Week Three Flashcards

1
Q

How are the cerebellum and basal ganglia involved in speech movements?

A
  • They both have bidirectional communication with the cerebral cortex
  • Help with motor control
  • Damage = errors in movement execution
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2
Q

Where does a lesion have to occur for ataxic dysarthria to occur?

A

In the cerebellum

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3
Q

What purpose does the cerebellum have in speech?

A
  • refinement
  • tempo/ rate
  • timing
  • steadiness
  • combining
  • sequencing movements
  • ensures smooth, well-timed, durationally and prosodically appropriate speech utterances
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4
Q

What are some causes of ataxic dysarthria

A
  • Degenerative disease (e.g., cerebellar degeneration)
    - Friedreich’s ataxia
  • Demyelinating (MS)
  • Stroke
  • Trauma
  • Tumour
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5
Q

What are some non-speech signs of ataxic dysarthria?

A

Ataxia
- can’t create smooth, fluid motions

Distrubed posture and gait
- wide stance when walking

Tremor
- Intention tremor

Dysdiadochokinesia
- Difficulty preforming rapidly alternating movements

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6
Q

What are the speech signs of ataxia dysarthria?

A
  • Most difficulty with articulation and prosody

Articulation
- imprecise consonants
- slurred
- Slow AMR’s with distortion (incorrect articulation /pa/ - /fa/) and irregular

Prosody
- Excess and equal stress (a little robotic
- Slow

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7
Q

What purpose does the basal ganglia have in speech?

A

Important in inhibition and excitation

Dopamine plays an important role in activating movements

Any imbalance in dopamine = degraded motor control performance

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8
Q

Where does a lesion occur to result in hypokinetic dysarthria?

A

Lesion occurs in the basal ganglia

Results in too much inhibition and too little excitation

= reduced mobility (hpokinesia) / too little movement

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9
Q

What are some causes of hypokinetic dysarthria?

A
  • Main cause is Parkinson’s Disease (occurs in 70-90% of PD cases)
  • Other causes include:
  • other degenerative diseases
  • Vascular
  • Traumatic
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10
Q

What are some non-speech signs of Parkinson’s disease?

A

Akinesia/hypokinesia (absence of/reduced movement)
- smaller movements
- Less spontaneous movements (bracing when fall)
- Reduction of automatic movements (arm swinging)
- mask-like facial expression

Tremor at rest

Slow to initiate and execute movement (bradykinesia)

Movement may to hard to stop (speedtrap)

Cognitive decline

Dementia

Depression

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11
Q

What are some speech characteristics of hypokinetic dysarthria?

A

Phonation/respiration
- reduced loudness
- breathy
- monopitch
- lack of stress

Articulation
- imprecise
- ARM’s very rapid and repeated syllables
fast

presenting complaints
- rapid
-mumbling
- difficulty initiating
- reduced loudness
- May very sensory deficits (lack of awareness)

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12
Q

Where does a lesion occur to result in hyperkinetic dysarthria?

A

Occurs in the basal ganglia

Increase of dopamine release

Too little inhibition and too much excitation

= excessive/involuntary/unwanted movements = hyperkinesia

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13
Q

What are some causes of hyperkinetic dysarthria?

A
  • Huntington’s disease
  • infectious
  • vascular
  • Trauma
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14
Q

What are some non-speech signs of hyperkinetic dysarthria?

A
  • Myoclonic jerks (sudden jerking of muscles)
  • Chorea (=dance)
  • Tremor (during action)
  • Dystonia (sudden and increased muscle contractions)
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15
Q

What are some speech characteristics of hyperkinetic dysarthria?

A

Phonation
Strained/breathy irregular
variation in loudness
voice stoppages

Resonance
nasal

Articulation

difficulty starting

Movements/muscle contraction
-Abnormal, irregular, unpredictable

Presenting complaints
- Inability to control or inhibit movements
- Shaky and tight voice
- Slurred and slow speech

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16
Q

Where does damage occur for apraxia of speech to occur?

A
  • Many different areas
  • Mostly left hemisphere
17
Q

What are the common causes of apraxia of speech?

A
  • most commonly Stroke
  • sometimes: tumour, trauma or degenerative diseases.
18
Q

what is apraxia of speech?

A
  • motor plan cannot be retrieved
  • impaired ability to correctly position the articulators
  • often have groping
  • need to construction new commands for each sound/syllable
19
Q

Can you have AOS and aphasia

A

95% of people with AOS also have aphasia (usually broca’s)

20
Q

What is non-verbal oral apraxia?

A

deficit in ability to sequence nonverbal, voluntary movements of the oral structures

  • can co-occur with AOS and aphasia
  • Doesn’t effect spontaneous of reflexive oral movements (cannot cough by command, but can reflexively)
  • limb apraxia is also a thing
21
Q

What are some speech characteristics of AOS?

A
  • primarily affects articulation and prosody
  • visible groping and can’t position the articulators
  • aware of mistakes and try to correct
  • slow rate
  • equal stress

Articultion errors:
-substitutions
- additios
- sound prolongations
- vowel sound prolongation