Acute medical presentation 2 Flashcards

1
Q

What key things should in include in a history for a collapsed patient?

A
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2
Q

What is the neurological approach?

A
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3
Q

What are the different parts of the brain where a problem could be?

A
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4
Q

What neurological problems could you get in the brainstem?

A
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5
Q

What neurological causes are there of impaired consciousness?

A
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6
Q

What metabolic causes are there of impaired consciousness?

A
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7
Q

What diagnoses should you always consider in a neurological patient?

A
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8
Q

What could a fluctuating GCS indicate?

A
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9
Q

What spontaneous movements could you observe in an unconscious patient?

A
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10
Q

What is the approach to the acute neurological evaluation?

A
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11
Q

What quick cognitive assessment would you do in a neurological evaluation?

A
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12
Q

What do you check for in the eyes for a neurological evaluation?

A
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13
Q

How do you check for brainstem reflexes?

A
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14
Q

What special tests could you conduct for a neurological evaluation?

A
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15
Q

How is DKA defined?

A
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16
Q

How is DKA managed?

A
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17
Q

How much fluid deficit are pts in DKA usually?

A

100ml/kg

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18
Q

Why do we give insulin in DKA?

A

Clear ketones, suppress ketogenesis and avoid avoid hypoglycaemia

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19
Q

How much insulin do you give in DKA?

A
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20
Q

When do you give dextrose in DKA?

A

when CBG <14

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21
Q

When would you switch to variable rate insulin infusion in DKA?

A

When ketones <0.6
pH >7.3
HCO3 >18

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22
Q

When do you measure hourly in DKA?

A

CBG
Blood ketones
Venous bicarbonate
K+

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23
Q

When is DKA resolved?

A

When blood ketones <0.3mmol/l
and venous pH >7.3

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24
Q

How long do you give IV insulin for in DKA, when patient has started back on usual sc insulin?

A

1 hour to make sure CBG’s are stable without huge variability.

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25
Q

signs of euglycaemic DKA?

A

Acidosis
Ketonaemia
normal/slightly raised CBG

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26
Q

Why does euglycaemic DKA occur?

A

As a result of insulin deficiency/inactivity accompanied by an increase in counter regulartory hormones.

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27
Q

What medications can cause Euglycaemic DKA?

28
Q

What are the characteristic features of HHS?

29
Q

How is HHS managed?

30
Q

How long does resolution of HHS take?

A

up to 72 hours

31
Q

What history and examination should you take for an endocrine problem?

32
Q

what is the pathophysiology of addisons disease (primary hypoadrenalism)?

33
Q

What is an adrenal crisis?

34
Q

what is the role of glucocorticoids?

35
Q

What is the role of mineralocorticoids?

36
Q

What are the causes of Addisons disease?

37
Q

What are the symptoms of addisons disease?

38
Q

What are the signs of addisons disease?

39
Q

What investigations do you do for addisons disease?

40
Q

What are the ACTH levels for adrenal failure?

41
Q

What is the normal cortisol response in a short synacthen test?

42
Q

What is the management of adrenal crisis?

43
Q

What is the regular treatment and education of addisons disease?

44
Q

What do thyroid hormones do?

45
Q

What are the clinical features of thyrotoxicosis?

46
Q

What are the poor prognostic features of a thyroid storm?

47
Q

What would the TSH be in patients with acute thyrotoxicosis?

A

Low (except TSHoma)

48
Q

How would you establish a cause of acute thyrotoxicosis?

49
Q

What antithyroid drugs would you commence in acute thyrotoxicosis?

50
Q

What is the definitive treatment for acute thyrotoxicosis?

51
Q

What precipitating factors contribute to thyroid storm?

52
Q

How do you manage thyroid storm?

53
Q

What is malignant HTN?

54
Q

What is the difference between HTN urgency and HTN emergency?

55
Q

What complications could arise from a HTN emergency?

56
Q

What are the signs of malignant HTN?

57
Q

What is the criteria for LVH?

A

S in V1 + R in V5 or V6 (whichever is larger) >35mm (7 large squares)

58
Q

What investigations would you do in malignant/accelerated HTN?

59
Q

When would you admit a patient with high BP?

60
Q

How would you treat a HTN emergency?

61
Q

What is the drug class of nifedipine?

A

dihydropyridine calcium channel blocker

62
Q

What is there a risk of if MAP is decreased too quickly?

A

Organ infarction (CNS)

63
Q

What are the endocrine secondary causes of HTN?

64
Q

What are the clinical features of a phaeochromocytoma?

65
Q

How is phaeochromocytoma managed?

66
Q

What is the reticular system?

A

Essence of awareness/consciousness