Week 15 Flashcards

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1
Q

Conversion to LTM

A
  • suggestions that STM components are converted into their corresponding LTM components (e.g. phonological STM –> phonological LTM)
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2
Q

Phonological LTM

A
  • supports out ability to identify spoken words
  • when we are born we have no associations between words, the sounding of the words need to be stored
  • phonological STM patients have difficulty learning new words in LTM –> have a poor vocabulary
  • difference between acquired cases (that still have a wide vocab but cannot add new words to LTM) and developmental cases
  • double dissociation proves there are subsets of this LTM (pure word deafness vs auditory agnosia –> separates out words from environmental/non=language sounds)
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3
Q

Visual LTM

A
  • supports our ability to identify visual information, including written words, objects, faces etc.
  • poor visual STM found to lead to poor route knowledge in LTM
  • double dissociation proves there are two types of visual LTM (one for words and one for faces)
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4
Q

Semantic LTM

A
  • our knowledge of meaning and function of words and objects, plus semantic memory supports inferences
  • semantic STM deficits lead to difficulty in learning new concepts and problems in understanding and producing complex meaningful sentences
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5
Q

Procedural memory

A
  • learning skills over many trials to acquire new skills (e.g. riding a bike)
  • amnesic patients are often quite skilled in procedural memory despite deficits in episodic memory (e.g. patient HM)
  • double dissociation as PD patients selectively have their procedural learning affected but their episodic memory is fine
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6
Q

Episodic memory

A
  • links memories from various LTM systems to restore a record of a personal event
  • involves visual, phonological + semantic STM and LTM systems which are integrated to form a single long term memory of a particular event
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7
Q

Single dissociation

A
  • an experimental manipulation or neurological impairment that affects performance on task 1 more than task 2
  • suggesting different areas of the brain are active in different tasks
  • but this does not necessarily prove the link as there may be other explanations
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8
Q

Double dissociation

A
  • an additional experimental manipulation or neurological impairment that affects performance on task 2 but not on task 1
  • rules out any other secondary arguments and can therefore prove links that systems work independently on different activities
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9
Q

Dissociations between episodic LTM and phonological STM

A
  • anterograde amnesic patients have poor episodic memory. but good functioning phonological STM
  • whereas phonological STM patients have poor phonological STM but good episodic memory
  • double dissociation suggests that STM and episodic memory are different systems
  • patients with deficits in their phonological STM may have deficits in their phonological LTM but their episodic memory can be supported by other subsystems
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10
Q

Encoding episodic memory

A
  • more important to have full attention at the encoding stage rather than at the retrieval stage
  • proven experimentally, that it is more important to pay more attention at the encoding stage than at the recall stage
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11
Q

Maintenance rehearsal

A
  • keeping information active in the STM by relying on the phonological loop
  • thought that the longer you store something in STM the more likely it is to pass into LTM –> found to not be true, just maintaining something in STM does not necessarily encode it in LTM
  • not enough to store it if it hasn’t been given a meaningful explanation within the brain
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12
Q

Elaborative rehearsal

A
  • encoding the meaning of information generally leads to better episodic memory storage of it
  • level-of-processing framework: the idea that the more deeply you encode a memory (and the more meaning you give it) the longer the memory will be retained
  • shown that digit span can improve when using elaborative encoding technique (by giving each digit meaning)
  • how we encode things is affected by our background knowledge and existing memories so people may remember different things from the same experience
  • utilises the frontal lobe (link to existing semantic knowledge and relationships between things) plus visual cortex of occipital lobe (converting information into mental pictures)
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13
Q

Picture superiority effect

A

we encode pictorial information much better than verbal information

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14
Q

Concreteness effect

A

words with a “concrete” meaning (e.g. car, house) are much better remembered than abstract words (e.g. truth, betrayal) –> links to pictorial effect as concrete words can be imagined in picture format

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15
Q

Dual code theory

A
  • information is better recalled when stored in at least two forms (verbal/linguistic code and a mental image code)
  • methods include mnemonics and method of loci
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16
Q

Consolidation

A
  • the process of converting memories into a format resistant to forgetting
  • encoding cannot be the full store of memory as amnesic patients can encode to deep levels but are still unable to remember
17
Q

Short-term consolidation

A
  • occurs over seconds and minutes
  • involves the hippocampus linking and integrating information from all various LTM systems to form an episodic memory
  • short-term retrograde amnesia shows that brain trauma erases memories encoded a few minutes before the injury (i.e. despite the memories being encoded, there was a lack of short-term consolidation to form the full memory so it is not remembered)
18
Q

Long-term consolidation

A
  • occurs over months and years
  • belief that long-term consolidation leads to encoding directly in the cortex for storage (so damage to hippocampus will not erase old episodic memories as they have moved to cortex)
  • observed through temporally graded retrograde amnesia (explains why this extends several years back to memories that have not had the chance to be fully consolidated yet; but memories from long-term past are still present)
19
Q

Multiple-memory trace (MMT) hypothesis

A
  • this theory believes that episodic memories are always encoded in the hippocampus and do not move to the cortex
  • belief that very old memories have been recounted more often and therefore re-stored in the hippocampus
  • according to this theory, severity and extent of retrograde amnesia depends on the extent of the hippocampal lesion
  • fMRI showed that hippocampus is involved in both old and recent memories and that there is no evidence that information has been moved to the cortex
  • N.B. possibility that memories are copied over into the cortex but also still remain in hippocampus
20
Q

Retrieval

A

retrieval is less affected by divided attention than the encoding stage (suggesting an automatic component)

21
Q

Korsakoff amnesia

A
  • associated with frontal lesions
  • free recall is more affected than recognition memory
  • also suffer from confabulation (false memories but in the form of honest lying)
22
Q

Automatic retrieval

A
  • hippocampus can retrieve information relatively automatically with strong retrieval cues (automatic retrieval with clear cues)
  • memory associations within the hippocampus “pop out” in memory (sometimes correct but sometimes not - confabulations)
  • hippocampus cannot correct itself so we need another system to correct false memories
23
Q

Effortful/strategic retrieval

A
  • initiated in the frontal cortex and is required for free recall (i.e. if you are not given any strong retrieval cues so the hippocampus struggles)
  • helps prevent false/confabulated memories as it can monitor errors made by the hippocampus
  • frontal system is the “boss” of the memory system as it controls the information presented to the hippocampus at encoding and guides retrieval and monitors information retrieved from the hippocampus
  • analogous to central executive in working memory
24
Q

Encoding specificity

A
  • the effectiveness of a retrieval cue depends on how well it relates to the initial encoding strategy
  • explains both state and mood dependent memory (why retrieval success depends on environment)
  • may also explain childhood amnesia (why you do not remember the first years of your life as that was such a different encoding environment to anything we can imagine as an adult; but smell is a good retrieval cue as this does not change much!)
  • also related to the testing effect (practising retrieval, e.g. practise exam questions, improves memory) –> supports the fact that you are practising retrieving information so practising in the test environment
  • also explains amnesia between different personalities in multiple personality disorder
25
Q

Why are some memories forgotten?

A
  • poor encoding in the first place
  • presence of a poor retrieval cue
  • poor consolidation (e.g. in amnesia)
  • loss of storage (acquisition of new memories can interfere with and potentially erase previously stored memories + false memories can take up space)
26
Q

Memory distortion in encoding

A
  • various studies have shown that episodic memory for faces is better for own-race, own-gender etc. (relevant for eye-witness testimony)
  • may be partially explained by being an expert at the categories that are the same as yourself –> leading to better encoding
  • not innate but just based on learned visual experience (e.g. adoptives of one race that have moved country will recognise people of their “new” country better)
27
Q

Memory distortions at retrieval

A
  • different wording to retrieval cues can make a significant difference to the answers people give and the memories people retrieve
  • even when encoding stage was identical
  • note: unclear whether false memories occur at encoding or retrieval stage (e.g. critical lures)
  • also that semantic encoding of knowledge may lead to both useful encoding of true memories but also contribute to errors in memory
  • eyewitness memory itself may not be unreliable as long as the recall conditions are suitable (e.g. not asking suggestive questions, open vs closed ended questions)
28
Q

Retrieval induced forgetting paradigm

A
  • highlights possible mechanism for repression of memories, e.g. in traumatic events
  • initial test that memory is worse for words from a non-practised category compared to others (e.g. possible to focus and rehearse certain aspects of one side of a memory and forget another)
  • over time, induced forgetting goes away so all items are remembered to the same level
  • however it is also proven that inducing false memories is a high possibility so it is not necessarily that the memory has been repressed but may also be false
29
Q

Explicit memory

A

the conscious retrieval of content from past experiences

30
Q

Implicit memory

A

refers to the unconscious influences of past experiences on later behaviour and performance

31
Q

Priming

A

enhanced ability to think of a stimulus as a result of recent exposure to the stimulus

32
Q

Blocking

A

occurs when stored information is temporarily inaccessible (i.e. we know we have the memory but are unable to retrieve it)

33
Q

Absentmindedness

A

results from failures of attention, shallow encoding and influence of automatic behaviours

34
Q

Memory’s mindbugs (Schacter’s seven sins)

A
  • transience, blocking, absentmindedness, misattribution, suggestibility, bias and persistence
  • thought of as the cost we pay for the benefits that allow memory to work as well as it does most of the time