3.2 Chronic Respiratory Disorders Flashcards

1
Q

COPD

A
  • Chronic Obstructive Pulmonary Disease
  • Preventable and treatable slowly progressive respiratory disease
  • Relates to airway obstruction or airflow obstruction involving airway, pulmonary parenchyma or both
  • Not fully reversible
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

COPD

A
  • Third leading cause of death in United States
  • Systemic disease resulting from chronic inflammation from damage to airways and damage to parenchyma.
  • Chronic Bronchitis and Emphysema
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Chronic Bronchitis

A
  • Chronic productive cough lasting at least 3 months in 2 consecutive years.
  • Airways thicken, diameter narrows
  • Mucus may plug airways
  • Alveoli damaged (fibrosed) and patient is more susceptible to respiratory infection because of altered alveolar macrophages.
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Emphysema

A
  • Abnormal permanent enlargement of air spaces to the terminal bronchioles.
  • Surface area responsible for gas exchange is destroyed resulting in trapped air and hyperinflation of lungs. (Destruction of alveolar walls)
  • Dead space, decreased surface area and hypoxemia
  • Increased pulmonary artery pressure which can lead to right side heart enlargement and signs and symptoms of heart failure (Cor Pulmonale)
  • Edema, Liver and Spleen Congestion, JVD
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

COPD with Aging

A
  • Accelerated changes of aging such as decrease in function

- Chronic inflammation leads to scarring, narrowing of airways, hypersecretion of mucus

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

COPD Manifestations

A
  • Genetic factors that predispose people to COPD
  • Forced exhalation of air due to loss of elastic recoil
  • Airway obstruction related to increased mucus, edema, bronchospasm.
  • Destroyed lung tissue and inflamed airways
  • Hypoxemia and Hypercarbia
  • CO2 is around 50 in COPD Patients.
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

COPD Patients

A
  • Rely on decreased oxygen to breath
  • Normal respiratory system relies on increased CO2
  • ## This is why it is dangerous to supply too much oxygen for COPD Patients. It diminishes their drive to breathe
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Assessment of COPD

A
  • Health history, risk factors, family history, physical examination
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Pulmonary Function Tests

A
  • Helps confirm diagnosis of COPD
  • Determines z severity, and monitors disease progression
  • Spirometry evaluates airflow obstruction
    FEV - Forced Expiratory Volume
    FVC - Forced Vital Capacity
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

FEV (Forced Expiratory Volume)

A
  • FEV 1 (in one second)
    Mild - 80%
    Very Severe - 30%
    Barrell Chest - Primarily with emphysema and chronic hyperinflation of lungs
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

3 Primary Symptoms of COPD

A
  • Chronic Cough
  • Sputum Production
  • Dyspnea (interferes with everyday eating)
  • Patients tend to lean forward and use accessory muscles to breathe.
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

COPD Patients Needs

A
  • They need high calorie, easy to eat food.
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Treatment for COPD Patients

A
  • Reduce risk of their symptoms
  • Major risk factor is environment which is modifiable
  • Most important is smoking
  • Use of smoking cessation has biggest impact on reducing risk of COPD
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Treatment for COPD Patients

A
  • Manage exacerbations

- Provide supplemental oxygen, long term use of oxygen has been shown to improve patients quality of life

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Reduce Risk Factors of COPD

A
  • Promote flu and pneumococcal vaccines
  • Prevents infections, viruses, and exacerbations
  • Pulmonary Rehab, bronchodilators for short term and steroids for long term.
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Surgeries

A

Bullectomy - Bullae are removed (large air spaces)
Lung Volume Reduction - Dead space, tissue is removed
Lung Transplant - Not enough lungs for all COPD Patients

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

Nursing Management

A
  • Assess patient. Review ABG’s Chest X-Ray, Pulmonary Function Test
  • Goal is to achieve airway clearance from bronchospasms and sputum
  • Medications, physiotherapy, respiratory treatment, increase fluid to thin secretions, teaching them to huff and cough, improve breathing techniques such as using pursed lip breathing (slow and control rate and depth of respirations)
  • Improve activity tolerance, pace activities throughout the day.
18
Q

Education

A
  • Use of MDI (Metered Dose Inhaler)
  • Use of Acapella (Flutter Device) which loosen mucus in airways.
  • ## Percussion on patients back to help loosen mucus
19
Q

Complications of COPD

A
  • Respiratory Insufficiency and failure
  • Risk of pneumonia, chronic atelectasis (flattening of alveoli)
  • Pneumothorax (air in thorax)
20
Q

Asthma

A
  • Overlap syndrome (both asthma and COPD)
  • Coughing, Wheezing, SOB
  • Mostly diagnosed as a child
  • Inflames and narrows airways and is triggered by something environmental like air quality, exercise or pet dander
  • Genetic Condition
  • Some improve over time and some completely overcome the disease.
  • Hyperresponsive (allergy strongest predisposing factor)
21
Q

COPD

A
  • Diagnosed as an adult (older than 40)
  • Lifestyle factors are a big factor
  • ## Symptoms milder than asthma but constant and can become progressively worse
22
Q

Asthma (cont)

A
  • Attacks occur at night or early morning.
  • Bronchoconstriction that narrows airways, mucosal edema, excessive mucus.
  • Increased heart rate
  • tachycardia, diaphoresis, chest tightness, central cyanosis around mouth and fingertips,
  • Severe sign is hypoxemia
23
Q

Care Plan Asthma

A
  • Prevention of attacks
  • Identify Triggers
  • Peak flow monitor to measure airflow during expiration
  • Figure 24.8 zones of asthma attack flow measurement.
    Green - 80-100% personal best
    Yellow - 60-80%
    Red - Less than 60% (call 911)
24
Q

Terminology

A

Antagonist - Block effect
Agonist - Produce effect
Adrenergic Amines - Stimulate sympathetic nervous system and activate epinephrine and norepinephrine
Alpha receptors - vascular smooth muscle
Beta Receptors - Bronchioles of lungs, arteries of skeletal muscle

25
Alpha receptors
- Responsible for constriction of blood vessels
26
Beta Receptors
- Dilation of blood vessels
27
Terminology
- Acetylcholine and muscarinic receptors are for parasympathetic nervous system that help slow heart rate and relax muscles. - Cholinergic is acetylcholine
28
Cholinergic
- Drugs that inhibit, enhance, or mimic actions of neurotransmitter acetylcholine are called cholinergic drugs -
29
Acetylcholine
- Chief neurotransmitter for parasympathetic nervous system | - Contracts smooth muscles, dilates blood vessels, increases body secretions, slows heart rate.
30
Medication Treatment for Asthma/COPD
- Corticosteroids used for both to decrease - Antibiotics, mucolytics, and antitussives can help thin mucus and cough up can be used for both - Quick relief medication can be short acting Beta Adrenergic agonist (albuterol and atrovent) inflammation Asthma - Leukotriene modifiers (Singulair) used to treat allergies. Mast Cell Stabilizers - Prevent release of histamine.
31
COPD Pathophysiology
- COPD accelerates normal lung aging such as vital capacity and forced expiratory volume. - Airflow limitations are progressive and associated with abnormal inflammation
32
Risk Factors COPD
- Environmental exposure - Cigarette smoking most important - Second hand smoke - Exposure to dust, chemicals, air pollution
33
Clinical Manifestations COPD
- Chronic Cough, Sputum Production, Dyspnea - Worsen over time - Dyspnea worse with exercise, and is persistent - Weight loss common due to dyspnea affecting eating. - Use of accessory muscles to breathe - Risk of respiratory insufficiency and infections - Barrel Chest - Increased CO2 retention, Respiratory Acidosis -
34
Diagnostic Tests for COPD
- Spirometry - Evaluate airflow obstruction. Ratio of FEV1 to FVC. - ABG's - Especially important in advanced COPD. - Screening for Alpha1-antitrypsin deficiency. Preformed for patients under 45 with history of COPD. - Key factors for determining diagnosis is patient history, severity of symptoms, responsiveness to bronchodialators
35
Complications of COPD
- Respiratory insufficiency and Failure - May need ventilator support - Pneumonia, chronic atelectasis, pneumothorax, pulmonary arterial hypertension (right side heart failure)
36
Treatment COPD
- Avoid environmental exposure to smoke. | -
37
ASTHMA Pathophysiology
- Reversible airway inflammation that leads to long term airway narrowing - Narrowing due to Broncho restriction, airway edema, airway hyperresponsiveness, airway remodeling. - Caused by mast cells -
38
Manifestations Asthma
- Cough, Dyspnea, Wheezing - Night or Early Morning - Cough with or without mucus production - Generalized chest tightness and dyspnea - Diaphoresis, Tachycardia, hypoxemia, central cyanosis.
39
Asthma Diagnostic Testing
- Improve with bronchodilators - Pulmonary function normal between exacerbations - Daily Peak Flow Monitoring is important Measures severity.
40
Complications of Asthma
Status Asthmaticus, Respiratory Failure, Pneumonia, Atelectasis. Airway obstruction can lead to hypoxemia - Give fluids -