abomasal disorders Flashcards

1
Q

what part of the stomach is the abomasum?

A

glandular stomach

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2
Q

how many times does the abomasum contract a day?

A

18-20x

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3
Q

which part of the stomach predominates in pre-ruminant calf?

A

abomasum

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4
Q

what results in reduced motility of the abomasum?

A
hypoCa
hypoCl
hypoNa
hypoK
metabolic acidosis
ketosis and acidosis
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5
Q

list the various abomasal dxs

A

check slide 10

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6
Q

where is the abomasum normally located?

A

right ventrum

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7
Q

what types of abomasal displacements are there?

A

left (most common)
right - dilation & volvulus
(also cranial btwn liver & diaphragm but v rare)

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8
Q

explain DA pathogenesis

A
  1. reduced abomasal motility: reduced plasma Ca2+ conc.
  2. gaseous distention
  3. displacement: L/R
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9
Q

when does LDA occur?

A

during transition period

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10
Q

risk factors for LDA

A

breed: dairy > beef
gender: female > male
yield: higher > lower
genetics: motilin gene influences abomasal motility, can affect abomasal displacement risk

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11
Q

external risk factors for LDA

A

immediate pp period
diet
concurrent dx (pp dx)
pp dx can be related to transition diet

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12
Q

pp risk factors

A
rumen reduces in size in the lat month before calving = cranioventral part of abdomen is more empty than normal 
allows abomasum (if filled w gas) to move across the left because less obs c.f. peri-partum period 
1st 4-6wks pp
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13
Q

dietary risk factors

A

pre-calving diet
reduction in DMI before calving which can result in: ketosis, hepatic lipidosis, NEB
high grain intake pre-calving = high CHO intake
low crude fibre <17%

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14
Q

prevention for LDA

A
focus on transition period
max. DMI 
prevent pp dx
treat ketosis & other conditions promptly (cow w NEB higher risk of LDA)
min. stress
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15
Q

what is the target LDA incidence for a herd?

A

<3%

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16
Q

when should you start investigating causes of LDA in herd?

A

if overall annual LDA incidence > or = 2%
cluster cases in short time
client concerns

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17
Q

what should you start with for herd approach regarding LDA?

A

transition management:

  • diet
  • stocking density
  • calving management
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18
Q

how to diagnose LDA?

A

hx and CE

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19
Q

what would the hx be like for a cow w LDA?

A

depressed feed intake/anorexia
drop in milk pdtn
recent calving
transition period problems

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20
Q

clinical findings on CE for LDA?

A

abnormal faeces
decreased rumination sounds +/- decreased rumen size
auscultation & ballottement of abdomen: pinging
+/- dehydration (skin tent/sunken eyes)
+/- concurrent dx
HR & RR norm/increased, occasional sinus arrhythmia

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21
Q

other diagnostic methods for LDA

A

abomasocentesis: 10/11th ICS, pH 2-3, no protozoa, small vol
US: last 3 ICS on LHS, ventral to dorsal

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22
Q

DA prognostic categories: group 1

A

gas distension only
HR wnl
excellent px: conservative, no fixation needed

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23
Q

DA prognostic categories: group 2

A

gas distention + <20% fluid
HR < 100bpm
good px: percutaneous fixation - “closed surgery”

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24
Q

DA prognostic cat: group 3

A

gas distension + >/= 20% fluid
HR >/= 100bpm
mod (guarded px): surgical - open (traditional)/laparoscopic

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25
Q

conservative techniques for LDA

A

medical management

cast and roll

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26
Q

explain cast and roll technique

A

cast cow onto RHS
method 1: roll over onto LHS + ballottement of ventral abdo
method 2: roll over dorsum then tilt cow 45deg alternately L&R several times before rolling onto LHS
hold cow in LLR for 5-10mins before returning to standing/sternal

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27
Q

cons of cast & roll technique?

A

high recurrence rate (>80%)

rolling risks

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28
Q

explain percutaneous fixation technique

A

roll & toggle: ‘Grymer/Sterner technique’

blind suture: utilises large suture instead of plastic toggle

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29
Q

what is the prep for roll & toggle?

A

roll to dorsal recumbency: cast on RHS
clip from xyphoid to umbilicus
LA blebs
be quick

30
Q

advantages of roll & toggle?

A

quick & easy to perform
min specialist eq needed
inexpensive
closed technique = reduced risk of abd contamination
reasonable success rate (75% 60d survival)

31
Q

disadvantages of roll & toggle?

A
blind technique: no visualisation of abomasum/abdomen/incorrect fixation
rolling risks 
2+ assistants needed
only suitable if abdomasum freely mobile
CI: adhesions present
32
Q

potential complications of roll & toggle?

A
incorrect fixation: omentum/rumen/SIs
infection & fistulation
peritonitis
suture breakage = recurrence of DA
abomasal obstruction
abomasal rupture
33
Q

what are the pre-op meds for LDA sx?

A

abs: broad spectrum - open sx
analgesia: NSAIDs - all sx, care if ulceration
Ca
fluid therapy as needed (can give oral fluid therapy post-op, don’t give pre-op)

34
Q

what are the pre-op prep for LDA sx?

A

adequate restraint +/- sedation
regional anaesthesia
pre-op clip & scrub: chlorhexidine/iodine, sx spirit

35
Q

what are the various regional anaesthesia methods?

A

line block
inverted L
paravertebral - distal/prox

36
Q

list the fixation techniques for DA

A

right flank approach (standing): pyloropexy, omentopexy (typically)
left flank approach (standing): abomasopexy
paramedian approach (recumbent): abomasopexy

37
Q

what can be used as a landmark for finding omentum?

A

“sow’s ear” - close to pylorus

38
Q

what are you suturing for omentopexy?

A

suturing greater omentum to body wall at incision site - locate as close to pylorus as poss.

39
Q

what are you suturing for pyloropexy?

A

muscular part of pylorus to body wall @ ventral aspect of incision site

40
Q

how to avoid stenosis for pyloropexy?

A

locate sutures about 5cm cranial to pylorus

avoid entering pyloric lumen!

41
Q

what else might be done tgt w pyloropexy?

A

omentopexy

42
Q

potential complications for sx techniques?

A
difficulties finding landmarks
difficulty moving abomasum 
peritonitis 
recurrence (if omentopexy performed too far from pylorus >8cm or omentopexy breakdown) 
pyloric stenosis
43
Q

compare omentopexy vs pyloropexy

A

success rates abt same: 86-94%
redisplacement rate higher in omentopexy (3.6-4.2% vs 3%)
omento = indirect vs plyoro = direct fixation
risk of omental breakdown in thin cows
risk of penetration of pyloric lumen = pyloric stenosis
pyloro can result in reduced abomasal motility

44
Q

explain right paramedian approach

A
dorsal recum: xylazine
right of midline (rectus abdominis)
fixation of fundus
allows max visibility of abomasum: ulceration
good visibility of abdomen
good success rate (80-95%)
45
Q

what are you suturing for paramedian abomasopexy?

A

suture abomasal serosa and muscularis layer to internal rectus sheath & peritoneum

46
Q

potential complications for abomasopexy?

A

fistulation @ suture site
wound dehiscence: herniation (can be fatal)
risk of recumbency
CI in preg cows

47
Q

where is the incision made for LHS abomasopexy?

A

10-15cm close to lat rib & ventral

utrecht technique

48
Q

what are the complications for LHS abomasopexy?

A

peritonitis
ventral fistulation
milk vein damage
failure to fix abomasum

49
Q

what are the advantages of laparoscopic techniques?

A
high success rate
visibility of abd
min invasive
short procedure
small wound size
quick post-op recovery
50
Q

what are the disadvantages of laparoscopic techniques?

A

specialist eq: initial cost/maintenance
further training
adjustment to laparoscopic ‘view’
higher costs for farmer

51
Q

what are the potential complications of laparo tech?

A
reoccurrence (2-3%): rupture of suture
rupture of abomasum
reduced abomasal motility
peritonitis 
milk vein perforation 
incorrect fixation (1 step)
risks of recumbency (2 step)
52
Q

differentiate between RDA vs AV

A

RDA:

  • liver not displaced
  • no palpable twist
  • rotation not observable from RHS of animal (during sx)
  • serosal surface may/may not be covered w omentum (following incision)

AV

  • liver displaced medially
  • palpable twist
  • rotation observable from RHS
  • serosal surface not covered w omentum
53
Q

name the 3 different types of AV

A
  1. abomasal volvulus (60% of cases): twist @ omasal-abomasal junction
  2. omasal-abomasal volvulus (OAV) (40% of cases): twist @ reticulo-omasal junction
  3. reticulo-omasal-abomasal volvulus (ROAV): v rare, twist @ jn of rumen & reticulum, PM dx
54
Q

RDA epidemiology

A

adult
female
dairy cattle
less common in: calves, males, beef

55
Q

risk factors of RDA?

A

pp risk: lower than LDA
high grain diet in early lactation
iatrogenic: after LDA roll

56
Q

approach to RDA + volvulus

A

severe life threatening

immediate tx = sx

57
Q

consequences of RDA + volvulus?

A

severe dehydration, shock, death
hypoCl
hypoK
metabolic alkalosis

58
Q

diagnosis of RDA?

A

same as LDA

59
Q

diagnosis of RAV?

A

same as RDA +
increased thirst
tachycardia (>100/min, worse px)
abdo pain
palpable distended viscus on rectal (adults)
palpable distended viscus behind last rib (calves): painful to palpate

60
Q

what are the surgical sites for RDA/RAV?

A

20cm incision, 4cm caudal to last rib

start 10cm distal to transverse processes

61
Q

define erosion

A

superficial defects, mucosal membrane

62
Q

define ulcer

A

defects including muscular layer

63
Q

types of abomasal ulceration?

A

primary: cause unknown, high yielding dairy cows, veal calves
secondary: DA, abomasal impaction, other dx

64
Q

ulcer classification

A

type 1: non-perforating ulcers w/o bleeding: no/min. CS
type 2: ulcers w bleeding into abomasal lumen: melena
type 3: perforating ulcers w local peritonitis: no melena, CS may be min., US
type 4: perforating ulcers w generalised peritonitis: no melena, CS non-spe, rectal exam & US
decreasing px 1-4

65
Q

pathogenesis of abomasal ulcers

A

injury to gastric mucosa
H+ ions diffuse from lumen to mucosa
pepsin diffuses into mucosa
imbalance btwn ulcerogenic & protective mechanisms

66
Q

CS of abomasal ulcers?

A

v variable & non-specific: no CS in type 1 ulcer, melena in type 2, septic shock w type 4
decreased feed intake & rumination
cranial abdo pain
anaemia

67
Q

diagnosis of abomasal ulcers?

A
challenging! 
CS
haem & biochem
faecal occult blood test (type II) 
abdominocentesis & peritoneal fluid analysis (type III & IV) 
US (types III & IV)
68
Q

tx of abomasal ulceration?

A

diet
oral antacids (magnesium oxide, aluminium hydroxide)
cimetidine, ranitidine, omeprazole NOT licensed in food producing animals
sx in perforating ulcers
supportive therapy
blood transfusion
NSAID v controversial (can make ulceration worse but only licensed analgesia for cattle)

69
Q

what types of abomasal impactions are there?

A

primary: pp dairy cattle, 2dary to hypomotility
secondary: TRP
dietary: sand, poor Q roughage, beef cattle, cold weather

70
Q

tx of abomasal impactions?

A

5L mineral oil mixed w 10L water: tube into rumen, SID for 3-5d
sx correction: R paramedian, abomasotomy
px = guarded to good