wk 5 to midterm Flashcards
1-factors realted to longevity of tooth/restoration
2-prognosis/predictable longevity
3-mechanical failure
4-biological failure
5-clinical success
-perserve what remains before replacing
Least complicated to Most: all ceramic, single crowns, FPD—highest percentage of failure= ceramic tho
-time inc, restorations dec
-new products arent necessarily better
2-estimation of likelihood of a favorable outcome for disease
- good fair or poor
- prognosis for the disease & treatment
- how much cost, how long it last—Ans= it depends
3-material, denist, and patient
4-patient and dentist…could be due to time or recurrent caries
5-material, dentist, patient & tooth
1-material
2-clinical success
3-prognosis is compromised
4-methamphetamine
1–Level of technique sensitivity :silver amalgam= least technique sensitive = 92% survival
–sucesptibility to fatigue: resin-composite & cereamic inlays (for smiling not chewing)
Survival rate materials: Gold (91), amalgam (72),
composite (56)
2-have knowledge, skill, passion, comm, averison to risk…pay attention to detail
patient also= have oral hygiene, diet, systemic health, emds, drugs, resistance to disease & occlusal
3-compromised w/ bruxing patient
4-methamphetamine reduces salivary flow, excessive consumption of sodas & poor oral hygiene
1-focus on tooth for success rate
2-prognosis vs outcome
3-posterior resin restorations
4-length vs gingival health
5-pulpal health vs esthetics
6-structural integrity vs esthetics
7-contours vs gingival health
1-position in arch, occlusal road, amt of remaining tooth, perio support & endo therapy
—veneer= 30% tooth removal, crown= 72%, adhesive= 27% posterior
2-effectiveness of clinical parameters in developing accurate porgnosis
3-preventative measures by taking away minimal tooth structure & filling groove w/ resin & sealant for minor caries
4-retention, esthetics & prevention—dont violate bio width…extension for prevention of caries= extension for promotion of perio disease
5-assessment of periapical & clinical status of crowned teeth over 25 yrs
6-loss of tooth structure= inc tooth flexure= potential failure due to fatigue
7-embrasure form vs esthetics
1-mandible as a lever
2-summary
1-bite force= related to lenght of the lever arm between fulcrum & pt of resistance
- posterior locations= 8-9xs load of anterior locations
- antes law= combine peri-cemental area of abutment teeth =/greater than to pericemental area to be supplied—bridges that didnt follow this= low survival
2-patients have right to know prognosis of therapy
- EBD for restorative dentistry is less than adequate
- determining accurate prognosis= multiple factors: materials, tooth, dentist & patient
- imp properties of materials= level of technique sensitivity & susceptibility to failure due to fatigue
- most important dentist factor= attention to detail
- factor that most affects longevity of restoration= operator
- amt of tooth leftover= prognosis of tooth restoration complex
Endontic isolation
1-benefits
2-contraindications
3-precaution
4-frames
5-colors of dam
6-RD clamp
1-clean & visible field
- protexts patients from aspirating & protects dentist
- reduces cross contamination
- dec conversation & keeps tongue/soft tissue out of the way
2-patient w/ upper resp infection, congestion of nasal passage
3-dont obstruct airway (nose)…holes should be made if has upper resp infection
single tooth isolation= mark tooth before
-allergy to latex=latex free rubber dam
4-holds rubber damn
prevents damn from folding over isolated teeth
maintains tension in dam so lips & cheeks are retracted
5-darker colors= better visual contrast
lighter colors= illuminating operating field
6-anchors rubber damn to tooth, retracts gingiva
1-ISO color coding
2-reamer
3-file
4-k file
5-k flex file
6-flex r file
1-‘what you really blue gets better’
white, yellow, red, blue, green black
15-140= repeating colors
15-60= inc in inc of 5
60-140= inc in inc of 10
2-taper/pointed/spiral cutting edge
- enlarge root canals by rotary action
- sizing & finishing already drilled hole
- clockwise reaming action
3-tapered/pointed
spiral w/ cutting edges
enlarge root canal by rotation or filing action
4-square shaped blank—90degree—rasping/filing motion
5- rhombus blank—2 acute= sharpness & cutting efficience 2 obtuse= more space for debris removal & flexibility
5-non cutting tip, reducing ledge formation, canal transport
triangular crossection= flexibility in curved canals
-cut during counter clockwise rotation
1-hedstrom file-h file
2-broach
3-spreader
1-spiral as buttress threaded screw= cutting on pulling stroke
-remove old root fillins & to plane the canal walls…poor fracture resistance on rotation
2-tapered & pointed—thin flexible & fragile
-sharp projections to remove pulp tissue & cotton woll dressings
3-compress filling material against sides of canal to make room for additional gutta percha cones
1-therapeutic interventions
2-goals of periodontal therapy
3-zone 1
4-zone 2
5-zone 3
6-zone 4
1-systemic factors (smoking), plaque control
scaling/root planing, extraction
occlusal treatment
host modulation
flap surgery
2-to eliminate microbial etiolgy & risk factors fro periodontitis
- arresting progression of the disease
- perserving dentition
3-soft tissue pocket wall—determine morphologic features & persistence of inflammation
4-tooth surface—presence of alterations or deposits on cementum surface
5-underlying bone—establish shape & height &&&detects bony defects
6-attached gingiva—presence or absence of an adequate band
1-zone one—soft tissue pocket wall
2-zone two—tooth surface
3-zone 3—underlying bone
4-zone 4—attached gingiva
1-biofilm= subgingival—first line of resistance
-inflam mediators & cells aggregate in pocket to fight off insult & damage surrounding tissue
2-loss of attachment, diseased cementum= contaminated w/ endotoxin & calculus deposits
-cementum surface becomes more calcified after attachement loss
3-bone & defect morph affect outcomes of interventions
-surgical procedures are for predictability in pocket reduction/regeneration of lost tissue
4-presence or absence of adequate band attached gingiva
1-personal oral hygiene
2-scaling & root planing
3-resin blonded splint
4-occlusal adjustment
5-perio surgery
1-daily mechanical disruption of plaque biofilm…improves superficial gingival health
2-removes calcified deposits, bacterial products & biofilm from tooth surfaces
- effectiveness by pocket depth & operator skill
- outcome better by patient oral hygiene skills
3-economic way to stabilize tooth segments w/ compromised support
-use w/ advanced mobility only if patient has discomfort
4-reshaping of teeth to improve function
- dec interferences
- relieve perio involved teeth to allow for healing of perio ligament
5-perio surgery doesnt eliminate disease—but dec risk factors & maximizing prognosis factors
—long term health & success= dependent on compliance
1-CDMI treatment planning
2-phase 1
3-initial therapy
1-disease control & risk management-1
- reconstructive & restorative-2
- ongoing maintenance care & support-3
2-systemic—tobacco & glycemic
local factors—caries management, extractions, scaling, oral hygiene instruction
3-assess compliance
- are teeth restorable
- probe more once anesthetized
- clarify patient needs & preferences & get plan
1-materia alba
2-calculus
3-etiologic signifcany
4-plaque
1-food debris, dead cells & bacteria—plaque under it
2-mineralized bacterials plaque that forms on surfaces of deeth
- formed by deposition of Ca & P from saliva & fluid
- rough/porous surface for plaque & toxins to grow
- classify by composition & location
- either supra/sub gingival
3-positive correleation between calculus & gingivits
young= perio & plaque but older= perio & calculus
4-plaque= gingival inflammation = pocket formation= sheltered area for plaque & bacterials accum.
subgingival calc= product rather than cause of inflamm
1-supragingival calc
2-subgingival calc
3-inorganic comp
4-how does calc form
5-precip
6-epitactic nucleation
1-coronal to margin & visible in cavity
- white in color, hard consistency & easily detached
- color via food & tobacco
- generalized throughtout mouth or on just 1—esp lingual of mand incisor
- buccal of max molars & lingual of mand anterior
- saliva form parotid flows over facial of molars via stensens duct & whartons (subman) over lingual incisors
2-below margin & not visible found by instrument
- hard & dense & dark color —source is via crevicular fluid not saliva
- can make up supragingival calc
3-enamel>calc>dentin>bone>cementum
4-precipitation & epitatic nucleation (seeding)
5-rise in degree of saturation of Ca & P
rise in pH= precip of CaP by lowering constant
-calcification on inner surface of plaque—calc made in layers that are then separated by cuticle
6-conc of Ca & P in saliva & fluid isnt high enough to preciptate spontaneous—presence of matrix= essential for hydroxyapatite crystal
—sooo small foci of calcification enlarge & coalesce to make a mass
1-how does calc attach
2-morph
3-exceptions
4-supraging
5-subging
1-acquired pellicle
- irregularities
- directly to cementum
2-crusty, ledge, smooth, finger, islands
highly caclified interproximal can be seen in radiographs
3- not all plaque becomes calcified & plaque isnt necessary for calcification
4-minerals from saliva, near salivary ducts, whiteish
5-miners from sulcular fluids, by gingival inflam, brown & black color, found anywhere…higher Ca to P
