endocrine system (A01)

A

endocrine + nervous system work closely together to regulate various physiological processes in human body

endocrine system works much more slowly than nervous system but has widespread effect

endocrine communicates chemical messages to organs of body messages- hormones regulate body’s growth, metabolism + sexual development + function

Hormones are released from glands in body- major glands of endocrine system are pituitary, pineal, adrenals, reproductive organs + thyroid

pituitary gland- called master gland bc it controls several other hormone glands in your body including thyroid + adrenals, ovaries + testicles

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23
Q

endocrine system: hormones (A01)

A

Chemicals that circulate in bloodstream + influence target organs in order to regulate bodily activity

produced in large amounts but disappear quickly- effects are very powerful

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24
Q

glands adrenal: adrenaline FUNCTION (A01)

A

Triggers fight-or-flight response in stressful situation by increasing heart rate, dilating pupils

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25
Q

glands testes: testosterone FUNCTION (A01)

A

development of testes in womb

surge of testosterone during puberty is also responsible for secondary sexual characteristics eg. facial hair + deepening voice

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26
Q

glands ovaries: oestrogen +
progesterone FUNCTION (A01)

A

help to regulate menstrual cycle

Oestrogen is involved in repairing + thickening uterus lining whilst progesterone maintains uterine lining

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27
Q

glands pineal: melatonin FUNCTION (A01)

A

Regulates sleep-wake cycle

High levels of melatonin cause drowsiness when daylight is low

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28
Q

Fight or Flight Response Including Role of Adrenaline (A01)

A

experience an acute stressor hypothalamus directs sympathetic branch of ANS to send neurotransmitters to adrenal medulla

results in release of adrenaline to bloodstream- release of adrenaline as well as noradrenaline causes “fight or flight” response by triggering several physiological reactions

reactions include an activation of emergency functions such as increased heart rate + blood pressure so that oxygen is pumped to muscles to enable increased physical activity

Non-emergency bodily processes eg. digestion are suppressed here leading to ‘dry mouth’ + changes in stomach activity often associated w/these situations

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29
Q

Fight or Flight Response Including Role of Adrenaline: sympathetic state (A01)

A

Increases heart rate
Increases breathing rate
Dilates pupils
Inhibits digestion
Inhibits saliva production
Contracts rectum

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30
Q

Fight or Flight Response Including Role of Adrenaline: parasympathetic state (A01)

A

Decreases heart rate
Decreases breathing rate
Constricts pupils
Stimulates digestion
Stimulates saliva production
Relaxes rectum

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31
Q

Fight or Flight Response: Adrenaline + Noradrenaline (A01)

A

Increase:
heart rate + blood pressure
glucose release
respiration + perspiration-sweat
blood coagulation

Decrease:
digestion

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32
Q

Fight or Flight Response: may be different in females (A03) (1)

A

P: fight or flight response may be different in females

E +E: Taylor et al found that females adopt ‘tend + befriend’ response in stressful or dangerous situations Women are more likely to protect their offspring + form alliances with other women rather than fight an adversary or flee

L: suggests that research to fight or flight response is gender bias as biological processes that occur during stress may only apply to males

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33
Q

Localisation of Function in Brain (A01)

A

specific functions have specific locations w/in brain

certain area of brain is damaged through illness or injury associated function will also be affected

brain is divided into 2 symmetrical halves called left + right hemispheres

outer layer of both these hemispheres is called cerebral cortex

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34
Q

Localisation of Function in Brain: auditory centres (A01)

A

human brain has 2 primary auditory cortices 1 in each hemisphere

primary auditory cortex in both hemispheres receives info from both ears via 2 pathways that transmit information about what sound is + its location

located in temporal lobe either side of brain

Damage to this area may produce partial hearing loss more extensive damage more extensive loss

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35
Q

Localisation of Function in Brain: motor centres (A01)

A

Movement is centred on motor cortex of brain which sends messages to muscles via brain stem + spinal cord

motor cortex is responsible for generating voluntary motor movements

located at back of frontal lobe in both hemispheres – motor cortex in each hemisphere controls movement in opposite side of body

Damage to this area may result in loss of control over fine movements

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36
Q

Localisation of Function in Brain: somatosensory centres (A01)

A

referring to sensation of body
somatosensory cortex lies next to motor cortex in brain

somatosensory area is where sensory information from skin is represented

perceives touch so number of neuronal connections needed dictates amount of somatosensory cortex needed for that area of body

located at front of parietal lobe in both hemispheres

somatosensory cortex on 1 side of brain receives sensory information from opposite side of body

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37
Q

Localisation of Function in Brain: visual centres (A01)

A

brain has 2 visual cortices 1 in each hemisphere

visual cortex is in occipital lobe which is at back of brain- main visual centre

located in occipital lobe at back of brain-each eye sends info from right visual field to left visual cortex + from left visual field to right visual cortex

means that damage to left hemisphere eg. can produce blindness in part of right visual field of both eyes

specifically an area called Area V1 which seems to be necessary for visual perception

individuals w/damage to that area report no vision of any kind

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38
Q

Localisation of Function in Brain BROCA + WERNICKES AREAS: Language centres (A01)

A

left hemisphere + that is where most language processing in most of population is situated

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39
Q

Localisation of Function in Brain BROCA AREAS: Language centres (A01)

A

identified area of brain responsible for speech production

located in small area in left frontal lobe

Damage to Broca’s area causes Broca’s aphasia which is characterised by speech that is slow, laborious + lacking in fluency

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40
Q

Localisation of Function in Brain WERNICKES AREAS: Language centres (A01)

A

found that patients who had damage in an area close to auditory cortex had specific language impairments

included inability to comprehend language + anomia which is when someone struggles to find word they need

but Wernicke noticed that these people did have fluent speech when they could access words quickly

led Wernicke to suggest that area now called Wernicke’s area was important for understanding language + accessing words

Patients who have Wernicke’s aphasia will often produce nonsense words as part of content of their speech- small area in left temporal lobe

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41
Q

Localisation of Function in Brain: Localisation supported by case study evidence (A03) (1)

A

P: Localisation supported by case study evidence

E: 1861 man who became known as “Tan” since this was only word he could speak met famous surgeon Paul Broca at hospital in France Shortly after meeting Tan died + Broca performed his autopsy During autopsy Broca found lesion in region of his brain

E: Broca concluded that Tan’s aphasia was caused by damage to this region + that this particular brain area-controlled speech- That region of brain was later renamed Broca’s area in honour of doctor

L: increases validity of theory of localisation as it appears damage to specific brain regions result in specific deficits

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42
Q

Localisation of Function in Brain: supporting evidence from brain scan research (A03) (2)

A

P: supporting evidence from brain scan research

E + E: Peterson et al used brain scans to demonstrate how Wernicke’s area was active during listening task + Broca’s area was active during reading task Tulving et al have shown that episodic + semantic memories were recalled from different sides of prefrontal cortex whilst procedural memory is associated w/cerebellum

L: positive as there is wide range of evidence to support idea that different areas of brain have different functions

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43
Q

Localisation of Function in Brain: evidence against localisation of function comes from animal research (A03) (3)

A

P: evidence against localisation of function comes from animal research

E: Lashley removed between 10-50% of cortex in rats that were learning maze + found that no area was more important than any other in terms of their ability to learn maze

E: problem as it suggests that higher cognitive processes eg. learning are not localised but distributed in more holistic way involving entire brain

L: reduces validity of theory of localisation

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44
Q

Localisation of Function in Brain: Evidence from plasticity studies fails to support localisation of function (A03) (4)

A

P: Evidence from plasticity studies fails to support localisation of function

E + E: When brain has become damaged through illness or accident + particular function has been lost rest of brain is able to reorganise itself to recover function eg. Turk et al discovered patient who suffered damage to left hemisphere but developed capacity to speak in right hemisphere

L: suggests that functions are not localised 1 + brain can adapt following damage to certain areas

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45
Q

Hemispheric lateralisation (A01)

A

idea that 2 halves of brain are functionally different + that certain mental processes + behaviours are mainly controlled by 1 hemisphere rather than other

human brain has 2 hemispheres which are bridged by corpus callosum-‘bridge’ which is bundle of fibres is communication pathway so that 2 hemispheres can exchange information

brain is contralateral in most people so parts of left hemisphere deal w/right side of body + right hemisphere does same for left side of body

46
Q

Hemispheric lateralisation: left hemisphere (A01)

A

language processing is done in left hemisphere

for many people if they have stroke on left side of their brain their speech is affected

bc Broca’s + Wernicke’s areas are found on left side of brain

47
Q

Hemispheric lateralisation: right hemisphere (A01)

A

dominant for facial recognition

case study of woman who had right hemisphere damage highlighted that right hemisphere also seems more adept at spatial relationships

woman would often get lost even in familiar situations

suggests that right hemisphere deals w/spatial information

48
Q

Hemispheric lateralisation: supporting evidence (A03) (1)

A

P: supporting evidence

E + E: Fink used PET scans to identify which brain regions were active during visual processing task when Ps were asked to attend to global elements of an image regions of right hemisphere were more active When required to focus in on finer detail specific areas of left hemisphere were more dominant

L: suggests tasks such as visual processing are feature of connected brain

49
Q

Hemispheric lateralisation: Evidence from plasticity studies fails to support lateralisation (A03) (2)

A

P: Evidence from plasticity studies fails to support lateralisation

E +E: When brain has become damaged through illness or accident + particular function has been lost rest of brain is able to reorganise itself to recover function eg. Turk et al discovered patient who suffered damage to left hemisphere but developed capacity to speak in right hemisphere

L: suggests that functions are not lateralised + brain can adapt following damage to certain areas

50
Q

Hemispheric lateralisation: contradictory evidence against lateralisation also comes from animal research (A03) (3)

A

P: Contradictory evidence against lateralisation also comes from animal research

E: Lashley removed between 10-50% of cortex in rats that were learning maze + found that no area was more important than any other in terms of their ability to learn maze

E: problem as it suggests that higher cognitive processes eg. learning are not lateralised but distributed in more holistic way involving entire brain

L: reduces validity of theory of lateralisation

51
Q

Hemispheric lateralisation: ‘lateralisation’ may be further complicated by age (A03) (4)

A

P: ‘lateralisation’ may be further complicated by age

E: means that research has shown lateralisation of function may change throughout an individual’s lifetime

E: eg. Szflarski who found that language became more lateralised to left hemisphere as children developed to adolescents but after age of 25 lateralisation decreased w/each decade of life

L: problem as it suggests hemispherical lateralisation is much more complex process than many realise w/many questions remaining as to why this happens

52
Q

split brain studies VERBAL RECOGNITION: procedure (A01)

A

visual image eg. picture of pencil was presented to left visual field via tachistoscope

Ps were asked to describe what they had seen- would then be repeated using right visual field

53
Q

split brain studies TOUCH RECOGNITION: procedure (A01)

A

patients’ hands were screened so they could not see objects in front of them

Ps would be asked to pick up an object using their right hand + then asked to describe what they had felt- would then be repeated using left hand

54
Q

split brain studies VERBAL RECOGNITION: findings (A01)

A

picture of an object was shown to patient’s right visual field patient could easily describe what was seen

but if same object was shown to left visual field patient could not describe what was seen + reported that there was nothing there

bc for most people language is processed in left hemisphere + therefore when picture was presented in left visual field this was processed by right hemisphere which has a lack of language centres to be able to describe it

allows us to infer that in normal brain messages from right hemisphere would have been relayed ‘across hemispheres’ to language centres in left hemisphere to describe it

55
Q

split brain studies TOUCH RECOGNITION: findings (A01)

A

patients could not verbally describe objects projected in their left visual field they were able to select matching object from grab-bag of different objects using their left hand

objects were placed behind screen so as not to be seen

left hand was also able to select an object that was most closely associated w/object presented to left visual field

each case patient was not able to verbally identify what they had seen but could nevertheless ‘understand’ what object was using right hemisphere + select corresponding object accordingly

56
Q

split brain research: support idea lateralisation is well controlled (A03) (1)

A

P: support idea lateralisation is well controlled

E: means methodology can be praised for using highly standardised procedures conducted in controlled environment to control possible extraneous variables

E: eg. images were flashed up for 1-tenth of second to ensure there was not time to spread info across both sides of visual field + subsequently both sides of brain

L: positive as it ensured research measured what it intended to give evidence high internal validity

57
Q

split brain research: evidence for lateralisation was flawed (A03) (2)

A

P: split-brain research evidence for lateralisation was flawed

E: means that findings came from very unusual + limited sample of people who were not well matched to control group eg. only 11 split-brain Ps took part in all variations of basic procedure all of whom had history of epileptic seizures + had received drug therapy for different amounts of time which may have caused unique changes in each of their brains

E: Some of Ps may also have experienced more disconnection of 2 hemispheres than others as part of their surgery-control group consisted of Ps w/no history of epilepsy making them poorly matched

L: problematic as it brings conclusions of research + support for lateralisation into doubt

58
Q

Methods of Studying Brain: functional magnetic resonance imaging (fMRI) (A01)

A

technique for detecting changes in blood oxygenation + flow in brain

brain area is more active it consumes more oxygen + to meet this increased demand blood flow is directed to the active area

FMRI produce 3-D images showing which part of brain is involved in particular mental process

eg. P might be asked to alternate between periods of doing task + relaxed state

resulting fMRI data can then be used to identify which areas of brain were being used when doing each task

fMRI images show activity approximately 1-4 seconds after an event occurs + are thought to be accurate w/in 1-2 mm

59
Q

fMRI: less invasive than other scanning techniques (A03) (1)

A

P: less invasive than other scanning techniques

E+ E: they don’t use radiation or require instruments to be inserted into brain

L: fMRI scans are virtually risk-free

60
Q

fMRI: good spatial resolution (A03) (2)

A

P: good spatial resolution of 1-2 mm

E + E: Spatial resolution refers to smallest measurement that scanner can detect- Greater spatial resolution allows psychologists to investigate brain regions w/greater accuracy

L: helps researcher’s pinpoint specific responses + exact source of brain activity

61
Q

fMRI: machines are expensive (A03) (3)

A

P: machines are expensive

E+ E: to buy + maintain + they require trained operators

L: makes research expensive + difficult to organise

62
Q

fMRI: Low temporal resolution (A03) (4)

A

P: Low temporal resolution

E+ E: refers to the accuracy of scanner in relation to time fMRI scans have temporal resolution of 1-4 seconds which is worse than other techniques eg. EEG/ERP which have temporal resolution of 1-10 milliseconds

L: Consequently psychologists are unable to predict w/high degree of accuracy onset of brain activity

63
Q

Methods of Studying Brain: electroencephalogram (EEG) (A01)

A

measure electrical activity w/in brain via electrodes that are fixed to an individual’s scalp using skull cap

scan recording represents brainwave patterns that are generated from action of millions of neurones providing an overall account of brain activity

can be anything from 2 to 3 electrodes to over hundred Electrodes measure activity of cells immediately under electrode so using more electrodes gives fuller picture

scan recording represents brainwave patterns that are generated from action of millions of neurones providing an overall account of brain activity

‘Real-time’ recording of brain activity

64
Q

Methods of Studying Brain: event-related potentials (ERPs) (A01)

A

issue w/EEG is that it only provides measure of general brain activity means that brain response to single stimulus or event of interest is not usually visible in EEG recording

possible to tease out + isolate specific sensory, cognitive + motor responses w/in EEG data

stimulus such as picture or sound is presented many times to P

Using statistical averaging technique, random or background brain activity from original EEG recording is filtered out leaving only event-related potentials

ERP is 1 of most widely used methods in cognitive neuroscience research to study physiological correlates of sensory, perceptual + cognitive activity associated w/ processing information

65
Q

EEG + ERP: less invasive than other scanning techniques (A03) (1)

A

P: less invasive than other scanning techniques

E+ E: they don’t use radiation or require instruments to be inserted into brain

L: EEG + ERP scans are virtually risk-free

66
Q

EEG + ERP: cheaper methods in comparison to fMRI scanning (A03) (2)

A

P: cheaper methods in comparison to fMRI scanning

E+ E: more widely available to researchers

L: Consequently this could help psychologists to gather further data on functioning human brain leading to greater understanding of sleeping + disorders eg. Alzheimer’s

67
Q

EEG + ERP: High temporal resolution (A03) (3)

A

P: High temporal resolution

E+ E: advantage of EEG/ERP technique is that it has good temporal resolution as it takes readings every millisecond meaning it can record brain activity in real time

L: leads to an accurate measurement of electrical activity when undertaking specific task

68
Q

EEG + ERP: poor spatial resolution (A03) (4)

A

P: poor spatial resolution

E+ E: EEGs/ERPs only detect activity in superficial regions of brain Consequently EEGS + ERPs are unable to provide info on what is happening in deeper regions of brain

L: making this technique limited in comparison to fMRI which has spatial resolution of 1-2 mm

69
Q

Post-mortem examinations (A01)

A

technique involves analysing person’s brain following their death

psychological research individuals whose brains are subject to post-mortem are likely to be those who have rare disorder + have experienced unusual deficits in mental processes or behaviour during their lifetime

Areas of damage w/in brain are examined after death as means of establishing likely cause of affliction person suffered

may also involve comparison w/ ‘normal’ brain to ascertain extent of difference

70
Q

Post-mortem examinations: ethical issues (A03) (2)

A

P: ethical issues in relation to informed consent + whether patient provides consent before his/her death

E+ E: many post-mortem examinations are carried out on patients w/severe psychological deficits who would be unable to provide fully informed consent + yet post-mortem examination has been conducted on his brain

L: raises severe ethical questions surrounding nature of such investigations

71
Q

Post-mortem examinations: provide detailed examination of structure of brain that is not possible w/other scanning techniques (A03) (1)

A

P: provide detailed examination of structure of brain that is not possible w/other scanning techniques

E+ E: Post-mortem examinations can access areas such as hypothalamus + hippocampus which other scanning techniques would struggle to reach

L: post-mortem examinations provide researchers w/insight into these deeper brain regions which often provide useful basis for further research

72
Q

Post-mortem examinations: issue of causation (A03) (3)

A

P: issue of causation

E+ E: deficit patient displays during their lifetime may not be linked to deficits found in brain- deficits reported could have been the result of another illness + therefore psychologists are unable to conclude that deficit is caused by damage found in brain

L: Other confounding factors include medication person may have been taking throughout their lives length of time between death + post mortem + age of person at death

73
Q

plasticity (A01)

A

brain’s capacity to change or adapt bc of new learning or brain trauma

well known that child’s brain changes during infancy as they acquire new knowledge + encounter new experiences but it is now known that this ability of brain does stop in childhood + that new neural connections can be made at any time of life

74
Q

functional recovery (A01)

A

Much recovery after trauma is due to anatomical compensation brought about by intensive rehabilitation

brain learns to compensate for lost functions

brain can be taught to learn how to use working faculties + function to compensate for ones that are lost forever

75
Q

functional recovery-brain after recovery: AXON SPROUTING (A01)

A

growth of new nerve endings which connect w/other undamaged nerve cells to form new neuronal pathways

When axon is damaged its connection w/neighbouring neurone is lost

some cases other axons that already connect w/that neurone will sprout extra connections to neurone replacing ones that have been destroyed

compensating for loss of neighbour- brain can re-wire itself by forming new synaptic connections close to area of damage

occurs for most part two weeks after damage happens

helps replace function but only if damaged axon + compensatory axons do similar job

If not problems can occur w/ function

76
Q

functional recovery-brain after recovery: NEURONAL UNMASKING (A01)

A

some of brain’s neurones are ‘dormant’

These neurones are alive but are not doing their specific function eg. they may fail to send messages to muscle

but when brain area becomes damaged these dormant areas become ‘unmasked’

unmasking of dormant neurones opens connections in regions of brain that are not normally activated which in time gives way to development of new structures

77
Q

functional recovery-brain after recovery: RECRUITMENT OF HOMOLOGOUS AREAS (A01)

A

opposite side of brain to perform specific tasks

eg. would be if Broca’s area was damaged on left side of brain similar area on right-side of brain would carry out its function

78
Q

factors affecting recovery of brain after trauma: age (A01)

A

deterioration of brain in old age + this therefore affects extent + speed of recovery

study by Marquez de la Plata et al found that following brain trauma older patients regained less function in treatment than younger patients + they were also more likely to decline in terms of function for 5 years following trauma

79
Q

factors affecting recovery of brain after trauma: gender (A01)

A

research to suggest that women recover better from brain injury as their function is not as lateralised

Ratcliffe et al examined 325 patients w/brain trauma for their level of response for cognitive skills to rehabilitation

patients were 16-45 years old at injury received rehabilitation at care facility + completed follow-up 1 year later

None of them had learning problems prior to trauma

When assessed for cognitive skills women performed significantly better than men on tests of attention/working memory + language whereas men outperformed females in visual analytic skills

80
Q

factors affecting recovery of brain after trauma: physical exhaustion, stress + alcohol consumption (A01)

A

function is recovered in an individual it is important to remember that often function is used w/considerable effort + although person can do task they are often fatigued by effort

Other factors such as stress + alcohol consumption can affect ability to use any function that has been regained

81
Q

factors affecting recovery of brain after trauma: evidence to support plasticity (A03) (1)

A

P: evidence to support plasticity

E+ E: Maguire et al used an MRI scanner to scan brains of London taxi drivers + found they had significantly more volume of grey matter in posterior hippocampus than matched control group- volume of this area was also positively correlated w/amount of time they had been taxi driver

L: positive as it supports idea that human brain can adapt as result of learning + experience

82
Q

factors affecting recovery of brain after trauma: plasticity has contributed to treatment + rehabilitation of brain injury patients (A03) (3)

A

P: Understanding processes involved in plasticity has contributed to treatment + rehabilitation of brain injury patients

E+ E: Following illness or injury to brain recovery tends to slow down after few weeks so forms of physical therapy are usually performed to maintain improvements in functioning

L: shows that although brain may have capacity to ‘fix itself’ to point this process requires further intervention if it is to be completely successful

83
Q

factors affecting recovery of brain after trauma: evidence to support plasticity also comes from animal studies (A03) (2)

A

P: evidence to support plasticity also comes from animal studies

E+ E: evidence of neuroplasticity
comes from an animal study conducted by Hubel + Wiesel- this study kittens had 1 of their eyes sewn up + brain’s cortical responses were analysed- found that area of visual cortex associated w/shut eye was not idle but continued to process info from open eye

L: positive as it supports idea that brain can change or adapt as result of experience

84
Q

factors affecting recovery of brain after trauma: functional recovery is affected by individual differences (A03) (4)

A

P: functional recovery is affected by individual differences

E: appears that certain individuals may have more of an ability to recover from brain trauma than others eg. Elbert et al showed that adults require far more intensive training than children after brain trauma

E: Schneider et al found that patients w/college education were seven times more likely than those who didn’t finish high school to be disability-free 1 year after brain injury

L: suggests no. of factors contribute to brain plasticity + recovery from brain trauma which makes it complex area to study

85
Q

biological rhythms (A01)

A

natural cycle of change in our body’s chemicals or functions

rhythms are governed by 2 things: body’s internal body clocks + external changes to environment

86
Q

biological rhythms: cardiac rhythms (A01)

A

cycle in biological or psychological activity that occurs once every 24 hours

most obvious example is sleep-wake cycle which occurs once every day

eg. include core body temp also follows daily rhythm as it is highest at around 4pm + lowest at around 4am

87
Q

cardiac rhythms: hormone production (A01)

A

Hormone release follows circadian rhythm eg. production + release of melatonin from pineal gland in brain follows circadian rhythm w/ peak levels occurring during hours of darkness

activating chemical receptors in brain melatonin encourages feelings of sleep

dark more melatonin in produced + when it is light again production of melatonin drops + person wakes

88
Q

cardiac rhythms: sleep-wake cycle (A01)

A

circadian rhythm not only dictates when we should be sleeping but also when we should be awake

Light + darkness are external signals that determine when we feel need to sleep + when to wake up

circadian rhythm also dips + rises at different times of day so our strongest sleep drive usually occurs in 2 ‘dips’ between 2-4 am + between 1-3 pm

89
Q

cardiac rhythms: practical application (A03) (1)

A

P: practical application

E+ E: Research into circadian rhythms provides an understanding of the consequences that occur when they are disrupted eg. night workers experience reduced concentration around 6am

L: leading to an increase in mistakes + accidents

90
Q

cardiac rhythms: small sample size (A03) (2) UNFINISHED

A

P: small sample size

91
Q

biological rhythms: infradian rhythms (A01)

A

last more than 24 hours eg. include menstruation breeding, hibernation + seasonal affective disorder

92
Q

infradian rhythms: support by theory of evolution (A03) (1)

A

P: support by theory of evolution

E+ E: eg. may have been advantageous for women to menstruate together + become pregnant at same time -In social group this would allow babies who had lost their mothers during childbirth to have access to breast milk thereby improving their chances of survival

L: suggests that synchronisation of menstrual cycle is an adaptive strategy

93
Q

infradian rhythms: menstrual cycle (A01)

A

woman’s menstrual cycle is monthly infradian rhythm that is governed by monthly changes in hormones that regulate ovulation

typical cycle lasts 28 days

During each cycle rising levels of hormone oestrogen cause ovary to release an egg

After ovulation hormone progesterone helps womb lining to grow thicker readying womb for pregnancy

If pregnancy does not occur egg is absorbed to body womb lining comes area + leaves body

94
Q

infradian rhythms: research (A01)

A

McClintock: 29 women w/irregular periods were studies

Samples of pheromones were gathered from 9 of women at different stages of their menstrual cycles via cotton pad placed in their armpits

pads were then rubbed on upper lip of other Ps

Stern + McClintock found that 68% of women experienced changes to their cycle which brough them closer to cycle of their ’odour donor’

95
Q

infradian rhythms: limitations to synchronisation studies (A03) (2) UNFINISHED

A

P: limitations to synchronisation studies

E+ E: eg. there are many factors that may cause menstrual cycle to change, including stress, extreme dieting + exercising

96
Q

ultradiation rhythms: stages of sleep (A01)

A

Stages 1: this is light sleep where person may be easily woken- alpha brain waves occur

Stage 2: Light sleep continues
Sleep spindles also occasionally occur

Stages 3 + 4: deep sleep or slow wave sleep
Delta waves occur w/lower frequency + higher amplitude difficult to wake someone at this point.

Stage 5: body is paralysed yet brain activity closely resembles that of awake brain
brain produces theta waves + eyes move around- Dreams are most often experienced during REM sleep

97
Q

biological rhythms: ultradiation rhythms (A01)

A

Ultradian rhythms are biological rhythms lasting less than 24 hours

eg. of ultradian rhythms include eye blinking, heartbeats, sleep patterns, breathing, pulse, appetite + digestion

When we sleep we go through 5 distinct stages that span about 90 minutes

cycle starts at light sleep progressing to deep sleep + then REM sleep

repeats itself about every 90 minutes throughout night typical person will go through about 5 full cycles in full night’s sleep

98
Q

ultradiation rhythms: improved understanding of age-related changes in sleep (A03) (1)

A

P: improved understanding of age-related changes in sleep

E+ E: eg. sleep scientists have observed that SWS reduces w/age leading to reduced alertness in elderly

L: suggests that knowledge of ultradian rhythms has practical value

99
Q

ultradiation rhythms: ignores individual differences (A03) (2)

A

P: ignore individual differences

E+ E: Tucker, 2007, found large differences in duration of each sleep stage in many Ps

L: makes it difficult to describe ‘normal sleep’

100
Q

endogenous pacemakers + sleep wake cycle: suprachiasmatic nucleus (SCN) (A01)

A

SCN is tiny bundle of nerve cells located in hypothalamus plays an important role in maintaining circadian rhythms eg. sleep/wake cycle SCN is damaged or destroyed, sleep becomes erratic

SCN lies just above the optic chaism receives information about light directly from this structure continues even when our eyes are closed

enabling biological clock to adjust to changing patterns of daylight whilst we are asleep

our biological clock is running slow then morning light automatically adjusts clock putting its rhythm in step w/world outside

synronisation of SCN by changes in light is known as entrainment

101
Q

endogenous pacemakers (A01)

A

internal body clocks that regulate many of our biological rhythms

102
Q

endogenous pacemakers + sleep wake cycle: pineal gland + melatonin (A01)

A

SCN passes info on day length + light to pineal gland

During night pineal gland increases production of melatonin chemical that induces sleep and is inhibited
during period of wakefulness

Melatonin has also been linked to seasonal affective disorder (SAD)

103
Q

endogenous pacemakers + sleep wake cycle: supporting evidence (A03) (1)

A

P: supporting evidence

E: support idea that SCN is key endogenous pacemaker in sleep-wake cycle comes from Morgan Studying hamsters it was found that if SCN was removed circadian sleep-wake cycle completely disappeared

E: Transplanting SCN cells from foetal hamsters to these hamsters helped to re-establish sleep-wake cycle if hamster was given transplanted SCN from mutant strain of hamster w/shorter sleep-wake cycle of 20 hours it will adopt the same activity patterns as mutant donor

L: animal studies support view that SCN is vital in maintaining sleep-wake cycle

104
Q

endogenous pacemakers + sleep wake cycle: case study of Siffre lends further support for role of SCN (A03) (2)

A

P: case study of Siffre lends further support for role of SCN

E+E: Siffre lived underground in cave in Texas for 6 months w/X external cues such as daylight- was found that his sleep-wake cycle settled to ‘free-running’ rhythm of around 25 hours- study suggests endogenous pacemakers such as SCN are important in controlling sleep-wake cycle

L: Despite absence of any exogenous zeitgebers Siffre was able to maintain relatively normal cycle

105
Q

endogenous pacemakers + sleep wake cycle: practical application (A03) (3)

A

P: real world application for role of endogenous pacemakers in circadian rhythms

E+ E: eg. our circadian rhythms affect when drugs are most affective therefore when prescribed drugs it is now advised that they are taken at different times of day to maximise their effects

L: suggests that we have an internal clock that controls our 24-hour sleep-wake cycle as medication is advised to be taken around this cycle

106
Q

exogenous Zeitgebers + sleep wake cycle: social cues (A01)

A

mealtimes, bedtimes + social events also act as zeitgeber

Research also suggests that adapting to local times for eating + sleeping is an effective way on entraining circadian rhythms + beating jet lag when travelling long distances

107
Q

endogenous pacemakers + sleep wake cycle: contradictory evidence (A03) (4)

A

P: contradictory evidence

E+ E: Folkard studied university student Kate Aldcroft who volunteered to spend 25 days in controlled environment of laboratory- During her time in lab she had no access to daylight or other zeitgebers that might have reset SCN end of 25 days her sleep-wake cycle had extended to 30 hours

L: reduces validity of role of endogenous pacemakers in sleep-wake cycle as it appears that exogenous zeitgebers are required to synchronise SCN

108
Q

exogenous Zeitgebers + sleep wake cycle: light (A01)

A

important zeitgeber in humans

It helps to maintain sleep–wake cycle by resetting SCN

Receptors in SCN are sensitive to changes in light levels during day + use this information to synchronise activity of body’s organs + glands

Light resets internal biological clock each day keeping it on 24-hour cycle

109
Q

exogenous Zeitgebers + sleep wake cycle: supporting evidence (A03) (1)

A

P: supporting evidence

E+ E: role of exogenous zeitgebers in circadian rhythm control comes from Campbell and Murphy- found that shining light on back of participants’ knees shifted circadian rhythm

L: implies that natural light plays role in ‘entraining’ our biological clocks to keep sleep-wake cycle in synchrony w/outside world

110
Q

exogenous Zeitgebers + sleep wake cycle: contradictory evidence (A03) (3)

A

P: contradictory evidence

E+ E: Miles studied young man who was blind from birth Despite exposure to social cues such as regular mealtimes his sleep/wake cycle remained abnormal

L: suggests that social cues alone are not effective in resetting biological rhythm

111
Q

exogenous Zeitgebers + sleep wake cycle: practical application (A03) (2)

A

P: practical application

E+ E: Research on exogenous zeitgebers has led to treatments for jet lag eg. Burgess et al found that exposure to bright light prior to an east-west flight reduced jet lag Ps who were exposed to bright light felt sleepier 2 hours earlier in evening + woke 2 hours earlier in morning eg. closer to local times conditions they would find after an east-west flight

L: huge benefits to the economy as preventing jet lag increases productivity eg. people are more likely to return to work earlier if they are less sleepy