REPRODUCTIVE Flashcards

1
Q

Erectile Dysfunction

A

Inability to attain or maintain an erect penis

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2
Q

Etiology and pathophysiology

Erectile Dysfunction

A

Results from physiological or psychological factors

Normal physiological age-related changes are associated with erectile function

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3
Q

Clinical manifestations and complications

Erectile Dysfunction

A

Self-report of problems associated with sexual function

Major complications: inability to perform sexually; personal issues

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4
Q

Diagnostic studies

Erectile Dysfunction

A

Thorough sexual, health, psychosocial history
-Erection Quality Scale
Physical examination
Further examination and testing based on findings

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5
Q

Collaborative care

Erectile Dysfunction

A
Oral drug therapy
Vacuum constriction devices
Intraurethral devices
Penile implants
Sexual counseling
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6
Q

Nursing Management

Erectile Dysfunction

A

Emotional support for both patient and partner
Confidentiality
Counselling and therapy for both patient and partner
Provide a support system and accurate information

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7
Q

Structural Abnormalities: Uterus

A

Normal position of uterus
Slightly anteverted and anteflexed
Cervix downward and posterior

Retroflexion of uterus
Uterus tipped posteriorly
May be excessively curved or bent
Marked retroversion may cause back pain, dysmenorrhea, dyspareunia
In some cases, infertility may occur.

Uterine displacement or prolapse
First-degree prolapse if cervix drops into the vagina
Second-degree prolapse if cervix lies at opening to the vagina
Body of uterus is in the vagina
Third-degree prolapse if uterus and cervix protrude through the vaginal orifice
Early stages of prolapse may be asymptomatic.
Advanced stages cause discomfort, infection, and decreased mobility.

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8
Q

Rectocele

A

Protrusion of the rectum into the posterior vagina

May cause constipation and pain

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9
Q

Cystocele

A

Protrusion of bladder into the anterior vagina

May cause UTIs

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10
Q

If severe, conditions are treated surgically to increase the support of the pelvic ligaments.

A

.

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11
Q

Infertility

A

Inability to achieve conception despite 1 year of frequent, unprotected intercourse
Express concern and tactfulness with patient

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12
Q

Physical causes

A

Pretesticular, testicular, post-testicular

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13
Q

Cause may be a female condition, male condition, or a combination of both - 33% of cases involve male factors

A
Associated with hormonal imbalances
Age of parents
Structural abnormalities
Infections
Chemotherapy
Workplace toxins 
Other environmental factors
Idiopathic
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14
Q

Amenorrhea (absence of menstruation)

A

May be primary or secondary
Primary form may be genetic.
Secondary form usually hormonal imbalance

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15
Q

Dysmenorrhea

A

Painful menstruation caused by excessive release of prostaglandins as a result of endometrial ischemia
Usually begins a few days prior to menses & lasts a few days
NSAIDs offer relief.

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16
Q

Premenstrual syndrome

A

Approx 1 week prior to menses

Pathophysiology not completely known; may be several forms

Breast tenderness, weight gain, abdominal distension or bloating, irritability, emotional liability, sleep disturbances, depression, headache, fatigue

Treatment is individualized and may include exercise, limiting salt intake, use of oral contraceptives, diuretics, or antidepressant drugs.

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17
Q

Menorrhagia

A

Increased amount and duration of flow

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18
Q

Metrorrhagia

A

Bleeding between cycles

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19
Q

Polymenorrhea

A

Short cycles of less than 3 weeks

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20
Q

Oligomenorrhea

A

Long cycles of more than 6 weeks

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21
Q

Prostatitis

A

Infection or inflammation of the prostate gland

E. coli

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22
Q

Occurs in:

Prostatitis

A
Young men with UTIs
Older men with prostatic hypertrophy
In association with STDs
With instrumentation such as catheterization
Through bacteremia
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23
Q

Signs and symptoms

Prostatitis

A
Both acute and chronic forms manifested by dysuria, urinary frequency, and urgency
Decreased urinary stream
Acute form includes fever and chills
Lower back pain 
Leukocytosis
Abdominal discomfort
Systemic signs include fever, malaise, 
Anorexia
Muscle aches
Acute Bacterial – Fever, chills, back pain, perineal pain, acute urinary symptoms
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24
Q

Diagnostic studies

Prostatitis

A

Urinalysis and urine culture are indicated
White blood cell count and blood cultures in presence of fever
PSA test to rule out prostate cancer; levels may be elevated with prostatic inflammation
Microscopic evaluation and culture of expressed prostate secretion

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25
Q

Nursing and Collaborative Management

Prostatitis

A
Antibiotics
Pain management
Treat acute urinary retention
Prostatic massage
High fluid intake
Management of fever
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26
Q

Benign Prostatic Hyperplasia

A

Hyperplasia of prostatic tissue
Palpated on DRE

Occurs in up to 50% of men > 65 years
Most common urological problem in males
Etiology - not completely understood
Related to estrogen–testosterone imbalance

Compression of urethra and urinary obstruction
Does not predispose to prostatic carcinoma

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27
Q

Benign Prostatic Hyperplasia

S&S

A
Symptoms usually gradual in onset
Early symptoms usually minimal because bladder can compensate 
Worsen as obstruction increases
•	Obstructed urinary flow
•	Hesitancy in starting flow
•	Dribbling
•	Decreased flow strength
•	Increased frequency and urgency
•	Nocturia
•	Dysuria occurs if infection is present.
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28
Q

Diagnostic studies

BPH

A
History and physical examination
Digital rectal examination (DRE)
Urinalysis with culture
Prostate-specific antigen (PSA) level
Serum creatinine 
Transrectal ultrasonography (TRUS) scan
Uroflowmetry 
Cystourethroscopy
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29
Q

Collaborative care

BPH

A
Goals:
•	Restore bladder drainage 
•	Relieve symptoms
•	Prevent and treat complications
Watchful waiting
Dietary changes
Timed voiding schedule
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30
Q

Drug therapy:

BPH

A

Offers symptomatic relief of BPH
• slow enlargement dutasteride (Avodart)
• Smooth muscle relaxers tamsulosin (Flomax)
• Reduces progression of hypertrophy Combination of finasteride (Proscar) and doxazosin (Cardura)

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31
Q

Invasive therapy

BPH

A

Transurethral resection of the prostate (TURP)
Transurethral incision of the prostate (TUIP)
Prostatectomy

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32
Q

Minimally invasive therapy

A

Transurethral microwave thermotherapy (TUMT)
Transurethral needle ablation (TUNA)
Laser prostatectomy
Intraprostatic urethral stents

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33
Q

Benign Prostatic Hyperplasia: Complications

A

Leads to frequent infections - Urinary tract infection (UTI) and sepsis, Incomplete bladder emptying with residual urine provides medium for bacterial growth

Continued obstruction - distended bladder, dilated ureters, hydronephrosis, and renal failure if untreated.

Acute urinary retention: common complication is indication for surgical intervention

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34
Q

Endometriosis

A

Presence of normal endometrial tissue in sites outside of the endometrial cavity. Ectopic endometrium responds to cyclical hormone changes. Bleeding leads to inflammation and pain

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35
Q

MANI AND ETIO

A

Wide range of clinical manifestations and severity

Etiology - not well understood

36
Q

Infections: Pelvic Inflammatory Disease (PID)

A

Infection of uterus, fallopian tubes, and/or ovaries

Acute or chronic

37
Q

PID Causes:

A
  • Ascending infection from lower reproductive tract
  • —Untreated cervicitis
  • —Chlamydia and gonococcal infection
  • Bacteremia
  • Sexually transmitted diseases
  • Nonsterile abortions,
  • Childbirth.
38
Q

Infections: PID: Manifestations

A

Pelvic pain is usually first sign (Lower abdominal)

Increased temperature - fever, chills, 
Guarding
Nausea and vomiting
Leukocytosis
Purulent vaginal discharge may be present.
39
Q

PID

treatment

A

Treatment usually requires aggressive antibiotic therapy in hospital.

40
Q

Infections: PID: Complications

A

Scarring of tubes increases risk of infertility and ectopic pregnancy.

Potential acute complications:
Peritonitis
Pelvic abscesses
Septic shock
Infertility
Chronic Pelvic Pain
41
Q

Benign Tumours of the Female Reproductive System

A

Leiomyomas (Fibroids)

Cervical polyps

Benign ovarian tumours
(Polycystic ovary syndrome (PCOS)

42
Q

Leiomyoma (fibroids)

A
•	Benign tumor of the myometrium 
•	Common during the reproductive years
•	Classified by location 
•	Usually multiple, well-defined, unencapsulated masses
     o	Abnormal bleeding may occur.
     o	May interfere with implantation
•	Often asymptomatic until large growth
•	Hormonal therapy or surgery
43
Q

STI: Bacterial

Chlamydial infections

A
  • most common STIs
  • Chlamydia trachomatis

Males
• urethritis = dysuria, itching, white discharge from penis
• epididymitis = painful, swollen scrotum, usually unilateral, fever
• inguinal lymph nodes swollen

Females
• Often asymptomatic until PID or infertility develops
• Newborns may be infected during birth.

44
Q

Gonorrhea

A

Neisseria gonorrheae
Many strains have become resistant to penicillin and tetracycline.
Second most frequently occurring STI

Direct physical contact with infected host

Males 20-24
Inflamed urethra
Some males are asymptomatic.

Females 15-19
Frequently asymptomatic
PID and infertility are serious complications.

May infect the eyes of the newborn, causing irreversible damage and blindness

May spread systemically to cause septic arthritis

45
Q

STIs: Syphilis

A

Treponema pallidum,

Primary stage:
Presence of chancre at site of infection
--Genital region
--Anus
--Oral cavity
Painless, firm, ulcerated nodule
Occurs about 3 weeks after exposure
Lesion heals spontaneously but client is still contagious

Secondary stage:
If untreated, a flulike illness occurs, with a widespread symmetrical rash—self-limited but client remains contagious

Latent stage:
May persist for years
Transmission may occur.

Tertiary syphilis—irreversible changes
Gummas in organs and major blood vessels
Dementia, blindness, motor disabilities

46
Q

STIs: Syphilis (Cont.)

A

Organism can be transmitted to fetus in utero

Baby born with tertiary syphilis changes that are not reversible

Treatment is usually antimicrobial drugs.

Increase in antibiotic resistant strains causing an increase in prevalence

47
Q

STIs: Viral Infections

Genital herpes—herpes simplex

A

• Caused by HSV-2 or HSV-1
• HSV-1 possible with oral sex
• Lesions similar to HSV-1
• Recurrent outbreaks of blister-like vesicles on the genitalia
o Preceded by tingling or itching sensation
o Lesions are extremely painful.
• After acute stage, virus migrates back to dorsal root ganglion
• Infectivity greater when symptoms are present

48
Q

Sexually Transmitted Infections

A

Can be bacterial or viral

Usually start as lesions on genitals or mucous membranes and can spread to other areas

49
Q

Contributing factors to STI rates

A
Earlier reproductive maturity 
Longer sexual life span
Greater sexual freedom
Media emphasis
Lack of barrier methods during sexual activity
50
Q

oral contraceptives

A

Oral contraceptive effects on acidity of vaginal/cervical secretions promote growth of certain organisms, causing STIs.

51
Q

GonorrheaEtiology and Pathophysiology

A

Easily killed by drying, heating, or washing with antiseptic
Incubation period: 3 to 8 days
Provides no immunity to subsequent reinfection

52
Q

Men

and gonorrhea

A
Initial site infection is urethra.
Symptoms
Develop 2 to 5 days after infection 
Dysuria
Profuse, purulent urethral discharge 
Unusual to be asymptomatic 

complications:
Include prostatitis, urethral strictures, and sterility
Often seek treatment early, so less likely to develop complications

53
Q

Women and gonorrhea

A

Mostly asymptomatic or have minor symptoms
Vaginal discharge
Dysuria
Frequency of urination

After incubation:
Redness and swelling occur at site of contact.
Greenish, yellow purulent exudate often develops.
May develop abscess

Disease may remain local or may spread by tissue extension to uterus, fallopian tubes, and ovaries.

complications:
Include pelvic inflammatory disease (PID), Bartholin’s abscess, ectopic pregnancy, and infertility
Usually asymptomatic, so seldom seek treatment

54
Q

Gonorrhea

Clinical Manifestations

A

Anorectal gonorrhea:
Usually from anal intercourse
Few symptoms
Include soreness, itching, and discharge of anus

Orogenital:
Few symptoms
Gonococcal pharyngitis can develop.

55
Q

Gonorrhea and babies

A

Eye infections in newborns
Instillations of prophylactic erythromycin (0.5%) ophthalmic ointment or silver nitrate (0.1%) aqueous solution
Untreated infants develop permanent blindness.

56
Q

Gonorrhea

Diagnostic Studies

A

Must have culture to confirm diagnosis

History and physical examination
Laboratory tests
Gram-stained smear to identify organism
Culture of discharge
Nucleic acid amplification test 
Testing for other STIs
57
Q

Gonorrhea

Collaborative Care

A

Drug therapy:
Treatment generally instituted without culture results
Treatment in early stage is curative.
Most common- Oral dose of cefixime (Suprax)

All sexual contacts of patients must be evaluated and treated.
Patient should be counselled to abstain from sexual intercourse and alcohol during treatment.
Re-examine if symptoms persist after treatment.

58
Q

Syphilis

A

Mainly due to men who have sex with men

59
Q

SyphilisEtiology and Pathophysiology

A

Caused by Treponema pallidum
Spirochete bacterium

Enters the body through breaks in skin or mucous membranes
Facilitated by abrasions that occur during sexual intercourse

Complex disease in which many organs and tissues can become infected
Causes production of antibodies that react with normal tissues
Not all exposures cause disease.

Destroyed by drying, heating, or washing
Incubation 10 to 90 days
Spread in utero after 10th week of pregnancy(still birth or baby can die)

60
Q

HIV-infected patients with syphilis appear to be at greatest risk for clinically significant central nervous system (CNS) involvement and may require more intensive treatment with penicillin than do other patients with syphilis.

A

.

61
Q

Syphilis

Clinical Manifestations

A

Variety of signs/symptoms can mimic another disease.

Primary stage 
Chancres appear.
Painless indurated lesions 
Occur 10 to 90 days after inoculation
Lasting 3 to 6 weeks
62
Q

Syphilis

Complications

A

Occur most often in late syphilis
Gummas can produce irreparable damage to bone, liver, or skin.
Aneurysm may press on structures such as intercostal nerves, causing pain.

Sudden attacks of pain
Loss of vision and sense of position

63
Q

Syphilis

Diagnostic Studies

A
History, including sexual history
PE 
Examine lesions.
Note signs/symptoms.
Dark-field microscopy
Serological testing 
Testing for other STIs
64
Q

Syphilis

Collaborative Care

A

Drug therapy:
Benzathine penicillin G (Bicillin)
Aqueous procaine penicillin G
Recurring or persistent symptoms after drug therapy are re-treated.

65
Q

Chlamydial Infections

A

Most prevalent bacterial STI in Canada

Incidence is 2 times higher in women than in men.

Major contributor to:
PID
Ectopic pregnancy
Infertility in women
Nongonococcal urethritis in men
66
Q

Chlamydial Infections

Etiology and Pathophysiology

A

Caused by Chlamydia trachomatis
Gram-negative bacteria
Largely underreported because infected persons are asymptomatic
Transmitted during vaginal, anal, or oral sex

67
Q

Risk factors

of chlamydia

A

Women and adolescents
New or multiple sexual partners
Sexual partners who have had multiple partners

68
Q

Chlamydial Infections

Clinical Manifestations

A

“Silent disease”
Symptoms may be absent or minor.
Infection often is not diagnosed until complications appear.

Men:
Urethritis- Dysuria and Urethral discharge
Proctitis- Rectal discharge and Pain during defecation
Epididymitis- Unilateral scrotal pain and Swelling

Women:
Cervicitis-Mucopurulent discharge and Hypertrophic ectopy
Urethritis- Dysuria and Pyuria
Bartholinitis-Purulent exudate
Perihepatitis- Fever, nausea, vomiting, right upper quadrant pain
PID

69
Q

Chlamydial Infections

Diagnostic Studies

A
Laboratory tests:
Nucleic acid amplification test (NAAT)
Direct fluorescent antibody (DFA)
Enzyme immunoassay (EIA)
Testing for other STIs
Culture for chlamydia

Cervical or urethral discharge less purulent, watery, and painful in chlamydia than in gonorrhea

70
Q

Chlamydial InfectionsCollaborative Care

A
Drug therapy:
Doxycycline (Vibramycin)
100 mg bid for 7 days
Azithromycin (Zithromax)
1 g in single dose

Abstinence from sexual intercourse for 7 days after treatment
Follow-up care for persistent symptoms
Treatment of partners
Encouraging use of condoms

71
Q

Genital Herpes

A

Not a reportable disease in most provinces and territories
True incidence difficult to determine
One of the most common STIs in North America

72
Q

Genital Herpes

Etiology and Pathophysiology

A

Caused by herpes simplex virus (HSV)

Enters through mucous membranes or breaks in the skin during contact with infected persons

HSV reproduces inside cell and spreads to surrounding cells.

Virus enters peripheral or autonomic nerve endings.

Ascends to sensory or autonomic nerve ganglion, where it is dormant

Recurrence when virus descends to initial site of infection

Persists for life

Virus sheds even in absence of lesion.

73
Q

Genital Herpes

Diagnostic Studies

A

History and physical examination
Viral isolation by tissue culture
Antibody assay for specific HSV viral type

74
Q

Genital HerpesCollaborative Care

A
Drug therapy:
Inhibit viral replication 
Suppress frequent recurrences
Acyclovir (Zovirax)
Valacyclovir (Valtrex)
Famciclovir (Famvir)
Not a cure, but shorten duration and healing time and reduce outbreaks
75
Q

Genital Warts

A

Highly contagious
Of adult population in Canada, at least 70% have at least one genital HPV infection over lifetime

Caused by human papillomavirus (HPV)
Highly contagious
Frequently seen in young, sexually active adults

76
Q

Genital Warts

Clinical Manifestations

A

White to grey and pink-fleshed coloured
May form large cauliflower-like masses
Itching may occur with anogenital warts.
Bleeding on defecation may occur with anal warts.

77
Q

genital warts and babies

A

Rapid growth with pregnancy

Transmitted to newborn

78
Q

Genital Warts

Diagnostic Studies

A

Diagnosis on basis of appearance of lesions
May be confused with other diseases

Primary goal: removal of symptomatic warts

Serological and cytological tests:
HPV DNA test to determine if women with abnormal Pap test results need follow-up
Identify women who are infected with high-risk HPV strains

Recurrences and re-infection possible
Careful long-term follow-up advised
Vaccine to prevent cervical cancer, precancerous genital lesion, and genital warts due to HPV

79
Q

Nursing Management

Nursing Assessment

A
Subjective data:
Past medical history, including sexual history
Medication use
IV drug use
Nausea/vomiting 
Dysuria
Urethral discharge
Burning lesions
Vaginal discharge
Presence of genital or perianal lesions
80
Q

Objective data

A
Fever
Visual assessment of lesions, warts, rash
Purulent rectal discharge
Proctitis
Urethral and cervical discharge
Laboratory findings
81
Q

Nursing Management

Nursing Diagnoses

A

Risk for infection
Anxiety
Ineffective health maintenance

82
Q

Nursing Management

Planning

A

Patient with STI will:
Demonstrate understanding of mode of transmission and risks imposed
Complete treatment and follow-up
Notify or assist in notification of sexual contacts
Abstain until infection is resolved
Demonstrate knowledge of safer sex practices

83
Q

Nursing Management

Nursing Implementation

A

Discuss practices with all patients.

Screen for cervical cancer.

Teach to inspect partner’s genitals.

Some protection if void immediately after intercourse; wash genitalia and adjacent areas with soap and water

84
Q

Nursing Management

Nursing Implementation

A

Proper use of condoms

Avoiding sexual contact with HIV-infected persons

Establishing risk of contracting STI

Compassion and respect

Screening programs

Locating and examining all contacts of person with STI for testing and treatment

Abstinence during treatment period, condoms afterward

Avoid oral-genital contact.

85
Q

Nursing Management

Evaluation

A
Patient with STI will:
Demonstrate modes of transmission
Use appropriate hygienic measures
Experience no re-infection
Demonstrate compliance with follow-up protocol