Abnormalities of Teeth Flashcards

1
Q

systemic factors that influence enamel

A

birth trauma
chemicals
chromosomal anomalies
infections
inherited diseases
malnutrition
systemic disease
neurologic disease

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2
Q

local factors that influence enamel

A

physical trauma
electrical burn
radiation
local infection

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3
Q

which teeth are most affected - turner tooth

A

facial of premolars and central incisors

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4
Q

why are premolars affected by turner tooth

A

usually because abscess under mandibular 1st molars (premolars are pelow)

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5
Q

why are central incisors often impacted by turner tooth

A

trauma of central incisors

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6
Q

molar incisor hypomineralization ethnic predilection

A

norther europeans (unknown cause)

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7
Q

clinical features of molar incisor hypomineralization

A

-white, yellow, or brown enamel
-soft and porous
-enamel chips easily
-teeth very sensitivity and difficult to anesthetize

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8
Q

hypoplasia cuased by antineoplastic therapy

A

-age at time of therapy
-form and dose of therapy
-can lead to hypodontia, microdontia, radicular hypoplasia, enamel hypoplasia, mandibular hypoplasia

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9
Q

mottled enamel (dental fluorosis)

A

white, chalky demineralization
occasional pitting
caries resistance

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10
Q

optimal levels of fluoride in water

A

0.7 ppm

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11
Q

attrition

A

tooth to tooth contact
physiologic
occlusal and incisal edges + proximal surfaces
wear is prominent in bruxism

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12
Q

abrasion

A

pathological wearing of tooth structure
external agent (ex. improper brushing)

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13
Q

erosion

A

non-bacterial chemical loss
food / drink / chemical / GERD

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14
Q

perimolysis

A

erosion from gastric contents

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15
Q

abfraction

A

occlusal stress - flexural - fracture
usually cervical enamel

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16
Q

internal resorption

A

uncommon
pink tooth of mummery

OUTLINE OF PULP CANAL IS WIDENED

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17
Q

treatment for internal resorption

A

endo therapy prior to perforation

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18
Q

external resorption

A

common
caused by trauma, cysts, tumors, inflammation

OUTLINE OF PULP CANAL IS NOT WIDENED

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19
Q

causes of extrinsic staining

A

chromogenic bacteria
ferric sulfide
coffee / tea / tobacco
gingival hemorrhage
medications
stannous fluoride
chlorohexidine

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20
Q

congenital erythropoietic porphyria

A

-porphyrin deposition in teeth
-red/brown discoloration
-red fluorescence

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21
Q

hyperbilirubinemia

A

-from RBC breakdown
-erythroblastosis fetalis
-biliary atresia
-green discoloration

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22
Q

calcific metamorphosis

A

only impacts dental pulp, no pulp seen in radiographs

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23
Q

lepromatous leprosy

A

pink / red discoloration

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24
Q

dental amalgam

A

gray / black

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25
medications causing intrinsic staining
tetracyclines (yellow / brown) minocycline (blue / grey)
26
extrinsic stain managemetn
mechanical removal
27
intrinsic stain management
chemical removal, cosmetic restorations
28
most common impacted teeth
17 / 32 1 / 16 6 / 11
29
treatment options for impacted teeth
observation orthodontically assisted eruption transplantation extration
30
ankylosis
cessation of eruption after emergence fusion with alveolar bone tooth below occlusal plane
31
hypodontia
reduction in number of teeth
32
anodontia
complete lack of tooth formation
33
oligodontia
lack of 6+ teeth
34
pseudo-oligodontia
failure of eruption
35
natal teeth problems
no lip seal problems with suckling trauma to breast trauma to infant tongue
36
microdontia (definition)
small teeth
37
microdontia is most often found in
lateral incisors (peg laterals) and maxillary third molars
38
macrodontia (definition)
large teeth
39
pituitary gigantism (+association)
increase in amount of growth hormone (generalized macrodontia)
40
macrodontia and jawbone size
teeth might be normal size, maxilla + mandible are small
41
unilateral macrodontia is associated with
hemifacial hyperplasia (half of face is large, teeth on one side larger)
42
germination
single enlarged tooth or joined double tooth. tooth count is normal
43
fusion
single enlarged tooth, fused teeth share cementum and dentin.
44
is the tooth count normal with fusion?
no, reduced by one
45
is the tooth count normal with germination?
yes!
46
concrescence (definition)
fusion of roots by cementum two fully formed teeth, joined along root surface by cementum
47
concrescence is related to
trauma and crowding
48
how does fusion affect extraction of teeth?
if trying to extract tooth + root is fused to adjacent tooth, that will pose problems
49
cusp of carabelli
palatal surface of mesiopalatal cusp of maillary permanent first molar
50
what is most important to remember about the cusp of carabelli?
must restore when doing restorative procedures
51
talon cusp
enlarged cingulum
52
talon cusps are most common in which teeth?
maxillary lateral and central incisors
53
dens evaginatus (leong's premolar)
additional cusp in the center of occlusal surface
54
which teeth do dens evaginatus affect the most?
primarily affects premolars
55
what 3 clinical implications are there for dens evaginatus?
1. early wear 2. pulp exposure 3. interferes with occlusion
56
shovel shaped incisors
most often found in maxillary incisors no clinical implications
57
dens in dente / dens invaginatus
tooth inside a tooth
58
dens invaginatus clinical complications
1. tooth more prone to caries 2. decay not visible
59
enamel pearl location
CEJ of buccal root furcation of molars
60
are enamel pearls more common in the maxilla or mandible?
maxilla
61
clinical importance of enamel pearls
loss of periodontal attachment inflammatory cysts
62
enamel pearls + extraction
must engage pearls during extraction
63
cervical enamel extensions
enamel should stop at cervical margin. if enamel goes more apical, introducing furcation to oral cavity
64
buccal bifurfaction cyst
typically occur in children (when molar is erupting), swelling and severe pain
65
taurodontism
enlargement of body and pulpal chamber of premolar or (usually) molar teeth, apical displacement of pulpal floor
66
which dimension of the pulp chamber increases in taurodontism?
occluso-apical dimension
67
is the furcation more apically or coronally positioned in taurodontism?
apically positioned
68
clinical ipmlications of taurodontism
incidental finding no furcation to grasp if extracting
69
hypercementosis
excessive deposition of cementum (normal PDL space, intact lamina dura)
70
hypercementosis is often seen with ______
paget's disease
71
dilaceration
abnormal angulation (bend) in a tooth root
72
how can dilceration happen?
truama masses (tumors) in way of developing root/crown
73
dilacerations are more likely to _______ during extraction
fracture
74
supernumerary roots / extra roots most commonly seen in
third molars
75
clinical implications of supernumerary roots
supernumerary roots tend to be super slender and thin (difficult in endodontic therapy and extractions)
76
amelogenesis imperfecta
hereditary disorder of enamel
77
3 steps of enamel formation
1. deposition of organic matrix 2. mineralization of matrix 3. maturation
78
hypoplastic amelogenesis imperfecta
inadequate deposition of enamel matrix
79
size of teeth in hypoplastic amelogenesis imperfecta
small
80
morphology of enamel in hypoplastic amelogenesis imperfecta
rough type with enamel pitting, smooth type with no pitting OPEN CONTACTS
81
hypomineralized amelogenesis imperfecta
inadequate mineralizaiton
82
size of teeth in hypomineralized amelogenesis imperfecta
normal size
83
teeth with hypomineralized amelogenesis are more prone to _____
caries
84
morphology of enamel with hypomineralized amelogenesis imperfecta
enamel is soft and cheesy, lost early
85
early wear staining with hypomineralized amelogenesis imperfecta
stains easily black / brown in color
86
hypomaturation type of amelogenesis imperfecta
mineralized enamel fails to mature (water of mineralization is retained, hydoxyapatite crystals fail to enlarge)
87
morphology of enamel with hypomaturation type of amelogenesis iperfecta
not hard enough, easily penetrated with explorer tip, chips away from underlying dentin
88
characteristic feature of hypomaturation type of amelogenesis imperfecta
mottled or snow capped enamel surface
89
dentinal disorders
hereditary disorders of the dentin related to osteogenesis imperfecta
90
type I dentinogenesis imperfecta
associated with osteogenesis imperfecta
91
type II dentinogenesis imperfecta
isolated to teeth
92
radiogrpahic features fo dentinogenesis imperfecta (3)
1. crowns are bulbous 2. cervical constriction 3. absence of pulp chamber and canals
93
clinical features of dentinogenesis imperfecta
teeth appear translucent, opalescent
94
expected complications of dentinogenesis imperfecta
implants cannot be used! be careful, even with routine procedures (easy to fracture bone)
95
type I dentin dysplasia
radicular type (rootless teeth) roots very short, pulps obliterated, periapical lesions
96
radiographic differences between type I dentin dysplasia and dentinogenesis imperfecta
no bulbous tooth or cervical constriction in dentin dysplasia
97
expected complicaitons of type I dentin dysplasia
periodontal concerns (periapical pathologies)
98
type II dentin dysplasia
coronal type enlarged pulps, thistle tube appearance
99
expected complications with type II dentin dysplasia
pulp stones
100
ghost teeth of regional odontodysplasia
affects enamel, dentin, and pulp manage with extraction