ADULT LANG Flashcards
decreased lean body mass
normal aging
decreased elasticity and flexibility of muscles
normal aging
decreased total body water
normal aging
increased fat tissue
normal aging
heart muscles thicken; valves enlarge and stiffen; muscular contraction force decreases
normal aging
less efficient oxygen use
normal aging
lower ability to remain physically active
normal aging
atherosclerosis
fat hardening
decreased lung capacity, elasticity, cilia, vital capacity, and cough
normal aging
primary concern of WHO
how is quality of life affected by health conditions
6 main aspects of health related quality of life
physical, psychological, level of independence, social relationships, environmental, spiritual
body structure
the anatomy of the body
body function
the physiology of what is actually affected
activities/ participation
activities you may NOT do
personal factors
age, gender, race, education, confidence, self esteem WITHIN the human
environmental factors
where you live, friends, family, social economic status
capacity
individuals ability to execute a task or action in a regular environment
performance
individuals ability to execute a task or action in current environment or actual contest of what they live in
A FROM
Living with Aphasia: Framework for Outcome Management
severity of aphasia (understanding other people, speaking, reading, writing)
body function and structure
communication and language environment (services, systems, policies, attitudes of others to you and the aphasia, help with communication and conversation)
environmental factors
H&P
history and physical
PHO
personal history of
PMH
past medical history
CMH
current medical history
S in SOAP
subjective observation, what you notice about your patient
O in SOAP
objective observation, DATA and facts
A in SOAP
assessment your analysis and summary similar to clinical impressions
P in SOAP
plan, steps and measures to be taken
TIA
transient ischemic attack
at risk of bigger stroke
brief focal cerebral event in which symptoms develop suddenly; part of the brain that has temporarily become ischemic
TIA
a transient episode of neurological interruptions cause by temporary brain ischemia without acute infarction
TIA
RND
reversible ischemic neurological deficit
stroke that gets completely better in 1 to 3 weeks
stroke
sudden and severe onset of a prominent and frequently persistent neurological deficit
an illness resulting from damage to the parenchyma (brain tissue) of the brain; brain death occurs (infarction)
stroke
occlusive stroke
ischemic
blockage that causes low blood flow to the brain
causes of occlusive stroke
thrombotic occlusion - stationary blockage along the arterial wall
embolic occlusion - particle that breaks away/ floats around in bloodstream
hemorrhagic stroke
rupture of blood vessel anywhere in the brain or skull
hemorrhagic stroke can occur in 4 different places
parenchyma (intracerebral), subarachnoid/ subdural/ epidural space (intracranial)
vertebral arteries
two arteries on each side of the side
basilar artery
vertebral artery branches into; supplies the brainstem
problems w basilar artery
CN III - XII
diplopia (double vision)
hearing problems
dysarthria (slurred speech CN 12), dysphagia, dysphonia (speech production CN10), hemiplagia (no mvmt), hemiparesis (weakness), respiratory
cerebellum
vertebral artery
cerebellum problems
ataxia - balance and coordination, ataxic dysarthria, cognitive problems
ANTERIOR lesion, nonfluent aphasia
MOTOR output
POSTERIOR lesion, fluent aphasia
SENSORY input
global aphasia
mixed aphasia
nonfluent
poor comprehension
poor repetition
poor naming
conduction aphasia
very poor word recognition
fluent speech
paraphasic speech
good comprehension
poor writing
poor word finding
good self awareness
lesion for conductive aphasia
arcuate fasciculus, supramarginal gyrus
Wernicke’s aphasia
fluent, rapid speech
paraphasias
impaired comprehension
impaired repetition
impaired naming
no self awareness
lesion of wernicke’s aphasia
posterior portion of the first temporal gyrus of the L hemisphere
Anomic aphasia
fluent , grammatically well-formed often circumlocutionary speech
good comprehension and repetition
anomic aphasia lesion
temporal, parietal injury
transcortical sensory aphasia
fluent
good repetition
poor comprehension
poor reading
poor writing
transcortical sensory aphasia lesion
watershed lesion cutting off Brocas Wernickes complex, posteriortemporalparietal junctoin
transcortical motor aphasia
absence of spontaneous speech
good comprehension
excellent repetition
good reading comprehension
transcortical motor aphasia lesion
multifocal or diffuse pathology anterior and posterior association cortex
transcortical mixed aphasia
nonfluent with echolalia
poor comprehension
good repetition
good articulation
transcortical mixed aphasia lesion
multifocal or diffuse pathology anterior and posterior association cortex
Brocas aphasia
nonfluent, effortful speech
intact auditory comprehension
literal pharaphsia
poor repetition
cortex
gray matter (groups of neurons)
white matter
myelinated pathways (motor and sensory)
extrathalamic
basal ganglia
extrathalamic damage
basal ganglia
parkinson’s disease
huntington’s disease
thalamic damage
high mortality rate
increased or decreased thresholds of sensations
confused with broca’s aphasia
anterior syndrome
confused with transcortical sensory aphasia
thalamic
alert
awake and aware
lethargic
drowsy/ groggy
obtunded
requires continued stimulation
stuporous
maximal stimulation (sternal rub or ear pinch)
comatose
no voluntary responses no matter the stimulation
