Week 5 - Pain Management Flashcards

1
Q

What is Pain?

A
  • an unpleasant, subjective sensory & emotional experience associated with actual or potential tissue damage or described in terms of such damage
  • a sensory & emotional experience of discomfort
  • Pain is “whatever and whenever the person says it is” (McCaffery)
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2
Q

Magnitude of the Pain Problem

A
  • Unrelieved persistent pain is an epidemic (in North America)
  • Pain-related disabilities account for >15% of total disabilities in Canada
  • Prevalence of pain increases with age. (Up to 85% of older people in
    LTC experience chronic pain)
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3
Q

Factors Influencing Pain Perception

A

1) Affective - emotions, suffering

2) Behavioural - behavioural responses

3) Cognitive - beliefs, attitudes, evaluations, goals

4) Sensory - pain perception

5) Physiological - transmission of nociceptive stimuli

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4
Q

Physiological Dimension of Pain

A

1) Transduction
1. Noxious stimuli causes cell damage with the release of sensitizing chemicals
- prostaglandins
- Bradykinin
- Serotonin
- Histamine
2. These substances activate nociceptors and lead to generation of action potential

2) Transmission: action potential continues from:
1. Site of Injury
2. Spinal cord to brainstem and thalamus
3. Thalamus to cortex for processing

3) Perception
- conscious experience of pain

4) Modulation
- neurons originating in the brainstem descend to the spinal cord and release substances (ie. endogenous opioids) that inhibit nociceptive impulses

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5
Q

Gate Control Theory

A

Large Nerve Fibers
- send brain information (ie. about being touched or feeling pressure on your skin)
- sends messages to your brain very quickly
- Stimulating large fiber activity can help “close the gates” by getting to your brain first

Small Nerve Fibers
- send your brain information about pain, temperature
- activity moves a bit more slowly
- Pain signals (carried by small fibers) are not allowed in = alters perception of pain

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6
Q

Acute Pain

A
  • Suden onset
  • Identifiable cause (injury, disease, surgery, psychological distress)
  • Short-lived, as recovery occurs
  • Usually treatable or will resolve on its own
  • S & reflect sympathetic NS stimulation
  • Vital signs: Increase pulse, RR, BP
  • Integumentary: Diaphoresis, pallor
  • Psychological: Anxiety, confusion
  • Goal: pain control with eventual resolution
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7
Q

Persistent (Chronic Pain)

A
  • Gradual or sudden
  • Cause not always known
  • Long-term (beyond expected healing time)
  • Unpredictable- constant or episodic
  • Predominantly behaviour S & S
  • Change in affect
  • Fatigue
  • Withdrawal from social interaction
  • Decrease physical activity
  • Goal: minimize pain, enhance function & QoL
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8
Q

Nociceptive Pain

A
  • Normal processing of stimuli that damages normal tissues or has
    potential to do so

2 Types
1) Somatic Pain- aching or throbbing from bone, joint, muscle, skin,
connective tissue
- responds to nonopioid & opioids

2) Visceral Pain-pain arising from internal
organs

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9
Q

Neuropathic Pain

A
  • Caused by damage to nerve cells or changes in CNS, abnormal processing
    of stimuli
  • Burning, shooting, stabbing
  • Often sudden, intense, lingering or short-lived
  • Central or peripheral origin
  • Common cause: inflammation, trauma, metabolic disease (diabetes)
  • Doesn’t respond well to opioids alone, requires adjuvant analgesics
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10
Q

Nursing Assessment - Determine client’s perspective of pain:

A
  • History of Pain
  • Meaning of pain
  • Physical Effects
  • Emotional Effects
  • Social Effects
  • Client Expectations
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11
Q

Nursing Assessment - OPQRSTUV & OLD CARTSS

A

OPQRSTUV
- Onset
- Provoking/palliating
- Quality
- Region of body, radiation
- Severity
- Timing, treatment
- Understanding of the pain
- Value- what level of pain is acceptable

OLD CARTSS
- Onset, including past history, what was happening at time of onset
- Location, including radiation
- Duration
- Characteristics
- Aggravating factors
- Relieving factors
- Timing
- Severity
- Self-perception (understanding), acceptable level

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12
Q

Pain Scales

A

Coloured Faces
- When using the faces, children might just pick the happy face because they like it

Faces
- Works for ppl who do not speak English, have dysphagia etc

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13
Q

Pain Management

A

1) Patient &
caregiver teaching
vital, especially
self-management

2) Pharmacological +
non-pharmacological
approach required

3) Treatment is
based on patient’s
& family’s goals

4) Team approach patient family, HCT

5) Use self-reports
when possible.
- Use non-verbal
assessments PRN

6) Routine Assessment - physical & psychosocial

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14
Q

Pharmacological Management

A
  • Equianalgesic dose = dose of one analgesic that produces pain-relieving
    effects equivalent to those of another analgesic
  • Important when substituting one analgesic for another due to ineffectiveness, intolerable side effects or route of opioid administration is changed
  • Fast-acting medications should be used for breakthrough pain, long acting analgesics are more effective for constant pain (ie. cancer, chronic)
  • Schedule to prevent or control pain rather than waiting until pain is
    moderate to severe
  • Administer medication before procedures or activities that are
    expected to produce pain
  • Patient with constant pain should receive analgesics around the clock
  • Rationale: control pain before it starts, usually result in lower analgesic
    requirements
  • Titration- adjusting dose up or down
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15
Q

Routes of Analgesic Administration

A

Variety of routes allowing for:

  • Targeting of a particular anatomical source of pain
  • Achievement of therapeutic blood levels rapidly
  • Avoidance of some adverse effects through localized administration
  • Provide analgesia if unable to swallow
  • Oral
  • Sublingual and buccal
  • Intranasal
  • Rectal
  • Transdermal
  • Parenteral
  • Intraspinal
  • PCA
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16
Q

The Analgesic Ladder

17
Q

Mild Pain (1-3/10)

A
  • Nonopioid analgesics (ASA, NSAIDs, Acetaminophen)
  • Ceiling effect- increasing dose beyond upper limit does not decrease
    pain
  • No tolerance or physical dependence
  • Side effects: GI upset, bleeding (ASA, NSAIDs), nephrotoxicity (NSAIDs
    chronic use), liver toxicity (Acetaminophen- >4000 mg/day or if liver disease present)
18
Q

Mild to Moderate pain (4-6/10)

A
  • Opioid + NSAIDs + adjuvant
  • Codeine, Tramadol- may have Acetaminophen added (Tylenol #3,
    Tramacet)
  • Opioids depress CNS
  • Side effects: nausea, constipation, dizziness, sedation
  • Pay attention to dose of Acetaminophen if using opioid with Acetaminophen in it.
    (Max dose Acetaminophen = 4000 mg/24 hours)
19
Q

Moderate to Severe Pain (4-10/10)

A
  • Moderate to severe pain or when step 2 meds ineffective
  • Morphine commonly used, fentanyl, hydrophone (Dilaudid), methadone,
    oxycodone
  • Long-acting Morphine (MS Contin) for continuous, round-the-clock therapy
    for extended period
  • Side effects: CNS depressant- nausea, vomiting, constipation, respiratory depression, sedation, itching, confusion, hallucinations, urinary retention
    less common
  • Most s.e. decrease with continued use (except constipation)
  • NO Meperidine (Demerol)- not responsive to Naloxone, high incidence of neurotoxicity (seizures)
20
Q

What other meds should be ordered for patient receiving opioids?

A
  • Anti-emetic
  • Stool softener/laxative
21
Q

Adjuvants (Coanalgesics)

A
  • Enhance effects of opioids & nonopioids
  • Possess analgesic properties
  • Counteract adverse effects of other analgesics

Includes
- Corticosteroids (anti-inflammatory; to reduce pain)
- Antidepressants
- Antiseizure
- Muscle relaxant
- Anaesthetics
- Psychostimulants
- Cannabinoids

22
Q

Age-Related Considerations

A

Older adults

  • Metabolize meds more slowly, at greater risk for higher blood levels &
    adverse effects
  • NSAIDs associated with higher risk of GI bleeds
  • Polypharmacy: greater risk for drug interactions
  • Cognitive impairment, ataxia increase when opioids, antidepressants,
    anticonvulsants used

Children
- can not metabolize drugs as well

23
Q

If you give a patient with cognitive impairment (dementia) an opiod, what are the risks?

A
  • opioids make people confused - difficult to know if the confusion is their dementia or a side effect of the drug
  • opioids can increase the fall risk of dementia patients who already have high risk of falls
24
Q

Tolerance

A

Tolerance: need for an increased dose of opiod to maintain the same degree of analgesia

Tolerance Management
- increased dose
- change to another drug in same class
- add medication from a different class to augment pain relief

25
Q

Tolerance VS. Addiction

A
  • Physical dependence= an expected physiological response to ongoing exposure
    to pharmacological agents
  • Manifested by a withdrawal syndrome when the drug is abruptly decreased
  • Slowly wean (decrease dose) to avoid withdrawal symptoms
  • Addiction (Substance misuse or substance use disorders)- neurobiological
    conditions due to use of a substance in high doses or inappropriate situations
    that can impact one’s health or create social problems
26
Q

Non-pharmacological Methods of Pain Management: Physical Interventions

A
  • Massage- works on principle that large fibers are stimulated, closing pain gate
  • Heat & cold application
  • Acupuncture
  • Tens
  • Exercise
27
Q

Non-pharmacological Methods of Pain Management: Behavioral/Cognitive Interventions

A
  • Provide sense of control, coping skills
  • Relaxation and imagery
  • Hypnosis - causes relaxation
  • Distraction, reframing
  • Patient education
  • Psychotherapy
  • Support Groups
28
Q

Opioid Crisis and Role of Nurse

A
  • use non-pharmacological interventions
  • think about care biases
  • provide health teaching (so clients are aware of risks)