leftovers Flashcards

1
Q

1-obturation

2-endodontic obturation

3-how far into the canal

4-ideal obturant

A

1-use of material & techniques to fill entire root canal system in 3D manner

2-to prevent reinfection of root canals that have been clean, shaped, disinfected by instrumentation, irrigation & medication procedures

  • cant cleanse & shape but can isolate & seal
  • stops coronal leakage
  • blocks influx of H20 & nutrients
  • entombs surviving microbes

3-at termination of where you instrumented

4-easily introduced into canal

  • seal canal laterally & apically
  • not shrink after being inserted
  • radiopaque
  • bactericidal
  • not stain tooth
  • non toxic
  • sterile
  • easily removed
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2
Q

1-solid obturants

2-semisolid obturants

3-pastes

4-silver points

5-siglets

A

1-silver points

  • gutta percha
  • carrier based

2-pastes—
zinc oxide eugenol
plastics sargenti paste (N2)

3-lack of length control (overfills/underfills)
-unpredictable sealability—voids, shrinkage, & solubility

4-corresponds to last file size used in prep

  • *contraidicated**—corrodes spontaneously w/ serum & blood
  • –irreversible staining
  • –lack plasticity—doesnt conform to canal
  • –complicated apical surgery
  • –induce inflam root resorption

5-used to remove silver points

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3
Q

1-gutta percha

2-does gutta percha alone seal the canals

3-good stuff about GP

A

1-form of rubber
-A-form—natural (brittle)
-B-form—synthetic (root canal & malleable)
-easily introduced into canal—seals canal laterally & apically
PROS= plasticity, easy to manage, inert (low toxicity), & self sterilizing (doesnt promote growth of bacteria)
CONS= lack of adhesion to dentin (doesnt seal)
can rebound & pull away from canals

2-no

3-easily introduced into canal
radiopaque
bactericidal
not staining
non toxic
be sterile
easily removed

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4
Q

1-sealers

2-ZnOE based

3-Plastic Epoxy

4-Glass Ionomer

5-CaOH2

6-obturation technique

A

1-provides a fluid tight seal between tooth & core material
functions= antimicrobial agent
filling discrepancies between material & dentin walls
binding agent between filling material & dentin walls
lubricant
gives radiopacity

2-roth selaers
-standard…but doesnt bind to dentin & is slow sletting

3-binds to dentin but it stains & is solvent insolubility

4-binds to dentin but is too hard

5-is antimicrobial but doesnt have long term stability

6-cold compaction or warm compaction

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5
Q

1-lateral condensation

2-warm vertical condensation

A

1-preselected master gutta percha cone filled into canal to WL & accessory cones are in laterally w/ spreader until filled to CEJ
-fitted (snug/tug back) to WL
-spread inserted alongside of MC 1-2 mm short of WL
-accesory cone into space made by spreader
-spreader inserted until resistance is met (repeat until filled)
ADV= length control, easy to remove, & easy techniqe
DISADV= cant obturate irregularities & insertion cant generate forces—vertical root fracture

2-preselected master gutta percha is fitted into at WL

  • MC cone is seared off, compacted to make room for plasticized GP segments
  • –fit GP MC
  • –fit system B plugger
  • –seat sealer coated MC to WL
  • –sear of MC w/ system B, leaving 5 mm GP
  • –condense heated MC plug w/ a cold plugger
  • –condense heated MC plug w/ a cold plugger
  • –back fill the canal w/ GP expressed from the obtura
  • –GP back fill is condensed w/ cold plugger
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6
Q

verrucal papillary lesions

1-infections

2-reactive

3-idiopathic

A

1-squamous papilloma
verruca vulgaris
condyloma acuminatum
focal epithelial hyperplasia

2-inflammatory papillary hyperplasia

3-verruciform xanthoma
sebhorrheic keratosis

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7
Q

1-squamous papilloma

2-verruca vulgaris

3-condyloma acuminatum

A

1-benign proliferation caused by HPV 6 & 11

  • 20-50 yr old…equal gender
  • soft palate, tongue & lips
  • some pedunculated
  • koliocytes (clear cells w/ pyknotic)
  • tx w/ surgical excision

2-benign prolif caused by HPV 2, 4, 6, 40

  • skin & hands of kids
  • vermillion border, lips & tongue
  • cutaneous horn—accum of keratin on a wart
  • prolif of sqamous epi finger projections
  • tx= liquid N2, lactic acid, surgery

3-benign prolif epi caused by HPV 2, 6, 11, 53 & 54

  • STI—genital warts
  • 20% of sexually transmitted disease
  • mouth & larynx, genitalia & perianal
  • clustered lesion
  • labial mucosa, soft palate & tongue frenum
  • epi projections are broad & blunt
  • surgical excision
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8
Q

1-focal epithelial hyperplasia

2-inflammatory papillary hyperplasia

3-verruciform xanthoma

4-seborrheic keratosis

A

1-benign prolif caused by HPV 13 & 32

  • native americans & multiple members of family
  • poor, crowded living, poor hygiene
  • usually kids
  • labial, buccal, & lingual mucosa
  • multiple lesions, flat or papillary, usually clustered
  • tx= spontaneous regression—surgical
  • abrupt acanthosis of epi= koilocytes & mitosoid cells

2-bc of ill fitting denture, poor denture hygiene or always wearing denture

  • candida albicans?
  • usually in palatal vault
  • papillary growths hyperkeratosis inflammation
  • tx= removal of denture to help w/ early lesions
  • –antifungals help, excision for larger/older lesions

3-hyperplastic condition of mouth, skin & genitalia
-unknown etiology
-soft, painless, sessile mass w/ white & yellow/papillary surface
surgical excision

4-skin of face, trunk & extremities
patients >40
macules that become fissured= verrucous plaques
-no oral lesions
-stuck on appearance
-dermatosis papulosa nigra on black ppl
-no tx needed
-horn cysts= keratin filled invaginations

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9
Q

1-goals of perio therapy

2-intial therapy goals

3-summary of initial therapy goals

A

1-to alter/eliminate microbial etiology & contributing risk factors for perio…stopping progression of disease & perserving dentition in state of health, comfort & function w/ appropriate esthetics & prevent recurrence of perio
-regeneration of perio attachment apparatus where needed

2-arrest/attenuate progress of perio disease
induce positive changes in subg bacterial flora
resolving inflammation
create environ that permits gingival tissue to heal
improving/maintaining attachment level
dec pocket depth
prep tissues for surgical procedures

3-dec bioburden mass to tissues

  • minimize impact of systemic factors
  • eliminate/control local risk factors
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10
Q

1-dec bioburden challenge to tissues

2-minimize impact of systemic factors

3-elim/control local risk factors

A

1-intensive training of patient in appropriate tecniques for self-care & professional removal of calc deposits & bacterial products from tooth surface

  • calc deposits always are covered w/ living bacterial biofilms that are associated w/ continuing inflammation if not removed
  • facilitating oral hygiene procedures

2-certain systemic diseases can inc risk of periodontitis & severity—systemic & glycemic control

3-plaque development & retention

  • –oral hygiene (dec bioburden mass)
  • –calculus (maintains bioburden mass)
  • –overcontoured restorations (plaque rention)
  • occlusal trauma factors
  • local environmental risk factors that can inc risk of developing periodontitis in sites
  • plaque rention in a site allow damage over time to periodontium
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11
Q

1-anticipated outcomes w/ initial therapy

2-phase 1 therapy

3-initial therapy/hygenic phase

A

1-reducing/eliminating inflammation

  • reducing probing depths
  • improving clinical attachment

2-management of acute problems (perio abscess, NUG, endo-perio)

  • control of local etiology (plaque, 2ndary factors & occlusal forces)
  • –dec bacterial load & improve bacterial flora
  • prep teeth for biologically healthy root surfaces
  • minimize plaque harboring factors
  • re-eval 4-6 wks after completion of initial therapy

3-plaque removal/control
scaling/root planing—calc debridement
minimize plaque retentive areas (restorations)
selective extraction
adjust occlusal discrepancies
re-eval 4-6 wks

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12
Q

1-patients role

2-effects of oral hygiene

3-effects of oral hygiene by professional cleanings on subg

4-effects of personal oral hygiene on subg

5-effect of adv perio therapy w/o personal hygiene

6-why Scaling/root planing

A

1-cotherapist in treatment…train for self care

2-daily deplaquing will improve superficial gingival health (reduce inflammation)

  • toothbrushing can reach up to 1 mm subg, but can alter up to depth of 3 mm
  • remocal of supraging plaque= limited response
  • in pockets over 5 mm oral hygiene alone isnt effective in altering subg biofilm

3-dec # of spirochetes & bacteriodes after 3 wks of professional daily plaque control

4-no impact when probing depth >5 mm

5-ineffective

6-dec quantitiy of organisms below a critical mass & composition of remaining bacterial flora to one associated w/ health

  • removal of root surface elements (calc-plaque-endotoxin)
  • biologically acceptable root surface
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13
Q

1-microbial shift

2-scaling root planing

3-scaling

4-root planing

A

1-effected by removal of bacterial by scaling root planing ===smooth root surface, supports effective oral hygiene & removal of toxins

2-reduce contributing factors like calc & overhanging restorations

  • removal of calcified deposits, bacterial products (endotoxins) & biofilm from tooth surfaces & infected perio pockets
  • outcome enhanced by patient oral hygiene

3-process by which plaque & calc are removed from suprag & subg tooth surfaces

4-process by which residual embedded calc & cementum are removed from roots to produce smooth, hard, clean surface

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14
Q

1-alterations in exposed cementum

2-how much to root plane

3-outcome after phase 1 therapy

A

1-hypermineralized surface zone
changes in organic matrix
endotoxins cytotoxic in tissue culture

2-deposits of calc on root surfaces in cementum irregularities
scaling is insufficient bc some cemetum has to be removed (planing)
-bacterial toxins dont strongly adhere to diseased root surfaces so tooth smoothness isnt required, BUT is is a clinical indicator of calc removal available at present

3-reduction of clinical inflam (erythema, edema & bleeding)

  • prep of bio acceptable tooth surfaces
  • microbial shift to less pathogenic flora
  • reduction of probing depth
  • gain of clinical attachment
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15
Q

1-whats a template

2-trimming

3-used for

4-shades

5-prep

A

1-matrix

  • helps form lingual wall
  • determines incisal edge: position, length, thickness & shape
  • made of poly vinyl, silicone, or clear vacuform

2-trim mesio-distally
-no material overlapping facial surface

3-incisal edge restorations, diastema closure, or peg lateral build up

4-A1 Dentin & BL Light Enamel

5-bevel anterior w/ 2 mm bevel w/ an 8 bur

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16
Q

1-aim of pulp therapy

2-approach of pulp therapy

3-normal

4-reversible pulpitis

5-irreversible pulpitis

6-necrotic

7-clinical data

A

1-preserve primary teeth until normal exfoliation
maintain arch integrity & health of teeth/supporting tissues
-maintain vitality of tooth affected by caries or trauma

2-prelim data gathering & interpretation focused on determining health of pulp upon which available therapeutic options are chosen

3-symptom free & responsive to vitality testing

4-caries, cracks, restorative procedures, trauma may cause exaggerated response to thermal stimuli, diasspears on removal of stimuli

5-symptomatic/asymptomatic, spontaneous pain…exaggerated response to thermal tests that lingers on removing stimuli

6-death of pulp

7-hx of pain elicited from parent of care giver since children vary in response to reporting pain (major clinical tool)
—intentionally eliciting pain can scare the kid

17
Q

1-pain

2-clinical examination

3-soft tissue changes

4-palpation

5-percussion

6-mobility

A

1-presence/absence
spontaneous/provoked
intermittent/prolonged

2-visual
palpation
percussion
mobility
vitality testing

3-redness of buccal mucosa
drainage through thin buccal bone
sinus tract/parulis

4-fluctations felt by palpating…swollen mucobuccal fold
-bone destruction

5-bite stick response for kids
not recommended bc have resorbing roots/furcation involvements

6-normal exfoliation vs path mobility
compare w/ contralateral

18
Q

1- pulp testing

2-non vital pulp therapy

3-vital pulp therapy

4-indications for vital pulp therapy

A

1-heat test= unreliable…chances of pulp damage bc of highly placed pulp horns

  • *cold** test= reliable…intrapulpal pressure=indicatory
  • *EPT**= unreliable bc of undeveloped odontoblast nerve fibres of pulp & plexus of raschkow at pulp dentin border

2-pulpectomy, extraction & space maintainer

3-pulpotomy & indirect pulp therapy

4-large carious lesion close to pulp where exposure might result in caries removal

  • absence of soft tissue path…like sinus tract, path mobility & spontaneous pain
  • absence of radio changes like furcation involvement, resoprtion or PA pathology
  • restorable tooth
  • atleast 1/3 of root remaining
19
Q

1-prim vs perm tooth morph

2-vital pulp therapy for primary teeth

3-non vital pulp for young perm teeth

A

1-greater molar root curvature

  • inc # of accessory canals in floor of pulp chamber
  • large pulp relative to crown size, w/ pulp horns coser to tooth surface (mesial more than distal)
  • flat ribbon shaped canals

2-protective liner, indirect pulp treatment
direct pulp capping &&& pulpotomy

3-liner, indirect pulp treatment, direct pulp cap
partial pulpoptomy, apexo, pulpectomy & apexification

20
Q

1-pulpotomy

2-indications

3-contraindications

A

1-involves amputation of coronal pulp which has been affected by caries/ exposure
remaining vital radicular pulp tissue surface is treated w/ clinically successful medicament
maintain asymptomatic primary tooth until exfoliation
bc of speech, mastication & space maintenance

2-deep carious lesion w/ pulp exposure

  • absence of soft tissue path (sinus tract), path mobility & spontaneous pain
  • absence of radio changes of furcation radiolucency, internal/external resorption, PA path
  • 1/3 of root remaining

3-hx of spontaneous/ unprovoked pain

  • fistula.draining sinus
  • pulp hemorrhage
  • PA radiolucency
  • path resoprtion & mobility
21
Q

1-tx success based on

2-pulpotomy medicaments/agents

A

1-dx of vitality status of pulp

  • radiographic results
  • clinical assessment of teeth to make sure they’re healthy & reversibly inflamed

2-formocresol

  • *ferric sulfate**
  • *mineral trioxide aggregate**
22
Q

1-formocresol

2-ferric sulfate

3-MTA—mineral trioxide aggregate

A

1-19% diluted
-glycerine w/ H20 w/ buckleys
-treats pulp stumps & not agent for hemostasis
(get hemostasis by damp cotton pellets)
-formaldehyde makes it bactericidal & reversibly inhibits enzymes
-creates superficial layer of fixation while preserving vitality of deeper radicular pulp
—-apply: soak cotten pellet, blot pellet in dry gauze, place pellet over stumps & pack w/ dry pellet…leave for 4-5 min to stop bleeding & then remove

2-hemostatic agent= astringedent
—apply: following hemorrhage contorle, infuser is applied for 10-15 s, pulp chamber rinsed w. h20 & dried before placing ZOE

3-available as proroot as powder & then mix liquid
—-apply: powder w/ water on mixing pad
1/4th powder needed for 1 pulpotomy (can be reused), h20 cant be reused, place moist cotton pellet in there then thick mix of MTA over pulp stumps w. thickness of 2 mm. coat the MTA w/ RMGI so it gives compressive forces for restoration placement

23
Q

1- indirect pulp therapy

A

1-if has large carious lesion

  • some caries must be left behind in tooth to avoid exposure
  • dont reenter tooth to remove caries
  • remove superficial/peripheral caries w/ high speed & peripheral walls are cleaned, leaving caries over pulp
  • large slow round removes caries over pulp
  • once affected dentin is reached, stop there
  • affected dentin remineralizes if theres a seal, can leave 1-2 mm over pulp
  • infected dentin covered w/ base that ends on sound dentin & gives a seal over affected dentin left behing—remineralize & get harder
  • reinforced ZOE like IRM or RMGI recommended for IPT
  • tooth restored w/ stainless steel crown
24
Q

1-follow up

2-pulpotomy

3-access pulp chamber

4-restorations

A

1-take PA every 6 mo

  • look for uniform narrowing of canals & signs of resoprtion
  • gold: no change between pre-op & followup radiograph

2-removes coronal pulp, leaving healthy pulp tissue in root canals/radicular part
-minimize contamination by removing superficial caries & cleaning walls before exposing pulp

3-wide access opening makes it possible to visualize chamber & remove rissues down to orifices, removing ledges that hide pulpal tissue

  • cusps tips are ood for location & # of pulp horns
  • pulp must bleed when roof is removed= vital pulp
  • 1st =high speed = access opening & remove roof
  • 2nd=large slow round= remove pulp tissue
  • 3rd=small & moist cotton pellets= control hemorrhage & clean chamber—uncontroled= inflammation & vital pulpotomy is contraindicated
  • –pack ZOE against pulp stumps w/ damp pellet
  • –condense ZOE onto floor & then restore tooth w/ final restoration at same appt to avoid failure of pulpotomy

4-pulpotomized tooth has been weakened by caries & tooth removal= full coverage stainless steel crown used for protection & seal

25
Q
A