REPRODUCTIVE Flashcards

1
Q

Erectile Dysfunction

A

Inability to attain or maintain an erect penis

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2
Q

Etiology and pathophysiology

Erectile Dysfunction

A

Results from physiological or psychological factors

Normal physiological age-related changes are associated with erectile function

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3
Q

Clinical manifestations and complications

Erectile Dysfunction

A

Self-report of problems associated with sexual function

Major complications: inability to perform sexually; personal issues

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4
Q

Diagnostic studies

Erectile Dysfunction

A

Thorough sexual, health, psychosocial history
-Erection Quality Scale
Physical examination
Further examination and testing based on findings

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5
Q

Collaborative care

Erectile Dysfunction

A
Oral drug therapy
Vacuum constriction devices
Intraurethral devices
Penile implants
Sexual counseling
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6
Q

Nursing Management

Erectile Dysfunction

A

Emotional support for both patient and partner
Confidentiality
Counselling and therapy for both patient and partner
Provide a support system and accurate information

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7
Q

Structural Abnormalities: Uterus

A

Normal position of uterus
Slightly anteverted and anteflexed
Cervix downward and posterior

Retroflexion of uterus
Uterus tipped posteriorly
May be excessively curved or bent
Marked retroversion may cause back pain, dysmenorrhea, dyspareunia
In some cases, infertility may occur.

Uterine displacement or prolapse
First-degree prolapse if cervix drops into the vagina
Second-degree prolapse if cervix lies at opening to the vagina
Body of uterus is in the vagina
Third-degree prolapse if uterus and cervix protrude through the vaginal orifice
Early stages of prolapse may be asymptomatic.
Advanced stages cause discomfort, infection, and decreased mobility.

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8
Q

Rectocele

A

Protrusion of the rectum into the posterior vagina

May cause constipation and pain

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9
Q

Cystocele

A

Protrusion of bladder into the anterior vagina

May cause UTIs

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10
Q

If severe, conditions are treated surgically to increase the support of the pelvic ligaments.

A

.

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11
Q

Infertility

A

Inability to achieve conception despite 1 year of frequent, unprotected intercourse
Express concern and tactfulness with patient

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12
Q

Physical causes

A

Pretesticular, testicular, post-testicular

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13
Q

Cause may be a female condition, male condition, or a combination of both - 33% of cases involve male factors

A
Associated with hormonal imbalances
Age of parents
Structural abnormalities
Infections
Chemotherapy
Workplace toxins 
Other environmental factors
Idiopathic
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14
Q

Amenorrhea (absence of menstruation)

A

May be primary or secondary
Primary form may be genetic.
Secondary form usually hormonal imbalance

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15
Q

Dysmenorrhea

A

Painful menstruation caused by excessive release of prostaglandins as a result of endometrial ischemia
Usually begins a few days prior to menses & lasts a few days
NSAIDs offer relief.

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16
Q

Premenstrual syndrome

A

Approx 1 week prior to menses

Pathophysiology not completely known; may be several forms

Breast tenderness, weight gain, abdominal distension or bloating, irritability, emotional liability, sleep disturbances, depression, headache, fatigue

Treatment is individualized and may include exercise, limiting salt intake, use of oral contraceptives, diuretics, or antidepressant drugs.

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17
Q

Menorrhagia

A

Increased amount and duration of flow

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18
Q

Metrorrhagia

A

Bleeding between cycles

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19
Q

Polymenorrhea

A

Short cycles of less than 3 weeks

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20
Q

Oligomenorrhea

A

Long cycles of more than 6 weeks

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21
Q

Prostatitis

A

Infection or inflammation of the prostate gland

E. coli

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22
Q

Occurs in:

Prostatitis

A
Young men with UTIs
Older men with prostatic hypertrophy
In association with STDs
With instrumentation such as catheterization
Through bacteremia
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23
Q

Signs and symptoms

Prostatitis

A
Both acute and chronic forms manifested by dysuria, urinary frequency, and urgency
Decreased urinary stream
Acute form includes fever and chills
Lower back pain 
Leukocytosis
Abdominal discomfort
Systemic signs include fever, malaise, 
Anorexia
Muscle aches
Acute Bacterial – Fever, chills, back pain, perineal pain, acute urinary symptoms
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24
Q

Diagnostic studies

Prostatitis

A

Urinalysis and urine culture are indicated
White blood cell count and blood cultures in presence of fever
PSA test to rule out prostate cancer; levels may be elevated with prostatic inflammation
Microscopic evaluation and culture of expressed prostate secretion

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25
Nursing and Collaborative Management | Prostatitis
``` Antibiotics Pain management Treat acute urinary retention Prostatic massage High fluid intake Management of fever ```
26
Benign Prostatic Hyperplasia
Hyperplasia of prostatic tissue Palpated on DRE Occurs in up to 50% of men > 65 years Most common urological problem in males Etiology - not completely understood Related to estrogen–testosterone imbalance Compression of urethra and urinary obstruction Does not predispose to prostatic carcinoma
27
Benign Prostatic Hyperplasia S&S
``` Symptoms usually gradual in onset Early symptoms usually minimal because bladder can compensate Worsen as obstruction increases • Obstructed urinary flow • Hesitancy in starting flow • Dribbling • Decreased flow strength • Increased frequency and urgency • Nocturia • Dysuria occurs if infection is present. ```
28
Diagnostic studies | BPH
``` History and physical examination Digital rectal examination (DRE) Urinalysis with culture Prostate-specific antigen (PSA) level Serum creatinine Transrectal ultrasonography (TRUS) scan Uroflowmetry Cystourethroscopy ```
29
Collaborative care | BPH
``` Goals: • Restore bladder drainage • Relieve symptoms • Prevent and treat complications Watchful waiting Dietary changes Timed voiding schedule ```
30
Drug therapy: | BPH
Offers symptomatic relief of BPH • slow enlargement dutasteride (Avodart) • Smooth muscle relaxers tamsulosin (Flomax) • Reduces progression of hypertrophy Combination of finasteride (Proscar) and doxazosin (Cardura)
31
Invasive therapy | BPH
Transurethral resection of the prostate (TURP) Transurethral incision of the prostate (TUIP) Prostatectomy
32
Minimally invasive therapy
Transurethral microwave thermotherapy (TUMT) Transurethral needle ablation (TUNA) Laser prostatectomy Intraprostatic urethral stents
33
Benign Prostatic Hyperplasia: Complications
Leads to frequent infections - Urinary tract infection (UTI) and sepsis, Incomplete bladder emptying with residual urine provides medium for bacterial growth Continued obstruction - distended bladder, dilated ureters, hydronephrosis, and renal failure if untreated. Acute urinary retention: common complication is indication for surgical intervention
34
Endometriosis
Presence of normal endometrial tissue in sites outside of the endometrial cavity. Ectopic endometrium responds to cyclical hormone changes. Bleeding leads to inflammation and pain
35
MANI AND ETIO
Wide range of clinical manifestations and severity | Etiology - not well understood
36
Infections: Pelvic Inflammatory Disease (PID)
Infection of uterus, fallopian tubes, and/or ovaries | Acute or chronic
37
PID Causes:
- Ascending infection from lower reproductive tract - ---Untreated cervicitis - ---Chlamydia and gonococcal infection - Bacteremia - Sexually transmitted diseases - Nonsterile abortions, - Childbirth.
38
Infections: PID: Manifestations
Pelvic pain is usually first sign (Lower abdominal) ``` Increased temperature - fever, chills, Guarding Nausea and vomiting Leukocytosis Purulent vaginal discharge may be present. ```
39
PID | treatment
Treatment usually requires aggressive antibiotic therapy in hospital.
40
Infections: PID: Complications
Scarring of tubes increases risk of infertility and ectopic pregnancy. ``` Potential acute complications: Peritonitis Pelvic abscesses Septic shock Infertility Chronic Pelvic Pain ```
41
Benign Tumours of the Female Reproductive System
Leiomyomas (Fibroids) Cervical polyps Benign ovarian tumours (Polycystic ovary syndrome (PCOS)
42
Leiomyoma (fibroids)
``` • Benign tumor of the myometrium • Common during the reproductive years • Classified by location • Usually multiple, well-defined, unencapsulated masses o Abnormal bleeding may occur. o May interfere with implantation • Often asymptomatic until large growth • Hormonal therapy or surgery ```
43
STI: Bacterial | Chlamydial infections
* most common STIs * Chlamydia trachomatis Males • urethritis = dysuria, itching, white discharge from penis • epididymitis = painful, swollen scrotum, usually unilateral, fever • inguinal lymph nodes swollen Females • Often asymptomatic until PID or infertility develops • Newborns may be infected during birth. •
44
Gonorrhea
Neisseria gonorrheae Many strains have become resistant to penicillin and tetracycline. Second most frequently occurring STI Direct physical contact with infected host Males 20-24 Inflamed urethra Some males are asymptomatic. Females 15-19 Frequently asymptomatic PID and infertility are serious complications. May infect the eyes of the newborn, causing irreversible damage and blindness May spread systemically to cause septic arthritis
45
STIs: Syphilis
Treponema pallidum, ``` Primary stage: Presence of chancre at site of infection --Genital region --Anus --Oral cavity Painless, firm, ulcerated nodule Occurs about 3 weeks after exposure Lesion heals spontaneously but client is still contagious ``` Secondary stage: If untreated, a flulike illness occurs, with a widespread symmetrical rash—self-limited but client remains contagious Latent stage: May persist for years Transmission may occur. Tertiary syphilis—irreversible changes Gummas in organs and major blood vessels Dementia, blindness, motor disabilities
46
STIs: Syphilis (Cont.)
Organism can be transmitted to fetus in utero Baby born with tertiary syphilis changes that are not reversible Treatment is usually antimicrobial drugs. Increase in antibiotic resistant strains causing an increase in prevalence
47
STIs: Viral Infections | Genital herpes—herpes simplex
• Caused by HSV-2 or HSV-1 • HSV-1 possible with oral sex • Lesions similar to HSV-1 • Recurrent outbreaks of blister-like vesicles on the genitalia o Preceded by tingling or itching sensation o Lesions are extremely painful. • After acute stage, virus migrates back to dorsal root ganglion • Infectivity greater when symptoms are present
48
Sexually Transmitted Infections
Can be bacterial or viral | Usually start as lesions on genitals or mucous membranes and can spread to other areas
49
Contributing factors to STI rates
``` Earlier reproductive maturity Longer sexual life span Greater sexual freedom Media emphasis Lack of barrier methods during sexual activity ```
50
oral contraceptives
Oral contraceptive effects on acidity of vaginal/cervical secretions promote growth of certain organisms, causing STIs.
51
GonorrheaEtiology and Pathophysiology
Easily killed by drying, heating, or washing with antiseptic Incubation period: 3 to 8 days Provides no immunity to subsequent reinfection
52
Men | and gonorrhea
``` Initial site infection is urethra. Symptoms Develop 2 to 5 days after infection Dysuria Profuse, purulent urethral discharge Unusual to be asymptomatic ``` complications: Include prostatitis, urethral strictures, and sterility Often seek treatment early, so less likely to develop complications
53
Women and gonorrhea
Mostly asymptomatic or have minor symptoms Vaginal discharge Dysuria Frequency of urination After incubation: Redness and swelling occur at site of contact. Greenish, yellow purulent exudate often develops. May develop abscess Disease may remain local or may spread by tissue extension to uterus, fallopian tubes, and ovaries. complications: Include pelvic inflammatory disease (PID), Bartholin’s abscess, ectopic pregnancy, and infertility Usually asymptomatic, so seldom seek treatment
54
Gonorrhea | Clinical Manifestations
Anorectal gonorrhea: Usually from anal intercourse Few symptoms Include soreness, itching, and discharge of anus Orogenital: Few symptoms Gonococcal pharyngitis can develop.
55
Gonorrhea and babies
Eye infections in newborns Instillations of prophylactic erythromycin (0.5%) ophthalmic ointment or silver nitrate (0.1%) aqueous solution Untreated infants develop permanent blindness.
56
Gonorrhea | Diagnostic Studies
Must have culture to confirm diagnosis ``` History and physical examination Laboratory tests Gram-stained smear to identify organism Culture of discharge Nucleic acid amplification test Testing for other STIs ```
57
Gonorrhea | Collaborative Care
Drug therapy: Treatment generally instituted without culture results Treatment in early stage is curative. Most common- Oral dose of cefixime (Suprax) All sexual contacts of patients must be evaluated and treated. Patient should be counselled to abstain from sexual intercourse and alcohol during treatment. Re-examine if symptoms persist after treatment.
58
Syphilis
Mainly due to men who have sex with men
59
SyphilisEtiology and Pathophysiology
Caused by Treponema pallidum Spirochete bacterium Enters the body through breaks in skin or mucous membranes Facilitated by abrasions that occur during sexual intercourse Complex disease in which many organs and tissues can become infected Causes production of antibodies that react with normal tissues Not all exposures cause disease. Destroyed by drying, heating, or washing Incubation 10 to 90 days Spread in utero after 10th week of pregnancy(still birth or baby can die)
60
HIV-infected patients with syphilis appear to be at greatest risk for clinically significant central nervous system (CNS) involvement and may require more intensive treatment with penicillin than do other patients with syphilis.
.
61
Syphilis | Clinical Manifestations
Variety of signs/symptoms can mimic another disease. ``` Primary stage Chancres appear. Painless indurated lesions Occur 10 to 90 days after inoculation Lasting 3 to 6 weeks ```
62
Syphilis | Complications
Occur most often in late syphilis Gummas can produce irreparable damage to bone, liver, or skin. Aneurysm may press on structures such as intercostal nerves, causing pain. Sudden attacks of pain Loss of vision and sense of position
63
Syphilis | Diagnostic Studies
``` History, including sexual history PE Examine lesions. Note signs/symptoms. Dark-field microscopy Serological testing Testing for other STIs ```
64
Syphilis | Collaborative Care
Drug therapy: Benzathine penicillin G (Bicillin) Aqueous procaine penicillin G Recurring or persistent symptoms after drug therapy are re-treated.
65
Chlamydial Infections
Most prevalent bacterial STI in Canada Incidence is 2 times higher in women than in men. ``` Major contributor to: PID Ectopic pregnancy Infertility in women Nongonococcal urethritis in men ```
66
Chlamydial Infections | Etiology and Pathophysiology
Caused by Chlamydia trachomatis Gram-negative bacteria Largely underreported because infected persons are asymptomatic Transmitted during vaginal, anal, or oral sex
67
Risk factors of chlamydia
Women and adolescents New or multiple sexual partners Sexual partners who have had multiple partners
68
Chlamydial Infections | Clinical Manifestations
“Silent disease” Symptoms may be absent or minor. Infection often is not diagnosed until complications appear. Men: Urethritis- Dysuria and Urethral discharge Proctitis- Rectal discharge and Pain during defecation Epididymitis- Unilateral scrotal pain and Swelling Women: Cervicitis-Mucopurulent discharge and Hypertrophic ectopy Urethritis- Dysuria and Pyuria Bartholinitis-Purulent exudate Perihepatitis- Fever, nausea, vomiting, right upper quadrant pain PID
69
Chlamydial Infections | Diagnostic Studies
``` Laboratory tests: Nucleic acid amplification test (NAAT) Direct fluorescent antibody (DFA) Enzyme immunoassay (EIA) Testing for other STIs Culture for chlamydia ``` Cervical or urethral discharge less purulent, watery, and painful in chlamydia than in gonorrhea
70
Chlamydial InfectionsCollaborative Care
``` Drug therapy: Doxycycline (Vibramycin) 100 mg bid for 7 days Azithromycin (Zithromax) 1 g in single dose ``` Abstinence from sexual intercourse for 7 days after treatment Follow-up care for persistent symptoms Treatment of partners Encouraging use of condoms
71
Genital Herpes
Not a reportable disease in most provinces and territories True incidence difficult to determine One of the most common STIs in North America
72
Genital Herpes | Etiology and Pathophysiology
Caused by herpes simplex virus (HSV) Enters through mucous membranes or breaks in the skin during contact with infected persons HSV reproduces inside cell and spreads to surrounding cells. Virus enters peripheral or autonomic nerve endings. Ascends to sensory or autonomic nerve ganglion, where it is dormant Recurrence when virus descends to initial site of infection Persists for life Virus sheds even in absence of lesion.
73
Genital Herpes | Diagnostic Studies
History and physical examination Viral isolation by tissue culture Antibody assay for specific HSV viral type
74
Genital HerpesCollaborative Care
``` Drug therapy: Inhibit viral replication Suppress frequent recurrences Acyclovir (Zovirax) Valacyclovir (Valtrex) Famciclovir (Famvir) Not a cure, but shorten duration and healing time and reduce outbreaks ```
75
Genital Warts
Highly contagious Of adult population in Canada, at least 70% have at least one genital HPV infection over lifetime Caused by human papillomavirus (HPV) Highly contagious Frequently seen in young, sexually active adults
76
Genital Warts | Clinical Manifestations
White to grey and pink-fleshed coloured May form large cauliflower-like masses Itching may occur with anogenital warts. Bleeding on defecation may occur with anal warts.
77
genital warts and babies
Rapid growth with pregnancy | Transmitted to newborn
78
Genital Warts | Diagnostic Studies
Diagnosis on basis of appearance of lesions May be confused with other diseases Primary goal: removal of symptomatic warts Serological and cytological tests: HPV DNA test to determine if women with abnormal Pap test results need follow-up Identify women who are infected with high-risk HPV strains Recurrences and re-infection possible Careful long-term follow-up advised Vaccine to prevent cervical cancer, precancerous genital lesion, and genital warts due to HPV
79
Nursing Management | Nursing Assessment
``` Subjective data: Past medical history, including sexual history Medication use IV drug use Nausea/vomiting Dysuria Urethral discharge Burning lesions Vaginal discharge Presence of genital or perianal lesions ```
80
Objective data
``` Fever Visual assessment of lesions, warts, rash Purulent rectal discharge Proctitis Urethral and cervical discharge Laboratory findings ```
81
Nursing Management | Nursing Diagnoses
Risk for infection Anxiety Ineffective health maintenance
82
Nursing Management | Planning
Patient with STI will: Demonstrate understanding of mode of transmission and risks imposed Complete treatment and follow-up Notify or assist in notification of sexual contacts Abstain until infection is resolved Demonstrate knowledge of safer sex practices
83
Nursing Management | Nursing Implementation
Discuss practices with all patients. Screen for cervical cancer. Teach to inspect partner’s genitals. Some protection if void immediately after intercourse; wash genitalia and adjacent areas with soap and water
84
Nursing Management | Nursing Implementation
Proper use of condoms Avoiding sexual contact with HIV-infected persons Establishing risk of contracting STI Compassion and respect Screening programs Locating and examining all contacts of person with STI for testing and treatment Abstinence during treatment period, condoms afterward Avoid oral-genital contact.
85
Nursing Management | Evaluation
``` Patient with STI will: Demonstrate modes of transmission Use appropriate hygienic measures Experience no re-infection Demonstrate compliance with follow-up protocol ```