3Cardiac Flashcards
Blood flows which direction
Which side of heart has higher pressure?
Path of least resistance
Left side has more pressure
What does this mean:
PVR<SVR
Pulmonary vascular resistance is lower so blood can get to the lungs
Systemic vascular resistance is higher (get blood out to rest of body)
Two things fetus hearts have that adults dont?
Foramen ovale:
-connects right and left atrium
Ductus arteriosus:
-connects aortic arch and pulmonary artery
higher pulmonary resistance during fetal life
What is happening with the ventricles
This is why what vital is high or low
Larger RV
PVR drops and SVR increases causing the LV to develop/strengthen
BP lower (d/t LV muscle being weak)
HR higher (meet metabolic and oxygen requirement)
Little cardiac reserve causes what
Heart muscle is fully developed by when
LCR: risk for Heart Failure
Fully developed heart muscle by 5y/o
Assessment of ped heart 6
-Family hx (HD/HTN/cholesterol/defects)
-Prenatal care
-Birth hx
-Feeding difficulties (cant eat is a sign in infants)
-Activity intolerance (preschooler not able to keep up)
-Growth and development issues (need more calories to keep up with heart working so hard)
Assessment (look) for cardiac
Visible pulsation (chest)
Edema
JVD/engorgement
Clubbing
Skin color
WOB
Assessment (touch) cardiac
-Skin (temp, moisture, pitting edema)
-Cap refill
-Pulse (central vs peripheral)
-Palpate liver (kids with cardiac issues tend to have enlarged livers)
Where to feel central pulses on different ages
Under 1(babies): brachial
5yr old(over 1): carotid/femoral
Assessment (listen) cardiac
Vital signs
Auscultate lung sounds
Auscultate heart sounds:
-apical x1 min
APE vs MAN
Aortic area
Pulmonic area
Tricuspid area
Mitral or apical area
Aortic: Right 2nd ICS RMCL
P: Left 2nd ICS LMCL
T: Right 4th ICS
M or Apical: Left 5th ICS
S1 vs S2
(where best heard)
(What it represents happening)
S1: 4-5th ICS LMCL
-systole/contraction (hearing mitral valve shut)
S2: 2nd ICS
-diastole/relaxation (hearing aortic/pulmonic valve shut
CHF (congestive heart failure)
Early signs
Poor feeding/ Diaphoretic with feedings
activity intolerance
Tachypnea
CHF
Right (3) vs Left sided (2)
Right: (backs up systemically)
-JVD
-Swelling/Edema
-Enlarged liver
Left: (backs up into lungs)
-pulmonary congestion
-increased WOB
Therapeutic management of CHF
-improve cardiac function (2 meds)
Digoxin (increases myocardial contractility)
-monitor s/s of toxicity
-monitor K levels
ACE-inhibitors
-monitor BP
-(-pril)
Digoxin
Toxicity s/s
K levels explained
Toxicity:
-halos (vision)
-bradycardia
-N/V
(Baby cant tell you this so HR is a good way to check)
K levels:
-digoxin binds to potassium receptors
-if hyperkalemic=causes digoxin to be less effective
-if hypokalemic=digoxin toxicity