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Biopsychology Flashcards

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1
Q

Motor cortex

A

Back of frontal lobe in both hems
Controls volun behav
Left hem controls movement in right side v.v.
Damage = loss of fine control

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2
Q

Somatosensory cortex

A

Processes info e.g., heat, pressure and touch
In both hems separate from motor by sulcus
Each part = allocated area - some = m than one

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3
Q

Visual cortex

A

If seen in left visual field processed in right hem v.v.
In occipital lobe
Damage in left hem = blindness in right eye v.v.

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4
Q

Auditory cortex

A

Temporal lobes
Process auditory info
Perception of sounds in environment signal from a.c. to other areas to process high level info

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5
Q

Broca’s Area

A

Lang processing
Left hem
Found area for speech production
Damage = aphasia = slow laborious speech - lacks fluency

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6
Q

Wernike’s Area

A

Language comprehension
Did research into patients who could speak ok but lacked ability to understand & comprehend speech
Language = fluent but meaningless

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7
Q

Case Studies

A

Clive Wearing had procedural and semantic no episodic

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8
Q

Brain Scan Evidence

A

Peterson et al (1988)
Used brain scans to show Wernike’s area = active in reading task - did functions

Tulving et al (1994)
Revealed semantic and episodic = dif areas of pre frontal cortex

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9
Q

Neurological Study

A

Dougherty et al (2002)
Reported on 44 ppl w/ OCD had eingulotomy at post surgery follow up = 32 weeks 1/3 met criteria for positive response & 14% for partial response

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10
Q

Holistic Theory

A

Lashley
Removed areas of cortex (10-50%) in rats learning a maze & found isn’t 1 section that = m NB - need whole cortex

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11
Q

Plasticity

A

When brain = damaged & a func = compromised/lost rest of the brain reorgs self to recover func - suggests not stuck in order

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12
Q

CNS

A

Made of brain: cerebral cortex what allow high level functioning = 2 hems
Spinal cord: extension of the brain - reflex actions

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13
Q

PNS

A

Transmits messages to and from the CNS
Subdivided into ANS & SNS

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14
Q

ANS

A

Governs vital function e.g., breathing, stress, heart rate and digestion
Sub divided into sympathetic and parasympathetic
Sympathetic = fight / flight - increased heart rate = for action
Parasympathetic = returns to resting state = rest and digest - blood goes back to digestion

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15
Q

SNS

A

Controls muscle movement
Receives info from sensory receptors

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16
Q

Endocrine System

A

Controls vital function
Slower than NS = m effective = widespread
Done via glands - uses hormones as chemical messengers

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17
Q

Pineal Gland

A

Releases melatonin
Responsible for NB bio rhythms e.g., sleep wake cycle

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18
Q

Thyroid Gland

A

Releases thyroxine - metabolism

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19
Q

Testes

A

Testosterone
Male sex characteristics
Muscle growth

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20
Q

Ovaries

A

Oestrogen
Reproduction and menstruation

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21
Q

Pituitary Gland

A

Controls and stimulates hormone release in other glands
Controlled and stimulated by the hypothalamus

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22
Q

Adrenal Gland (medulla)

A

Adrenaline and noradrenaline
Fight / flight response

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23
Q

Adrenal Gland (cortex)

A

Cortisol
Stimulates glucose release = energy and immune system

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24
Q

Glands and Hormones

A

Behaviour as a result of hormones
Glands secrete into blood - helps growth etc can increase / decrease cell processes

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25
Q

Fight / flight

A

Stress response system = acute / chronic stressors
Co-ord by hypothalamus - detects threat
= arousal of the ANS - SNS - stress hormone - F/F - sympha adrenal medullary system (SAM) ANS - PNS - normal
SNS & SAM = sympathomedullary pathway
Physiological changes = arousal for f/f acute response = automatic
PNS & SNS = antagonistic

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26
Q

Behaviour not limited to 2 responses (f/f AO3)

A

Gray (1988) doesn’t take into account freezing = hyper vigilant until decide correct response
Doesn’t fully explain complied cognitive and biological factors

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27
Q

Doesn’t fully explain females (f/f AO3)

A

Taylor et al (2002) adopt a tend & befriend tactic = m likely to protect offspring and make alliances
F/f = limited bc doesn’t explain
M recent research = beta bias assumes women respond same as men

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28
Q

Fight / Flight negative impact on health

A

Modern life don’t need to run so high blood pressure and flow can damage heart and lead to heath disease
= maladaptive now

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29
Q

Motor Neuron

A

Short dendrites and long axon
Connect CNS to effectors in CNS
Control muscle movement
When stimulated release neurotransmitters to receptors on muscles - trigger response = movement

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30
Q

Relay Neuron

A

Short dendrites and short axon
Connect motor and sensory / other relay
In brain and spinal cord
Allow sensory and motor neurones to communicate
Fast reflex bypass brain

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31
Q

Sensory Neuron

A

Long dendrites and short axon
In receptors e.g., eyes, ears, skin
Carry nerve impulses to spinal cord and brain and translate to sensations e.g., vision
Don’t all reach brain some stop at spinal cord
Quick reflex action

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32
Q

Firing of a neuron

A

Resting neuron = negatively charged when activated by stimulus in cell =positively charged for split second
Action potential = passing through when positively charged allows electrical impulse to move through

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33
Q

Synaptic Transmission

A

Process = 1 neuron communicates with another and sends information down axon as an electrical impulse = action potential - when at end need to - to neuron / tissue

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34
Q

Process of Synaptic Transmission

A

Vesicles release neurotransmitters to synaptic cleft
Neurotransmitter binds to receptor and activate
Excess = taken up by presynaptic neuron
Enzymes released to breakdown remaining
Vesicles replenished with new and reused neurotransmitter

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35
Q

Neurotransmitters

A

Chemical messengers found in brain, spinal cord and some glands
Transmit sign, across synaptic cleft - communicates between neurons
Only certain ones fit receptors = specialised function

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36
Q

Excitation

A

Increased likelihood of something happening
E.g., adrenaline causes excitation - increases neurons positive charge makes firing m likely

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37
Q

Inhibitation

A

Decreased likelihood of something happening
E.g., serotonin inhibits neighbouring neurons = - charge = less likely

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38
Q

Summation

A

Effect of neurotransmitters at synapse depends on number of factors:
1. How much = released
2. Action on postsynaptic neuron e.g., E/I - brings closer/further from AP threshold
Inputs at same time add together if E trigger AP and transmission of nerve impulse = summation
Temporal: 2 / m APs arrive in rapid succession along single presynaptic neuron and add together

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39
Q

Plasticity

A

Brains apparent ability (in infancy) to change & adapt physically & func
In infancy brain grows - peaks 15,000 synaptic connections at 2-3 (Gopnick 1999) = 2x adult brain
Originally thought that brain moves out of critical period & changes stop
Now think learning and experience changes the brain

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40
Q

Synaptic Pruning

A

As we age unused connections = deleted and used = strengthen

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41
Q

Maguire et al (2000)

A

Aim: investigate func of hippocampus in spatial memory
Ptps: 16 male London TD’s comp to MRI scans of 50 male non TDs
Hippocampus = bigger in TDs & volume = correlated w/ time as TDs

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42
Q

Function Recovery

A

After physical injury / trauma e.g., stroke unaffected areas comp for damage = ex of plasticity
Healthy areas take over damaged roles
= quick at start and then slows

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43
Q

Axonal Sprouting

A

Growth of new nerve endings to connect w/ undamaged nerve cells
Form new neural pathway can unmask neural pathways

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44
Q

Reformation of Blood Vessels

A

Blood vessels = refed to ensure brain funcs in affected areas

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45
Q

Recruitment of Homologous Areas

A

Sim area on opposite side = used
E.g., Broca’s Area = damaged so Wernike’s Area carries out func
Can shift back

46
Q

Case Study - P

A

Gabby Giffords = shot & put in waking coma
In months made progress w/ physical rehab - could walk under supervision, control 1 arm & leg, could read, understand & speak short phrases
5% of ppl recover

47
Q

Individual differences

Recovery

A

Age
How quick = in therapy
Level of education
Seriousness
Pre-existing conditions
Whether = localised

48
Q

Practical Applications

A

Understanding the plasticity process = contrib to neuro rehab after illness/ injury
Spontaneous recovery slows after a few weeks is physical therapy is needed to increase func movement
Electrical therapy to counter deficits in motor / cognitive func

49
Q

Negative Plasticity

A

Brains ability to rewire can have cons
Medina et al (2007) decreased cognitive func = increased dementia risk
60-80% of amputees get phantom limb syndrome - experience sensations due to cortical reorg in somasen cortex - Ramachrandran & Hirsten

50
Q

Animal Research

A

Early evidence of plasticity & func recovery = study by Hubel & Wise (1963) - sewed 1 eye of a kitten shut & found the visual cortex of the shut eye = active & processed info from open eye

51
Q

Age & Plasticity

A

Func plas decrease w/ age
Ladina Bezzola et al (2012) found 40hrs of golf training = changes to the neural movement in ptps age 40-60 using fMRI observed desired motor cortex activity in novice players comp to control = m efficient after training

52
Q

Cognitive Reserve

A

Evidence suggests that a persons education attainment may influence how well brain func adapts after injury
Schneider et al (2014) discovered M time brain injury patients spent in education increased chance of dis ability free recovery = 40% in M than 16 yrs of education and 10% if less than 12 years

53
Q

Hemispheric Lateralisation

A

Idea that the 2 1/2s of the brain = func dif & each hem has func specialisations

54
Q

Split Brain Studies

A

Sperry & Gazzaniga (1967) = 1st to investigate hem lat
SB patients have procedure that cuts their corpus callosum to treat severe epilepsy

55
Q

Procedure

A

Describe: pic presented to L/R visual field & describe
Tactile: object placed in L/R hand describe / pick sim object
Drawing: pic in L/R visual field had to draw

56
Q

Describe

A

Could describe if in R VF - L hem = lang prod
In L VF couldn’t R hem often said = nothing there

57
Q

Touch Recognition

A

R: could verbally describe & identify with sim object
L: couldn’t describe but could guess similar object

58
Q

Composite Word

A

If 2 words presented 1 to each VF simultaneously could select object with their left and say the word

59
Q

Drawing

A

R: attempted = not as clear
L: better at visual motor tasks = clearer R hem = superior for motor tasks

60
Q

Matching Faces

A

R hem = superior in facial recognition when asked to match faces processed in R hem = consistently selected
L: ignored 1/2
L hem = superior in verbal description & right in matching

61
Q

Support for Hemispheric Lateralisation

A

Rogers et al (2004) found domestic chickens brain lat allowed them to do 2 tasks simultaneously e.g., find food & watch for predators
1 hem doing 1 and 1 the other

62
Q

Positives of the Method

A

Used standardised procedure - ppl stare at fixed spot and shown image for 10th of a millisecond so eye can’t move image to other visual field - only one hemisphere receives so = good validity

63
Q

Problems w/ Generalisation

A

Ideographic - SB = rare & ptps = hard to find so = small number & not easily generalisable to population

64
Q

Theoretical Basis

A

Prompted theoretical debate about level of communication between hems in normal func & nature of consciousness
Pucetti (1988) argued we are all in 2 minds

65
Q

Differences in Function Could be Overstated

A

Mod neuro scientists argue distinction is less clear between hems & = constant com when performing everyday tasks
Any behaviours associated with 1 can be done by the other

66
Q

FMRI

A

Functional Magnetic Resonance Imaging
Detects changes in blood oxygen & flown as a result of neural activity
Active brain areas consume more oxygen
Produces 3D activation maps showing larger amounts of oxygen in brain areas

67
Q

FMRI Evaluation

A

Unlike PET scans doesn’t use radiation = virtually risk free & non invasive & straight forward
High spatial resolution - mm
= expensive, have to stay still, poor temp res = 5 second lag, only measure blood flow not neurons so not what kind of activity

68
Q

EEG

A

Electroencephalogram
Measure electricity in the brain via electrodes on the scalp, scan recording reps brainwave patterns generated from neuron action = overall account of brain activity
Used for diagnosis of conditions e.g., epilepsy chara by unusual arrhythmic patterns

69
Q

EEG Evaluation

A

NB for epilepsy diagnosis, helped understand sleep stages, = v high temporal resolution 1 millisecond
Gen info not pinpointed to source & doesn’t distinguish between sim areas

70
Q

ERP

A

Event Related Potentials
Isolate specific neural response associated with sensory/cognitive/motor events
Statistical averaging tech used to filter out extraneous activity so only response to stimulus = left
Triggered by specific events
Research shown ERPs linked to cognitive processes e.g., perception & attention

71
Q

ERP Evaluation

A

Partly addresses EEG issues = m specificity in measurement of neural processes, good temp res led to widespread use of measurement of cognitive func & can identify many different types to describe precise roles
Lack of subordination in method between different studies dif to confirm findings, dif to get pure data

72
Q

Post Mortem

A

Analysis of brain after death in psych likely bc have rare disorder & unusual deficits - damage = examined to establish cause & = comp to norm

73
Q

Post Mortem Evaluation

A

Vital for understanding key processes - Broca & Wernike, improves medical knowledge & gens hypothesis
Causation = issue observed damage might not = cause & = ethical issues - consent before might not = informed e.g., HM no mental ability to for consent

74
Q

Bio Rhythms

A

= any cyclical change in level of bodily chemicals / func

75
Q

Circadian Rhythms

A

Occur over approx 24 hours

76
Q

Sleep Wake Cycle

A

EPs: suprachiasmatic nucleus = bundle of nerve fibres above the optic chiasm, stims pineal gland to release melatonin - EZ & EP interact & melatonin wakes up

77
Q

Siffre’s Cave

A

2 months in a cave, no tech, clock, calendar / sun
Slept & ate when body told him to
After 1st 62 days resurfaced 17th Sept thought = 20th Aug
Settled into 24hr & 30 min cycle
2nd time = 24hrs
Lacks pop val & eco val & control - had artificial light

78
Q

Aschoff & Wever’s Bunker (1976)

A

Ptps in WW2 bunker for 4 weeks - no natural light
25-27 hr SWC 1 = 29hr - EP controls

79
Q

Folkard’s Cave (1985)

A

12 ptps iso from natural light for 3 weeks manip clocks day = 22 hrs
No one could adjust comfy shows strength of CR = free running ?s EZ

80
Q

Methodological Problems

Duffy (2001)

A

found some ppl = larks & others owls
Changes w/ age

81
Q

Practical Application CRs

A

Knowledge of CR troughs (Boivan 1996) & desynchronisation helped inform workplaces how to avoid accidents caused by these e.g., Chernobyl
Research has helped decrease stress of shift work - can cause heart disease (Knutson 2003) - fixed / phase delay not advance

82
Q

Czeisler (1999)

A

Found individual sleep wake cycles can vary 13-65 hrs also showed you can use dim light to adjust from 22-28 hrs meaning Siffre’s lamp could = confounding

83
Q

Infradian Rhythms

A

Take longer than 24hrs to complete

84
Q

SAD

A

Depression w/ seasonal pattern = circannual rhythm = yearly
Attributed to long periods of darkness in winter - melatonin = secreted longer so decrease serotonin

85
Q

Menstrual Cycle

A

1st day womb lining sheds & ends before next period
Increased oestrogen levels during menstruation = ovulation
Progesterone thickens for menstruation

86
Q

Entrainment

A

Menstrual synchrony = McClintock - 29 women w/ irregular periods
Gathered armpit sweat from 9 & rubbed on upper lip of 20
68% experienced change in cycle = more in line w/ donor

87
Q

McClintock Meth Problems

A

Confounding variables could change bc of other factors e.g., stress, diet, exercise, chance
= small sample & reliant on self report
Trevathan (1993) failed to replicate in small samples

88
Q

Evolutionary Basis (menstruation)

A

= good to fall pregnant same time = col care e.g., breast feeding = m likely to survive
Valid = ? By Shank (2004) as ovulating at same time have to comp for best male & resources - not synch would = adaptive

89
Q

Ultradian Rhythms

A

Happen more than once in 24 hrs

90
Q

Stages of sleep

A

4-5% = light sleep - muscle activity slows = occasional twitch
45-55% = breathing & heart rate slow and body temp decreases
4-6% = deep sleep = slow delta waves
12-15% = v deep sleep = rhythmic breathing, lim muscle movement, delta waves, dream
20-25% = REM sleep rapid eye movement, brainwaves speed up, dream, muscles relax, heart rate increases & breathing = rapid & shallow

91
Q

Dement & Kleitman (1957)

A

Monitored brainwave activity of 9 ptps using EEG
REM activity correlated w/ dreaming
Replication found sim findings REM =NB

92
Q

Basic Rest Activity Cycle

A

Suggested = 90 minute rhythm best violinists 3 seshs = 90 min / less w/ breaks

93
Q

Randy Gardner

A

Some sleep stages = m NB
Awake for 264hrs = blurred vision & disorganised speech
Slept 15 hrs = 25% lost sleep = recovered 70% of stage 4 sleep 50% of REM & v little of the others - allows flexibility

94
Q

Endogenous Pacemakers

A

Internal body clocks that regulate bio rhythms

95
Q

SCN

A

Information from eye stims melatonin released
Have own regular rhythms
M NB EP

96
Q

Pineal Gland

A

Small structure in the brain
Secretes melatonin
Regulates bio rhythms & controlled by SCN

97
Q

Melatonin

A

SCN passes info on day length & light to PG during night releases melatonin
Induces sleep = inhibited in awake periods
Could cause SAD

98
Q

Chipmunks

A

DeCoursey (2000) destroyed the SCN in some chipmunks
Found = more active at night & m likely to be eaten

99
Q

Mutant Hamsters

A

Ralph - if mutant hamsters are bred w/ CR of 20hrs & their SCN is put in a normal hamster then have 20hr CR

100
Q

Binkley (1977)

A

Chickens = v responsive to light - out hat over their eyes did not what up at dawn

101
Q

Not just SCN

A

Body temp rhythm persists when both SCNs are removed - suggests = another
Maybe = ventromedial hypothalamus
Aldcroft (1996) spent weeks in a cave after 25 days her temp adapted 24hr rhythm but sleep = 30hr

102
Q

Problem w/ Animal Studies

A

Gen prob
Ethical issue - chipmunks put at more risk when returned home
Does the discovery justify

103
Q

Exogenous zeitgebers

A

External cues that may affect / entrain our bio rhythms
E.g., light on sleep wake cycle

104
Q

Light

A

Can reset SCN plays role in s/w cycle
Also has role in controlling hormone secretion & blood circulation

105
Q

Campbell & Murphy (1998)

A

Demoed that light may be detected by skin receptor in other areas not just eyes
15 ptps woken up at various times via light shone on the backs of their knees
Produced variation in sleep wake cycle of up to 3hrs which suggests light = powerful EZ

106
Q

Social Cues

A

Mealtimes, radios, clocks
Human infants born w/ random s/w cycle
At about 6 weeks Cars begin
By 16 weeks = m entrained schedules imposed by mealtimes

107
Q

Research w/ Blind ppl

A

NBance of light as a EZ - have different sleep patterns 1 man blind from birth Had CR of 24.9 hrs had to take stimulants and sedatives to set rhythm - Miles et al (1977)
Czeisler (1995) discovered some respond to exposure to very bright light w/ decreased melatonin levels - suggests = 2 pathways eyes & SCN

108
Q

Campbell & Murphy Method Issues

A

Haven’t been replicated
May have been some light to eyes = CV
Doesn’t show effect of other EPs

109
Q

Influence may be overstated

A

Miles et al (1997) blind man = exposed to social cues & didn’t change
Ppl in arctic regions have normal pattern even tho in summer = no dark

110
Q

Interactionist

A

Only in extreme circums are EPs free running
Total iso studies eg Siffre = rare & lack val
In real life EP & EZ interact wouldn’t make sense to separate in research

Psychology (11 decks)

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