Cardiac II Flashcards

1
Q

What are the types of congestive heart failure

A

Systolic dysfunction- Heart can’t generate enough force to pump blood (low cardiac output, anything below 40% ventricular volume)

Diastolic dysfunction- can’t dilate adequately to fill with enough blood

Left sided heart failure
Right sided heart failure

High output failure- cardiac output is normal but demand for blood flow is high

Low output failure- cardiac output is low but demand for blood is normal

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2
Q

Pathogenesis of congestive heart failure

A
  • Increased hydrostatic pressure
  • renin angiotensin aldosterone system activation
  • sodium retention
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3
Q

What is diastolic heart failure

A

a condition caused by increased resistance to the filling of one or both ventricles

will have no time to relax

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4
Q

What is acute rheumatic fever, what causes it and main concern

A

Autoimmune inflammatory process due to group A strep inf

.3-3% of time will develop rheumatic heart disease

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5
Q

Clinical presentation of acute rheumatic fever (symptoms)

A
  • Sore throat
  • Polyarthritis (usually symmetrical and involves large jts)
  • Sydenham chorea (neuro involunatry monts)
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6
Q

Jones criteria for dx of rheumatic fever

A

1 required criteria and 2 major and 0 minor (or 1 maj and 2 minor)

Required- Evidence of strep
Major- Carditis, poly arthritis, chorea, erythema marginatum,, subcutaneous nodules

Minor- Fever, arthralgia, prev fever

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7
Q

What kind of cells form in acute rheumatic fever

A

Formation of small giant cell granulomas called aschoffs bodies mainly in left atrium and ventricle

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8
Q

When does chronic rheumatic valve disease occur and what does it target

A

develops up to 30 years after first attack of RF

attacks mitral valve in 50% of cases

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9
Q

What is endocarditis and clinical manifestation

A

inflamation of the innermost layer of heart

  • new cardiac murmur
  • osler nodes (raised tender lesions on hand)
  • Roth spots (pale lesions in retina)
  • Janeway lesions (erythematous lesion palm/sole)
  • Splinter hemmorrages
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10
Q

What is the characteristic lesion in infective endocarditis

A

Vegatations- occur on valve leaflets or chordae tendineae

NEVER occur on the atheromatous plaques in the aorta

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11
Q

Endocarditis cause in prostatic valves and elterly

A

prosthetic- staphylococci

Elderly- S auerus

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12
Q

What is the etiology of mitral valve stenosis and what is it + complications

A

Rheumatic heart disease is the cause of almost all cases of mitral stenosis

atrial fibrillation
thrombus in left atrium
pulmonary venous hypertension

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13
Q

What is the pathology of mitral incompetence

A

post inflammatory scarring of the mitral valve, holds the cusps in a partly open pos
increased rigidity and fusion of cusps often cause combo of stenosis and incompetence

-results in left ventricular function and failure

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14
Q

What is papillary mm ischemia

A

Loss of contractility of papillary mm as a result of mitral incompetence
-following infarction, rupture of infarcted papillary mm may cause sudden and severe incompetence with rapidly fatal left ventricular faulure

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15
Q

Main causes of aortic stenosis 92)

A

idiopathic calcified aortic stenosis

rheumatic heart disease

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16
Q

main complications of aortic valve stenosis

A

concentric left ventricular hypertrophy-> left ventricular failure

17
Q

Pathology of tricuspid valve stenosis and comon causes (2)

A

similar to mitral valve (both stenosis or incompetence)

mc- infective endocarditis + IV drug

18
Q

What are cardiomyopathies and 3 classifications

A

associated w mechanical or electrical dysfunction of heart

  • hypertrophic cardiomyopathy
  • congestic cardiomyopathy
  • restrictive cardiomyopathy
19
Q

What is hypertrophic cardiomyopathy, etiology and symtoms

A

HCM is a genetic disorder that causes myocardial hypertrophy that is inappropriate and asymmetrical and occurs in the absence of obvious inciting hypertrophic stim

s/s- Sudden death, angina, dyspnea, syncope, dizziness

20
Q

What is dilated (congestive) cardiomyopathy

A

Progressive, irreversible, disease causing global systolic contractile dysfunction w heart

  • dialated chancers unable to contract to maintain outoput
  • idiopathic process
21
Q

Causes of dilated cardiomyopathy

A

usually genetic

22
Q

What is restrictive cardiomyopathy

mc causes

A

The myocardium is infiltrated w a material that results in impaired ventricular filling

causes- amyloidosis, hemochromatosis, collagen deposition

23
Q

main causes of myocarditis, complications

A

Viral inf- enterovirus

complications- sudden death- lethal arryhymias

24
Q

what is transudate pericardial disease

A

Transudate is an extra cellular fluid and a result of increased fluid pressure or low oncotic pressure

  • low pro conc
  • low spec gravity
  • clear
  • low lactate dehydrogenase
25
Q

What is an exudate pericardial disease

A

Exudate is an inflammatory fluid

  • cloudy apperence
  • increased specific gravity
  • increased pro
  • increased inflammatory cells
26
Q

Symptoms of pericarditis

A

Sharp retrosternal chest pain, mainly left sided, abrupt onset, pain worsens in inspiration an dreleievd when leaning forward

27
Q

Serous pericarditis features

A
  • Minimal inflammation
  • no exudation of fibrin
  • fluid collection
28
Q

Fibrinous pericarditi features

A

Increased strands of fibrin

29
Q

What is pericardial effusion, types (4) and symptoms

A

Accumulation of fluid in pericardial sac

  1. Transudate (CHF, nephrotic syndrome)
  2. Exudate- TB, Pleural empyema
  3. Hemorrhagic- traumatic
  4. Malignant- metastatic fluid accumulation

s/s- pain, dyspnea, pericardial friction rib etc

30
Q

what is cardiac tamponade and presentation

A

compression of the heart due to increase in fluid within pericardiac sac

pres- slow development of Dyspnea, fatigue
signs- pulses paradoxes, pulses electrical activity

31
Q

histology of cardiac tumors

A

3/4 of primary heart tumors are benign, half of which are myxomas

32
Q

mc primary cardiac tumor

A
Atrial myxoma (benign)
in left atrium