14. Dr. Stephens CIS lectures spinal cord and brainstem lectures (pre-reading and lectures) Flashcards
What are the deficit patterns that are seen with dorsal root injuries?
Diminished sensation or reflex from associated sensory or motor dermatome
What are the deficit patterns that are seen with an injury to the fasciculus gracilis?
Ipsilateral loss of proprioception and no 2 point discrimination of the lower limb
What are the deficit patterns that are seen with an injury to the fasciculus cutaneous?
Ipsilateral loss of the proprioception and no 2 point discrimination to the upper limb
If the lateral corticospinal tract is damaged, what happens?
ipsilateral spastic paralysis
If the lateral reticulospinal tract is damaged, what happens?
Contralateral loss of pain and temperature 2 sensory dermatomes below the lesion
If the lateral reticulospinal tract is damaged, what happens?
There is loss of autonomic functions including bowel and bladder incontinence
If there is damage to the anterior white commissure, what happens?
Bilateral pain and temperature anesthesia
If there is damage to the anterior horns, what happens?
Ipsilateral LMN paralysis
If there is decreased proprioception to to L3-S4 dermatomes of the right leg, where is the associated lesion?
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Fasciculus gracilis
If there is hyperreflexia, hypertonia, paralyzed muscles to the left lower extremity including clonus, what is involved in the deficit?
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These muscles are in spastic paralysis and the lateral corticospinal tract is involved
If there is loss of pain and temperuare of the L3-S4 dermatomes, what is the tract that is involved?
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Lateral spinothalamic tract
If there is a loss of pain and temperature to C5-C6 region, what is the deficit? What is this called?
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Anterior white commissure deficit and it is called syringomyelia
If the following clinically presents, what would your diagnosis be?
Upper extremity: bilateral areflexemia, atonia, paretic and atrophied muscles
Lower extremity: bilateral hyperreflexemia, hypertonia paralyzed muscle strength with clonus and bilateral positive babinski reflexes
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ALS: LMN and UMN paralysis
Describe how spinal shock would present clinically using the following picture as a guide to the injury
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Bilateral loss of pain and temperature below L1 and loss of proprioception and 2 point discrimination below L1
Upper extremity would be WNL
Lower extremity: areflexemia, atonia, paralysis
If there is a loss of proprioception and 2 point discrimination from L2-S5 in the right lower extremity with hyperreflexemia, hypertonia, and paralysis
In addition to decreased pain and temperature in the left lower extremity from L4-S5, what could be happening?
Brown Sequard syndrome
What causes brown sequard syndrome?
Lesions in the SC, hemisections. The loss of proprioception and 2 point discrimination occurs on the same side of the lesion and the loss of pain and temperature occurs on the opposite side of the lesion
In this clinical presenation of Brown Sequard syndrome, where would the lesion be? (red is loss of pain and temperature and green is a loss of proprioception and 2 point discrimination)
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The lesion would be in the right spinal cord
A patient presents with left sided spastic hemiplegia including
LUE: hyperreflexemia, hypertonia, paralysis and a positive Hoffmans sign
LLE: hyper reflexemia, hypertonia, paralysis and clonus
positive Babinksis sign
Which tract is involved with these sx?
Corticospinal tract
If a patient presents with L sided proprioceptive hemianesthesia, where would you suspect the damage is?
Medial Lemniscus: an issue with PCMLS
If a patient presents with deficits of the spinal lemniscus, what would you expect to see in that patient?
Loss of pain and temp to an entire side of the body (hemianalgesia)
If there is a lesion at 1, what would you expect to see?
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CONTRALATERAL spastic hemiplasia because there is a lesion of the corticospinal tract
If there was a lesion at 2, what would you expect to see?
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Contralateral loss of proprio/2 point discrimination due to a lesion of the medial lemniscus
If there was a lesion at 3, what would occur?
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Contralateral hemianalgesia, due to a lesion of the spinal lemniscus
If there was a lesion at 4, what would occur?
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Ipsilateral hemianalgesia in the face due to a lesion in the descending tract of V
If there was a lesion at 5, what would you expect to see clinically?
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Ipsilateral Vestibular signs (nystagmus, vertigo, nausea, etc.) due to a lesion of the Vestibular nuclei
If there was a lesion at 6, what would you expect to see clinically?
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Ipsilateral cerebellar signs due to a lesion in the inferior cerebellar peduncle
Positive Romberg, ataxia, dysmetria, etc
Review the picture on page 10 of the DSA for Stephens
Review for 5 minutes (Staci this is what you do so I am just putting this here for you, youre welcome)
If a patient presents with a lesion in 3 and 4, what would you expect to see clinically?
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Ipsilateral loss of pain and temperature to the face
Contralateral loss of pain and temperature to the body due to the involvement of the descending tract of CN V, and spinal lemniscus alternating hemianalgesia
With dysphagia and dysphonia secondary to CN X involvement
Also known as lateral medullary syndrome or Wallenburgs syndrome
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If there is a lesion at 4, what would you expect to see?
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Lesion is at the right fasciculus gracilis which would result in ipsilateral loss of proprioception to the lower extremity
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What are the 7 clinically important structures that are located within the spinal cord?
sensory
- dorsal roots
- posterior columns
- lateral spinothalamic tract
- anterior white commissure
motor
- lateral corticospinal tract
- anterior horn
- lateral reticulospinal tract
What are the 8 clinically important structures in the brainstem?
sensory
- spinal lemniscus
- medial lemniscus
- trigeminal lemniscus
- lateral lemniscus
- descending tract of CN V
motor
- corticospinal tract
- corticobulbar tract
special
- medial longitudinal fasciciulus
Spastic hemiplegia indicates involvement of which of the following?
a. Corticospinal tract
b. Lateral reticulospinal tract
c. Spinal lemniscus
d. Genu of internal capsule
e. Ventral roots
a. corticospinal tract
Supranuclear facial palsy indicates involvement of which of the following?
a. Facial nerve
b. Corticobulbar tract
c. Corticospinal tract
d. Posterior limb of internal capsule
e. Rubrospinal tract
B
1.
2.
3.
Corticobulbar fibers originate in the head region of precentral gyrus,
Course through the genu of the internal capsule and cerebral peduncles as uncrossed CBT
• Unilateral lesions of uncrossed CBT result in contralateral supranuclear facial palsy
Decussate in lower pons (between V and VI) and descend in the lower brainstem as crossed CBT
• Unilateral lesions below the decussation may result in some ipsilateral cranial nerve palsies.