11 - Common Clinical Growth and Development Issues Flashcards

1
Q

local causes of clinical growth and development issues

A
  1. Ankylosis
  2. Impaction
  3. Supernumerary tooth
  4. Trauma
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2
Q

systemic conditions causing clinical growth and development issues

A
  1. Low birthweight/premature
  2. Down syndrome
  3. Apert syndrome
  4. Chondoectodermal dysplasia
  5. Cliedocranial dysplasia
  6. Albright’s hereditary osteodystrophy
  7. Osteogenesis imperfecta
  8. Achondroplasia
  9. Gardner syndrome
  10. Hypothyroidism
  11. Hypopituitarism
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3
Q

what causes primary failure to erupt

A

Malfunction of eruption mechanism with non-ankylosed teeth
Failure of affected tooth to move through eruption path cleared for it
Teeth may partially erupt yet remain submerged or not erupt at all
Abnormal or complete lack of response to orthodontic forces

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4
Q

treatment of primary teeth failing to erupt

A

varies but often extraction and prosthodontic replacement

Successful treatment depends on multi-specialty collaboration: Pediatric/General dentist, Orthodontist, OMFS, Prosthodontist, etc.

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5
Q

what are common clinical growth and devlopment issues that lead to premature exfoliation of primary teeth

A
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6
Q

local causes that lead to accelerated permanent tooth eruption

A

early loss of primary teeth

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7
Q

systemic causes that lead to accelerated permanent tooth eruption

A
  1. Chondoectodermal dysplasia
  2. Hemifacial hypertrophy
  3. Osteogenesis imperfecta
  4. Hyperthyroidism
  5. Precocious puberty
  6. Sturge-Weber syndrome
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8
Q

what is a group of conditions where there is abnormal development of the skin, hair, nails, teeth or sweat glands

A

ectodermal dysplasia

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9
Q

any tissues that are embryologically derived from ___ may potentially be affected in ectodermal dysplasia

A

ectoderm

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10
Q

T/F: many different types/subcategories of ectodermal dysplasia is each caused by a specific genetic mutation

A

TRUE

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11
Q

what individuals may exhibit thin sparse hair, poor vision, and intolerance to heat due to lack of functional sweat glands

A

pt with ectodermal dysplasia

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12
Q

describe cleft lip

A

lip does not completely form

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13
Q

what is upper lip derived from

A

upper lift is derived from the median nasal process and maxillary process

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14
Q

when do median nasal process and maxillary process merge (lack of merge results in cleft lip)

A

5 weeks gestation

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15
Q

what is cleft palate

A

roof of mouth does not completely form leaving an opening that can extend into nasal cavity

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16
Q

what is the palate derived from

A

median nasal process and palatal shelves of maxillary process

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17
Q

when do median nasal process and palatal shelves of maxillary process merge

A

8-12 weeks gestation

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18
Q

prevalence of:
- cleft lip
- cleft palate
- clef lip with or without cleft palate

A

Cleft lip: 1 in 700 in US
Cleft palate: 1 in 1,000 in US
Cleft lip with or without cleft palate: 1 in 300-500 in US

19
Q

what are common dental findings for patients with cleft lip and/or palate

A

Congenitally missing teeth
Supernumerary teeth
Malformed teeth
Transposed teeth or teeth not in the correct position
Rotated teeth
Tipped teeth
Delayed eruption
Premature eruption
Enamel hypoplasia
Dentin defects
Class III malocclusions common (midface deficiency)
Narrow maxillary arch
Anterior or posterior crossbite

20
Q

what are complications patients with cleft lip and/or palate have

A

Higher risk for caries due to presence obturators, retainers, palatal expanders, and other orthodontic devices

Anatomic variation may cause difficulty with oral clearance and oral hygiene

Patients likely to undergo multiple surgeries at young age - resultant scar tissue may restrict oral opening and cause difficulty with oral hygiene procedures and dental restorative care

Patients likely to undergo multiple surgeries at a young age and may have learned aversion to healthcare providers

21
Q

who is part of treatment team for cleft lip and/or palate

A

Most successful treatment is from multidisciplinary healthcare team: Pediatrician, Geneticist, Plastic Surgeon, Oral Surgeon, Pediatric/General Dentist, Orthodontist, Audiologist, Speech Pathologist, and others

22
Q

who serves as dental home for patients and help to coordinate oral health care

A

pediatric/general dentist

23
Q

prevention is important in what patients

A

patients with congenitally missing teeth

because arch length and alveolar bone preservation critically important

24
Q

surgical management of cleft lip and/or palate at:

0-3 months

A

Pre-surgical infant orthopedics is used to narrow the cleft by reducing the gap in alveolar segments and lip (ex: Nasoalveolar molding, lip taping)

25
Q

surgical management of cleft lip and/or palate at:

3-6 months

A

Cheiloplasty (surgical lip closure)

26
Q

surgical management of cleft lip and/or palate at:

10-18 months

A

Palatoplasty (surgical palate closure)

27
Q

surgical management of cleft lip and/or palate at:

2-5 years

A

Possible lip and nose revision and/or second palate surgery for speech

28
Q

surgical management of cleft lip and/or palate at:

6-11 years

A

Possible alveolar bone graft surgery

29
Q

surgical management of cleft lip and/or palate at:

12-18 years

A

Possible lip revision, possible jaw surgery, possible septorhinoplasty (nose surgery)

30
Q

surgical management of cleft lip and/or palate at:

Adult

A

Final surgeries

31
Q

Crouzon syndrome is also called what

A

craniofacial dysostosis (Class III)

32
Q

what is a genetic syndrome with autosomal dominant pattern of inheritance with links to increased paternal age. pt has premature fusion of the sutures of the bones of the skull in particular the middle face and sockets

A

Crouzon syndrome

33
Q

what patient has:

wide-set bulging eyes, small beaked nose, and an underdeveloped maxilla (midface deficiency)

A

Crouzon syndrome

34
Q

what patient has:

constricted dental arches, Class III malocclusion, dental crowding, ectopic eruption of teeth in the maxilla, delayed tooth eruption, dental crossbites

A

Crouzon Syndrome

35
Q

Prevalence of Crouzon Syndrome

A

16 in 1 million births

36
Q

what is the results of hypoplasia of the mandible that occurs before the 9th week of development

A

Pierre Robin Sequence (Class II)

37
Q

Prevalence of Pierre Robin Sequence

A

4 in 100,000 births

38
Q

who has mandibular micrognathia, cleft palate, glossoptosis, feeding problems, respiratory compromise possibly requiring tracheostomay, and have concomitant Velocardiofacial

A

Pierre Robin Sequence

39
Q

what disease has heart defects, palate defects, facial defects, and learning disabilities

A

Velocardiogacial syndrome

40
Q

what are dental findings of Pierre Robin Sequence

A

Class II malocclusion, dental crowding, and limited opening

41
Q

what is an inherited congenital craniofacial condition, with unknown cause and prevalence of 1 in 25,000-50,000 births

A

Treacher Collins Syndrome

42
Q

what disease has mandibular micrognathia, cleft palate, external ear abnormalities, choanal atresia (nasal tissue blocking airway), eye problems, feeding problems, respiratory compromise possibly requiring tracheostomy

A

Treacher Collins Syndrome

43
Q

what disease has Class II malocclusion, 30% with concomitant cleft lip and/or palate, large fish-like mouth

A

Treacher Collins Syndrome