age changes in oral tissues Flashcards
aging
aging & its effects depend on both
genetic factors
environmental factors
general effects of aging
-restricted flow of blood in the lungs
-reduced strength of the heartbeat
-increasing difficulty to digest & break down fats & carbohydrates especially if the food is not chewed properly due to poor dentition
-reduced ability of the immune system to produce disease fighting antibodies
people > 45 yrs old more prone to illnesses / diseases
cellular senescence
– reduced cell multiplication thus may cause cells to function inappropriately
EFFECTS OF AGING ON ORAL TISSUES
ENAMEL DENTINE CEMENTUM PULP SALIVARY GLANDS ORAL MUCOSA FACE
aging in enamel
attrition, erosion, abrasion
aging in dentine
primary dentine, secondary dentine scleroticdentine
PRIMARY DENTINE
forms next to enamel & circumference of the pulp
& continues until completion of the root
SECONDARY DENTINE
forms after the completion of root as the
tooth comes into occlusion
secondary dentine increases in thickness
with age
SCLEROTIC DENTINE
-Associated with physiological process
1. Dentinal tubules become occluded by minerals with age (Deposition occurs from the root apex to the cervical) 2. Dentine becomes translucent (The increase in translucency occur linearly with age) 3. Sclerotic / Translucent Dentine (can be used in age estimation)
What leads to the formation of sclerotic dentine? (1)
DIAMETER OF TUBULES GET CONSTRICTED / SMALLER WITH AGE,
Odontoblasts become crowded as they move towards the pulp. As a result some odontoblasts may disapear and the tubules become progressively occluded with the deposition of minerals to form intratubular/peritubular dentine. Theory - calcium enters the damaged process and precipitates with the phosphates (ATP & ADP) in cytoplasm. (Avery)
What leads to the formation of sclerotic dentine? (2)
- poor transmission of impulse to the
- pulp thus inefficient response towards a stimulus
- changes in the cytoplasmic content of the odontoblasts thus numbers of organelles diminishes
effect of aging on cementum
Thickening of CE at the root apex a result of compensatory tooth eruption in response to attrition/wear at occlusal surface,
hypercementosis
note abt cementum
life-long deposition occur at very slow rate increased thickness (3-folds) between the age of 17 – 70 yrs with aging the surface of the Ce becomes irregular many lacunae becomes empty
CEMENTUM hypercementosis
may result from chronic localised periapical inflammation problem during extraction procedure Paget’s disease – presence of hypercementosis in all teeth
pathological sequence of cementum
constriction of the apical foramen / becomes narrower with age