week 2 Flashcards

1
Q

Hydrostatic Pressure

A

Force within a fluid compartment

Major force that pushes water out of vascular system at capillary level

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2
Q

Oncotic Pressure

A

Osmotic pressure exerted by colloids in solution

Protein is a major colloid.

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3
Q

Fluid Shifts AND EDEMA

A

Plasma-to-interstitial fluid shift results in edema.

Elevation of hydrostatic pressure
Decrease in plasma oncotic pressure
Elevation of interstitial oncotic pressure

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4
Q

Effects of Edema (cont’d.)

A

-Functional impairment:
Restricts range of joint movement
Reduced vital capacity
Impaired diastole

-Pain:
Edema exerts pressure on nerves locally.
Headache with cerebral edema
Stretching of capsule in organs (kidney, liver)

-Dental practice:
Difficult to take accurate impressions
Dentures do not fit well

-Edema in skin:
Susceptible to tissue breakdown from pressure
Impaired arterial circulation
Ischemia leading to tissue breakdown

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5
Q

Fluid Movement Between ECF and ICF

A

-Water deficit (increased ECF)
Associated with symptoms that result from cell shrinkage as water is pulled into vascular system

-Water excess (decreased ECF)
Develops from gain or retention of excess water

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6
Q

Fluid Spacing

A

First spacing
Normal distribution of fluid in ICF and ECF

Second spacing
Abnormal accumulation of interstitial fluid (edema)

Third spacing
Fluid accumulation in part of body where it is not easily exchanged with ECF

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7
Q

Hypothalamic Regulation

A
  • Osmoreceptors in hypothalamus sense fluid deficit or increase.
  • -Stimulates thirst and antidiuretic hormone (ADH) release
  • -Result in increased free water and decreased plasma osmolarity
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8
Q

Pituitary Regulation

A

Under control of hypothalamus, posterior pituitary releases ADH.
Stress, nausea, nicotine, and morphine also stimulate ADH release.

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9
Q

Adrenal Cortical Regulation

A

Releases hormones to regulate water and electrolytes
Glucocorticoids: Cortisol
Mineralocorticoids: Aldosterone

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10
Q

Renal Regulation

A

Primary organs for regulating fluid and electrolyte balance

-Adjusting urine volume: Selective reabsorption of water and electrolytes. Renal tubules are sites of action of ADH and aldosterone.

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11
Q

Cardiac Regulation

A

Natriuretic peptides are antagonists to the RAAS.

  • -Produced by cardiomyocytes in response to increased atrial pressure
  • -Suppress secretion of aldosterone, renin, and ADH to decrease blood volume and pressure
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12
Q

Gastrointestinal Regulation

A

Oral intake accounts for most water.

Small amounts of water are eliminated by gastrointestinal tract in feces.

Diarrhea and vomiting can lead to significant fluid and electrolyte loss.

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13
Q

Insensible Water Loss

A

Invisible vaporization from lungs and skin to regulate body temperature

  • -Approximately 600 to 900 mL/day is lost.
  • -No electrolytes are lost.
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14
Q

Age-Related Considerations

A

Structural changes in kidneys decrease ability to conserve water.

Hormonal changes lead to decrease in ADH and ANP.

Loss of subcutaneous tissue leads to increased loss of moisture.

Reduced thirst mechanism results in decreased fluid intake.

Nurse must assess for these changes and implement treatment accordingly.

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15
Q

Age-Related Considerations

A

Structural changes in kidneys decrease ability to conserve water.

Hormonal changes lead to decrease in ADH and ANP.

Loss of subcutaneous tissue leads to increased loss of moisture.

Reduced thirst mechanism results in decreased fluid intake.

Nurse must assess for these changes and implement treatment accordingly.

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16
Q

Fluid and Electrolyte Imbalances

A

Common in most patients with major illness or injury

  • -Directly caused by illness or disease (burns or heart failure)
  • -Result of therapeutic measures (IV fluid replacement or diuretics)
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17
Q

Extracellular Fluid Volume Imbalances

A

ECF volume deficit (hypovolemia)

  • -Abnormal loss of normal body fluids (diarrhea, fistula drainage, hemorrhage), inadequate intake, or plasma-to-interstitial fluid shift
  • -Treatment: Replace water and electrolytes with balanced IV solutions.

Fluid volume excess (hypervolemia)

  • -Excessive intake of fluids, abnormal retention of fluids (HF), or interstitial-to-plasma fluid shift
  • -Treatment: Remove fluid without changing electrolyte composition or osmolality of ECF.
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18
Q

Nursing Management Nursing Diagnoses

A
-Hypovolemia
Deficient fluid volume
Decreased cardiac output
Risk for deficient fluid volume
Potential complication: Hypovolemic shock
-Hypervolemia
Excess fluid volume
Risk for imbalanced fluid volume
Ineffective airway clearance
Risk for impaired skin integrity
Disturbed body image
Potential complications: Pulmonary edema, ascites
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19
Q

Fluid Deficit―Dehydration

A

-Insufficient body fluid
Inadequate intake
Excessive loss
Both

-Fluid loss often measured by change in body weight

-Dehydration more serious in infants and older adults
Water loss may be accompanied by loss of electrolytes and proteins (e.g., diarrhea).

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20
Q

Causes of Dehydration

A
  • Vomiting and diarrhea
  • Excessive sweating with loss of sodium and water
  • Diabetic ketoacidosis (Loss of fluid, electrolytes, and glucose in the urine)
  • Insufficient water intake in older adults or unconscious persons
  • Use of concentrated formula in infants
21
Q

Effects of Dehydration

A
  • Dry mucous membranes in the mouth
  • Decreased skin turgor or elasticity
  • Lower blood pressure, weak pulse, and fatigue
  • Decreased mental function, confusion, loss of consciousness
22
Q

Manifestations of Dehydration

A
Decreased skin turgor and dry mucous membranes
Sunken eyes
Sunken fontanelles in infant 
Lower blood pressure, rapid weak pulse
Increased hematocrit
Increased temperature
Decreasing level of consciousness
Urine―low volume & high specific gravity
23
Q

Nursing Management Nursing Implementation

A
I & O
Monitor cardiovascular changes.
Assess respiratory changes.
Daily weights
Skin assessment

Neurological function:
LOC,PERLA, Voluntary movement of extremities, Muscle strength, Reflexes

24
Q

Calcium (Ca)

A
Excess:
Hypercalcemia
Thirst
CNS deterioration
Increased interstitial fluid
Deficit:
Hypocalcemia
Tetany
Chvostek’s, Trousseau’s signs 
Muscle twitching
CNS changes
ECG changes
25
Q

Magnesium (Mg)

A
EXCESS:
Hypermagnesemia 
Loss of deep tendon reflexes (DTRs)
Depression of CNS
Depression of neuromuscular function

DEFICIT:
Hypomagnesemia
Hyperactive DTRs
CNS changes

26
Q

Sodium (Na)

A
EXCESS:
Hypernatremia
Thirst
CNS deterioration
Increased interstitial fluid

DEFICIT:
Hyponatremia
CNS deterioration

27
Q

Potassium (K)

A
EXCESS:
Hyperkalemia
Ventricular fibrillation
ECG changes
CNS changes
DEFICIT:
Hypokalemia 
Bradycardia
ECG changes 
CNS changes
28
Q

Sodium

A

-Imbalances typically associated with parallel changes in osmolality

  • Plays a major role in:
  • -ECF volume and concentration
  • -Generation and transmission of nerve impulses
  • -Acid-base balance
29
Q

Hypernatremia

A

Elevated serum sodium occurring with water loss or sodium gain

Causes hyperosmolality leading to cellular dehydration

Primary protection is thirst from hypothalamus.

Manifestations
Thirst, lethargy, agitation, seizures, and coma

Impaired LOC

Produced by clinical states
Central or nephrogenic diabetes insipidus

30
Q

Hyponatremia

A

Results from loss of sodium-containing fluids or from water excess.

Manifestations
Confusion, nausea, vomiting, seizures, and coma

31
Q

Potassium

A

Major ICF cation

Necessary for
Transmission and conduction of nerve and muscle impulses 
Cellular growth
Maintenance of cardiac rhythms
Acid-base balance
Sources 
Fruits and vegetables (bananas and oranges)
Salt substitutes 
Potassium medications (PO, IV)
Stored blood
32
Q

Hyperkalemia

A

High serum potassium caused by
Massive intake
Impaired renal excretion
Shift from ICF to ECF

Most common in renal failure

Manifestations:
Cramping leg pain
Weak or paralyzed skeletal muscles
Ventricular fibrillation or cardiac standstill 
Abdominal cramping or diarrhea
33
Q

Hypokalemia

A

Low serum potassium caused by:

  • Abnormal losses of K+ via the kidneys or gastrointestinal tract
  • Magnesium deficiency
  • Metabolic alkalosis
Manifestations
Most serious are cardiac.
Skeletal muscle weakness (legs)
Weakness of respiratory muscles
Decreased gastrointestinal motility
Impaired regulation of arteriolar blood flow
34
Q

Calcium

A

Obtained from ingested foods
More than 99% combined with phosphorus and concentrated in skeletal system
Inverse relationship with phosphorus
Bones are readily available store

Functions::
Transmission of nerve impulses
Myocardial contractions
Blood clotting
Formation of teeth and bone
Muscle contractions
35
Q

Hypercalcemia

A
High serum calcium levels caused by:
Hyperparathyroidism (two-thirds of cases)
Malignancy 
Vitamin D overdose
Prolonged immobilization
Manifestations:
Decreased memory
Confusion
Disorientation
Fatigue
Constipation
36
Q

Hypocalcemia

A

Low serum Ca levels caused by:

Decreased production of PTH
Acute pancreatitis 
Multiple blood transfusions 
Alkalosis
Decreased intake

Manifestations:

Positive Trousseau’s or Chvostek’s sign
Laryngeal stridor
Dysphagia
Tingling around the mouth or in the extremities

37
Q

Magnesium

A

50% to 60% contained in bone.

Coenzyme in metabolism of protein and carbohydrates

Factors that regulate calcium balance appear to influence magnesium balance.

Acts directly on myoneural junction
Important for normal cardiac function

38
Q

Hypermagnesemia

A

High serum Mg caused by:
Increased intake or ingestion of products containing magnesium when renal insufficiency or failure is present

Manifestations:
Lethargy or drowsiness
Nausea/vomiting
Impaired reflexes
Somnolence 
Respiratory and cardiac arrest
39
Q

Hypomagnesemia

A
Low serum Mg caused by::
Prolonged fasting or starvation
Chronic alcoholism
Fluid loss from gastrointestinal tract
Prolonged parenteral nutrition without supplementation
Diuretics
Manifestations::
Confusion
Hyperactive deep tendon reflexes
Tremors
Seizures 
Cardiac dysrhythmias
40
Q

Normal Saline (NS)

A
Isotonic 
No calories
More NaCl than ECF
30% stays in IV (most)
70% moves out of IV
Expands IV volume
--Preferred fluid for immediate response
--Risk for fluid overload higher
Does not change ICF volume
Blood products
Compatible with most medications
--
41
Q

Acid-Base Imbalance

A

Acidosis
Excess hydrogen ions
Decrease in serum pH

Alkalosis
Deficit of hydrogen ions
Increase in serum pH

42
Q

Respiratory Acidosis –

A

lungs can not get enough 02

Acute problems
Pneumonia, airway obstruction, chest injuries
Drugs that depress the respiratory control center

Chronic respiratory acidosis
Common with COPD

43
Q

Respiratory Alkalosis –

A

Blowing off too much C02

Hyperventilating
Caused by anxiety, high fever, overdose of aspirin
Head injuries
Brainstem tumor

44
Q

Metabolic Acidosis – 3 D’s

A
  1. Excessive loss of bicarbonate (HCO3) ions to buffer hydrogen
    - -Diarrhea―loss of HCO3 from intestines

2.Diabetic acidosis

3.Renal disease or failure Dialysis
Decreased excretion of acids
Decreased production of HCO3 ions

45
Q

Metabolic Alkalosis

A

Increase in serum bicarbonate ion

  • Loss of hydrochloric acid from stomach
  • Hypokalemia
  • Excessive ingestion of antacids
46
Q

Effects of Acidosis

A
-Impaired nervous system function
Headache
Lethargy
Weakness
Confusion
Coma and death

-Compensation
Deep rapid breathing
Secretion of urine with a low pH

47
Q

Effects of Alkalosis

A

Increased irritability of the nervous system causes:

Restlessness
Muscle twitching
Tingling and numbness of the fingers
Tetany
Seizures
Coma
48
Q

Treatment of Imbalances

A

Treatment of underlying cause

Immediate corrective measures to include fluid and electrolyte replacement or removal
Caution is required when adjusting fluid levels to ensure appropriate electrolyte balance.

Addition of bicarbonate to the blood to reverse acidosis

Modification of diet to maintain better electrolyte balance

49
Q

Hydrogen Ion and pH Scale

A
  1. 8= DEATH
  2. 35=ACIDOSIS AND INCREASE H+
  3. 4= NORMAL
  4. 45= ALKALOSIS AND DECREASE H+
  5. 8= DEATH