Week 6 Cardiac Flashcards
Three layers of the heart
Endocardium
Myocardium
Epicardium
4 chambers of the heart
Right atrium
Left atrium
Right ventricle
Left ventricle
Atrioventricular valves include
tricuspid- right
Mitral- left
Aortic and Pulmonic are
Semilunar Valves
Heart has own circulation
Coronary arteries
Heart and electricity
Hemodynamics and CArdiac conduction
Order of the valves with the blood flow
TPMA
Tricuspid
Palpating
Mitral
Artery
Self stimulating cells located along
Autorhythmic cells
SA node- 60-100
Internodal pathways
Causes electrical stimulation of atrium
Connection between SA node and AV node
AV Node
Secondary pacemaker
40-60
Acts as relay delay to coordinate
contractions between atrium and ventricles filing
Bundle of HIS
Atrioventricular Bundle
Purkinje Fibers
Causes electrical stimulation
30-40 bpm
use if both av and sa are malfunctioned
Pressures within the right side of the heart and pulmonary vascular system are
Significantly lower than left side of heart and systemic vascular system
Where does venous circulation accumulates?
Coronary Sinus
Then dumps back into the right atrium
Ventricular contraction creates what type of blood flow?
Upward
through venous coronary circulation
Blood that has been pushed into coronary circulation and is allowed to circulate during diastole
Atrial Systole and Ventricular Systole
Management of CAD
MONA
Morphine
Oxygen
Nitroglycerin
Aspirin
STEMI
ST elevation
Troponin is specific to cardiac muscle damage
Dialysis pt can have slightly elevated
Troponin due to inability to filter proteins
View EKG
EKG first
O2
Aspirin
Morphine
Average SV is
60-130ml
Preload
Volume of blood within the ventricles at the end of diastole, immediately prior to systole
Afterload
Resistance of systemic or pulmonary blood pressure; Systemic VAscular resistance- left side of the heart
Pulmonary Vascular Resistance
Right side of the heart
Preload and SV have a ______________ relationship
Direct
Afterload and SV have a __________________ relationship
Inverse
Contractility and SV have a direct relationship
Increased by ionotropic drugs and decreased by beta adrenergic blockers
SV
Stroke Volume
Amount of blood ejected with each heartbeat
Preload
Degree of stretch of cardiac muscle fibers at end of diastole- how much blood fills the ventricles
Afterload
Resistance to ejection of blood out of ventricles into arteries -diameter or arteries
Contractility
Ability of myocardium to contract in response to electrical conduction impulse
Ejection Fraction
Percent of blood that is ejected with each heartbeat
CO
Cardiac Output
Amount of blood pumped by ventricles in liters per minute
CO=SVxHR
Normal EFis
55% to 65%
CO at rest is
4-6 l/min
HR direct relationship with CO
Control of heart rate
Autonomic Nervous System
Sympathetic innervation of
Beta 1 receptors- increases hr
Parasympathetic Innervation of
Stimulation of Vagus nerve which SLOWS HR
Baroreceptors
Located in the Aortic Arch and bilateral Internal Carotid artery sinuses
Elevated BP
Increases parasympathetic activity
Lowered BP enhances activity
Sympathetic Activity
HTN sends impulse to the
Cerebral Medulla to activate parasympathetic activity and lower HR
Hypotension causes
Decreased amount of signals so that sympathetic responses are enhanced to increase HR and vasoconstriction
The volume of blood in the heart just before the ventricles begin to contract
Preload
Low BP baroreceptors trigger
Sympathetic nerve fibers to increase HR and vasoconstrict by release of catecholamines
Increases Beta adrenergic receptors
High BP baroreceptors trigger
Parasympathetic nerve fibers to decrease HR- vagus nerve and vasodilate
Decrease alpha agonist
Preload is compared with what law
Frank Starling Law
Afterload affected
By diameter of pulmonary artery and systemic arteries
Greater stretch of cardiac muscle will cause a greater degree of shortening - stronger contraction and increased stroke volume
Frank Starling
Preload
Degree of stretch on ventricular cardiac muscle = Left Ventricular End Diastolic Pressure
Resistance to eject blood out of ventricles
Afterload
Afterload increases….
Stroke volume decreases and lowers CO
Increased by catecholamines
Epi and Norepi
Contractility
Contractility influencing factors
Increased by sympathetic Nervous System
Increased by meds
Decreased by hypoxemia and acidosis
Decreased by some meds
If LVEF is or less the patient has what?
40%
Has left ventricular dysfunction and requires tx of HF
Measured by use of echocardiogram
Nursing considerations for Cardiac
Cardiac Assessment
Investigative Health History
Clinical Manifestations
Lab tests
Diagnostics Tests
Cardiac Assessment
Inspection
Palpitation
Auscultation
Inspection of cardiac
General appearance
Skin, nails, and mucous membranes
Anterior chest wall
JVD
Heart Rhythm on monitor
Palpation
Pulses
Cap Refill
Skin Turgor
Warmth
Moisture
Edema
Non pitting edema due to
Lymphatic disorders and obstruction
Pitting Edema
Increased hydrostatic pressure
Excess Fluid
Decreased Oncotic Pressure
Edema grading
0= no edema
1+= Slight, 2mm
2+= Mild edema, 4mm indentation, disappears in 10-25 seconds
3+= Moderate edema, 4-6mm, disappears over a minute
4+= Severe edema, disappears after several minutes
Relaxation of all 4 chambers
Diastole
Systole
Contraction of atrium just prior to ventricles