week 3 (neuro, sensorineural) Flashcards

1
Q

Nursing management MG

A

Meals coinside with the peak anticholinesterase meds

suction available

impaired vision (tape/patch eye closed)

avoid stress factors (stress, infections, high temp., vigorous exercise).

Rest throughout day

Food considerations (soft, small, etc.)

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2
Q

Myasthenic Crisis

A

Temporary exacerbation of symptoms triggered by infection. As a nurse, you should monitor for respiratory weakness, dysphagia, and avoid any sedative medications.

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3
Q

Cholinergic crisis (MG)

A

We treat MG with anticholinergic medications. However, the patient can become overmedication and go into cholinergic crisis. You will see respiratory impairment, muscle weakness, and excessive pulmonary secretions. To treat this, stop the medications.

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4
Q

What are we concerned about with Guillain-Barre Syndrome?

A

respiratory function

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5
Q

What is Amyotropic Lateral Sclerosis?

A

ALS - degenerative disease that results in the loss of both upper and lower motor neurons.

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6
Q

ALS - s/s

A

Variable depending on where you are at in the progression of the disease.
a. Spasticity - no loss of bladder/bowel control/ respiratory function

b. ultimately, respiratory function is impaired.

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7
Q

ALS treatment

A

Nothing specific - manage symptoms.

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8
Q

What medications can be given to ALS patients who have trouble with spasticity?

A

Baclofen, dantrolene sodium, diazepam

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9
Q

ALS nursing management

A

guide them on end-of-life care

Supportive therapy

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10
Q

Ischemic stroke: cause

A
Large artery thrombosis 
Small, penetrating artery thrombosis 
Cardiogenic emboli
Cryptogeni 
other
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11
Q

Hemorrhagic stroke: cause

A

Intracerebral hemorrhage
Subarachnoid hemorrhage
Cerebral aneurysm
Arteriovenous malformation

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12
Q

Ischemic stroke s/s

A

Numbness or weakness of the face, arm, leg especially on one side of the body. You may also see slurred speech or difficulty with word finding or comprehension

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13
Q

Hemorrhagic stroke s/s

A
"Exploding headache"
Decrease LOC
N/V
Visual changes
Seizures
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14
Q

What is the BE FAST acronym for strokes?

A
Balance loss
Eye blur 
Face droop
Arm weakness
Speech difficulty
Time to call 911
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15
Q

Risk factors for ischemic stroke

A
HTN
Smoking
Diabetes
A-fib
Obstructive sleep apnea
Family
Race/ethnicity 

The list goes on and on

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16
Q

Hemiplegia

A

paralysis on one side of the body or part of it

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17
Q

Hemiparesis

A

weakness to one side of the body or part of it

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18
Q

Aphasia

A

Inability to express oneself or to understand language

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19
Q

Dysarthria

A

difficulty in speaking

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20
Q

Dysphasia

A

impaired speech

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21
Q

Aphasia: expressive, receptive, global

A

expressive aphasia(inability to express oneself)

receptive aphasia(inability to understand language)

global (mixed) aphasia (combo of the 2)

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22
Q

Apraxia

A

inability to perform a previously learned action

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23
Q

Homonymoushemianopsia

A

blindness in half of the visual field in one or both eyes - temporary or permanent

The affected side of vision corresponds to the paralyzed side of the body.

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24
Q

agnosia

A

loss of the ability to recognize objects through a particular sensory system; it may be visual, auditory, or tactile

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25
Q

What might you see if someone is having a left hemisphere stroke

A
Paralysis or weakness on right side of body
Right visual field deficit
Aphasia
Altered intellectual ability
Slow, cautious behavior
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26
Q

What might you see if someone is having a stroke in their right hemisphere

A
Paralysis/weak on left side of body
Left visual field deficit 
Spatial-perceptual deficits
Increased distractibility
Impulsive/poor judgement 
Lack of awareness of deficits
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27
Q

Stroke prevention

A

Decrease risk factors and know medical conditions that increase the chance of a stroke (HLD, a-fib, diabetes, HTN).

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28
Q

What do we do when someone is discharged of a stroke?

A

We have to follow through with core measures such as medications (medications such as Plavix, -statins, BP medications, Aspirin).

HOWEVER, if someone had a hemorrhagic stroke, we want to be careful with aspirin or plavix because it can cause more bleeding. Depends on what the Dr. wants.

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29
Q

Why would someone get a CT and MRI scan when they come in for a stroke

A

CT - to see if hemorrhagic or ischemic

MRI - tells exactly where stroke has occurred

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30
Q

How do we assess someone who has a stroke

A

NIH scale (0-42)
The higher the score, the worse the client is
If you assess and their score goes up, call provider

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31
Q

Reperfusion therapies: tPA - when is it given

A

within 3 hours of last seen normal

may be up to 4.5 with no history of DM, or previous stroke, not on anticoagulants, and NIH less than 25

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32
Q

tPA dosing

A

weight based dose 0.9 mg/kg with a maximum dose of 90 mg

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33
Q

How much tPA is given at a time?

A

10% given over 1 minute

Remaining is given over 1 hour

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34
Q

What are things to considered once tPA is given

A

Assess for bleeding
Once given, no anticoagulants for 24 hours
Must go to ICU for 24 hours
No inserting any tubes for 24 hours (unless emergent)

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35
Q

What do we want in regard to reperfusion for an ischemic stroke

A

for the first 24 hours we want that patient to have permissive hypertension (HIGH BP) because the more blood flow we can force to brain better chance of perfusion. After 24 hours, immediately bring BP down

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36
Q

Reperfusion therapies: Thrombectomy with or without intra arterial alteplase

A

Give within 6 hours LSN

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37
Q

Reperfusion therapies: Endovascular therapy

A

Now recommended that patients with acute ischemic stroke

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38
Q

Endovascular therapy: criteria that needs to be met

A

Prestroke status of no deficits

Acute ischemic stroke receiving IV t-PA within 4.5 hours of onset according to guidelines from professional medical societies

Causative occlusion of the internal carotid artery or proximal middle cerebral artery

Age ≥18 years

NIHSS score of ≥6

ASPECT (a radiologic assessment of the CT scan) score of ≥6, and treatment can be initiated (groin puncture) within 6 hours of symptom onset

Patients eligible for t-PA should receive IV t-PA even if endovascular treatments are being considered (Powers et al., 2015). Thrombolytic therapy should not be delayed.

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39
Q

Reperfusion therapies: Carotid Endarterectomy (CEA)

A

surgery to treat carotid artery diseases (i think a stent is inserted)

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40
Q

CEA complications - incision hematoma: intervention

A

Monitor neck discomfort and wound expansion. Report swelling, subjective feelings of pressure in the neck, difficulty breathing.

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41
Q

CEA complications - hypertension: intervention

A

Keep in mind that risk is highest in the first 48 hours after surgery. Check blood pressure frequently, and report deviations from baseline. Administer medications, as prescribed, to reduce hypertension. Observe for and report new onset of neurologic deficits.

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42
Q

CEA complication - postoperative hypotension: intervention

A

monitor blood pressure and observe for s/s of hypotension

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43
Q

CEA complication - hyperperfusion syndrome: intervention

A

Observe for severe unilateral headache improved by sitting upright or standing.

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44
Q

CEA complication - intracerebral hemorrhage: intervention

A

monitor neurological status, and report any changes in mental status or neurological functioning immidiately

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45
Q

Ischemic stroke intervention:

A

Mobility and preventing joing deformities

  • prevent should adduction
  • position the hands and fingers
  • changing positions
  • exercise program
  • preparing for ambulation
Preventing shoulder pain
Self-care
Bladder and bowel control (training)
Nutrition (dysphagia)
Improve thought process and communication
Skin integrity
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46
Q

Ischemic stroke: preventing should adduction

A

To prevent adduction of the affected shoulder while the patient is in bed, a pillow is placed in the axilla when there is limited external rotation; this keeps the arm away from the chest. A pillow is placed under the arm, and the arm is placed in a neutral (slightly flexed) position, with distal joints positioned higher than the more proximal joints (i.e., the elbow is positioned higher than the shoulder and the wrist higher than the elbow). This helps to prevent edema and the resultant joint fibrosis that will limit range of motion if the patient regains control of the arm

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47
Q

Ischemic stroke: changing positions

A

The patient’s position should be changed every 2 hours. To place a patient in a lateral (side-lying) position, a pillow is placed between the legs before the patient is turned. To promote venous return and prevent edema, the upper thigh should not be acutely flexed. The patient may be turned from side to side, but if sensation is impaired, the amount of time spent on the affected side should be limited.
If possible, the patient is placed in a prone position for 15 to 30 minutes several times a day. A small pillow or a support is placed under the pelvis, extending from the level of the umbilicus to the upper third of the thigh. This position helps promote hyperextension of the hip joints, which is essential for normal gait and helps prevent knee and hip flexion contractures. The prone position also helps drain bronchial secretions and prevents contractural deformities of the shoulders and knees. During positioning, it is important to reduce pressure and change position frequently to prevent pressure ulcers

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48
Q

Ischemic stroke: establishing exercise program

A

The affected extremities are exercised passively and put through a full range of motion four or five times a day to maintain joint mobility, regain motor control, prevent contractures in the paralyzed extremity, prevent further deterioration of the neuromuscular system, and enhance circulation. Exercise is helpful in preventing venous stasis, which may predispose the patient to venous thromboembolism (VTE). VTE includes deep vein thrombosis (DVT) and pulmonary embolus (PE).

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49
Q

Ischemic stroke: preparing for ambulation

A

As soon as possible, the patient is assisted out of bed and an active rehabilitation program is started. The patient is first educated to maintain balance while sitting and then to learn to balance while standing. If the patient has difficulty in achieving standing balance, a tilt table, which slowly brings the patient to an upright position, can be used. Tilt tables are especially helpful for patients who have been on bed rest for prolonged periods and have orthostatic blood pressure changes.

If the patient needs a wheelchair, the folding type with hand brakes is the most practical because it allows the patient to manipulate the chair. The chair should be low enough to allow the patient to propel it with the uninvolved foot and narrow enough to permit it to be used at home. When the patient is transferred from the wheelchair, the brakes must be applied and locked on both sides of the chair.
The patient is usually ready to walk as soon as standing balance is achieved. Parallel bars are useful in these first efforts. A chair or wheelchair should be readily available in case the patient suddenly becomes fatigued or feels dizzy.
The training periods for ambulation should be short and frequent. As the patient gains strength and confidence, an adjustable cane can be used for support. In general, a three- or four-pronged cane provides a stable support in the early phases of rehabilitation

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50
Q

Ischemic stroke: positioning the hands and fingers

A

The fingers are positioned so that they are barely flexed. The hand is placed in slight supination (palm faces upward), which is its most functional position. If the upper extremity is flaccid, a splint can be used to support the wrist and hand in a functional position. If the upper extremity is spastic, a hand roll is not used, becauseit stimulates the grasp reflex. In this instance, a dorsal wrist splint is useful in allowing the palm to be free of pressure. Every effort is made to prevent hand edema.

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51
Q

Hemorrhagic stroke nursing intervention: optimizing tissue perfusion

A

The patient is closely monitored for neurologic deterioration resulting from recurrent bleeding, increasing ICP, or vasospasm. A neurologic flow record is maintained. The blood pressure, pulse, level of consciousness (an indicator of cerebral perfusion), pupillary responses, and motor function are checked hourly. Respiratory status is monitored, because a reduction in oxygen in areas of the brain with impaired autoregulation increases the chances of a cerebral infarction. Any changes are reported immediately.
Implementing Aneurysm Precautions.Cerebral aneurysm precautions are implemented for the patient with a diagnosis of aneurysm to provide a nonstimulating environment, prevent increases in ICP, and prevent further bleeding. The patient is placed on bed rest in a quiet, nonstressful environment, because activity, pain, and anxiety are thought to elevate the blood pressure, which may increase the risk for bleeding. Visitors may be restricted (American Association of Neuroscience Nurses [AANN], 2009;Hickey, 2014).
The head of the bed is elevated 30 degrees to promote venous drainage and decrease ICP. Any activity that suddenly increases the blood pressure or obstructs venous return is avoided. This includes the Valsalva maneuver, straining, forceful sneezing, pushing oneself up in bed and acute flexion or rotation of the head and neck (which compromises the jugular veins). Stool softeners and mild laxatives are prescribed. Both prevent constipation, which can cause an increase in ICP. Dim lighting is helpful, because photophobia (visual intolerance of light) is common. The purpose of aneurysm precautions should be thoroughly explained to both the patient (if possible) and family. Intermittent pneumatic compression devices are prescribed to decrease the incidence of DVT resulting from immobility. The legs are observed for signs and symptoms of DVT (tenderness, redness, swelling, warmth, and edema), and abnormal findings are reported.

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52
Q

Hemorrhagic stroke nursing interventions: relieving anxiety

A

Sensory stimulation is kept to a minimum for patients on aneurysm precautions. For patients who are awake, alert, and oriented, an explanation of the restrictions helps reduce the patient’s sense of isolation. Reality orientation is provided to help maintain orientation.
Keeping the patient well informed of the plan of care provides reassurance and helps minimize anxiety. Appropriate reassurance also helps relieve the patient’s fears and anxiety. The family also requires information and support.

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53
Q

Hemorrhagic stroke nursing interventions: monitoring and managing potential complications - vasospasm

A

The patient is assessed for signs of possible vasospasm: intensified headaches, a decrease in level of responsiveness (confusion, disorientation, lethargy), or evidence of aphasia or partial paralysis. These signs may develop several days after surgery or on the initiation of treatment and must be reported immediately. The calcium channel blocker nimodipine should be given for prevention of vasospasm, and fluid volume expanders in the form of triple-H therapy may be prescribed as well

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54
Q

Hemorrhagic stroke nursing interventions: monitoring and managing potential complications - Seizures

A

Seizure precautions are maintained for every patient who may be at risk for seizure activity. Should a seizure occur, maintaining the airway and preventing injury are the primary goals. Medication therapy is initiated at this time

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55
Q

Hemorrhagic stroke nursing interventions: monitoring and managing potential complications - hydrocephalus

A

Blood in the subarachnoid space or ventricles impedes the circulation of CSF, resulting in hydrocephalus. A CT scan that indicates dilated ventricles confirms the diagnosis. Hydrocephalus can occur within the first 24 hours (acute) after subarachnoid hemorrhage or several days (subacute) to several weeks (delayed) later. Symptoms vary according to the time of onset and may be nonspecific. Acute hydrocephalus is characterized by sudden onset of stupor or coma and is managed with a ventriculostomy drain to decrease ICP. Symptoms of subacute and delayed hydrocephalus include gradual onset of drowsiness, behavioral changes, and ataxic gait. A ventriculoperitoneal shunt is surgically placed to treat chronic hydrocephalus. Changes in patient responsiveness are reported immediately.

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56
Q

Hemorrhagic stroke nursing interventions: monitoring and managing potential complications - rebleeding

A

The rate of recurrent hemorrhage is approximately 1% to 5% per patient per year after intracerebral hemorrhage (Hemphill et al., 2015). Hypertension is the most serious and modifiable risk factor, which shows the importance of appropriate antihypertensive treatment.
Aneurysm rebleeding is the highest during the first 2 to 12 hours after the initial hemorrhage (Connolly et al., 2012) and is considered a major complication. Symptoms of rebleeding include sudden severe headache, nausea, vomiting, decreased level of consciousness, and neurologic deficit. Rebleeding is confirmed by CT scan. Blood pressure is carefully maintained with medications. The most effective preventive treatment is to secure the aneurysm if the patient is a candidate for surgery or endovascular treatment.

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57
Q

Hemorrhagic stroke nursing interventions: monitoring and managing potential complications - hyponatremia

A

After subarachnoid hemorrhage, hyponatremia is found in 10% to 30% of patients. Hyponatremia has been found to be associated with the onset of vasospasm (Connolly et al., 2012). Laboratory data must be checked frequently, and hyponatremia (defined as a serum sodium concentration less than 135 mEq/L) must be identified as early as possible. The patient’s primary provider needs to be notified of a low serum sodium level that has persisted for 24 hours or longer. The patient is then evaluated for syndrome of inappropriate antidiuretic hormone (SIADH) or cerebral salt-wasting syndrome. SIADH is described inChapter 13. Cerebral salt-wasting syndrome occurs when the kidneys are unable to conserve sodium and volume depletion results. The treatment most often is the use of IV hypertonic 3% saline.

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58
Q

Described the anatomy of the eyes in relation to the cranial nerves

A

The four rectus muscles and two oblique muscles are innervated by cranial nerves (CN) III, IV, and VI.

Normally, the movements of the two eyes are coordinated, and the brain perceives a single image.

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59
Q

Tears: role

A

protect the cornea and sclera with lubrication, nutritional support, moisture, and protection from microbes

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60
Q

The external eye contain immunoglobulins that do what?

A

contribute to the increase in tearing during allergic responses

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61
Q

What should the nurse do in the absence of the patient’s corneal reflex?

A

protect the patient’s affectedeye from injury by lubricating it with artificial tears to prevent drying.

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62
Q

Conjunctiva

A

provides a barrier to the external environment and nourishes the eye.

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63
Q

What do the goblet cells of the conjunctiva do

A

secrete lubricating mucus

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64
Q

What does the bulbar conjunctiva do and what does the palpebral conjunctiva do

A

Bulbar conjunctiva: covers sclera
Palpebra conjunctiva: lines the inner surface of the upper and lower eyelids

The junction of the 2 of known as the fornix

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65
Q

Describe the sclera

A

dense, fibrous structure that helps maintain the shape of the eyeball and protects the intraocular contents from trauma

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66
Q

Describe the sclera across age groups

A

children can have slightly blue
adults can have dull white
elderly can have slightly yellow
African Americans can have muddy brown

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67
Q

Describe the cornea

A

a transparent, avascular, domelike structure that covers the iris, pupil, and anterior chamber. It is the most anterior portion of the eyeball and is the main refracting surface of the eye.

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68
Q

What is the uvea

A

consists of the iris, the ciliary body, and the choroid. The iris, or colored part of the eye, is a highly vascularized, pigmented collection of fibers surrounding the pupil

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69
Q

What is the pupil

A

The pupil is a space that dilates and constricts in response to light.

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70
Q

Describe the lens of the eye

A

Directly behind the pupil and iris

A colorless and almost completely transparent biconvex structure

Avascular, no nerve or pain fibers

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71
Q

What does the lens enable the eye to do

A

Enables focusing on near vision and refocusing for distance vision (ACCOMODATION)

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72
Q

What is the ocular fundus

A

the largest chamber and contains the vitreous humor (clear, gelatinous substances)

73
Q

What is the innermost surface of the fundus?

A

retina

74
Q

the retina is an extension of what?

A

optic nerve

75
Q

where do the retina and optic nerve neet?

A

optic disc

76
Q

What are rods responsible for?

A

night vision or vision in low light

77
Q

What do cones do?

A

Provide the best vision for bright light, color vision and fine detail

78
Q

Visual acuity depends on what?

A

a healthy, functioning eyeball and act intact visual pathways

79
Q

What does the visual pathway consist of?

A

retina, optic nerve, optic chiasm, optic tracts, lateral geniculate bodies, optic radiations, and the visual cortex area of the brain. The pathway is an extension of the CNS

80
Q

Optic nerve purpose

A

transmit impulses from the retina to the occipital lobe of the brain

81
Q

What is the blind spot in each eye?

A

The optic nerve head, or optic disc

82
Q

Gerontological considerations of the eye on slide 8

A

go look

83
Q

There is more information on the anatomy and physiology on the eye in the beginning of the sensorineural powerpoint

A

go look if it is on the blueprint

84
Q

Eye assessment consists of what

A
ask about...
changes in vision
pain or discomfort
past history
family history
social history
85
Q

Presbyopia

A

the term used for impaired near vision and is often found in middle-aged and older persons. A specially designed handheld card is held 14 in away from the patient’s eyes, and the patient is asked to read the chart. Presbyopic people are commonly able to read the chart when it is held farther away

86
Q

Testing and diagnostics of eyes

A

slide 10

87
Q

Ear assessment

A

Hearing

Balance and equilibrium

88
Q

Ears - gerontological considerations on slide 12

A

go look

89
Q

What 3 characteristics are important when evaluating hearing

A

frequency, pitch, intensity

90
Q

Hearing: frequency

A

the number of sound waves emanating from a source per second, measured as cycles per second, or Hertz (Hz).

91
Q

The normal human ear perceives sounds ranging in what frequency

A

from 20 to 20,000 Hz

92
Q

pitch

A

term used to describe frequency

– a tone with 100 Hz is considered of low pitch, and a tone of 10,000 Hz is considered of high pitch

93
Q

What are hearing tests

A

Whisper test
Weber test
Rinne test

94
Q

Emmetropia

A

normal vision

95
Q

Myopia

A

nearsighted

96
Q

hyperopia

A

farsighted

97
Q

astigmatism

A

irregularity in the curve of the cornea

98
Q

What is glaucoma

A

Condition cause by optic nerve damage

99
Q

Glaucome: patho

A

increased IOP caused by congestion of aqueous humor in the eye

100
Q

Types of glaucoma

A

wide-angle (complete blockage)

narrow-angle (partial blockage)

101
Q

Glaucoma s/s

A

Blurred vision or “halos” around lights
Difficulty focusing
Difficulty adjusting eyes in low lighting
Loss of peripheral vision, aching or discomforted around the eyes, and headache

102
Q

Glaucoma management

A

No cure - prevent damage to optic nerve

Beta blockers –> decrease the production of aqueous humor with resultant decrease in IOP

103
Q

What is an example of a BB that you can give someone with glaucoma and what are nursing considerations

A

Timolol

- check BP, HR, stop it from going systematic

104
Q

Risk factors for glaucoma

A
Family history of glaucoma
African American race
Older age (over 60 years of age)
Diabetes mellitus
Cardiovascular disease
Migraine syndromes
Nearsightedness (myopia)
Eye trauma – increased pressure 
Prolonged use of topical or systemic corticosteroids
105
Q

Progression of Glaucoma: Initiating events

A

Precipitating factors include illness, emotional stress, congenital narrow angles, long-term use of corticosteroids, and use of mydriatics (i.e., medications causing pupillary dilation)

106
Q

Progression of glaucoma: structural alterations in the aqueous outflow system

A

Tissue and cellular changes caused by factors that affect aqueous humor dynamics lead to structural alterations

107
Q

Progression of glaucoma: functional alterations

A

Conditions such as increased IOP or impaired blood flow create functional changes

108
Q

Progression of glaucoma: optic nerve damage

A

Atrophy of the optic nerve is characterized by loss of nerve fibers and blood supply. This fourth stage inevitably progresses to the fifth stage

109
Q

Progression of glaucoma: visual loss

A

Progressive loss of vision is characterized by visual field defects

110
Q

Cataracts

A

Cloudiness in the eyes

111
Q

Types of cataracts

A

senile
nuclear
posterior subcapsular

112
Q

Cataracts: s/s

A

painless, blurry vision

decreased visual acuity is directly proportional to cataract density

113
Q

Cataract risk factors: aging

A

Loss of lens transparency
Clumping or aggregation of lens protein (which leads to light scattering)
Accumulation of a yellow-brown pigment due to the breakdown of lens protein
Decreased oxygen uptake
Increase in sodium and calcium
Decrease in levels of vitamin C, protein, and glutathione (an antioxidant)

114
Q

Cataract risk factors: associated ocular conditions

A

Retinitis pigmentosa
Myopia
Retinal detachment and retinal surgery
Infection (e.g., herpes zoster, uveitis)

115
Q

Cataract risk factors: toxic factors

A

Corticosteroids, especially at high doses and in long-term use

Alkaline chemical eye burns, poisoning

Cigarette smoking

Calcium, copper, iron, gold, silver, and mercury, which tend to deposit in the pupillary area of the lens

116
Q

Cataract risk factors: nutritional factors

A

reduced levels of antioxidants
poor nutrition
obesity

117
Q

cataract risk factors: physical factors

A

Dehydration associated with chronic diarrhea, use of purgatives in anorexia nervosa, and use of hyperbaric oxygenation

Blunt trauma, perforation of the lens with a sharp object or foreign body, electric shock

Ultraviolet radiation in sunlight and x-ray

118
Q

cataract risk factors: systemic diseases and syndromes

A
Diabetes mellitus
Down syndrome
Disorders related to lipid metabolism
Kidney disorders
Musculoskeletal disorders
119
Q

Cataract Surgery: pre-op considerations

A

No anticoagulant therapy to reduce risk of retrobulbar hemorrhage

Dilating drops are administered Q10min for 4 doses at least 1 hour before surgery

Dilating drops may be administered in operating room if eye not fully dilated

120
Q

Cataract post-op surgery: nursing considerations

A

Antibiotic, corticosteroid, anti-inflammatory drops may be administered prophylactically to prevent postoperative infection and inflammation.

Protective eye patch for 24 hours

Slight discharge in AM, redness and scratchy feeling is expected for a few days

121
Q

Retinal detachment

A

separation of retinal pigment epithelium from the sensory layer

122
Q

Rhegmatogenous retinal detachment

A

A hold or tear develops in the sensory retina allowing liquid vitreous to seep through the sensory Terina and detach

123
Q

Traction retinal detachment

A

Pulling force

Develop fibrous scar tissue from diabetic retinopathy, vitreous hemorrhage or retinapothy of prematurity

124
Q

Exudative retinal detachment

A

Result from production of serous fluid under the retina from the choroid

125
Q

Exudative retinal detachment: cause

A

– uveitis and macular degeneration

126
Q

Uveitis

A

inflammation in the middle part of the eye

127
Q

Retinal detachment: s/s

A

Shade of curtain coming across the vision of one eye

  • cobwebs
  • bright flashing lights
  • sudden onset of a great number of floaters
  • NO pain
128
Q

Retinal detachment: medical management

A

Visualization through dilatated fundus examination using indirect ophthalmoscope

Surgically reattach the sensory retina to the RPE

129
Q

Retinal detachment: nursing management

A
Assistance with walking/eating
Avoid heavy lifting (increase IOP)
Restrict reading
Sunglasses/eyepatch
Ice packs
Reevaluation in 6-8
130
Q

Retinal detachment: postoperative complications

A
Increased IOP
endophthalmitis (inflammation of the internal layer of the eye)
development of other retinal detachments
development of cataracts
loss of turgor of the eye.
131
Q

Retinal detachment: teaching

A

Patients must be taught the signs and symptoms of complications, particularly of increasing IOP and postoperative infection, such as eye pain, sudden change in vision, fever, lid swelling, or conjunctival and/or corneal injection (redness). Excessivepain, swelling, and bleeding must be reported immediately to the surgeon.

132
Q

Macular degeneration

A

Primarily central vision gone

133
Q

Wet macular degeneration

A

New growth of blood vessels beneath the retina

  • abrupt onset
  • vessels can leak fluid and blood
  • report straight lines appear crooked and distorted or letters in works appear broken
134
Q

Dry macular degeneration

A

Outer layers of the retina slowly break down

Gradual blurring of vision when trying to read

insidious onset and lead to mild vision loss (central)

135
Q

Macular degeneration: laser treatment

A

can cause retinal destruction

136
Q

macular degeneration: photodynamic therapy –> Verteprofin

A

photosensitive dye – must avoid bright sunlight, tanning booths, halogen lights, and the bright lights used in dental offices and operating room

137
Q

What should a patient bring with them to photodynamic therapy for macular degenerations

A

bring dark sunglasses, gloves, a wide-brimmed hat, long-sleeved shirt, and slacks

138
Q

Macular degeneration: nursing management:

A
education
assistive devices (large print, sunglasses, hat to decrease sunlight, walker, no driving, etc.)
139
Q

Orbital trauma: soft tissue injury and hemorrhage s/s

A

tenderness, ecchymosis, lid swelling, proptosis, hemorrhage

140
Q

proptosis

A

downward displacement of eyeball

141
Q

management of soft tissue injury and hemorrhage (orbital trauma)

A

thorough inspection, cleansing, and repair of wounds.

Cold compresses are used in the early phase, followed by warm compresses.

Hematomas that appear as swollen, fluctuating areas may be surgically drained or aspirated; if they are causing significant orbital pressure, they may be surgically evacuated.

142
Q

Orbital fractures: how are they detected?

A

by facial x-rays

143
Q

how can orbital fractures be classified

A

blowout, zygomatic or tripod, maxillary, midfacial, orbital apex, and orbital roof fractures.

144
Q

What do blowout fractures result from?

A

Blowout fractures result from compression of soft tissue and the sudden increase in orbital pressure when the force is transmitted to the orbital floor, the area of least resistance.

145
Q

What foreign bodies can not be tolerated?

A

Foreign bodies that enter the orbit are usually tolerated, except for copper, iron, and vegetable materials, such as those from plants or trees, which may cause purulent infection

146
Q

Assessment for foreign bodies in the eye

A

The nurse examines the corneal surface for foreign bodies, wounds, and abrasions and then the other external structures of the eye. Pupillary size, shape, and light reaction of the pupil of the affected eye are compared with the other eye. The nurse assesses ocular motility (ability of the eyes to move synchronously up, down, right, and left).

147
Q

Orbital trauma: splash injury tx

A

irrigate with NS
abx
tetanus antitoxin

148
Q

Foreign Bodies and Corneal Abrasions: tx

A

Removal of foreign body
Antibiotic ointment and eye patch applied
Exam eye daily

149
Q

What should you avoid if you have foreign body in eye or corneal abrasion

A

corticosteriors

150
Q

Penetrating injuries and contusions of the eyeball: treatment

A

abx
eye sheilf
topical corticosteroids

151
Q

What should be avoided if you have a penetrating injury or contusion of the eyeball

A

aspirin

152
Q

what are ocular burns considered a severe injury

A

the penetrate the ocular tissues rapidly and continue to cause damage long after the initial injury is sustained

153
Q

Ocular burns: treatment

A

RINSE - tap water irrigation should be started on site before transport to ER

ER will irrigate with NS or neutral solution

Local anesthetic instilled, and a lid speculum is applied to overcome blepharospasm

154
Q

How is particulate matter removed from an ocular burn?

A

using moistened, cotton-tipped applicators and minimal pressure on the globe.

155
Q

How long does the eye need to be irrigated with an ocular burn

A

until the conjunctival pH normalizes (between 7.3 and 7.6). The pH of the corneal surface is checked by placing a pH paper strip in the fornix

156
Q

Conjunctivitis

A

Inflammation of the conjunctiva

157
Q

conjuctivitis: s/s

A

Discharge, lymphadenopathy, irritation, scratching or burning sensation, fullness around the eyes, crusting of the eyelids, visual blurring and photophobia

158
Q

Bacterial Conjunctivitis

A

Manifests with an acute onset of redness, burning, and discharge.

Chlamydial conjunctivitis includestrachomaand inclusion conjunctivitis

159
Q

Viral Conjunctivitis

A

Discharge is watery, and follicles are prominent

160
Q

Conjuctivitis: treatment

A

education
abx
eye drops / ointment

161
Q

management of conjunctivitis?

A

Good handwashing
Do not share makeup
Throw away remaining makeup so you do not reinfect self
No contacts
No sharing towels, washcloether
Eyedrops should not tough eye – new bottle if it does

162
Q

Orbital Cellulitis

A

inflammation of the tissues surrounding the eye

163
Q

orbital cellulitis: patho

A

result from bacterial, fungal, or viral inflammatory conditions of contiguous structures, such as the face, oropharynx, dental structures, or intracranial structures

164
Q

Orbital cellulitis: common causative organisms

A

staphylococci and streptococci in adults andH. influenzaein children.

165
Q

orbital cellulitis: s/s

A

The symptoms include pain, lid swelling, conjunctival edema,proptosis, and decreased ocular motility. With such edema, optic nerve compression can occur and IOP may increase

166
Q

orbital cellulitis: treatment

A

Immediate administration of high-dose, broad-spectrum, systemic antibiotics is indicated. Cultures and Gram-stained smears are obtained. Monitoring changes in visual acuity, degree of proptosis, central nervous system function (e.g., nausea, vomiting, fever, cognitive changes), displacement of the globe, extraocular movements, pupillary signs, and the fundus is extremely important.

167
Q

Enucleation

A

removal of eyeball – will leave muscles/soft tissue in orbitals

168
Q

Evisceration

A

removal of interocular contents – usually because severe ocular trauma (car accident, burn, those in military)

169
Q

Exenteration

A

removal of ENTIRE contents including soft tissue that is surrounding the eye

170
Q

Tympanic membran perforation

A

eardrum ruptures (dive into pool, altitude), should heal spontaneously but over time if it doesn’t be concerned about INFECTION

171
Q

Otosclerosis

A

results from formation of new or abnormal spongey bone.

172
Q

Middle ear masses

A

rare

173
Q

what can help relieve motion sickness?

A

antihistamine

174
Q

Menieres disease

A

abnormal inner ear fluid that causes a balance or increase pressure (vertigo most common s/s)

175
Q

What can be given to decrease dizziness?

A

meclizine

176
Q

Tinnitus cause

A

a lot of time medications such as furosemide if you push too fast

177
Q

Benign Paroxysmal Positional Vertigo - treatment

A

bedrest

try to clean out ear cannal

178
Q

ototoxicity cause

A

usually from meds causing issues in cochlea or cranial nerve 8

179
Q

Acoustic neuroma

A

slow growing benign tumor of cranial nerve 8. something we monitor – not a lot you can do for it