3Cardiac Part 2 Flashcards
Defects that decrease pulmonary blood flow
Tetralogy of fallot
Tricuspid atresia
(Right to left shunt)
Hypoxemia
S/s
Polycythemia
Fatigue
Clubbing
Cyanosis
Exertional dyspnea
Polycythemia:
-kidney produce erythropoietin to stimulate bone marrow to produce more RBCs
What is polycythemia:
What we are worried about
Kidneys produce erythropoietin to stimulate bone marrow to produce more RBC’s
Over production—>
causes sluggish blood (thick)—>
Causes thromboembolism (clots)
Tetralogy of fallot (TOF)
Defects included
Pulmonary stenosis
Right ventricular hypertrophy (bc of stenosis)
VSD
Overriding aorta (more centered in middle)
Tetralogy of fallot (TOF)
S/s
Tx
Murmur
Cyanosis
TET spells
Tx:
-palliative repairs (blalock taussig BT shunt): connects subclavian artery to pulmonary artery
-complete repair (fix all defects)
Hypercyanotic episodes (TET spells)
-caused by
Crying
Feeding
Exercise
Straining
Hypercyanotic episodes (TET spells)
S/s 8 DLITTIPS
Diaphoresis
Loss of consciousness
Irritability and crying
Tachycardia
Tachypnea
Increased cyanosis
Poor tissue perfusion
Seizures (if last too long have no O2 to brain)
Hypercyanotic episodes (TET spells)
1st intervention
Tx
1st intervention:
-knee to chest position
(increase systemic pressure forcing blood to go to lungs)
Tx:
-reduce crying/calm child (pacifier, swaddle)
-give oxygen
-administer morphine or propranolol
Tricuspid atresia
What is it
Common to see
Failure of tricuspid valve to develop
Common to see VSD
Tricuspid atresia
S/s
Tx
Cyanosis
Marked cyanosis once PDA closes
Tachypnea
Difficulty feeding
Murmur
Tx:
-PFO
-palliative BT shunt
-several surgeries
Mixed defects
(significant mixing of oxygenated and deoxygenated blood)
Transposition of the Great Arteries/Vessels
Truncus Arteriosus
total anomalous pulmonary venous return (TAPVR)
Hypoplastic left heart syndrome (HLHS)
Transposition of the Great Arteries (TGA)
What is it
What is needed
Pulmonary artery and aorta swapped
-need PDA and PFO to be compatible with life other wise blood would be pumped out to rest of body and never oxygenated.
-RV pushing blood to aorta instead of pulmonary artery
Transposition of the Great Arteries (TGA)
S/s
Tx
Cyanosis
Tachypnea
Worsens as PDA closes
Tx:
Keep PDA open (other wise uncompatible with life)
-administer Prostaglandin E (IV) to keep open
-PFO stretched
-surgical repair 4-7 DOL (days of life)
Truncus Arteriosus
What is it
What may be present
One major artery leaves the heart with one valve
(Aorta and pulmonary artery are one)
VSD may be present (murmur)
Truncus Arteriosus
S/s
Tx
Cyanosis
Tachynea, retractions, crackles
Activity intolerance
Poor feeding/growth
Tx:
-surgical repairs: 1st few weeks of life (cant go home at first)
Total anomalous pulmonary venous return (TAPVR)
What is it
What does that lead to
Is it compatible with life (can it be)
Pulmonary vein should connect to left atrium, instead it connects to right atrium/superior vena cava
Right sided hypertrophy
Pulmonary HTN/edema
Incompatible with life unless: PFO/ASD present
Total anomalous pulmonary venous return (TAPVR)
S/s
Tx
Cyanosis
Murmur
Tachypnea
Hepatomegaly (bc it causes right sided HF if not treated)
Tx:
-surgical repair
Hypoplastic left heart syndrome (HLHS)
What is it
Goal
-Structures on L side underdeveloped
-Mitral/aortic valves closed or very narrow
-LV nonfunctional (minimal CO)
Goal:
-create the R side of heart to do all the pumping
Hypoplastic left heart syndrome (HLHS)
S/s
Tx
-Tachycardia, low BP (only using R side of heart)
-Pallor, progressive cyanosis
Tx:
-KEEP PDA OPEN (has to be open to be compatible)
-palliative care/surgery
-heart transplant
Cath procedures nursing management
Pre-procedure
Assessments (physiologic/psychosocial)
VS, H&H, capillary refills
baseline temp/color
Baseline pedal and popliteal pulses
Parents psychosocial needs