Brainscape's Knowledge GenomeTM


Top Flashcards (Certified)
All Flashcards

OP - Yield > 2 dental anomalies > Flashcards

2 dental anomalies Flashcards

1
Q

causes of environmental enamel defects

A

hypoplasia/diffuse and demarcated opacities; Turner hypoplasia; molar-incisor hypomineralization; RT effects; fluorosis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

exanthamatous fevers of childhood + teeth

A

if before 2yo, enamel of anterior teeth and 1st molars

if ~4-5yo – cuspids, bicuspids, 2nd molars

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

turner teeth

A

periapical inflammation/trauma/caries of overlying primary tooth -> hypoplasia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

periapical inflammation/trauma/caries of primary tooth risk of what

A

turner hypoplasia/turner teeth

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

cheese molars

A

molar incisor hypomineralization – northern europe

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

RT in childhood dental effects

A

hypodontia, microdontia, enamel/radicular/mandibular hypoplasia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

fluorosis look

A

white challky hypomineralized/mottled enamel

can stain yellow/brown

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

white challky hypomineralized/mottled enamel

can stain yellow/brown

A

fluorosis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

tooth/tooth contact –>

A

attrition (eg bruxism)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

mechanical external agent on tooth –>

A

abrasion (toothbrush)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

chemical agent on tooth –>

A

erosion (soft drinks)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

enamel loss from loading force

A

abfraction

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

chewing of abrasive substance

A

demastication = attrition + abrasion

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

erosion due to gastric secretions

A

perimolysis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

perimolysis

A

erosion due to gastric secretions

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

demastication

A

demastication = attrition + abrasion

chewing of abrasive substance

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

attrition

A

tooth/tooth contact -(eg bruxism)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

abrasion

A

mechanical external agent on tooth (eg toothbrush)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

internal resorption reasons

A

inflammatory or replacement

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

inflammatory internal resorption of coronal pulp

A

pink tooth of Mummery

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

pink tooth of mummery

A

inflammatory internal resorption of coronal pulp

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

rapid external resorption starting at cervical region

A

invasive cervical resorption

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

resorption of multiple teeth with no obvious cause

A

multiple idiopathic apical root resorption

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

congenital eryhtropoietic porphyria aka, inheritance, what’s wrong, teeth look

A

gunther disease
AR
porphyrin metabolism
red color of teeth under UV light (Woods lamp test)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
Q

alkaptonuria aka, inheritance, what’s wrong, why relevant

A

black urine disease
AR disorder of phenylalanine and tyrosine metabolism

ochronosis: blue/black discoloration in connective tissue, tendons, cartilage

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
26
Q

ochronosis

A

blue/black discoloration in connective tissue, tendons, cartilage – in alkaptonuria

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
27
Q

blue/black discoloration in connective tissue, tendons, cartilage

A

ochronosis – in alkaptonuria

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
28
Q

hyperbilirubinemia causes and why relevant

A

erythroblastosis fetalis and biliary atresia

can stain teeth

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
29
Q

green teeth why

A

chlorodontia – 2/2 biliverdin deposition

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
30
Q

chlorodontia

A

green teeth – biliverdin deposition

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
31
Q

biliverdin deposition

A

chlorodontia green teeth

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
32
Q

pink teeth why

A

trauma, internal resorption, leprosy

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
33
Q

leprosy teeth

A

pink teeth

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
34
Q

why are teeth impacted

A

barrier or lack of force –> tooth ceases to erupt

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
35
Q

eruption sequestrum

A

bone spicule overlying crown of a partially erupted tooth

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
36
Q

bone spicule overlying crown of a partially erupted tooth

A

eruption sequestrum

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
37
Q

ankylosis of tooth

A

cessation of eription after emergence 2/2 fusion of tooth w bone

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
38
Q

fusion of tooth w bone –>

A

ankylosis

stops eruption after emergence

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
39
Q

hypodontia genes

A

PAX9 molars
MSX1 distal tooth of each type
AXIN2 molars – assoc w colon polyps and colorectal ca

+ He-Zhao deficiency

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
40
Q

PAX9 hypodontia

A

molars

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
41
Q

MSX1 hypodontia

A

distal tooth of each type

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
42
Q

AXIN2 hypodontia

A

molars – assoc w colon polyps and colorectal ca

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
43
Q

supernumerary tooth of anterior maxilla

A

mesiodens

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
44
Q

mesiodens

A

supernumerary tooth of anterior maxilla

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
45
Q

distodens

A

aka distomolar

fourth molar

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
46
Q

fourth molar

A

distodens or distomolar

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
47
Q

dental transposition

A

normal tooth in abnormal location

assoc w hypodontia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
48
Q

teeth present at birth

A

natal – assoc w riga fede

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
49
Q

normal tooth in abnormal location

A

dental transposition

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
50
Q

diffuse microdontia 2 syndromes

A

Down and pituitary dwarfism

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
51
Q

diffuse macrodontia 4 conditions

A

gigantism, otodental syndrome, Klinefelter (XXY), pineal hyperplasia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
52
Q

unilateral macrodontia manifestation of

A

hemifacial hyperplasia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
53
Q

single enlarged tooth, normal tooth count

A

gemination

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
54
Q

gemination tooth count

A

single enlarged tooth, normal tooth count

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
55
Q

single enlarged tooth, missing one count

A

fusion

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
56
Q

gemination vs fusion

A

both have single enlarged tooth
gemination normal count
fusion missing one

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
57
Q

union of teeth by cementum only

A

concrescence

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
58
Q

concrescence

A

union of teeth by cementum only

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
59
Q

3 types of accessory cusps

A

cusp of carabelli, talon cusp, dens evaginatus

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
60
Q

cusp of carabelli

A

palatal surface of mesiolingual cusp of mx 1 molar

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
61
Q

accessory cusp on palatal surface of mesiolingual cusp of mx 1 molar

A

cusp of carabelli

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
62
Q

talon cusp where and when

A

anterior teeth

rubinstein-taybi, sturge weber. ellis van creveld

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
63
Q

rubinstein-taybi components (3)

A

talon cusps, mental retardation, broad thumbs and toes

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
64
Q

accessory cusp on mesiobuccal of MD molar

A

protostylid

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
65
Q

protostylid

A

accessory cusp on mesiobuccal of MD molar

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
66
Q

dens evaginatus

A

cusplike elevation in PM and molars
assoc w shovel-shaped incisors
100% of inuits and indians

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
67
Q

cusplike elevation in PM and molars

assoc w
demographic

A

dens evaginatus

assoc w shovel-shaped incisors
100% of inuits and indians

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
68
Q

dens invaginatus aka

and types

A

aka dens in dente (large invagination) or dilated odontome (–> anomalous tooth development)

coronal (types I, II, III) or radicular (similar to enamel pearls but inside)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
69
Q

enamel pearls inside the root

A

dens invaginatus radicular type

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
70
Q

types of ectopic enamel

A

enamel pearl (on root) and cervical enamel extension (on bifurcation of molars)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
71
Q

ectopic enamel on molar bifurcation

A

cervical enamel extension

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
72
Q

ectopic enamel on root and in root

A

on root: enamel pearl

in root: dens invaginatus radicular type

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
73
Q

enlargement of body and pulp chamber

A

taurodontism (mild, moderate, severe)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
74
Q

taurodontism

A

enlargement of body and pulp chamber

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
75
Q

hypercementosis vs cementoblastoma

A

latter has pain, expansion, and continuous growth

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
76
Q

genes in amelogenesis imperfecta (6)

A 
amelx
enam
klk4
mmp20
wdr72
dlx3
c4orf,,,
fam83h
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
77
Q

hypoplastic amelogenesis imperfecta what’s up and types (6 presentations)

A

type I
inadequate deposition of enamel matrix
generalized pitted, localized pitted, AD diffuse smooth, x-linked diffuse smooth, diffuse rough, enamel agenesis)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
78
Q

generalized thin hypoplastic amelogenesis imperfecta

A

joins all diffuse forms

smooth, rough, and enamel agenesis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
79
Q

diffuse smooth and rough enamel hypoplasia + enamel agenesis

A

generalized thin hypoplastic amelogenesis imperfecta

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
80
Q

hypomaturation amelogenesis imperfecta: what’s up and types

teeth look like

A

type II
defect of maturation of of crystal structure
(diffuse pigmented, x-linked, snow capped)
normal shape but discolored

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
81
Q

hypocalcified amelogenesis imperfecta what’s up and to what extent

A

type III
mineralization does not occur
diffuse

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
82
Q

defect of maturation of enamel crystal structure

A

type II

hypomaturation amelogenesis imperfecta

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
83
Q

enamel mineralization does not occur

A

type III

hypocalcified amelogenesis imperfecta

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
84
Q

inadequate deposition of enamel matrix

A

type I

hypoplastic amelogenesis imperfecta

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
85
Q

type I amelogenesis imperfecta

A

hypoplastic: inadequate deposition of enamel matrix

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
86
Q

type II amelogenesis imperfecta

A

hypomaturation: defect of maturation of crystal structure (normal shape but discolored)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
87
Q

type III amelogenesis imperfecta

A

hypocalcified: mineralization does not occur

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
88
Q

AI with taurodontism types

A

hypomaturation-hypoplastic (IVA, thicker enamel) and hypoplastic-hypomaturation (IVB)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
89
Q

type IVA amelogenesis imperfecta

A

hypomaturation-hypoplastic (IVA, thicker enamel)

AI with taurodontism

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
90
Q

type IVB amelogenesis imperfecta

A

hypoplastic-hypomaturation (IVB)

AI with taurodontism

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
91
Q

tricho-dento-osseous syndrome components

A

AI, taurodontia, kinky hair, osteosclerosis, brittle nails

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
92
Q

AI, taurodontia, kinky hair, osteosclerosis, brittle nails

A

tricho-dento-osseous syndrome

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
93
Q

dentinogenesis imperfecta mutation and types

A

DSPP
I = OI + opaslescent teeth
II = hereditary opalescent teeth
III = Brandywine isolate

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
94
Q

DSPP

A

dentinogenesis imperfecta and DD II

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
95
Q

Brandywine isolate

A

type III dentinogenesis imperfecta

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
96
Q

hereditary opalescent teeth

A

type II dentinogenesis imperfecta

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
97
Q

teeth of dentinogenesis imperfecta

A 
opalescent 
obliteration of root canals and chambers
bubous crowns
cervical constriction
x ray very important for dx
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
98
Q

shell teeth

A

normal thickness enamel, thin dentin, enlarged pulp in dentinogenesis imperfecta

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
99
Q

normal thickness enamel, thin dentin, enlarged pulp

A

shell teeth

dentinogenesis imperfecta

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
100
Q

dentin dysplasia type I aka, teeth

A

aka radicular DD
types Ia-Id
rootless teeth, no pulp, periapical RL, pulp stones

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
101
Q

rootless teeth, no pulp, periapical RL, pulp stones

A

dentin dysplasia type I

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
102
Q

dentin dysplasia histo

A

whorls of tubular dentin

stream flowing around boulders

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
103
Q

whorls of tubular dentin histo

A

stream flowing around boulders

dentin dysplasia type I

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
104
Q

stream flowing around boulders

A

whorls of tubular dentin dentin dysplasia type I

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
105
Q

fibrous dysplasia of dentin

A

radiodensity fills pulp chamber and canal

small foci of RL in pulp (vs dentinogenesis imperfetca) and roots have normal length (vs dentin dysplasia)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
106
Q

radicular dentin dysplasia is

A

type I

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
107
Q

coronal dentin dysplasia is

A

type II

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
108
Q

radiodensity fills pulp chamber and canal

small foci of RL in pulp – ddx

A

fibrous dysplasia of dentin – and not dentin dysplasia type I

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
109
Q

radiodensity fills pulp chamber and roots are normal length – ddx

A

fibrous dysplasia of dentin – and not dentin dysplasia type I

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
110
Q

dentin dysplasia type II aka and teeth

A

coronal dentin dysplasia
normal root length
also DSPP
deciduous teeth features of dentinogenesis imperfetca
permanent – normal color but flame/thistle shaped pulp and pulp stones

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
111
Q

flame/thistle shaped pulp and pulp stones in permanent dentition ddx and difference

A

dentin dysplasia type II
its primary teeth are like dentinogenesis imperfecta

pulpal dysplasia both dentitions

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
112
Q

pulpal dysplasia teeth

A

both dentitions flame/thistle shaped pulp and pulp stones

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
113
Q

regional odontodysplasia tissues, xrays, and histo

A

affects enamel, dentin, and pulp
ghost teeth on xray
enameloid conglomerates on histo

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
114
Q

ghost teeth on xray

A

regional odontodysplasia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
115
Q

enameloid conglomerates on histo

A

regional odontodysplasia and amelogenesis imperfetca

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
116
Q

cheese molars

A

molar-incisor hypomineralization

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
117
Q

white chalky enamel

A

fluorosis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
118
Q

amoxicillin and developing teeth

A

similar to fluorosis

affects 1st molars and mx central incisors

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
119
Q

cupped lesions on teeth

A

erosion (acid)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
120
Q

v shaped cervical lesions on teeth

A

abfraction

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
121
Q

tooth resorption types, why,, which more common

A

internal (by pulp cells) and external (by PDL cells, more common)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
122
Q

factors assoc w external resorption (4)

A

zoster, Pagets, cysts, tumors

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
123
Q

inflammatory internal resorption what happens

A

dentin replaced by granulation tissue

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
124
Q

pink tooth of mummery what is

A

inflammatory internal resorption in coronal pulp

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
125
Q

metaplastic internal resorption aka and what happens

A

aka replacement internal resorption
dentin replaced by bone
area is radiodense

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
126
Q

external resorption on xrays

A

moth eaten, usually apical

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
127
Q

which cells cause tooth resorption

A

dentinoclasts

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
128
Q

dentin replaced by granulation tissue

A

inflammatory internal resorption

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
129
Q

inner dentin replaced by bone

A

replacement/metaplastic internal resorption

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
130
Q

moth eaten apical root defects

A

external resorption

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
131
Q

rapid external resorption starting at cervix of teeth

A

invasive cervical resorption

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
132
Q

resorption of multiple teeth without obvious cause

A

multiple idiopathic apical root resorption

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
133
Q

how can gingival hemorrhage stain teeth

A

green from breakdown of Hb into biliverdin

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
134
Q

stannous fluoride staining

A

labial surfaces of anterior teeth and occlusal of posterior teeth

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
135
Q

chlorhexidine staining

A

interproximal surfaces near gingival margins

listerina and sanguinaria also

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
136
Q

gunther disease aka and effect on teeth

A

congenital erythropoietic pophyria
deposition of porphyrin
red teeth

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
137
Q

ochronosis what is??

A

blue/black teeth, connective tissues, tendons, cartilage

alkaptonuria (black urine disease: AR; phenylalanine and tyrosine metabolism)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
138
Q

chlorodontia why

A

green teeth
hyperbilirubinemia
2/2 biliverdin deposition

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
139
Q

pink teeth reasons (4)

A

trauma
inflammatory internal resoprtion
leprosy (mx incisors)
postmortem

140
Q

tetracycline and teeth

A 
incorporates into developing tooth
bright yellos to dark brown
UV diagnostic (yellow fluorescence)
141
Q

UV tetracycline teeth

A

yellow fluorescence

142
Q

minocycline teeth

A

blue gray discoloration of alveolus evident though thin mucosa

143
Q

impacted tooth what happens

A

eruption stops before emergence

144
Q

green teeth reasons

A

chlorodontia
hyperbilirubinemia
2/2 biliverdin deposition

145
Q

red teeth why

A

congenital erythropoietic porphyria )aka gunther dz

deposition of porphyrin

146
Q

blue black teeth why

A

ochronosis, alkaptonuria (AR, phenylalanine and tyrosine metabolism)

147
Q

blue gray alveolus discoloration

A

minocycline – seen through thin mucosa

148
Q

yellow/brown teeth why

A

tetracycline – yellow fluorescence under UV

incorporates into developing tooth

149
Q

eruption stops before emergence

A

impacted tooth

150
Q

bone spicule over crown of partially erupted tooth

A

eruption sequestrum

151
Q

eruption stops after emrgence – why? which teeth most common?

A

ankylosis
tooth fuses w bone
most common in md primary first molars

152
Q

total lack of teeth

A

anodontia – usually hypohydrotic ectodermal dysplasia

153
Q

lack of 1+ teeth

A

hypodontia

154
Q

lack of 6+teeth

A

oligodontia

155
Q

hypodontia vs oligodontia

A

hypo == lack 1+

oligo – lack 6+

156
Q

PAX9 and teeth

A

hypodontia, esp molars

157
Q

MSX1 and teeth

A

hypodontia, distal tooth of every type

158
Q

he zhao

A

hypodontia

159
Q

which hypodontia gene is assoc w colonic polyps and colorectal ca

160
Q

AXIN2

A

hypodontia, colonic polyps, colorectal Ca

161
Q

supernumerary tooth of anteiror maxilla

162
Q

fourth molar

A

distodens or distomolar

163
Q

paramolar location

A

supernumerary tooth lingual or buccal to molar

164
Q

types of supernumerary teeth

A

supplemental – normal size/shape

rudimentary – abnormal size/shape

165
Q

three looks of rudimentary supernumerary teeth

A

conical, tuberculate, molariform

166
Q

normal tooth in abnormal location – which teeth affetced

A

dental transposition

usually canines and first premolars

167
Q

dental transposition

A

normal tooth in abnormal location

usually canines and first premolars

168
Q

teeth present at birth – risks?

A

natal teeth – birth or shortly thereafter

assoc w riga fede dz

169
Q

relative microdontia

A

normal teeth bu macrognathia

170
Q

diffuse microdontia syndromes (2)

A

Down syndrome and pituitary dwarfism

171
Q

relative macrodontia

A

normal teeth but micrognathia

172
Q

diffuse macrodontia syndromes (4)

A

gigantism, otodental syndrome, XYY males, pineal hyperplasia

173
Q

unilateral macrodontia assoc w

A

hemifacial hyperplasia

174
Q

hemifacial hyperplasia and teeth

A

unilateral macrodontia

175
Q

otodental syndrome two main components

A

globe shaped molars and sensorineural deafness

176
Q

globe shaped molars and sensorineural deafness

A

otodental syndrome

177
Q

pituitary dwarfism teeth

A

diffuse microdontia

178
Q

Klinefelter males teeth

A

diffuse macrodontia

179
Q

pineal hyperplasia teeth

A

diffuse macrodontia

180
Q

single enlarged tooth; normal tooth count if anomalous counted as one

A

gemination

181
Q

single enlarged tooth; tooth count missin one if anomalous counted as one

182
Q

gemination vs fusion

A

gemination normal tooth count, fusion missing one (if enlarged counted as one)

183
Q

union of teeth only by cementum – why?

A

concrescence

developmental or post-inflammatory

184
Q

3 types of accessory cusps

A

cusp of carabelli, talon cusp, dens evaginatus

185
Q

cusp of carabelli

A

accessory cusp on palatal surface of mesiolingual cusp of mx 1 molar

186
Q

accessory cusp on palatal surface of mesiolingual cusp of mx 1 molar

A

cusp of carabelli

187
Q

protostylid

A

accessory cusp on mesiobuccal of md molar

188
Q

accessory cusp on mesiobuccal of md molar

A

protostylid

189
Q

talon cusp where and assoc

A

lingual ant teeth (esp mx lat incisor)

assoc w rubinstein-taybi syndrome

190
Q

rubinstein-taybi syndrome and teeth

other things?

A

talon cusp on mx lat incisor

other things: mental retardation, broad thumbs and toes

191
Q

cusplike elevation on occlusal of PM and molars – risks?

A

dens evaginatus

25% –> pulpal pathosis

192
Q

dens evaginatus – risks?

A

cusplike elevation on occlusal of PM and molars

25% –> pulpal pathosis

193
Q

shovel shaped incisors who

A

lingual surface looks like a scoop of a shovel
100% inuits/native americans
15% asians

194
Q

dens invaginatus

A

deep surface invagination of crown/root lined by enamel

most in mx lat incisors

195
Q

deep surface invagination of crown/root lined by enamel

A

dens invaginatus

most in mx lat incisors

196
Q

dens in dente

A

large invagination - dens invaginatus

197
Q

dilated odontome

A

dilated invigination disturbing tooth development

198
Q

dilated invigination disturbing tooth development

A

dilated odontome

199
Q

ectopic enamel names and locations

A

enamel pearl – root

cervical enamel extension – CEJ of molars bifurcation

200
Q

enlargement of body and pulp chamber – shape?

A

taurodontism

rectangular teeth

201
Q

taurodontism classifications

A

mild (hypotauro), moderate (meso), severe (hyper)

202
Q

pts w increased taurodontism

  • other local troubles
  • 4 syndromes
A

pts w hypodontia, cleft lip and cleft palate

assoc w ectodermal dysplasia, hypophosphatasia, Down, tricho-dento-osseous syndrome

203
Q

hypercementosis vs cementoblastoma

A

cementoblastoma painful, expansion, continuous growth

204
Q

generalized hypercementosis suggests what

205
Q

dilaceration what is and why

A

abnormal angulation/bend of root

idiopathic, trauma, of adjacent lesion

206
Q

abnormal angulation/bend of root

A

dilaceration

idiopathic, trauma, of adjacent lesion

207
Q

all teeth present and erupted bu enamel is v thin

A

hypoplastic amelogenesis imperfecta

208
Q

teeth normal shape but discolored and enamel is soft

A

hypomaturation amelogenesis imperfecta

209
Q

AMELX9

A

amelogenesis imperfecta

amelogenin

210
Q

ENAM

A

amelogenesis imperfecta

enamelin

211
Q

MMP20

A

amelogenesis imperfecta

enamelysin

212
Q

KLK4

A

amelogenesis imperfecta

kallikrein

213
Q

DLX3

A

amelogenesis imperfecta

group of genes

214
Q

AMBN

A

amelogenesis imperfecta

ameloblastin

215
Q

normal tooth shape but soft and very dark enamel

A

hypocalcified amelogenesis imperfecta

216
Q

amelogenesis imperfecta + taurodontism

A

hypomaturation-hypoplastic (IVa, thicker enamel) and hypoplastic hypomaturation (IVB)

217
Q

tricho-dento-osseous syndrome components (5)

A

amelogenesis imperfecta + taurodontism, kinky hair, osteosclerosis, brittle nails

218
Q

amelogenesis imperfecta + taurodontism, kinky hair, osteosclerosis, brittle nails

A

tricho-dento-osseous syndrome

219
Q

dentinogenesis imperfetca aka

A

hereditary opalescnet teeth

Capdepont’s teeth

220
Q

Capdepont’s teeth

A

dentinogenesis imperfetca

221
Q

dentinogenesis imperfetca vs osteogenesis imperfecta

A

dentinogenesis imperfetca DSPP, no systemic disorder

osteogenesis COL1A1/A2

222
Q

dentinogenesis imperfetca lewk

A

blue/brown and translucent

bulbous crowns, cervical construction, thin roots, obliteration of canals and chambers

223
Q

blue/brown and translucent teeth

A

dentinogenesis imperfetca

224
Q

bulbous crowns, cervical construction, thin roots, obliteration of canals and chambers

A

dentinogenesis imperfetca

225
Q

shell teeth

A

enamel normal thickness, thin dentin, very enlarged pulp

dentinogenesis imperfecta

226
Q

enamel normal thickness, thin dentin, very enlarged pulp

A

shell teeth

dentinogenesis imperfecta

227
Q

dentinogenesis imperfecta histo

A

shot tubules in granular dentin w calcifications

228
Q

dentin dysplasia types/subtypes

A

type I - radicular (Ia-Id)
rootless teeth, no pulp, periapical RL, pulp stones

type II – coronal
normal root length, DSPP mutation
deciduous teeth look like dentinogenesis imperfecta’
permanent normal color but flame/thistle shaped pulp and pulp stones

229
Q

flame/thistle shaped pulp and pulp stones

A

dentin dysplasia type II if only permanent

pulpal dysplasia if both dentitions

230
Q

rootless teeth

A

dentin dysplasia type I

231
Q

fibrous dysplasia of dentin ddx

A

radiodensity fills pulp chamber and canal
small foci of RL in pulp (vs dentinogenesis imperfecta)
roots normal length (vs dentin dysplasia)

232
Q

teeth no pulp and no roots

A

dentin dysplasia type I

233
Q

teeth no pulp but normal roots

A

fibrous dysplasia of dentin

234
Q

dentin-dysplasia like teeth – 5 systemic dzz

A 
calcinosis universalis
tumoral calcinosis
rhematoid arthritis
vitaminosis D
sclerotic bone + skeletal abnormalities
235
Q

dentin dysplasia type I histo

A

whorls of tubular dentin with peripheral layer of normal dentin
(stream flowing around boulders)

236
Q

(stream flowing around boulders)

A

dentin dysplasia type I

whorls of tubular dentin with peripheral layer of normal dentin

237
Q

dentin dysplasia type II histo

A

primary teeth like dentinogenesis imperfecta (short tubules in granular dentin with calcifications)
permanent teeth atubular dentin and pulp stones

238
Q

regional odontodysplasia aka and what is

A

ghost teeth

non-hereditary developmental anomaly of enamel, dentin, pulp

239
Q

what’s wrong in regional odontodysplasia

A

vascular, neural, growth abnormalities

some cases assoc w vascular nevi

240
Q

teeth in regional odontodysplasia

A

ghost teeth

v thin enamel and dentin around enlarged radiolucent pulp

241
Q

ghost teeth

A

v thin enamel and dentin around enlarged radiolucent pulp

regional odontodysplasia

242
Q

regional odontodysplasia histo

A 
enameloid conglomerates (focal collections of basophilic calcifications)
also seen in AI
243
Q

enameloid conglomerates

A

focal collections of basophilic calcifications)

regional odontodysplasia and amelogenesis imperfetca

244
Q

qualitative vs quantitative enamel defect

A

qualitative - variation in translucency – enamel opacity

quantitative – hypoplasia – pits and grooves

245
Q

decisuous teeth crown development

A

14th week in utero - 12 mo

246
Q

permanent teeth crown development

247
Q

enamel defect in permanent tooth 2/2 periapical disease or trauma of primary tooth

A

turner hypoplasia

248
Q

radioation dose to cause enamel hypoplasia

249
Q

mottled enamel

250
Q

attrition locations

A

occlusal surfaces, lingual max, labial mand

251
Q

abrasion location

A

prominent teeth, buccal surface

252
Q

abfraction location

A

cervical area or sublingual gingiva

facial of bicuspids

253
Q

perimolysis conditions

A

gastric acid erosion

GERD< bulimia

254
Q

erosion locations

A

facial/palatal maxilla
facial/occusal mandibular
central depression surrounded by enamel

255
Q

inflammatory internal resorption what happens

A

dentinal tubules replaced by granulation tissue

256
Q

replacement/metaplastic internal resorption what happens

A

dentinal tubules replaced by cementum or bone

257
Q

pink tooth of mummery what it, why pink

A

internal resorption affecting the coronal part, causes the blood
vessels to approach the surface and pink discoloration

258
Q

multiple areas of invasive external cervical resorption

occurs , cause unknown

A

Multiple idiopathic root resorption:

259
Q

rapid external resorption at neck of tooth

A

invasive cervical resorption

260
Q

moth eaten apical/mid portion of root

A

external resorption

261
Q

stannous fluoride cause of staining

A

tin (stannous) + bacteria hydrogen sulfide

262
Q

congeniral eryhtropoietic porphyria inheritance, reasons, color, additional color features

A

 AR
 Deposition of porphyrin in teeth
 Also appears in urine
 Red-brown color that exhibits a red fluorescence when exposed to Wood’s UV light

263
Q

red brown teeth and urine

A

congeniral eryhtropoietic porphyria

264
Q

alkaptonuria tissue phenomenon, inheritance, color features

A

(Ochronosis):
 AR
 Blue-black pigmentation in CT, sclera, teeth, cartilage

265
Q

blue-black cartilage and sclera

A

ochronosis in alkaptonuria

266
Q

Congenital erythropoietic porphyria aka and tooth color

A

Gunter disease
red brown teeth
red fluorescnece on Wood’s lamp test

267
Q

alkaptonuria tooth color

A

blue black in cervical areas

268
Q

jaundice tooth color

A

blue black in cervical area

green (chlorodontia) 2/2 biliverdin from bilirubin

269
Q

causes of hyperbilirubinemia in infants

A

erhtroblastosis fetalis

biliary atresia

270
Q

green teeth

A

chlorodontia
hyperbilirubinemia
IV ciprofloxacin for klebsiella in kids

271
Q

trauma tooth color when

A

pink 1-3 wks after trauma

272
Q

yellow teeth after trauma

A

pulp obliteration, calcific metamorphosis

273
Q

lepromatous leprosy teeth

A

pink/red maxillary incisors

274
Q

pink/red maxillary incisors

A

trauma or leprosy

275
Q

tetracycline teeth

A

yellow-brown

276
Q

minocycline teeth and other locations

A

gray on incisal hald

also deposits in thyroid / sclera / nails / bones / teeth

277
Q

cirpfloxacin scenario and teeth

A

IV for Klebsiella in kids

green teeth

278
Q

ankylosis most freq affected teeth

A

Mandibular primary 1st molar > Mandibular primary 2nd molar > Maxillary primary 1st
permanent molar

279
Q

pax9 teeth

A

AD / Oligodontia in permanent molars

280
Q

msx1 teeth

A

AD / Distal tooth of each type

Most common 2nd PM / 3RD M

281
Q

axin2 teeth

A

AD / 2nd + 3rd molars

+adenomatous polyps and colorectal ca

282
Q

chinese hypodontia gene

A

He Zhao deficiency

283
Q

pakistain hypodontia

284
Q

eda teeth

A

Non syndromic x –linked

Max + mand CI > LI > C > PM

285
Q

WNT10A teeth

A

Non syndromic

Max + mand lat. Incisors + 2nd PM

286
Q

hypodontia which teeth

A

3rd molars > 2nd PM > LI

287
Q

types of rudimentary supernumeraries

A

Conical; small peg shaped
Tuberculate; barrel shaped
Molariform; small PM-like or molar –like

288
Q

normal teeth erupt in abnormal site

A

Dental transposition:

289
Q

Dental transposition:

A

normal teeth erupt in abnormal site

290
Q

neonatal teeth: timing, what are they

teeth

A 
first 30 days of life / most are premature eruption of primary teeth
Mandibular incisors (85%) > maxillary incisors (11%) > posterior teeth (4%)
291
Q

Riga-Fede Disease:

A

New born with traumatic ulceration of anterior ventral

tongue during breast feeding due to neonatal teeth

292
Q

New born with traumatic ulceration of anterior ventral

tongue during breast feeding due to neonatal teeth

A

Riga-Fede Disease:

293
Q

5 macrodontia conditions

A 
 Gigantism
 Otodental syndrome : Globodontia + Sensory loss
 XYY
 Hyperinsulinemia
 Pineal Hyperplasia
294
Q

Globodontia + Sensory loss

A

Otodental syndrome

295
Q

2 microdontia conditions

A

Down syndrome

 Pituitary dwarfism

296
Q

Enamel pearl locations

A

Ectopic enamel on roots of max 3rd molars > mand molars

297
Q

Dense Invaginatus types:

A

 Type 1 = confined to crown ( Most common )
 Type 2 = extends below CEJ
 Type 3 = extends through root ( least common

298
Q

cervical enamel extension location

A

Mandibular molars most common but can occur at any molar

299
Q

globodontia look, association, dentition condition, radio look

A

 gigantic globe shaped teeth “ TIED END OF SAUSAGE”
 Ass w otodental syndrome
 PM usually missing
 Radiograph: short roots, bulbous crowns, pulp stones

300
Q

gigantic globe shaped teeth

A

globodontia

301
Q

TIED END OF SAUSAGE

A

globodontia

302
Q

lobodontia inheritance, affected teeth

A

AD

 Cuspids and PM fang-like cusps / Tooth size reduced

303
Q

Cuspids and PM fang-like cusps /

A

lobodontia

304
Q

three type families of amelogenesis imperfecta

A

Hypoplastic
 Hypomineralization (hypomaturation / hypocalcification)
 AI w taurodontism (hypomaturation / hypoplastic)

305
Q

cheesy enamel

A

hypocalcified AI

306
Q

hypocalcified AI enamel

307
Q

amelogenesis imperfecta similar to fluorosis

A

Hypomaturation

308
Q

tricho-dento-osseous syndrome inheritance and look

A

AD
 Kinky hair at birth but may straighten with age
 Osteosclerosis affects base of skull and mastoid process
 Short ramus and obtuse angle

assoc w AI

309
Q

8 amelogenesis imperfecta genes with products

A 
AMELX - amelogenin - 90%
ENAM - enamelin - 5%
MMP20 - enamelysin
KLK4 - kallikrein 4
FAM83H
C4orf26 - extracellular matrix
DLX3
WDR72 - protein scaffold
310
Q

most commonly affected genes in amelogenesis imperfetca with products

A

AMELX - amelogenin - 90%

ENAM - enamelin - 5%

311
Q

AMELX inheritance and type

A

x-linked amelogenesis imperfecta

hypomat/hypoplastic

312
Q

ENAM inheritance and type

A

AD, AR amelogenesis imperfecta

hypoplastic

313
Q

MMP20 inheritance and type

A

AR

Pigmented hypomaturation amelogenesis imperfecta

314
Q

KLK4 type

A

hypomaturation amelogenesis imperfecta

315
Q

FAM83H inheritance and type

A

AD

hypocalcified amelogenesis imperfecta

316
Q

C4orf26 inheritance and type

A

AR amelogenesis imperfecta

hypomineralized

317
Q

DLX3 type

A

Hypmat/hypoplastic amelogenesis imperfecta w taurodontism

Maybe ass w trich-dento-oseeous syndrome

318
Q

WDR72 inheritance and type

A

AR
amelogenesis imperfecta
hypomaturation

319
Q

amelogenin gene

320
Q

enamelin gene

321
Q

enamelysin gene

322
Q

kallikrein 4 gene

323
Q

scaffold gene in amelogenesis imperfecta

324
Q

extracellular matrix gene in amelogenesis imperfetca

325
Q

AD genes of amelogenesis imperfecta

A

ENAM and FAM83H

326
Q

x linked genes of amelogensis imperfetca

A

AMELX only

327
Q

tricho dento osseous syndrome gene

328
Q

AR genes of amelogenesis imperfetca

A

MMP20, C4orf26, WDR72

329
Q

hypoplastic amelogenesis imperfecta genes, inheritance, mechanism, presentations

A

AD: ENAM
AR: More severe

Inadequate deposition of enamel matrix
Generalized: pinpoints
Local: mid 1/3 of buccal

330
Q

Inadequate deposition of enamel matrix amelogenesis imperfecta

A

hypoplastic

331
Q

amelogenesis imperfecta what’s affected, teeth look, other mouth problems

A 
Both dentition
Pulpal calcification
Taurodontism
Delayed eruptions
Gingival overgrowth
Open bite occlusion
332
Q

hypomaturation vs hypocalcification amelogenesis

A

both are hypomineralization

Hypomaturation:
Enamel chips & fractures but no loss of enamel
during eruption
White opaque, mottled E
Resemble fluorosis
1- Pigmented
2- X-linked
3- Snow capped

Hypocalification:
Normal shape
Enamel soft and lost easily (cheesy) except cervical
portion (spared)

333
Q

amelogenesis:
Hypomat /hypoplastic vs Hypoplastic/hypomat
inheritance?

A

AI with taurodontism – AD

Hypomat /hypoplastic : pits only

Hypoplastic/hypomat : Enamel is thin and pits

334
Q

dentinogenesis imperfecta-like changes in systemic diseases (4)

A

 OI
 Ehlers-Danlos Syndrome
 Goldblatt syndrome
 Schimke immune-osseous dysplasia

335
Q

dentinogenesis imperfecta what is and aka

A

 Development of dentin is disturbed WITHOUT systemic disease
 Capdepont’s teeth/hereditary opalescent dentin: Also known as DGI
 Shell teeth: expanded pulp in DGI

336
Q

Systemic diseases in correlation with dentin dysplasia-like alterations:

A

 Calcinosis Universalis
 RA and hypervitaminosis D
 Sclerotic bone and skeletal anomalies
 Tumoral calcinosis

337
Q

OI w opalescent teeth inheritance and genes

A

AD

COL1A1, COL1A2

338
Q

DSPP disorders and inheritance

A

DI, dentin dysplasia 2

AD

339
Q

dentin dysplasia 1 inheritance and gene

A

AD, gene unknown!

340
Q

Dentinogenesis Imperfecta aka, injeritance, affected teeth, look

A

AKA:
Hereditary opalescent dentin
Capdepont’s teeth

AD

Both Dentition
Dec > per
Bulbous crowns
cervical constriction
early obliteration of pulp
shell teeth
hearing LOSS
341
Q

dentin dysplasias types and inheritance

A

DD1 – radicular
DD2 – coronal

both AD

342
Q

regional odontodysplasia aka, inheritance

age, affected teeth

A

ghost teeth
localized non hereditary
Both dentitions

Bimodal peak : 2-4 / 7-11 yrs
Max > mand
Ant > post
One qua > 2 quad

343
Q

otodental syndrome genetics, teeth, major points

A

AD / chr # 11q13
FGFR3

Globodontia (large teeth)
Hearing loss

344
Q

tricho-dento-osseous syndrome inheritance, possible mutation,
manifestations

A

AD – poss DLX3
hypoplastic/hypomaturation AI-like

Osteosclerosis base of skull & mastoid
process mainly
Kinky hair
Brittle nails
Short ramus and obtuse angle
345
Q

Enameloid conglomerate: what is and where

A

basophilic enamel like calcifications

regional odontodysplasia and amelogenesis imperfecta

346
Q

pathoses associated with regional odontodysplasia (9)

A 
 Ectodermal dysplasia
 Epidermal nevi
 Hypophosphatasia
 NF
 Rh factor incompatibility
 Ipsilateral facial hypoplasia
 Hydrocephalus
 Orbital Coloboma
 Vascular Nevi
347
Q

proposed causes of regional odontodysplasia (9)

A 
 Abnormal migration of neural crest cells
 Latent virus
 Local circulatory deficiency
 Local trauma
 Hyperpyrexia
 Malnutrition
 Medication
 Radiation
 Somatic

OP - Yield (19 decks)

Brainscape helps you reach your goals faster, through stronger study habits.
© 2025 Bold Learning Solutions. Terms and Conditions