A & V disease Flashcards

1
Q
A
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2
Q

what do we use the doppler for?

A

to measure velocity and direction of flow

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3
Q

where r the superficial and deep veins located in our body? how is the pattern of flow between them orientated?

A

deep veins r w/in the muscles of the calf!

superficial veins r w/in the subcutaneous fat!

Flow is always superficial to deep!

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4
Q

the veins that go from superficial to deep r called________

A

perforating veins

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5
Q

the deep fascia is tightly adhered to the calf muscles,

why is this clinically important?

A

cuz when the muscles contract, they contract w/ in the compartment, the fascia is drawn in tight, and the pressure rises w/ in the compartment!

this rise in pressure compresses the deep veins and pushes the blood back to the heart!

if there was no fascial compartment, then there would be no compartment for the pressure to rise in!

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6
Q

what r the superficial veins of our legs?

A
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7
Q

which of the 2 veins is constant in our body?

describe its route

A
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8
Q

what is the clinical significance of the long saphenous vein being anterior to the medial malleolus ?

A

that if a patient is in shock, and their veins r all empty cuz theyre losing blood, if u need to find that vein, u will ALWAYS be able to see it in front of the MEDIAL MALLELOUS!

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9
Q

bc of the effectivness of the calf msucle pump, it reduces the pressure in lower limbs by 80 % during excersize!

A

ok

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10
Q

what r varicose veins? pathophysiology ?

describe their appearence?

A

swollen and enlarged veins that usually occur on the legs and feet.

vein wall is weak> dilates>valve cusps seperate causing regurgitation of blod.

They may be blue or dark purple and are often lumpy, bulging or twisted in appearance.

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11
Q

symptoms of varicose veins?

how do u releive them?

A

• Heaviness

  • Tension
  • Aching
  • Itching

these symptoms occurs along the vein ITSELF

lie them down and put leg in an upward position

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12
Q

complications of Varicose Veins

why do they arise

A

can cause complications because they stop your blood flowing properly.

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13
Q

what is thrombophlebitis?

A

Phlebitis literally means ‘inflammation of a vein’ due to blood clotting inside it or the vein walls are damaged.

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14
Q

name the complication

describe its appearence?

A

Varicose eczema

  • itchy
  • red and swollen
  • dry and flaky
  • scaly or crusty
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15
Q

what is this complucation?

A

Lipodermatosclerosis

which causes your skin, usually around the calf area, to become hardened and tight, and you may find it turns a red or brown colour

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16
Q

what can lead to Venous ulceration?

A

VENOUS HYPERTENSION

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17
Q

what might cause ur calf muscle pump to fail?

A
  1. -contration failure>> due to obesity, immobility, reduced hip or ankle movement (maybe due toosteoarhtrids)
  2. deep vein incompetence
  3. superficial vein incompetence
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18
Q

explain the retrograde circuit u get if ur

A

if its competence, everytime blood ejected a proportion of blood keeps going back into the calf muscle pump, causing it to dilate> cant functions as a pump> venous hypertension> venous ulceration

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19
Q

Virchows triad

A

if its competence, everytime blood ejected a proportion of blood keeps going back into the calf muscle pump, causing it to dilate> cant functions as a pump> venous hypertension> venous ulceration

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20
Q

difference btw arterial thrombosis and venous thrombosis!

A

In arterial>> Changes in the lining of the vessel wall

In Venous>> Changes in the flow of blood

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21
Q

why does Stasis lead to Venous thrombosis?

A

u normally have to have some “else” combined with the stais for ex: surgery (trauma), oral contraceptive pill, dehydration, cancer

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22
Q
A
23
Q

In a survival mode, when u have a low flow, u want the ability for ur blood to clot!

thats why stasis can be benifcial somtimes!

A
24
Q

normally those with DVT always have somthiing else going on

A
25
Q

DVT

A

DVT usually occurs in a deep leg vein, a larger vein that runs through the muscles of the calf and the thigh.

• Produces an inflammatory response

– Calor

– Dolor
– Functio laesa

– Rubor
– Tumor

26
Q

Sign and symptoms of DVT

A

Symptoms
• Pain, cannot walk

  • Swelling
  • Blue-red skin discolouration

Signs
• Calf tenderness

  • Muscle induration
  • Skin warmth
  • Skin discolouration
  • Distended, warm superficial veins • Oedema

• Pyrexia

27
Q

association btw Surgery and DVT.

A
  • Immobility prior to surgery
  • No calf muscle pump during surgery
  • Immobility after surgery
  • Surgery is trauma. The body’s response trauma includes a ‘prothrombotic state’
28
Q

what causes acute limb ischemia?

if not reversed w/ in ___hrs the limb cannot be recovered.

A

embolism (from heart or abdominal aortic aneurysm) and trauma

Sudden onset

If not reversed within 6 hours the limb cannot be recovered and if not amputated the patient will die (hyperkalemia due to dead tissue releasing intracellular potassium)

29
Q

how long does it take for collateral circulation to take place?

A

weeks/ months

30
Q

what r the Symptoms/signs of acute ischaemia

(6p’s)

A
31
Q

how does an acute ischemic foot look like?

A

white

32
Q

what is fixed mottling?

A

it’ll get patchy cyanosis,

bc w/ in that leg, there will be little areas where the blood in the capillaries o2 is used up> redcued Hb> if u press it it wont blanche! bc vessels r thrombosed> walls r broken down> no flow!

MUST AMPUTATE ASAP

33
Q

chronic peripheral arterial disease divided into (2)

A

• Intermittent claudication (equivalent to stable angina)

• Critical ischaemia
– Rest pain (equivalent to unstable angina)
– Ulceration/gangrene (equivalent to myocardial infarction)

34
Q

Define intermittent claudication

A

Pain in the muscles of the lower limb when walking / exercise

35
Q

Intermittent Claudication

which muscles r most likely effected?

how is the pain releived?

A
  • The calf muscles
  • Pain relieved rapidly by stopping exercise for a few minutes, even whilst standing up
36
Q
A
37
Q
A
38
Q

where is the femoral artery felt?

A
39
Q

what 2 landmarks help locate the femoral pulse?

A
40
Q

how to u palpate for the popliteal pulse?

A
41
Q

how do u palpate for the Pedis pulses? where r they anatomically?

A

Dorsalis pedis> lateral to the extensor hallucis longus tendon

Posterior tibial> behind the medial mallelous!

42
Q

what r we feeling for here?

A

Dorsalis pedis

43
Q

what r we feeling for here?

A

posterioir tibial A.

44
Q

what is rest pain? what causes it? how is it relieved?

A

Pain in the FOOT that comes on when the patient goes to bed.

The ischaemia is so severe that at rest the foot skin, muscles, bones are ischaemic at rest.

relieved by dangling the foot out of the bed (restoring gravity)

45
Q

why does rest pain occur at night?

A
  1. u take away gravity
  2. ur warming the foot up> metabolic rate goes up> o2 demands go up
  3. CO goes down at night
46
Q

what happens if rest pain is untreated?

A

u get Dry Gangrene and ulceration!

47
Q

what is critical ischmeia?

A

(limb viability is threatened)

Rest pain (equivalent to unstable angina)

Ulceration/gangrene (equivalent to myocardial infarction)

48
Q

what is the most common place for ulceration due to ischemia in the legs/foot?

A

blood supply in front of tibai is not so great> takes ages for it to heal

49
Q

rest pain is equivalent to _______

Gangrene/ ulceration is equivalent to ______

A

Rest pain (equivalent to unstable angina)

Ulceration/gangrene (equivalent to myocardial infarction)

50
Q

what type of occlusion is by far the commonest finding in patients with claudication ?

A
51
Q

what is gangrene?

difference btw dry and wet gangrene

A

necrosis visible to the naked eye?

Dry gangrene= Exposed to air

Wet Gangrene= infection mixed with bacterial culture (exaudate)

52
Q

Young people with normal coronary vessels and uncontrolled thyrotoxicosis can develop angina or even myocardial infarction. Why/how?

A

their metabolic demands go up>> need more o2

53
Q

why do u get pain in ischemia?

A

When muscle is ischaemic the pH drops>>stimulates pain afferents (nociceptors) in muscle.

Substance P>>> a neuropeptide is released locally which then increases the sensitvity of pain afferents in muscle