anaethetics Flashcards
metformin on day of surgery
OD or BD = take as normal
TDS: miss lunchtime dose
assumes only one meal will be missed during surgery, eGFR> 60 and no contrast in procedure
reverse action of benzo eg midazolam
flumazenil
commonly used induction agents
propafol
sodum thiopentone
ketamine
etomidate
what to check before parathyroid surgery
methylene blue to identify gland
checklist before induction of anaesthesia
pt confirmed: site indentity, procedure, consent
site marked
anaesthesia safety check
pulse ox on pt
allergies?
difficut airway/asp risk?
risk of >500ml blood loss (kids7ml/kg)
3 phases of operation checklist
sign in - b4 anaethesia
time out - before incision
sign out - b4 pt leaves op
what to check before thyroid surgery
vocal cord check
sentinal node biopsy
radioactive marker/patent blue dye
surgery involving thoracic duct
consider administration of cream
pheochromocytoma surgery
alpha and beta blockage
surgery for carcinoid tumors
cover with octreotide
colorectal cases
bowel prep esp if left sided surgery
when is adrenaline with local contraindicated
pt taking MOAI or TCAs
tx for local anaesthetic toxicity
IV 20% lipid emulsion
increased risk of local anaesthetic toxicty
liver dysfxn
low protein state
how does lidocaine work
blocks Na channels in axon = disrupts action potential
paralytic ileus what is it and what can contribute
pseudo obstruction bc reduced bowel peristalsis post bowel surgery
deranaged electrolytes can contribute thefore check potassium mag and phos as might show this AND hypovolaemia before nausea and vom begins
mx of post op/paralytic ileus
nil by mouth initially –> small sips clear fluids
nasogastric tube if vomiting
IV fluids (correct any electrolytes also)
total parenteral nutrition for prolonged/severe cases
colonoscopy prep
laxative day before
no food 24hrs
sulfonyurea eg and day of surgery
gliclazide
omit on day of surgery
unless morning surgery in Pt who take BD = can have afternoon dose
hyponatraemic encephalopathy post op causes
hypotonic IV fluid eg 0.45% sodium chloride
also trauma and stress = SIADH
paeds patients fluids caution
dont use hypotonic (0.45%) solution in peads __> in risk of hepatic encephalopathy
when to not use adrenaline with local anaesthetic
on digits = ischaemia
patients with myasthenia gravis sensitive to what paralytic agents and why
non-depolorising agents (rocuranium)
bc they work by competitively antagonising nicotinic acetylcholine receptors in motor end plate
in MG less of these receptors bc autoimmune distruction = more sensitive to non-depolorising blockade
examples of non depolorising paralytics and reversal agents and SE
curoniums eg
rocuranium
vecuranium
tubacurarine
reverse with neostigmine (achesterase inhib) and sugammadex
hypotension
ASA I classification
normal healthy pt
healthy non smoking minimal alcohol use
ASA II classification
pt w mild systemic disease
without major functional limitations ie
current smoker, social alochol drinker, pregnancy, Obesity (BMI30-40), well controlled DM/HTN, mild lung disease
ASA III classification
pt w severe systemic disease
one or more mod to severe disease w big functional limit ie
poor control DM, HTN
COPD
morbid obese (bmi>40)
active hepitis
alcohol abuse
pacemaker
reduced ejection frac
ESRD - undergoing reg dialysis
hx >3months ago MI
CVD accidents
ASA IV classification
pr w severe systemic disease constant threat to life
<3mths MI
CVD accidents
ongoing cardiac ischaemia or severe valve dysfxn
severe red Eject frac
sepsis
DIC
ARD
ESRD not having red dialysis
ASA V classification
moribund pt not expected tto survive w/o op
ruptured AAA
massive trauma
intracran bleed w mass effect
ischaemic bowel in face of sign cardiac pathology
multiple organ system/dysfxn
ASA VI classification
pt declared brain dead whos organs being removed for donor purposes
depolorisng agents examples and how they work
succinycholine ie suxamethonum
binds to nictonic ach receptors (non compet) = persistant depolorization of motor end plate
thats why might get fasciculations