Week 3 Memory and Amnesia Flashcards

1
Q

definition of amnesia

A

acquired impairment of explicit long term mem

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2
Q

two types of amnesia + causes

A

neurogenic - brain lesions
psychogenic - psych factors

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3
Q

anterograde amnesia

A

Impaired recall/recognition of facts/personal episodes after brain damage

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4
Q

retrograde amnesia

A

Impaired recall/recognition of memories of facts/personal episodes before brain damage

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5
Q

what memory impaired/spared in amnesia

A

short-term + implicit learning + mem preserved
some long-term can be spared e.g. procedural/semantic

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6
Q

global amnesia

A

combined verbal and non-verbal mem disorder

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7
Q

pure amnesia

A

absence of associated cog impairments
affects recall and recognition

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8
Q

causes of amnesia

A

traumatic head injury (concussion)
stroke (ischemia)
substance abuse / alcoholism
alzheimer’s
vitamin deficiency / malnutrition
stress

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9
Q

diff between STM and LTM

A

STM - mem for info currently in mind + limited
LTM _ stored info not always consciously accessible, virtually unlimited

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10
Q

declarative LTM and two subgroups

A

declarative - explicit
two types:
events (episodic, personal exp)
facts (semantic, knowledge)

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11
Q

non-declarative LTM + 4 subgroups

A

implicit
4 types:
procedural (motor/cog skills)
perceptual representation system (perceptual priming)
classical conditioning (conditioned responses between 2 stim)
non-associative learning (habituation sensitization)

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12
Q

who was HM + disease + therapy

A

suffered epilepsy in 50s
radical therapy involving temporal lobectomy surgery to remove temporal lobes bilaterally, including hippocampus

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13
Q

what happened to HM post-surgery

A

severe anterograde amnesia caused by medial temporal damage

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14
Q

double dissociation

A

2 related mental processes shown to function independently

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15
Q

case KC + parts that were damaged + impact on memory

A

motorcycle accident = widespread brain damage
large bilateral hippocampal lesions
sharp dissociation between intact semantic and impaired episodic mem

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16
Q

why does location of brain damage impact the resulting deficit

A

different areas = different aspects of mem
diff lesions = diff patterns of spared/impaired mem functions

17
Q

what lesions result in episodic mem loss

A

hippocampal formation

18
Q

what lesions result in semantic mem loss

A

parahippocampal region

19
Q

how are declarative mem systems tested

A

intentional recollection
recall/recognition tasks

20
Q

how are non declarative memory systems tested

A

performance in priming tasks

21
Q

how are non declarative memory systems tested

A

performance in priming tasks

22
Q

what elements of mem linked to frontal lobes

A

prefrontal cortex of frontal lobes = maintenance/manipulation of info
short term working mem
encoding, retrieval, evaluation of info stored in long term
patients with PFC lesions = disorganized mem

23
Q

what are confabulations following PFC damage?

A

false/self-contradictory mems patients beleive are real
associated with damage to diff regions than in classical amnesia