Alterations of Veins, arteries and dysrhythmia Flashcards

1
Q

Desies of the veins are?

A
  1. Varicose veins
  2. Chronic venous insufficiency
  3. Venous stasis ulcers
  4. Thrombosis
  5. deep vein thrombosis
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2
Q

Varicose veins are

A
  • a vein in which blood has pooled
  • usually caused by damaged or incompetent valves
  • Pooling causes veins to be palpated, distended, and tortuous
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3
Q

Risk factors of varicose veins

A
  • age
  • family history
  • pregnant women
  • obesity
  • prolonged standing
  • wearing tight clothing around the thighs
  • crossing legs at the knees
  • deep thrombosis
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4
Q

Chronic venpus insuficensiy is

A
  • inadequate venous return over long period of time
  • progression of varicose veins
  • has the same risk factors
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5
Q

Venous stasis ulcer is

A
  • caused by venous insufficiency
  • high venouse pressures, circulatory stasis and tissue hypoxia leads to inflammatory response
  • leads to remodeling of the skin and ulcer development
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6
Q

Thrombosis formation is

A
  • formation of blood clot through
    1. Thrombosis: blood clot attached to a vessel wall. venous thrombi are very common
    2. Embolism: bolus of matter that is circulating in the blood
    3. Thromboembolism: detached thrombus
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7
Q

risk factors of thrombus

A

Triad of Virchow
- venous stasis
- venous endothelial damage
- hypercoagulable state

others
- cancer
- spina cord injury
- heart trauma
- immobility
- orthopedic surgery

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8
Q

Deep Vein Thrombosis

A
  • blood clot found in deep vein
  • found usually in the lower extermities
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9
Q

Clinical manifestations of deep vein thrombosis

A
  • unilateral
  • erythema
  • warmth
  • pain
  • tenderness with palpation
  • varicose veins
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10
Q

Disease of arteries are

A

1.orthostatic hypotension
2. Aortic aneurisms
3. Aortic dissection
4. Raynaud phenomenon
5. Atherosclerosis

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11
Q

Orthostatic hypotension is

Clinical manifastations?

A
  • decrease in the systole and diastolic blood pressure while standing due to lack of normal blood pressure compensation in response to gravity
  • can be acute or chronic

Clinical manifestation
- dizziness, blurry vision, and loss of vision
this are because there is lack of blood return to the brain and vasomotor compensation

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12
Q

Aortic aneurisms?
Clinical manifestation?
Risk factors?

A
  • dilation or outpouching of a vessel wall
  • most common thoracic and abdominal aorta

Risk factors:
- smoking
- atherosclerosis
- hypertension

Clinical manifestations
- asymptomatic unil rupture
- dysphagia and dyspnea

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13
Q

What is the difference between true and false aneurisms?

A

True aneurism affects all three layers of the arterial walls of the blood vessel while false aneurism is extravascular hematoma (clot) that communicates within the intravascular space

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14
Q

Aortic dissection is

A

is the tearing in the vessels and when blood gets into the wall of an artery

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15
Q

Raynaud phenomenon is ?
Clinical manifestations?

A
  • episodic vasospasm which is narrowing of the arteries by persistent contractions of the blood vessels

Clinical manifestations
- color changes in finger, toes, ears, and nose ( from white, blue, red)
- cold
- painful
- numbness

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16
Q

Atherosclerosis is ?
Arteriosclerosis?
Risk factors?

A
  • form of arteriosclerosis that is formed by the thickening and hardening caused by accumulation of lipid- laden macrophages in the arterial wall
  • plaque development

Arteriosclerosis is chronic disease of arterial system that is caused by abnormal thickening and hardening of the vessel walls

Risk Factors
- age
- family history …genetic
- tobacco use
- diabetics
- weight
- stress
- hyperlipidemia
- uncontrolled hypertension
- physical inactivity

17
Q

Peripheral Artery Disease (PAD)

A
  • progressive narrowing and degeneration of arteries of upper and lower extremities
  • usually affects lower extremities
  • most commonly results from atherosclerosis
18
Q

PAD clinical manifestation

A
  • severity depends on the site, degree of blockage, and amount of collateral circulation
  • intermittent claudication….. clamping, tired, burning in legs, aching
  • usually occurs with activity and goes’ away with rest
  • numbness, tingling, neuropathy
  • skin is thin, shiny, taut, hairless
    -elevation pallor: between legs is elevated because of gravity
  • dependent rubor( reactive hyperemia): causes redden position
  • decreased capillary refill
  • diminished or absent pulses depending on severity
  • thick toe nails
  • ulcers, gangrenous toes
  • cold
19
Q

common dysrhythmia is

A
  • abnormal physiological rhythm
  • consists of
    1. Normal sinus rhythm
    2. simious bradycardia
    3. Sinus tachycardia
    4. Arterial fibrillation
    5. Heat block
    6. Premature ventricular contractions
    7/ Ventricular fibrillation
    8. Ventricular Tachycardia
    9 . Sudden cardiac death
20
Q

Normal sinus rhythm is

A
  • SA nodes firing 60/100 beats/ min
  • follows normal conduction pattern
    -p and QRS wave is normal
  • PR interval is normal
21
Q

Sinus bradycardia

A
  • SA nodes fire < 60 beats/min
  • normal for athletes and during sleep
  • occurs during parasympathetic nerve stimulation and certain drugs
  • P wave is less and QRS is pointy
  • associated with some diseases
22
Q

Sinus tachycardia

A
  • SA nodes fire > 100 beats/ min
  • occurs during sympathetic stimulation or vagal inhibition
  • associated with physiologic and psychologic stressors
  • drugs increase heart rate
  • P wave is elevated
23
Q

Arterial fibrillation

A
  • paroxysmal or persistent
  • most common dysrhythmia
  • prevalence increases with age and heart disease
  • aka “ irregularly irregular pulse “
    causes decrease in CO2 and increase risk for stroke
  • has no pattern
24
Q

ventricular fibrillation

A
  • associated with acute MI, ischemia
  • unresponsive , pulseless, and apneic
  • if not treated causes death
    -can be treated by CPR and ACLS
    - defibrillation and drug therapy like ephenephrine and amiodarone
25
Q

Ventricular tachycardia

A
  • life threatening because it causes decrease on CO2 and possible development of ventricular fibrillation
26
Q

Sudden Cardiac Death (SCD)

A

caused by ventricular dysthymias
- ventricular tachycardia
- ventricular fibrillation

27
Q

Premature ventricular contractions are

A
  • associated with stimulus, electrolyte imbalances, hypoxia, and heat disease
  • not harmful for normal heat, but may reduce CO2 and lead to angina and heart failure in diseased heart
  • assess hemodynamic status
28
Q

heart Blocks are?

A

abnormal conduction of electrical impulses because of
- electrolyte imbalance
- ischemia or hypoxia
- infraction or MI

29
Q

Asystole

A
  • total absence of ventricular electrical activity ( flat line on ECG)
  • no ventricular contraction
  • pt is unresponsive, pulseless, apneic
  • result of advanced cardiac disease, severe conduction system problem, or end stage heart failure
  • has poor prognosis
  • treated with immediate CPR and ACLS measures, epinephrine, and intubation
30
Q

Pulseless electrical activity

A
  • no pulse but electrical activity can be seen on the ECG
  • poor prognosis unless underlying cause is quickly identified and treated
  • underlying cause can be

Hs Ts
Hypovolemia Toxins
hypoxia Tamponade
hypothermia Thrombosis
hypo/hyperkalemia tension pneumothorax
hypoglycemia trauma
hydrogen ion( acidosis)