Week 1 - Breathlessness ('dysponea') Flashcards

1
Q

What are common cardiorespiratory problems?

A

Dyspnoea (+/- cough, +/- cyanosis)
Sneezing/nasal discharge
Respiratory noise
Collapse, weak, exercise intolerance
Heart murmur +/- other clinical signs
Dysrhythmia +/- other clinical signs

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2
Q

What are the two respiratory noises that are heard?

A

Stridor
Stertor

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3
Q

What is a stridor?

A

Stridor is a high-pitched sound that results from rigid tissue vibrations.

Problem at the level of or below the larynx

It is typically associated with laryngeal or tracheal disease.
- Laryngeal paralysis
- Tracheal collapse

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4
Q

What is a stertor

A

Stertor is noisy breathing that occurs during inhalation.

It is a low-pitched, snoring type of sound that usually arises from the vibration of fluid, or the vibration of tissue that is relaxed or flabby.

Problem is above the larynx and noise created at the back of the throat.

It usually arises from airway blockage in the throat (pharynx).

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5
Q

What part of ‘signalment’ can impact the order of differentials the most?

A

Age

e.g An older dog would move neoplasia up the list, but a younger dog would move neoplasia down and grass seed FB up the list.

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6
Q

With a dyponeic animal, what should be assessed in the consult room?

A

Observe = clinically stable or emergency admission?

Condition
Breathing (rate, pattern, regularity, depth, effort)
MM colour (pale, cyanotic, normal)
Behaviours that are worrying to the owner.

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7
Q

What is a normal breathing pattern and effort?

A

Inspiratory phase is longer than expiratory.

Effort is minimal

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8
Q

How is the breathing pattern and effort effected in upper respiratory tract disease?

A

Slow respiratory rate

Exaggerated inspiratory effort (longer phase)

Inspiratory effort increased

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9
Q

How is the breathing pattern and effort effected in lower respiratory tract disease?

A

Fast and shallow breathes

Both phases of breathing altered.

Expiratory phase can become longer than inspiratory -> less elasticity -> harder to push air out.

Fibrotic lung disease = increases inspiratory effort due to reduced long compliance.

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10
Q

How is the breathing pattern and effort effected in pleural disease?

A

Loss of pleural adhesion increased required effort to breathe.

Inspiratory and expiratory effort increased.

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11
Q

Define tachypnea

A

Increased respiratory rate

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12
Q

Define hyperpnoea

A

Increased respiratory effort

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13
Q

Define orthopnoea

A

Dyspnoea in any position other than standing or erect sitting.

Lying down can push abdomen forward making it difficult to breathe.

Common cause: bilateral pulmonary oedema.

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14
Q

Define trepopnoea

A

Dyspnoea in one lateral recumbency but not the other.

Common cause: unilateral lung or pleural disease, unilateral airway obstruction, unilateral pleural effusion.

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15
Q

What three things should be done on a thoracic examination?

A
  1. Thoracic palpation
    - Look for presence of apex beat (displaces to R = mass in the L), rhonchi, masses deformities, pain (e.g rib fractures)
  2. Thoracic auscultation
    - hindered by purring/panting
    - both sides of stethoscope
  3. Normal sounds
    - Inspiratory = soft, low pitched
    - Expiratory = none or softer/lower pitched
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16
Q

What abnormal sounds could be heard in the lungs?

A

Crackles (rales) = dry or moist

Wheezes (high pitched)

Rhonchi (low pitched_

17
Q

What is a crackle indicative of?

A

Moist crackle = congestive heart failure (CHF), more prominent on inspiration, resp distress

Dry crackle = acute or chronic

18
Q

What is a wheeze or rhonchi indicative of?

A

Narrowing of airway (bronchi/trachea)

Most commonly expiration (but can be inspiration)

19
Q

When percussing the chest, what are you looking for?

A

Difference in tympanic sounds if there is a difference in density.

Can be normal, increased, decreased.

e.g Pleural effusion… percussion becomes dull below fluid line, normal above (as were air is).

20
Q

What are common respiratory tract investigations?

A

History
Clinical exam
Haem and biochem (Serum Pro-BNP concentration increases with heart failure)
Imagining (radiograph, CT, ultrasound, MRI)
Tracheal wash, BAL
Bronchoscopy
Lung FNA/Biopsy (RARE)

21
Q

What are the four areas that can cause dyspnoea?

A
  1. Upper respiratory tract
  2. Pleural Space
  3. Lungs
  4. Non-cardiorespiratory cause
22
Q

What 4 things characterise upper respiratory disease?

A
  1. Inspiratory difficulty
  2. Audible noise
  3. Mostly surgical
  4. Emergency tracheostomy
23
Q

What 4 things characterise pleural space disease?

A
  1. Characteristic respiratory pattern
  2. Muffled heart and lung sounds
  3. Ultrasound thorax -> identify fluid or air?
  4. Remove fluid
24
Q

What 2 things characterise lung disease?

A
  1. Stuff in the alveoli (blood, pus parasite, fluid ect).
  2. Stuff in the intersititium

Often, there is no respiratory noise.

25
Q

Out of the four areas that can cause dyspnoea, which could lead to a cough and cyanosis if severe?

A

Cough + cyanosis
1. URT (cyanosis most common)
2. Pleural space
3. Lung

26
Q

If there is a cough, where could the problem be?

A

There are cough receptors in any airway up to the terminal bronchioles.

So could be:
1. URT
2. Pleural space
3. Lung

27
Q

What 23things characterise non-cardiorespiratory problems?

A
  1. Often metabolic/physiologic
  2. Rapid, shallow breathing
  3. Rarely severe difficulty
28
Q

What are common clinical signs for metabolic/physiologic condition?

A

Open-mouthed
Panting
Shallow breathing

29
Q

What are some metabolic/physiologic conditions that can cause dyspnea?

A

Hyperthermia/heat stroke
Obesity
Excitement/fear/shock
Stress/pain
Parturition/eclampsia/false pregnancy
Anemia
Acidosis
CNS disease
Endocrine (hyperAC, hyperT4)
Neuromuscular

30
Q

What are some metabolic/physiologic commonly presents with acute onset dyspnoea?

A

Pulmonary thromboembolism
- Few radiographic signs
- Hyper-coagulable states (trauma, sepsis, HAC, hypo T4, IMHA)
- Pulmonary hypertension

Dog don’t form clots easily so something is prompting clot formation.