15. neurocognitive correlates of change in CBT Flashcards
notable areas of the brain, cognitive model of anx + depr, neurocognitive correlates of change in CBT
what’s the PFC?
prefrontal cortex
purpose of the PFC + category
what therapy targets this part of the brain?
- high order functioning
- reasoning, planning
CBT
what’s the ACC?
anterior cingulate cortex
what’s the purpose of the ACC + category?
- connects higher + lower order structures
- integration of info
- emotional info
what’s the purpose of the hippocampus? what order of functioning does it occupy?
- attaches emotion to memories
- lower order of functioning
what’s the purpose of the amygdala? what order of functioning does it occupy?
- it’s the center of emotional information
- lower order of cognition
what is top-down processing?
what therapy relies on this kind of processing?
slow, deliberate, explicit, strategic processing that uses rule-based knowledge
CBT – question-processing, targets PFC
what’s bottom-up processing?
automatic, effortless, implicit, pre-conscious processing based on salient features or stimulus/situational cues
what kinds of therapy targets bottom-up processing?
what part of brain is targeted?
- CBT too, but not as explicitly targeted
- relies on trickle-down effect
- targets ACC
which information processing biases relate to schemas?
- selective attention to fearful/mood-congruent stimuli
- selective memory for negative>positive material
- inability to disengage from negative material
- interpretation of ambiguous events as not positive
cognitive model of depr + anx
what is thought to lead to depressive rumination?
inability to disengage, pull attention away from negative material
how does CBT act in changing biased info processing?
what does this prove?
- reduction of negative cognition (cognitive mediation)
- reduction of attentional bias for threat in anx dis
- CBT + meds results in greater connections between positive schema content than med alone in tx of depr
- reduction of negative schema content after sad mood induction than meds alone in depr
it validates the cog model: to improve sx, need a change in cog
according to Linden, what is cognitive restructuring thought to do?
increase top down cognition control over negative emotion
neurocognitive correlates of change in CBT
what kind of reactivity is supposed to be decreased by medication?
bottom up reactivity to emotional stimuli
less reactive rather than restructuring
through which pathway does CBT produce sx reduction?
through its impact on higher order executive functions (problem solving, cognitive reappraisal, self-referential thinking)