Applying to everyday problems Flashcards
road accident facts
1.25 millions death each year (WHO 2015)
9th common cause of death
under reported
car manufacturers promote hands free phones is safe-distraction leads to death
Figures on driving and phone use
45% drivers text in UK according to RAC 2008
75% of 18-25 year olds text according to Pennay 2006 australia
Dingus 2016
naturalistic study
cars equipped with sensors and cameras
3,500 US drivers
case control method:compare what proceeds crash and non-crash events
mobile use increases likelihood of crashing 4 fold
using touch screen increases likelihood 5 fold
human visual system
visual acuity decreases at peripheral
eye makes 3 fixations a min
attention part of perception
change blindness-failure to detect visual change
inattentional blindness-failure to notice unexpected event when attention diverted to other aspect
theoretical model of attetnion
limited mental resources, selective attention required
distinction between automatic and controlled processing
Rasmussen: hierarchal model of driving: operational->tactical->strategic
Norman and shallice 1980
two systems in attention
lower level: contention scheduling
higher level: supervisory attentional system
automatic response to a trigger is lower level
if don’t get result from lower level, higher level relied on to get result
Hockeys compensation control theory
drivers may preserve performance on primary task at expense of secondary task
results in driving performance decreasing
Wickens multiple resource theory 1984 - task interference
task interference affected by 3 factors: mode of input (auditory, visual, tactile), type of coding (spatial, verbal), type of response (manual, visual)
Redelmeier and Tibshirani 1997 - phone bills and drivinh
case control study
itemised phone bills of 699 toronto drivers
driver reports crash, see if phone in use in moments leading up to crash
risk collision 3-6.5 times greater if phone in use
collision risk same for all types of drivers
Alm and Nilsson - braking responses
1994-braking reponse to symbol, RT increased from 0.95 to 1.3 when using phone
1995-emergency brake inresponse to car infront, RT increased from 1.6 to 2.2 when phone in use
Hancock, Lrsch and simmons -normal driving ….
normal driving is long periods of low demand interspersed with moments of crucial responses
RT in emergency braking slowed by dual task
Atchley 2017-difference between handheld or handsfree
no difference between hands free and hand held phone
81% phone studies show significant impairment
Strayer and Johnston 2001 - RT to red light and conversation
RT to red light
participnats listen to radio, converse over phone or undivided attention
active participation in conversing is problem
passive listening no affect on performance
loss of non-verbal cues requires more concentration to maintain convo
passengers adjust convo to driving conditions
Crundall, bains chapman and underwoods 2005 - phone distraction
participants drove in silence or conversed with passenger or on phone
phone just as distracting as mobile phone
Hyman unicycling clown 2010
watched students cross courtyard
noted how often change direction, acknowledgements
if on phone lack situation awareness, take longer to cross, change direction more, didnt see unicycling clown
recrate and nunes 2000- eye movement in driving
verbal and spatial imagery tasks while driving
both increased pupil size
both tasks reduced gaze distribution
spatial task produced more eye fixations
Briggs, hole and land 2011 - eye movement with phobics
eye movements in stimulated motorway driving
participants on phone, conversation about spiders
spider phobic more fixed view, cognitive tunnel vision
non phobic less fixations, lower cog tunnel vision
Briggs hole and land 2016-visual imagery
conversations involving visual imagery
imagery and perception share processing systems
imagery and ‘real world’ compete for attention
primary task: hazard perception
secondary task: sentence verification with imagery inducing or non imagery inducing statements
distracted participants detect fewer hazards
distraction increases with imagery induction
effects worse for high imageners
Drains on processing resources
driving-vehicle control, hazard perception, hazard detection, hazard perception, lane keeping
phones-problem solving, maintenance of convo, mental imagery, emotional effects of convo
immediate consequences:compensatory behaviour eg driving slower
ultimate consequences:increased RT to hazards, earlier onset fatigue
result in increased accident risk
psychological theories of driving
norman & shallice: theory of controlled and automatic processing-relying on contention scheduling
Hockeys compensatory control theory: compensate for impairment by using mirrors less, driving slower
wickens multiple resource theory: explain why driving and phone use interfere
why drivers use their phones
fail to notice own impairment, effects are cognitive as vehicle control not affected
self serving bias-better driver than everyone
faulty risk perception-illusion of control:self driving cars
protective beliefs-im a good driver
accidents are rare so no immediate penalties, every trip reinforces delusion they are safe
theory of planned behaviour
lieklihood of performing behaviour affected by:
perceived benefits-not wasting time
perceived costs-low accident risk
perceived acceptability-everyone does it
benefits and acceptability override costs
possible solutions to crashes
educate-make drivers aware
enforcement-detection and prosecution
engineering-ignition interlocks
future-driverless cars, drivers not distracted
driverless cars
semi autonomous:driver assisted systems, control driving for 5 seconds, there to support human ability, encourage laziness
fully autonomous:robot taxis,self driving cars dont require human driving
why mobile phones are problimatic
cognitive burden of maintaining convo
remote conversors unaware of driver situation
visual imagery competes with visual perception of surrounding
involve extended periods of distraction
whats clinical psychology
application of psychological theories to understand, prevent and alleviate distress
what do clinical psychologists do?
work in range of settings, with range of clients, all ages genders disabilities complete assessments formulate theories to undertsand suggets interventions communicate with clients and colleagues
key tasks of clinical psychologists
- assessment:process used to answer questions eg what led to problems
formulation: collaborative process, BioPsychoSocial focus, seek to explain problems
intervention: driven by formulation, acts that should bring about change
evaluation: feedback from clients, symptom reduction?
communication: consultation, research, teaching, training
clinical methods of assessment
clinical interview: one to one with client or carer
psychometrics: questionnaires, neuropsychological tests
self monitoring: diary and record forms to understand daily experiences
observation: observe behaviours and social interactions, completed by psychologist or teacher/carer
PioPsychoSocial model
- how different aspects influence illness
- interconnection between biology, psychology and socio-environmental factors
- biology: genetics, neurology, physiology
- psychology: emotion, thoughts, memory
- social factors: relationships, family, culture, society
cognitive formulation of panic
- trigger stimulation
- perceived threat
- apprehension
- body sensations
- interpretation of sensations as catstrophic
models of practice and training
scientist practitioner: training emphasis on scientific knowledge, research, critical skills
reflective practitioner: ability to reflect on work n themselves, how own personality and history affects practice
reflective practice
maintaining awareness of biases, what may influence application or professional knowledge, skills, experience eg cog bias
supervision, consultation and personal therapy to extend knowledge of self in practice
characteristics of depression
feeling saddness, hopelessness, dejection, anhedonia
motivational deficits: lack ambition
behavioural symptoms: appetite change
physical symptoms: sleep disturbance, wieght change
cognitive features: negative thoughts, low self esteem
onset of depression
episodes following significant life events or series day to day difficulties
can be due to no reasons
risk factors: early adversity, family or personal history, economic and educational disadvantage, negative life events, low levels of close social support
assessment od depression
questionnaire: Beck depression inventory, patient health questionnaire
clinical interview: understand difficulties, experiences, symptoms severity
depressive disorders
characterised by extended periods of clinical depression, cause distress to individual and impairment in social functioning
example of depressive disorders
persistent depressive disorder:mild symptoms over long periods of time
premenstrual dysphoric disorder:symptoms surface in time with period
disruptive mood dysregulation disorder
bipolar disorder: pattern of depressed and manic moods
biological theories of depression
genetic factors-twin concordance rates, 46% fro monozygotic and 20% dizygotic
structural and functional brain differences-lower level brain activation in areas associated with emotional regulation
neurochemical factors-lower levels of seratonin dopamine and norodrenaline
treating depression
drugs can alleviate symptoms
tricyclic drugs, monoamine oxidase inhibitors
psychological theories of depression
psychodynamic: result of unconscious process that unfolds and responds to loss
cognitive: not what happens but the view we take of it, maintained by negative schemas and negative triad
behavioural: results from lack of reinforcement from pleasurable or meaningful activities
attachment: early relationships with carers pave way to patterns of relationships may be depressogenic
systematic: individuals experiences cant be understood in isolation, depression is result of wider system
negative triad
self - biased, unrealistic view of worthlessness
world - unkind
future - hopeless
individual is left vulnerable to activation of mood congruent thoughts and feelings characteristic of depressoin
depression interventions
medication physical exercise psychological therapies and self help -2:1 preference for therapy over drugs choice dependent on nature of problem and training of clinician
cognitive behavioural therapy
work with client to identify and evaluate unhelpful thoughts/beliefs
diary of significant events associated with mood change
6-8 sessions for mild-moderate problems
16-24 sessions for severe to complex problems
behavioural activation
activity scheduling to help re-engage with pleasurable and meaningful activities to reinforce positivity
behavioural couple therapy
relationships where one or both has depression
improve communication, problem solving, promote acceptance
interpersonal psychotherapy
brief attachment focused therapy
grief and loss
changes in relationships
short term psychodynamic therapy
bring unconscious to conscious to understand psychic conflict
improve self knowledge on problems
counselling
relationship between client and therapist
talking therapy to help deal with emotions
couples, family and systematic approaches
problem is in system not the individual
focus on relationship dynamics, communication, conflict and functioning
mindfulness based cognitive therapy
preventing relapse
evaluating own work
for the individual:session by session to see if works
for service:numbers successfully treated, worldwide works
treatment effectiveness
majority of people can get over or learn to deal with depression if help is gotten early on
CBT has largest evidence base for helping
CBT as effective as anti depressants
whats a specifc phobia
excessive fear of specific and identifiable object or situation eg claustrophobia
can acquire a fear about anything
ICD-10 criteria for specifc fear
A-either 1) marked fear of object/situation not included in social or agora phobia or 2)marked avoidance of object/situation
B-atleast 2 symptoms of anxiety at same time
C-significant emotional distress and recognition they are unavoidable
D-symptoms restricted to feared object/situation or when thinking of it
what are symptoms of anxiety
autonomic arousal symptoms
symptoms concerning chest
symptoms concerning head
general symptoms
specific phobia sub divisions
animal type nature forces type blood, injection, injury type situational type other type
diagnostic controversies for specific phobias
quick method to decide where to focus treatment
useful way for researchers to describe participants clearly and reliably
reduce complex individuals into simple label
criteria is vague, what constitutes as unreasonable
prevalence of specific phobias
18% adults
ford 1999-1%children have one
fears are natural part of growing up
gender bias-25% women, 18% men due to reporting bias
fear in childhood
normal to experience fears not phobias
during infancy, fear stimuli in immediate environment eg noise
age 4-8 start to fear ghost and animals
pre adolescence fear physical injury
early adolescence fear social situation and criticism
phobias in children diagnosed with care only if real impairment is shown
how phobias acquired
heritability/genetics-25-45% phobias, gene environment interaction
prenatal stress
Mowrers two stage phobia theory
1-person learn to associate stimulus with aversive outcome, lead to fear response
2-person learns to avoid stimulus to reduce ear, relief felt leads to further avoidance
Rachmans 3 pathways to fear acquisition
1-direct conditioning or experience
2-learning through observation of others
3-transmission of fearful information
nature/nurture acquisition of phobia
shy child leads to overprotective parent
shy child becomes even more anxious
preparedness therapy
more genetically predisposed to acquire fears than others
born with tendency to learn to fear stimuli dangerous to ancestors
if fear stimulus likely to avoid it and survive and pass on ‘fear’ gene
more likely to learn phobia to fear relevant stimuli after experience
cognitive model of phobias
assume cognitive biases drive phobias and cause fear response, treat cognition then behaviour disappear
phobia come from thoughts? thoughts come from phobia?
phobia develop in children lacking language
phobias are learnt independently from cognition, cognition maintains fear
treating specific phobias
baseline assessment of clients difficulties
formulation of clients difficulties
establish goals of therapy
intervention
successfulness of cognitive therapy
improvements in relationships
improvements in confidence
cognitive therapy not changed since 1990s as effective
ICD-10 for oppositional defiant disorder
A-doesn’t meet criteria fro dissocial personality disorder, schizo, mania, depression, pervasive developmental disorder or hyper kinetic disorder
B-presence of four or more symptoms from criterion list, no more than 2 from severe
C-symptoms in B must be maladaptive and inconsistent with developmental level
D-atleast four symptoms must be present for atleast 6 months