18 facial pain Flashcards
causes of bell plasy
HSV/VZV reactivation, demyelination, nerve edema.ischemia, autoimmune damage, vasospasms of nerve-feeding vessels
rapid onset of bilateral facial paralysis consider what
guillain-barre and uveroparotid fever (sarcoid)
most freq distribution of trigeminal neuralgia
V2/V3
common trigger point for trigeminal neuralgia
nasolabial fold
1st line tx for trigeminal
carbamazepine (anticonvulsant)
neurosurgery complications
facial dysesthesia and anesthesia dolorosa (numbness/pain)
glossopharyngeal neuralgia what’s affected
9th nerve (tonsil radiating to ear)
pain felt in ear neuralgia
tympanic plexus – ddx from TMD
trigger point for glossopharyngeal neuralgia
in ear canal
postherpetic neuralgia timeline
persists 1-6 months after onset of rash
postherpetic vs trigeminal
postherpetic burns w episodic stabbing
tactile allodynia
light touch elicits pain
postherpetic neuralgia
age for postherpetic neuralgia
50% of pts over 50 and 75% of pts over 75
atypical facial pain characteristic
drawing aching or pulling pain
hot spot on bone scan – increased temperature, tenderness, or marros activity
cluster headache clinical, cause, demographics
pain of midface and upper face, esp around eyes attacks in temporal groups vascular cuase 80% smokers \+ fmailial influence
cluster headache aka
horton’s syndrome, histaminic cephalgia, migrainous neuralgia
alarm clock headache
cluster – same time everyday
cluster headahce vs migraine
migraine has aura and pt is reclusive
vs cluster – no aura and hyperactive
paroxysmal hemicrania characteristics
attacks last 2-30 min, up to 40x /day
high freq, short attacjs, responds to indomethacin
hemicrania continua
unilateral, chronic, daily similar to cluster
responds to indomethacin
chronic paroxysmal hemicrania aka and cayse
sjastaad syndrome
congestion of conjunctival blood vessles and increased intraocular pressure
attacks last 2-30 min, up to 40x /day
high freq, short attacjs, responds to indomethacin
paroxysmal hemicrania
migraine characteristics
paroxysmal, unilateral, 4-72h
migraine cause
vasoconstriction of brain arteries 2/2 serotonin reduction –> ischemia and dilation
aura characteristic of
migraine
visual hallucination before pain
sparks (scintillation), partial blindness, loss of light perception (scotome), loss of ability to express thoughts (aphasia)
temporal arteritis aka and cause
giant cell arteritis, cranial arteritis
multifocal autoimmune vasculitis of temporal artery
headache coincinding with heartbeat
headache coincinding with heartbeat
temporal arteritis
temporal arteritis oral symptoms
jaw claudication (pain w mastication), pain wearing hats (pressure), blindness
generalized muscle ache and stiffness after headache attack
polymyalgia rheumatica
temporal arteritis
temporal arteritis histo
chronic inflammation of tunica intima and media, narrowing of lumen from edema and prolif of tunica intima, necrosis, giant cells
chronic inflammation of tunica intima and media, narrowing of lumen from edema and prolif of tunica intima, necrosis, giant cells
temporal arteritis
myasthenia gravis mechanism
autoimmune attack on ACh receptors w progressive fatigue of skeletal muscle
pts have thymus hyeprplasia or thymoma and at least 1 other autoimmune diosrder
myasthenia gravis clinical
subtle but progressive weakness, most noted in head and neck
inability to focus eye, drooping eyelid (ptosis), double vision (diplopia), dysphagia, dysarthria (slurred speeech), difficulty chewing
jaws hang open
inability to focus eye, drooping eyelid (ptosis), double vision (diplopia), dysphagia, dysarthria (slurred speeech), difficulty chewing
jaws hang open
myasthenia gravis
myasthenia gravis diagnosis
elevated serum ACh receptor antibodies
elevated serum ACh receptor antibodies
myasthenia gravis
myasthenia gravis histo
degenerated muscle fibers – smaller, lose rounded cross-section look
degenerated muscle fibers – smaller, lose rounded cross-section look
myasthenia gravis
neurodegenerative disorders w progressive weakness and mm wasting
progressive muscular atrophy
progressive bulbar palsy
amyotrophic lateral sclerosis
most common lethal AR disorder
progressive muscular atrophy
(SMN gene)
childhood; no facial involvement