Welsh's Study Guide Aneurysms/PVD Flashcards

1
Q

What is PAD?

A

Peripheral Artery Disease. S/S Intermittent claudication. Pallor, no hair on lower extremeties, skin is shiny, sores that won’t heal and if it is bad enough you’ll have gangrenous tissue. Often the gangrenous tissue will be on the toes or between the toes.

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2
Q

What are the risk factors for PAD?

A

High blood pressure, smoking, high cholesterol, hypertension, diabetes, family history of PAD and obesity

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3
Q

What is atherosclerosis?

A

Hardening of the artieries. Happens when you get older. They are less flexible and have plaque build up.

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4
Q

What are the s/s of PAD

A

1

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5
Q

What is intermittent claudication? What relieves it?

A

Pain in the legs that is constant that happens when you do normal stuff. Remember, it is constant. It is NOT from working out hard. It is constant. IT IS RELIEVED BY REST.

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6
Q

What are the severe s/s of PAD.

A

1

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7
Q

What are the 6 P’s?

A

Pulse, pain, pallor, polikothermia, paralysis, parasthesia

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8
Q

What is the nursing diagnosis for Intermittent claudication and what are the goals?

A

Decreased tissue perfusion. Goal: cappilary refill < x seconds. THEY DO NOT CROSS LEGS OR WEAR TIGHT CLOTHING.

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9
Q

What are arterial ulcers? What are the s/s?

A

They are round with well defined borders. They are usually blue to black. They are painful. They are caused by arterial insufficiency. The tissue becomes necrotic.

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10
Q

What is angiography? What are the complications? What do you assess for after the procedure?

A

A catheter that is stuck into an area where dye is injected to help them diagnose problems. Complications include allergic reaction to dye, emboli, perforation, hemorrhaging or hematoma. After the procedure you’ll assess for temp, color, pulses.

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11
Q

What is the nonsurgical management for arterial ulcers?

A

Teach about positioning (do not cross legs, do not put feet up for long periods of time or keep them in one position), teach them to moderate exercise. Promote vasodilation. Talk to them about medication therapy. With arterial issues you want them to keep their feet BELOW THE HEART. (i.e. keep legs in a dependent position)

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12
Q

What drugs are used to treat arterial ulcers?

A

Vasodilators.

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13
Q

What is percutaneous transluminal angioplasty (PTA)?

A

It is when a baloon is used to try to open up an arterial occlusion. Sometimes they will try to use a stint to keep it open.

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14
Q

What is the surgical management for percutaneous transluminal angioplasty?

A

Arterial revascularization (a graft).

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15
Q

Where is outflow done? Complications?

A

Outflow is from below the femoral arteries. The grafts will be short grafts made from multiple vein segments. Complications include leaking,

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16
Q

Where is inflow done? Complications?

A

Inflow is above the femoral arteries. Big, long veins are normally used. Mr. Welsh said there are usually not many complications.

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17
Q

Post op care for PTA patients?

A

Deep Breething, Incentive Spirometer, Check pulse, temp, color, bp. If their bp drops suddenly it could be a bleeding issue. Teach patient to try not to move much. ALSO ASSESS SURGICAL SITE FOR INFECTION, BLEEDING, COMPARE THE LEGS FOR COLOR/TEMP, ETC., NOTIFY SURGEON IMMEDIATELY OF ANY BLEEDING AND ASSESS FOR PAIN! (WHAT KIND OF PAIN IT FEELS LIKE- SHARP RIPPING PAIN IS BAD. THROBBING MIGHT BE NORMAL)

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18
Q

What is arterial occlusion? What is the treatment?

A

Blocked artery. Treated with anticoagulants and surgery. This is an emergency situation!!

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19
Q

What is Buerger’s disease? S/S? Cure?

A

Inflammation of small arteries with thrombosis. Basically, the small arteries have a clot. Often occurs in smokers. S/S include: coolness, numbness, tingling. Usually found in men ages 20-35. It is exasterbated by cold, stress and nicotine. Diminished pulses in the affected areas. No cure- just treat symptoms.

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20
Q

What is Raynaud’s? S/S? Treatment? Teaching?

A

Vasospasms to the upper arteries. Common in the hands. S/S: Coldness, pain, pallor, “patriotic color changes” (i.e. red, white, blue). Cold, nicotine and stress exasterbate it. Teach patient to stay warm, reduce stress and not to smoke. This will be on the test** (especially stress management as part of the teaching)

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21
Q

What is PVD?

A

Peripheral Vascular Disease. Happens on the venous side.

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22
Q

Risk factors for PVD?

A

Risk factors include: smoking, obesity, family history of PVD, high cholesterol, High BP and all the same things as PAD.

23
Q

What are varicose veins? Risk factors? S/S? Treatment?

A

These are dilated, tortuous(?) veins. Usually caused by a weakness in the valve or vein structure. Risk factors include: Obesity, Pregnant women, Folks who have long periods of standing. S/S include: dilated, purple, bulging veins. Treatment includes: prevention, surgical stripping (removal of valve or whole vein),

24
Q

What is thrombophlebitis? Causes? Treatment?

A

Inflammation in the vein (usually caused by an IV). The area becomes warm and reddened. Edema can occur. Treatment includes removing the IV, elevate the area, moist, warm compress.

25
Q

What is a venous thrombosis?

A

Blood clot in the vein.

26
Q

What are the risk factors for venous thrombosis?

A

People on bed rest, people older than 40 who have had surgery (especially folks who have had immobilization surgery like having a cast, having a knee replacement, femur fracture, etc)

27
Q

S/S of deep vein thrombosis? S/S of superficial vein?

A

Deep: Edema, warmth/tenderness at site, positive Homan’s sign. Superficial: Pain, redness, warmth/tenderness.

28
Q

How are deep vein/superficial vein diagnosed?

A

Ultrasound of legs, chest x-ray, possibly a CT scan.

29
Q

Treatment for Deep vein/superficial vein thrombosis?

A

Anticoagulants, Elevate, bed rest, activity as tolerated (if they don’t become active they could get another one).

30
Q

DVT S/S? Prevention? Complication?

A

Positive Homan’s Sign, unilateral leg edema, pain, redness, warmth, fever (and, if it’s in the lower extremity they could have calf tenderness). Prevention: Early immobilization, TED hose or SCD’s. The biggest complication would be a PE.

31
Q

Nonsurgical Treatment for DVT?

A

Anticoagulants, antithrombolytics, elevate leg, encourage ambulation when tolerated.

32
Q

Surgical Treatment for DVT?

A

Thrombectomy. IVC filters

33
Q

What is a PE? S/S?

A

Pulmonary Embolism (a clot has broken loose, usually from a DVT but sometimes it can be a fat or air embolus). S/S include: Dyspnea, impending sense of doom, 02 sats low

34
Q

What are the complications of a massive PE?

A

1

35
Q

Priority Care for a PE?

A

Airway! Raise the head of the bed, put oxygen on them. Get them to where they can breathe?

36
Q

What is the treatment for PE?

A

Prevention (if they have DVT’s they’ll probably get a filter), anticoagulants, bed rest, elevate head, monitor vitals and assess respiratory system. Anticoagulants will be given.

37
Q

How does Heparin therapy help? What labs need to be monitored?

A

Heparin thins the blood BUT REMEMBER ANTICOAGULANTS DO NOT BUST UP THE CLOTS. They just stop them from getting bigger. Labs: PTT

38
Q

What are the complications?

A

Hemorrhage, bleeding.

39
Q

What is the antidote to Heparin?

A

Protamine Sulfate. This will be on the test.

40
Q

What is Lovenox? How is it given? Air bubble?

A

LMWH. It is a low molecular weight heparin that is given subcutaneously. (Heparin is given subcu or IV). You push any air bubbles. You do not get rid of them. This ensures they get ALL of the medicine. He said to remember this.

41
Q

What is Coumadin?

A

Warfarin. Blood thinner. Also used as rat poison. It is theraputic 3-4 days. Heparin and Coumadin can be given together until it becomes therapeutic (then you’ll stop the heparin) Coumadin inhibits clotting factors.

42
Q

Complications from Coumadin?

A

Bruising and hematomas come easy. Wounds take longer to clot. You’ll monitor PTINR. Keep an eye on H&H. People on coumadin are at a higher risk for bleeding.

43
Q

Teaching for Coumadin?

A

Wear a medical alert bracelet. LIMIT VITAMIN K FOODS, Monitor for bleeding.

44
Q

What is the antidote for Coumadin?

A

Vitamin K

45
Q

What is a CVI?

A

Chronic venous insufficiency. Caused by destruction of the valves.

46
Q

S/S of CVI?

A

Edema (usually in lower legs), increased pain, STATUS DERMATITIS (brown pigmentation), Redness, Venous ulcerations.

47
Q

Complications of CVI?

A

Cellulitis and infection

48
Q

What are venous ulcerations? S/S? Treatment? **this answer is not complete. Add to it.

A

Ulcerations caused by venous insufficiency. S/S: irregular border, superficial, pink wound bed.

49
Q

What is an AAA? When is it treated surgically?

A

Abdominal Aortic Aneurysm. It is a weakness in the aortic wall (it pooches out). It is not treated surgically until it is about 5-6 cm.

50
Q

S/S of an AAA? Complications?

A

S/S: PULSATING MASS, sudden severe pain. Complications: rupture. S/S of a rupture, pallor, tachycardia, low bp. Treatment: Graft.

51
Q

What is an aortic dissection? Complications?

A

It is when the aorta splits open. Complications: Instant death.

52
Q

Post op care/assessment for AAA repair?

A

Watch for hemorrhage, vitals, watch for infection, loc, temp, wbc count, wound care, keep everything clean!

53
Q

D/C teaching for AAA repair?

A

Avoid heavy lifting for 4-6 weeks, gradually increase activities, have them watch for infection and fever, have them inspect wound site, have them watch color. They will be tired, have decreased appetite and irregular bowel habits. Teach them that the repair can cause sexual dysfunction.

54
Q

Study specifically

A

3, 5, 8, 9, 10, 18, 20, 30, 33, 37, 38, 39, 40, 41, 42, 43, 45, 46, 48, 52. Study the difference between venous and arterial! He stressed this several times.