Anatomy: The Eye and Raised ICP Flashcards

1
Q

what is the normal intraocular pressure

A

12-22mmHg

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2
Q

what can cause raised ICP

A

brain tumour

head injury

hydrocephalus

meningitis

stroke

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3
Q

Monro-Kellie hypothesis

A

the cranial compartment is incompressible and the volume inside it is fixed - made up of brain tissue, blood and CSF

the cranium and its constituents create a state of volume equilibrium, such that an increase in the volume of one constituent must be compensated for by a decrease in the volume of another

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4
Q

what are the principle buffers in the IC cavity to increased volume

A

CSF and, to a lesser extent, blood volume

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5
Q

what damage can raised ICP cause in the cranial cavity

A

damage to tissues, shifts in tissues, herniation and constrictionof blood vessels

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6
Q

what are the signs and symptoms of raised ICP

A
  • Headache – worse on awakening and may wake them from sleep, exacerbated by cough, bending, sneezing etc.
  • Vomiting without nausea
  • Ocular palsy
  • Back pain
  • Papilloedema
  • Altered level of consciousness
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7
Q

what effect can raised ICP have on the eye

A

65-75% patients with ICP report visual problems

transient blurred vision

double vision

loss of vision

papilloedema

pupillary changes

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8
Q

is the effect of raised ICP on the eye uni or bilateral?

A

either

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9
Q

how is raised ICP transmitted to the eye

A

via the optic nerves - these are surrounded by cranial meninges, and found in the subarachnoid space

this becomes abnormally filled with CSF

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10
Q

what is seen on fundoscopy of papilloedema

A

bulging optic disc

disc margins are blurred and in places retinal vessels are concealed, because oedema has impaired the translucency of disc tissues.

optic cup is lost

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11
Q

describe the 3 layers of the meninges

A
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12
Q

which nerve innervates the dura mater

A

sensory supply from CNV

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13
Q

what is enclosed between the 2 layers of the dura mater

A

dural venous sinuses

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14
Q

what do the dural venous sinuses drain to

A

IJV at jugular foramen

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15
Q

where is the jugular foramen

A
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16
Q

describe the vascualture and innervation of arachnoid mater

A

avascular and no innervation

17
Q

arachnoid granulations

A

small projections of arachnoid mater into the superior sagittal sinus in dura that allow CSF to re-enter the venous circulation via the dural venous sinuses

18
Q

subarachnoid space

A

contains circulating CSF that acts to cushion the brain

also contains blood vessels

19
Q

pia mater

A

‘faithful mother’

thin and tightly adhered to the surface of the brain and spinal cord - follows the contours of the brain (gyri and sulci)

highly vascularised

20
Q
A
21
Q

which other nerve is susceptible to damage

A

CNIII

22
Q

what happens if CNIII is damaged

A

paralysis of motor innervation - LPS, SR and IR, MR, IO

paralysis of parasympathetic innervation of sphincter of pupil (ciliary ganglion and short ciliary nerve)

results in lose/slowness of pupillary light reflex, dilated pupil, ptosis and eye turned inferolaterally (down and out)

think, this is because LR and SO are not innervated by CNIII, LR pulls laterally, SO pulls down and laterally

23
Q

name the septas that dura mater forms

A

also, diaphragma sellae over pituitary fossa

the brain can herniate through these

24
Q

what is the purpose of the tentorial notch

A

to allow passage of brain stem

25
Q

how can CNIII be damaged

A

if raised ICP causes the medial temporal lobe to herniate through tentorial notch

26
Q

what action will not be able to be performed with CNIV palsy

A

eye cannot move inferomedially

  • diplopia when looking down
27
Q

what action will not be able to be performed with CNVI palsy

A

lateral rectus - eye cannot move laterally in horizontal plane