Wound Care Part 2 Flashcards

1
Q

what is HVPC?

A

twin peaked monophasic waveform with 2 single pulses having a short phase duration and long interpulse interval

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2
Q

in HVPC, each pulse is of short duration (typically <____usec) and a high peak voltage (up to ____V)

A

200, 500

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3
Q

evidence for application of HVPC mainly related to its use for what?

A

stimulation of wound healing, pain relief, and facilitated edema resolution

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4
Q

what are the physiological responses to ES?

A

increased blood flow

increased cell migration

increased collagen production

increased growth factor

bacteriostatic

decreased edema

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5
Q

what does bacteriostatic mean?

A

that it stops the bacteria from working

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6
Q

despite what Muskett tells us, can HVPC be used for ms strengthening?

A

no!!!

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7
Q

tensile strength of scars after HVPC are ____% better than wounds that healed naturally

A

50

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8
Q

what are the indications for HVPC for wounds?

A

stage 3-4 pressure ulcers

neuropathic foot ulcers that haven’t responded to standard care

LE ulcer due to poor blood supply (arterial insuffiency)

LE venous insufficient ulcers

wounds that haven’t responded to 30 days of standard care

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9
Q

what is “standard care” in wounds?

A

clean, moist, get rid of any nonviable tissue

can be done by the pt themself or the PT

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10
Q

what electrodes are used in HVPC for wound healing?

A

typically aluminum foil and gauze soaked in saline with an alligator clip to the foil cut to the size or just just larger than the wound

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11
Q

why is aluminum foil used in HVPC?

A

bc aluminum has little to no resistance, so the stim goes right to the wound

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12
Q

what are the HVPC parameters used during days 1-5 (or until the necrosis is removed)?

A

negative polarity

50-80pps

100-150 volts

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13
Q

what are the HVPC parameters used from day 4 to closure of the wound?

A

positive polarity

80-100pps

100-125 volts

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14
Q

if wound healing plateaus with HVPC, what should we do? why?

A

initiate negative polarity and alternate polarity daily bc negative is bacteriostatic/bactericidal and the plateau is likely due to bacterial growth

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15
Q

how often is HVPC done in inpatient?

A

45-60 minutes daily

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16
Q

how often is HVPC done in outpatient?

A

3x/week

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17
Q

what are the contraindications for HVCP for wounds?

A

basal or squamous cell CA

osteomyelitis

ion residues of iodine or silver

electronic pacing implants

directly over the heart or carotid sinus

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18
Q

what is the best diagnostic test for osteomyelitis?

A

radiographs

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19
Q

when a pt has osteomyelitis, what has to be done?

A

the infection has to be debrided then followed up with 6-8 weeks of antibiotics then either close or keep the wound open

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20
Q

what is negative pressure therapy?

A

negative pressure distributed over the wound surface by an airtight thin-film secondary dressing

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21
Q

what are the benefits of negative pressure therapy?

A

decreased interstitial edema

decreased bacterial count

increased capillary flow

moist wound healing

granulation tissue formation

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22
Q

t/f: once there is granulation tissue, you have to switch to white foam with negative pressure therapy

A

true

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23
Q

t/f: negative pressure therapy preps the wound bed for closure, it DOES NOT close the wound

A

true

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24
Q

what is the physiological response to negative pressure therapy?

A

increased cell proliferation by stretch activated ion channels

mechanical stree stimulates angiogenesis

increased blood flow and O2 delivery

removes edema fluid

decreased bacterial count

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25
Q

what are the indications for negative pressure therapy?

A

pressure ulcers stage 3-4

dehisced and traumatic wounds

diabetic, arterial insufficient, and venous insufficient ulcers

radiation ulcers

meshed grafts, flaps, and donor sites

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26
Q

is it preferred for negative pressure therapy to be continuous or pulsed? why?

A

continuous so that the pt doesn’t have to deal with the pain of the on/off suction

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27
Q

how long is negative pressure therapy?

A

22 hrs/day

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28
Q

what is the target pressure of negative pressure therapy?

A

it wound specific bw 50-125

ideally 50mmHg
most often 125 mmHg

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29
Q

how often should the dressing (sponge) be changed in negative pressure therapy normally? what if it is infected?

A

normally every 48-72 hrs

every 12-24 hrs if infected

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30
Q

t/f: we want the sponge in contact with all surfaces of the wound in negative pressure therapy, including tunneling and undermining

A

true

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31
Q

how long does it usually take to get the dressing set for negative pressure therapy?

A

~40 min

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32
Q

what are the limitations of negative pressure therapy?

A

fistulas to organs or body cavities

presence of >20% necrotic tissue

osteomyelitis

cancer in wound margins

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33
Q

how is negative pressure therapy applied?

A

sponge placed in wound bed

adherent cover dressing applied

suction attached

monitor the dressing adherence and type of drainage in the collection system

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34
Q

if there is necrotic tissue, with negative pressure we can put _____ over the necrotic tissue

A

mesh mat

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35
Q

what are the types of LE ulcers?

A

ischemic (arterial) ulcers

lymphatic ulcers

vasculitic ulcers

venous insufficiency ulcers

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36
Q

t/f: the likelihood of seeing lymphatic ulcers and vasculitic ulcers outside of a specialty wound clinic is low

A

true

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37
Q

_______ carry blood away from the heart to every fxning cell in the body, _______ carry blood to the heart

A

arteries, veins

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38
Q

t/f: we can’t treat the venous system without knowing what’s going on with the arterial system

A

true

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39
Q

when treating the vascular system, what should we also check?

A

the kidney fxn

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40
Q

how do we assess the CV system?

A

BP

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41
Q

how do we assess blood flow?

A

pulse palpation

capillary refill

rubor on dependency

ABI

Wells criteria for DVT

42
Q

when taking tissue cultures of a wound older than 60 days, what method are we using?

A

the 12 point swab method back and forth across the wound

43
Q

when taking tissue cultures of a wound older than 6 months, what method are we using?

44
Q

where are arterial insufficiency wounds usually found?

A

on the lateral ankle

on the webbed spaced of the toes

45
Q

t/f: there is no inflammatory stage for wound healing when a pt is on steroids (ie for RA)

46
Q

where are venous insufficiency wounds usually located?

A

on the medial ankle

47
Q

t/f: pain with wounds is dependent on the depth of the wound, with deeper being less painful bc there are no more exposed nerve endings

48
Q

what causes ischemic/arterial insufficiency wounds?

A

poor perfusion to the LE deprives the skin and tissue of oxygen, killing these tissues and causing an open wound to form

49
Q

what does an ischemic/arterial insufficiency wound look like?

A

pale, dry wound bed with well demarcated wound edges

50
Q

what causes pain with arterial insufficiency?

A

LE elevation

51
Q

what are the signs of poor perfusion?

A

cool skin

thick/thin, pale yellow nails

loss of leg hair

weak/absent pulses

hx of claudication and/or resting pain

52
Q

t/f: claudication is a true indicator that something vascular is wrong

53
Q

claudication pain is due to what?

A

decreased blood flow

54
Q

what causes claudication pain? what relieves it?

A

activity makes it worse
rest relieves it

55
Q

what causes venous insufficiency ulcers?

A

improper fxning of the venous valves (or incompitent perforators) of the legs

venous stasis

56
Q

______ insufficiency occurs in 70-90% of leg ulcer cases

57
Q

what is a venous stasis ulcer?

A

an ulcer caused by pooling of blood in veins that creates an inflammatory response in the body and cracks the veins leading to RBCs leaking out

58
Q

what is a classic signs of venous insufficiency caused by the pressure of pooling cracking vessels and leaking RBCs?

A

hemosiderin staining

59
Q

t/f: valves in the veins get more brittle with age and fluid stretching the vessel can cause backflow when the valves can’t overlap anymore

60
Q

what causes medial ankle ulcers?

A

venous insufficiency

61
Q

what do venous insufficiency wounds look like?

A

red, wet wound bed with irregular edges

heavy drainage

62
Q

what is the difference bw cellulitis and dermatitis?

A

dermatitis is inflammation of the dermis

cellulitis is inflammation of the cellular matrix surrounding the wound

63
Q

what is dermatitis a sign of?

A

venous insufficiency

64
Q

what can decrease venous insufficiency pain?

A

LE elevation

65
Q

what are the signs of venous insufficiency?

A

pitting edema

hemosiderin staining

varicosities

hx of DVT

66
Q

what factors in the Wells criteria increase risk of DVT?

A

LE trauma, surgery, or plaster

immobilization for >3 days or surgery within the last month

tenderness along the femoral or popliteal veins

entire limb swelling

non-varicose dilation of collateral superficial veins

calf >3cm bigger circumference 10 cm below tib tub

pitting edema

hx of confirmed DVT

malignancy

IV drug use

67
Q

what are vasculitic ulcers?

A

cutaneous vasculitis resulting from inflammation of superficial blood vessels

68
Q

where are vasculitic ulcers commonly located?

A

on the lateral side of the calf

69
Q

what are lymphatic ulcers?

A

ulcers that result when lymphedema causes damage to integument

70
Q

what do lymphatic ulcers look like?

A

small, oozing blisters or ulcer surrounded by fibrotic, thickened skin

71
Q

how do we assess limb and vascular status?

A

systemic BP

limb blood flow (ABI, pulses, capillary refill, rubor of dependency)

limb girth measurements

tissue cultures

lab values

72
Q

what locations should we measure with LE girth measurements?

A

at the met heads

arch

figure 8 ankle

10 cm above med mal

20 cm above med mal

73
Q

what are the chronic wound classifications (venous, arterial, traumatic, post-op wounds)?

A

partial thickness

full thickness

74
Q

what is a partial thickness wound?

A

breakdown of the epidermis and possibly penetrating into but not through the dermis

75
Q

what is a full thickness wound?

A

breakdown of the dermis into the subQ tissue through fascia, and may involve muscles, tendon, and/or bone

76
Q

what wounds are not classified as full or partial thickness?

A

pressure ulcers

diabetic foot ulcers

77
Q

what are adjunctive biophysical agents we can use for venous and arterial wounds?

A

compression

debridement

exercise and ambulation

dressings

non-contact low frequency US

78
Q

what is compression used for?

A

chronic VENOUS ambulatory hypertension

79
Q

what does compression do?

A

moves fluid from interstitial spaces through lymphatics

80
Q

what does the evidence say about elastic vs inelastic compression?

A

that elastic compression is better

81
Q

t/f: evidence suggests that there is probably a shorter time to complete healing venous leg ulcers in people wearing compression than those not wearing compression

82
Q

t/f: those using compression bandages/stockings are more likely to experience complete ulcer healing within 12 months compared with ppl with no compression

83
Q

what are the non stretch compression bandages?

A

gauze

paste bandage (unna boot)

84
Q

what are the types of compression?

A

non-stretch

short stretch

long stretch

compression stockings

hook and loop

intermittent/gradient sequential

pneumatic compression

85
Q

t/f: no bandaging/wrapping is necessary following compression

86
Q

what is the most ideal compression? why?

A

gradient sequential bc it mimics the natural muscles pump actions the best with most efficiency distally

87
Q

the goal is to not see >__cm increase in circumference girth measurements bw visits

88
Q

what is the first sign of chronic venous insufficiency?

A

aching legs

89
Q

what is the second sign of chronic venous insufficiency?

A

marks from socks

90
Q

how should we position a pt with compression?

A

in supine with the LEs elevated higher then the hip (higher than the heart is better)

91
Q

what are the indications for using compression?

A

venous insufficient ulcers

LE edema

lymphedema

92
Q

what are the limitations of compression (when would we not use compression)?

A

acute DVT

acute infection or untreated infection

acute CHF

arterial insufficiency

93
Q

t/f: if a wound is red, but not hot, it is likely chronically inflammed, not infected

94
Q

what is non-contact low frequency US?

A

ultra MIST device delivers low frequency 40kHz (no more no less) nonthermal ultrasonic energy w/o contract through fluid saline mist to the wound bed

95
Q

why is saline used in non-contact low frequency US?

A

bc energy travels faster through saline than through water or air

96
Q

how does non-contact low frequency US work?

A

the US energy creates a pressure wave that pushes on the cells below the wound surface that causes micro-strains that deform the cell membranes and create a physiologic responses resulting in promotion of healing

97
Q

does the 8 minute rule for billing apply to non-contact low frequency US?

98
Q

is 1mHz or 3mHz deeper US?

99
Q

how deep can non-contact low frequency US reach in tissues?

A

~9mm (enought to get through to subQ tissue)

100
Q

what are the benefits of non-contact low frequency US?

A

reduces a wide variety of bacteria (including biofilm!!!!)

reduces pro-inflammatory cytokines

stimulates angiogenesis through VEGF (vascular endothelial growth factor)

101
Q

all benefits of non-contact low frequency US occur within ____ days, so if there is still no response in this time frame, it is not the right tx for the pt

102
Q

as long as the pt is responding to non-contact low frequency US, how many sessions can we do?

A

up to 18 sessions at least 2x/week