WAD & Headache - Exam 2 Flashcards

1
Q

Alar ligamentous tears:
– runs from?
– weaker than?
– unique S&S?

A

– dens up and lateral to foramen magnum
– transverse ligament
– splinting, particularly with SB due to immediate tension on ligament. Possible cord S&S due to loss of dens stability

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2
Q

Transverse ligament tears:
– stronger than?
– keeps dens from moving ___ & contacting ___
– unique S&S?

A

– dens
– keeps dens from moving posterior and contacting cord
– splinting and likely cord S&S with forward nodding

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3
Q

what is a rim lesion?

A

horizontal tear of anterior annulus close to end plate - think of ligament rupture

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4
Q

rim lesion is often misdiagnosed as?

A

rare anterior disc herniation

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5
Q

what is the cause of rim lesion?

A

excessive hyperextension

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6
Q

unique S&S of rim lesion

A

splinting, particularly with extension due to tension on torn anterior annulus
pain with compression (end plate) and distraction (annulus)

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7
Q

what are symptoms of WAD?

A
  • trauma with acute neck and interscapular referred P!
  • potential trigeminocervical nucleus (TCN) symptoms
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8
Q

what would you notice in a scan for WAD?
– observation:
– ROM:
– resisted/MMT:
– neuro:
– stress tests:

A

– likely splinting
– limited with empty and painful end feels in several if not all directions
– weak and painful in several directions
– + findings, including cord or cranial n involvement
– + for involved tissue

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9
Q

what would a biomechanical exam show with immobilization/disuse?

A

joint hypomobility with accessory motion
fibrotic scarring

  • excessive scarring –> hypomobile
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10
Q

what would a biomechanical exam show if no prolonged immobilization or fibrotic scarring?

A

hypermobility due to laxity

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11
Q

what is trigeminocervical nucleus (TCN)?
-___________ may develop
-location?
-the interaction of?

A

nociplastic P! may develop
located at C2, 3
interaction of sensory nerve fibers of trigeminal nerve and upper cervical spinal nerves

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12
Q

in TCN, what areas experience symptoms from inflammation and/or sensitization?

A

head, face and neck

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13
Q

describe the symptoms at each of the 8 areas TCN causes symptoms
1 - mandibular n.
2. ophthalmic n.
3. maxillary n.
4. CV region (C1-3 spinal n.)
5. head (C1-3 spinal n)
6. face (C1-3 spinal n)
7. jaw (C1-3 spinal n)

A

1 - tongue - altered taste/tingling
- ear - P!/tinnitus/hypersensitivity to sound
2. eye - P!/conjunctivitis without red eye/visual deficits
3. tooth ache/P!
4. upper cervical P!
5. headache aka cervicogenic HA
dizziness aka cervicogenic dizziness
paresthesia’s
6. p!/paresthesia’s
7. TMJ P!

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14
Q

TCN also receives sensory input from what nerve? where is its nucleus?

A

vagus N
C3, 4

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15
Q

What does the vagus nerve promote with TCN? (3)
What symptoms of parasympathetic system dysfunction? (6)

A

calmness, relaxation, digestion

irregular HR, lack of sweating, dyspnea, nausea, indigestion, GI S&S
——coordination can be influenced too

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16
Q

general Rx for WAD:

A

1 - POLICED (soft collar)
2 - improve joint mechanics and stabilize
- cervical & thoracic JM/manipulation
- deep neck flexor and scapular stabilizer exercises
- if nociplastic P!: body awareness and stabilization exercises
– 90 minute sessions, 2x/wk, 10-16 weeks

17
Q

if a younger individual with a low disability at baseline presents with WAD, what is their prognosis?

A

~50% P! free at 6 months and 8/10 fully recovered

18
Q

true or false. following MVA, 50% report symptoms of WAD up to 2 years

A

true

19
Q

HA with other suspicious S&S (dizziness) require what type of referral?

all other HA - perform what?

A

urgent or even emergency

perform MSK scan and biomechanical exam

20
Q

primary HA are due to what? what are the 3 types of primary HA?

A

due to HA condition itself

tension, migraine, cluster

21
Q

what are secondary HA due to? what is the type?

A

another source
cervicogenic

22
Q

symptoms of tension headaches?

A

BAND
bilateral band-like tightness
anxiety/stress etiology
no migraine S&S - milder
dull pressure

23
Q

tension HA:
- prevalence:
- cause:

A
  • ~40% of HAs
  • anxiety/stress can have associated muscle tension
24
Q

what would a PT do for a tension HA?

A

address anxiety/stress
moderate quality evidence for MET plus biofeedback
oscillations or manipulations (tension doesn’t like prolonged tension)

25
Q

what are symptoms of a migraine?

A

POUNDS
pulsating
out of commission (mod-severe P! up to 3 days)
unilateral
N&V
dromes - constitutional S&S
sensational auras with visual and/or auditory sensitivity

26
Q

migraine:
- prevalence
- cause:

A
  • 10-15% of headaches
  • temporal artery vasodilation
    trigeminal n. nociplastic P! with CV dysfunction
27
Q

what would a PT do for a migraine?

A

address CV dysfunction
vasoconstriction of temporal arteries - caffeine
increase water intake - 1.5 L/day
2-3 mg melatonin prior to bedtime
nociplastic P! MET

28
Q

what are symptoms for a cluster HA?

A

CRUSHING
comes and goes
retro-orbital and temporal regions
unilateral
sudden and severe P!
horners syndrome
intense
grumpy

29
Q

how common are cluster headaches?

A

not common
< 1% of HA

30
Q

cervicogenic headaches:
- prevalence:
- pathophysiology:

A
  • 15-20%
  • C2 and/or C3 joint dysfunction due to trigeminal cervical influence
31
Q

what are symptoms of cervicogenic headaches?

A

unilateral
starting in neck/occipital region, progressing to fronto-ocular region
provoked by neck motion (movement of spine)
mild to mod pain
non-throbbing/pulsating

32
Q

what are signs of a cervicogenic headache?

A

limited and painful A/PROM
possible + combined motion
possible + neuro hypersensitivity
C2 and C3 joint dysfunction
- hypo and/or hypermobility: + linear stress test
+ cervical flexion rotation test
+ TTP (tenderness to palpation) in O-C3 region

33
Q

what should a PT do to treat cervicogenic headaches?

A

address cervical dysfunction – address what we find
dry needling - not recommended as stand alone Rx, use with MT and exercise