Week 11 - Cardiac Health Flashcards

1
Q

Blood Flow through the Heart

A

Body → Lungs

Right
1. SVC/IVC
2. Right Atrium
3. Tricuspid valve
4. Right ventricle
5. Pulmonary Valve
6. Pulmonary Artery

Left
1. Pulmonary Veins
2. Left Atrium
3. Mitral Valve
4. Left Ventricle
5. Aortic Valve
6. Aorta

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2
Q

Electrical Conduction through the Heart

A
  1. Sinoatrial Node
  2. Atrioventricular Node
  3. Bundle of His
  4. R/L Bundle Branches
  5. Purkinje Fibres
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3
Q

Coronary Vasculature (Arterial Supply to the Heart)

A

1) Right Coronary Artery (RCA)
2) Left Coronary Artery (LCA)
→Left Circumflex Artery (LCx)

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4
Q

Perfusion

A

Perfusion: flow of blood through circulatory system to oxygenate cells
& remove waste

Perfusion depends on:
→Sufficient cardiac output
→Sufficient blood pressure to move blood to periphery
→Complementary action of adequate coagulation to prevent hemorrhagic blood loss

  • Blood pressure influenced by CO (amount of blood pumped, peripheral
    vascular resistance)
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5
Q

Cardiac Output (CO)

A
  • Cardiac Output relies on: 1) Heart
    Rate & 2) Stroke Volume

Heart Rate: frequency with which heart pumps, based on electrical stimulation

Stroke volume: volume of blood in the heart when it pumps

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6
Q

Factors impacting CO

A

1) nervous system
2) fluid volume
3) heart muscle contraction
4) resistance in vessels

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7
Q

Cardiac Assessment

A

1) Health history - other illness that impact cardiac function
→ risk factors for heart disease
→ medications
→chest pain, exercise tolerance

2) Physical Assessment- vital signs
→ heart sounds
→ perfusion - cap refill, skin colour and temp, edema
→ respiratory assessment
→ other systems (Liver, kidney)

3) Bloodwork, ECG, echocardiogram
→ looks at structure of the heart (ex. size)

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8
Q

Risk Factors for Cardiovascular Disease

A

Modifiable
- Hypertension (blood pressure)
→Damage to vessels over time
→Left ventricular hypertrophy
Dyslipidemia
→ Blocks vessels, impacts blood flow
- Diabetes (keeping BG w/in normal range)
→Damage to vessels over time
- Lifestyle
→smoking, lack of exercise, obesity
- Sleep apnea

Nonmodifiable
- Age
- Family history/genetics
- Gender-3 to 2 ratio men to
women

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9
Q

In relation to Cardiovascular health, you should watch for:

A
  • Changes in vital signs
  • Fatigue
  • Changes in breathing (s.o.b., crackles)
  • Chest pain- Tissue hypoxia
    →Cardiac: squeezing, racing, palpitations, SOB, nausea, shoulder or jaw pain, anxiety, dizziness
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10
Q

Hypertension

A
  • Chronic condition where BP remains consistently elevated above target level

Diagnoses: 2 or more readings following initial screening

  • Major risk factor for heart attack & stroke
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11
Q

Primary VS. Secondary Hypertension

A

Primary Hypertension
- 95% of all cases
- No identifiable cause

Possible contributing factors
→↑SNS activity
→↑ Sodium-retaining hormone
→↑ Sodium intake
→↑ Alcohol consumption
→Diabetes Mellitus
→Obesity

Secondary Hypertension
- 5% of all cases
- Identifiable cause

Contributing Factors
→Kidney disease
→Neurologic disorder
→Drugs: estrogen replacement therapy, corticosteroids etc.
→Pregnancy

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12
Q

Classification of Hypertension

A

Normal
≤ 120-129 & ≤ 80-84

Pre HPTN
130-139 OR 85-89

HPTN
≥ 140 OR ≥ 90

Stage 1 HPTN
140-159 OR 90-99

Stage 2 HPTN
≥ 160 OR ≥ 100

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13
Q

Signs & Symptoms

A

-Often asymptomatic “silent killer”
- Severe and symptomatic hypertension can cause:
→Fatigue
→Dizziness
→Dyspnea
→Angina

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14
Q

Diagnosing Hypertension

A
  • Health history, including risk factors, medications

Systems Assessment
1) Neuro- headaches
- hypertensive retinopathy

2) Cardiac
→ chest pain, irregular, bounding pulse

3) Respiratory
→ shortness of breath

4) Gi
→ nausea, vomiting (hypertensive
crisis)

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15
Q

Other Diagnostic Test

A
  • Hematocrit
  • Serum K+ & Ca ++
  • Serum creatinine (kidney function)
  • HDL, LDL & triglycerides
  • ECG
  • Urinalysis
  • Blood glucose
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16
Q

Goals & Nursing Interventions

A
  • Prevention
  • Control and maintain BP < 140/90 mm Hg
  • In clients with diabetes or renal disease, BP goal is <130/80
  • Decrease risk of cardiovascular & renal complications
  • Develop and maintain lifestyle modifications, medication adherence
  • Patient education
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17
Q

Patient Education

A
  1. Knowledge
    →self-management, risk factors, managing hypotension
  2. Monitor
    →BP tracking, follow-up appointments
  3. Lifestyle
    →reduce risk, complications
  4. Medications
    →name, dose, side effects, frequency, adherence
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18
Q

Lifestyle Modification

A
  1. Maintain
    → healthy body weight (BMI 18.5-14.9)
  2. Eat
    → diet rich in fruits, veg, low-fat dairy products, low in saturated fat, reduced Na+, DASH diet
  3. Reduce
    → sodium intake
    → stress
  4. Quit
    →smoking
  5. Moderate
    → alcohol consumption (less than 1-2/day for men, no > 2 for women)
  6. Increase
    → exercise (at least 30 mins/day)
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19
Q

Pharmacological Intervention

A

Meds target
- Peripheral resistance
- Cardiac output
- RAAS System & fluid volume

  1. Angiotensin Converting Enzyme
    (ACE) Inhibitors
  2. Angiotensin Receptor Blockers
    (ARB)
  3. Beta Blockers
    → watch for bradycardia
  4. Calcium Channel Blockers
    → watch for bradycardia
  5. Diuretics
  6. Vasodilators (hydralazine)
20
Q

PRILs: Ramipril, Perindopril, Lisinopril

A

Therapeutic Class: Antihypertensives

Pharmacologic Class: ACE inhibitors

Action:
→vasodilate
→lower BP

Side Effects:
→Drowsiness
→dizziness
→headache
→persistent dry cough

Caution:
→Risk of angioedema (fluid below skin could block airway-emergency)

Nursing Considerations:
→ Monitor for hypotension (hold SBP >90)
→change position slow
→fall risk
→advocate for change in medication if client is having trouble coping
with dry cough SE
→metabolized by liver
→excreted by kidney

21
Q

Metoprolol, Carvedilol

A

→ pulse <60, do NOT give, bc it drops BP

Therapeutic Class: Antihypertensives & Anti Dysrhythmics

Pharmacologic Class: Beta Blockers

Action:
→decreases HR/cardiac contractility, lowers BP
→treats dysrhythmias, HF, HTN,
acute MI, CAD

Side Effects:
→Dizziness
→lightheadedness
→hypotension/ortho hypotension
→bradycardia
→dry mouth

Caution:
→May worsen CHF over time, therefore contraindicated in late stage

Nursing Considerations:
→Assess VS prior to admin (hold in HR<60. SBP<90)
→change position slowly
→medication may mask hypoglycemic symptoms—monitor in people
with type 1 or 2 diabetes

22
Q

Amlodipine, Diltiazem

A

Therapeutic Class: Antihypertensives & Anti Dysrhythmics

Pharmacologic Class: Calcium Channel Blockers

Action:
→decreases HR/cardiac contractility, cause vasodilation, lowers BP

Side Effects:
→Dizziness
→lightheadedness
→hypotension/orthostatic hypotension
→bradycardia
→peripheral edema
→flushing
→GI upset
→constipation

Caution: Avoid with CHF

Nursing Considerations:
→Risk for hypotension (hold SBP<90, HR<60)
→change position slowly
→monitor I/O due to risk for edema →metabolized by liver
→excreted by kidneys

23
Q

Furosemide (Lasix)

A

Therapeutic Class: Antihypertensives

Pharmacologic Class: Loop Diuretic

Action: lower BP by reducing fluid volume

Side Effects:
→ Dehydration
→Dizziness, headache
→hypotension/orthostatic
hypotension
→electrolyte imbalances (hypokalemia, hyponatremia, hypocalcaemia)

Caution:
→Use with caution in kidney disease, monitor kidney function

Nursing Considerations:
→Risk for hypotension
→monitor electrolytes
→daily weights
→I/O
→falls risk with older adults
→monitor kidney function

24
Q

Hydrochlorothiazide

A

Therapeutic Class: Antihypertensives

Pharmacologic Class: Thiazide Diuretic

Action: lower BP by reducing fluid volume

Side Effects:
→Dehydration
→dizziness, headache
→ hypotension/orthostatic hypotension
→electrolyte imbalances (hypokalemia, hyponatremia)

Caution:
→Contraindicated with impaired kidney function

Nursing Considerations:
→Risk for hypotension
→monitor electrolytes
→daily weights, I/O
→falls risk with older adults
→monitor kidney function

25
Q

Spironolactone

A

Therapeutic Class: Antihypertensives

Pharmacologic Class: Potassium Sparing Diuretic

Action: lower BP by reducing fluid volume

Side Effects:
→Dizziness, headache, →hypotension/orthostatic hypotension,
→electrolyte imbalances (hyperkalemia)
→photosensitivity
→N/V/D

Caution:
→Contraindicated with later stage kidney function

Nursing Considerations:
→Risk for hypotension
→monitor electrolytes
→daily weights
→I/O
→falls risk with older adults
→monitor kidney function

26
Q

Hypertensive Crisis

A
  • Acute marked increase in BP
  • Cause = abrupt stopping of medication, poor management of HTN

Urgency
→ High BP, headache, nosebleed, anxiety
→ Manifests in days → weeks
→ BP > 180/110mm Hg
→ No major organ damage Identified

Emergency
→ Manifests in hours to days
→ BP > 220/140mm Hg
→ Potential organ damage: Intracranial or subarachnoid hemorrhage
→ MI
→ left ventricular failure
→ renal failure
→ retinopathy
→ aortic aneurysm

27
Q

Nursing Care of a Patient with a Hypertensive Crisis

A

Goal:
→Decrease BP by 25% in 1 hour
→Achieve 160/100 BP in 2-6 hours

  • VS monitoring: Q 5 minutes
  • Systems Assessment: S & S of organ damage (remember findings
    associated with lack of perfusion!)
  • Bloodwork: Electrolytes (K, Mg, Cl, Na), urea, creatinine, lipids, glucose
  • Initiate IV Access
  • Administer IV antihypertensive medications
  • Urinalysis
  • 12 lead ECG
  • I/O
  • Administer O2 PRN
  • Encourage rest & quiet room
28
Q

Coronary Artery Disease

A
  • Narrowing or obstruction of the coronary arteries due to atherosclerosis

Atherosclerosis: Buildup of plaque in arterial walls
→Caused by injury to artery walls (due to HTN, diabetes, inflammation,
cholesterol)
→Plaque impacts perfusion=damage to body systems

29
Q

Progression of Coronary Artery Disease

A

1) Atherosclerosis: Plaque deposition in arteries
→hardened and thickened walls, restricted blood flow

2) Peripheral Artery Disease
(PAD): Inadequate oxygen supply to the extremities

2) Coronary Artery Disease (CAD): Inadequate oxygen supply to the myocardium

2a. Angina = chest pain due to
inadequate oxygen supply to myocardium

2b. Acute Coronary Syndrome
(ACS): Spectrum of acute myocardial infarction; results in STEMI, NSTEMI, & unstable angina

30
Q

Risk Factors for CAD

A

Modifiable
→Lifestyle (smoking, obesity, diet, exercise)

Non-modifiable
→Family history
→Age
→Gender (women after menopause)
→Genetics
→ need to consider other comorbidities (diabetes, dyslipidemia)

31
Q

Assessment for CAD

A

1) Health history

2) Labs & diagnostic tests:
Lipid Profile:
→Total cholesterol Normal = < 200 mg/dL
→LDL (“bad”) cholesterol Normal = < 100 mg/dL (high=risk)
→HDL (“good”) cholesterol Normal = ≥ 60 mg/dL (low=risk)

→Triglycerides Normal = < 150 mg/dL

→HbA1C: uncontrolled diabetes, high A1C = increased risk

→BG: uncontrolled diabetes, high BG = increased risk

32
Q

Nursing Priorities

A
  1. Reduce Cholesterol
    →Diet, medications
  2. Manage comorbidities (glucose, BP management)
  3. Health Promotion
    →Lifestyle changes (diet similar to diabetic diet-low in sugars, low in
    saturated fats, high in fiber)
    → Exercise
    →Stress reduction
33
Q

Statins: Atrovastatin, Rosuvastatin

A

Therapeutic Class: Antihyperlipidemic

Pharmacologic Class: HMG-CoA reductase inhibitors

Action: decrease LDL/triglycerides

Side Effects:
→Headache, dizziness
→GI upset
→insomnia
→MUSCLE PAIN

Caution:
→Do not take if liver disease present; may increase blood glucose level

Nursing Considerations:
→monitor lipids (**fasting)
→hepatotoxic

34
Q

Angina’s Mismatch of Supply & demand

A

Normal physiology
→ Oxygen demand is in balance with supply
→ When oxygen demand ↑, coronary
blood flow will ↑; increasing oxygen
supply, or vice versa

Angina’s Mismatch of Supply & demand
→ Coronary blood flow ↓, & oxygen supply is limited
→ Since heart muscles need oxygen, oxygen demand ↑ during exercise

Results:
→ ↑ Oxygen demand, & ↓ supply =
hypoxia (oxygen deficiency) → MI
or angina

35
Q

Angina VS. Myocardial Infarction

A

Angina
→ Ischemia of partial thickness of myocardial muscle
→ Causes chest pain
→ Due to stenosis, vasospasm,
thickening of heart wall
→ Goal: Relieve pain &reduce risk
of disease progression & MI

Myocardial Infarction
→Death of myocardia cells due to prolonged ischemia of full thickness of myocardial muscle
→ EMERGENCY Time is Tissue
→ Due to stenosis, plaque lodge
→ Goal: restore blood flow

36
Q

Stable VS. Unstable Angina

A

Stable Angina (exertional angina)
→ due to exertion/stress
→ stable pattern of onset and duration
→ inadequate blood supply
→ treated with stopping/ resting and nitroglycerin
→ will feel the same and last for the same amount of time
→ ST depression and or T wave inversion on ECG, or no changes at rest

Unstable Angina (Pre-infarction angina)
→plaque rupture, prone got thrombogeneis
→ can occur during at rest or very little stress/exertion
→ increased occurrence and severity over time
→ ST depression and T wave changes may occur on ECG
→ treated with nitroglycerin

Variant Angina (rare)
→ chest pain at rest
→ often occurs between midnight adn early morning
→ caused by temp spasms in the coronary arteries, leading to reduced blood flow
→ treatment: nitrates, calcium channel blockers

38
Q

Signs & Symptoms of Angina

A

Cardiac
→ Chest pain (pressure, squeezing)
→ irregular rhythm
→ bounding pulse
→ pain can radiate to left arm, jaw, shoulders, and back

Respiratory
→dyspnea
→ shortness of breath
tachypnea
crackles (more common with MI)

Neurological
→ anxiety
→ restlessness
→ dizziness

GI (more common with MI)
→ nausea, vomiting

Integumentary
→flush
→pale
→ diaphoretic

39
Q

Assessment of Angina

A
  • Health history
  • Vital signs
  • Bloodwork & diagnostic tests
  • Troponin, ECG, stress test, angiogram
40
Q

Troponin I

A
  • A cardiac biomarker lab value that measures a protein that is specific to the heart muscle; it is released when heart muscle cells are damaged

→Normal=<0.03 ng/mL
→ Elevates 3-6 hrs post MI, peak 12-16 hrs, remains high 5-9 days

  • Elevated troponin= MI
41
Q

Electrocardiogram

A
  • Investigation of cardiac electrical activity, used to identify rhythm
    changes that are indicative of an MI
  • Within 10 minutes of arrival with S&S to rule out Myocardial Infarction

** look at electrocardiogram on slide*

42
Q

Other Diagnostic Tests

A
  1. Exercise Tolerance Test (ETT, or “Stress Test”)
    →used to visualize activity of heart during exertion
  2. Coronary Angiogram
    →catheter to heart muscle with dye to visualize coronary blood flow (blockages) & identify areas of ischemia
43
Q

Pharmacological Interventions

A
  • Meds used for vasodilation, prevention/treatment of pain, reduce risk of clotting/atherosclerosis
  1. Nitrates (short acting: nitroglycerin)
  2. Nitrates (long acting, extended release, patches, etc.)
  3. Calcium channel blockers
  4. Beta Blockers
  5. Antihyperlipidemics (statins)
  6. Anti-coagulants (ASA)
44
Q

Nitroglycerin

A

Therapeutic Class: Vasodilator (short acting)

Pharmacologic Class: Nitrates

Action: d
→decreases BP
→decrease pre-load and afterload

Side Effects:
→Dizziness
→light headedness, headache, →orthostatic hypotension
→tachycardia
→hypotension
→flushing

Caution: avoid sildenafil (Viagara)→hypotension!

Nursing Considerations:
→health teaching on use
→monitor effects after
use
→Given sublingually to treat chest pain

45
Q

If someone experiences chest pain…

A
  1. Stop activity
  2. Sit down
  3. Assess pain
  4. Assess vitals (BP, pulse)
  5. Administer nitroglycerin sublingually (1 spray q 5 minutes up to 3 sprays)
    → watch for HYPOtension
  6. Reassess pain
  7. Reassess vitals
  8. If pain is unrelieved by 3 consecutive sprays or pain lasts longer than 15 minutes, notify MD