Arrythmias Flashcards

1
Q

How does A Fib appear on an ECG?

A

Irregularly irregular rhythm, no distinct P waves, narrow QRS complexes, variable R-R intervals.

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2
Q

What are common causes of atrial fibrillation?

A

Hypertension, valvular disease, heart failure, thyrotoxicosis, alcohol (“holiday heart”).

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3
Q

What is the most characteristic ECG finding of atrial flutter?

A

Sawtooth flutter waves, usually best seen in inferior leads (II, III, aVF); regular rhythm.

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4
Q

What is the typical atrial rate in atrial flutter?

A

Around 250–350 bpm, with a fixed ventricular response (e.g., 2:1 block).

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5
Q

How is supraventricular tachycardia (SVT) identified on ECG?

A

Narrow QRS complex tachycardia (>150 bpm), often no visible P waves due to overlap with T waves.

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6
Q

What maneuver can help diagnose or treat SVT?

A

Vagal maneuvers or adenosine administration (can transiently block AV node).

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7
Q

What ECG finding is diagnostic of ventricular tachycardia (VT)?

A

Wide QRS complex tachycardia (>120 ms), regular rhythm, no clear P waves.

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8
Q

What are some causes of VT?

A

Ischemic heart disease, cardiomyopathy, electrolyte imbalances (e.g., hypokalemia).

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9
Q

How can you distinguish VT from SVT with aberrancy?

A

VT has AV dissociation, capture/fusion beats, extreme axis deviation.

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10
Q

What does ventricular fibrillation (VF) look like on ECG?

A

Chaotic, disorganized electrical activity; no identifiable QRS complexes, P waves, or T waves

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11
Q

What is the clinical significance of VF?

A

Medical emergency requiring immediate defibrillation; patient is pulseless.

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12
Q

What is a key ECG feature of Torsades de Pointes?

A

Polymorphic VT with twisting QRS complexes around the isoelectric line; prolonged QT interval.

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13
Q

What are common causes of Torsades de Pointes

A

Prolonged QT (drugs, hypokalemia, hypomagnesemia, congenital long QT)

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14
Q

What ECG feature defines first-degree AV block?

A

PR interval >200 ms; every P wave followed by a QRS.

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15
Q

How does Mobitz type I (Wenckebach) AV block appear on ECG?

A

Progressive PR lengthening until a dropped QRS complex.

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16
Q

What is the ECG hallmark of Mobitz type II AV block?

A

Fixed PR interval with intermittent dropped QRS complexes; more dangerous than type I.

17
Q

What characterizes a third-degree (complete) heart block?

A

No relationship between P waves and QRS complexes; atria and ventricles beat independently.

18
Q

What is a junctional rhythm on ECG?

A

Bradycardia with narrow QRS, absent or inverted P waves (if present).

19
Q

What is a bundle branch block pattern on ECG?

A

Wide QRS (>120 ms); RBBB = RSR’ in V1, wide S in V6; LBBB = broad monophasic R in I/V6, deep S in V1.

20
Q

What is the defining ECG feature of pre-excitation syndromes like WPW?

A

Short PR interval (<120 ms), delta wave (slurred upstroke of QRS), wide QRS complex.

21
Q

What arrhythmia is most associated with WPW syndrome?

A

Paroxysmal SVT, especially AVRT (atrioventricular reentrant tachycardia).

22
Q

What rhythm is defined by irregularly irregular wide QRS complexes in a patient with WPW and AF?

A

Pre-excited atrial fibrillation—dangerous, can lead to VF.

23
Q

What is the normal PR interval?

A

120–200 ms.

24
Q

What is the normal QRS duration?

A

<120 ms (less than 3 small boxes)

25
Q

What does sinus bradycardia look like on ECG?

A

Normal P-QRS-T morphology with rate <60 bpm

26
Q

What does sinus tachycardia look like on ECG?

A

Normal rhythm with rate >100 bpm, P waves preceding each QRS.