W12 Perio classification continued Flashcards

1
Q

What is a healthy periodontium?

A

Absense of signs and symptoms of gingival and destructive periodontal diseases, or any tissue status outside normal range.

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2
Q

What does the periodontium look like in health? Clinical and histologically

A
CLINICALLY
Colour: pink, resilient in consistency.
Gingival margin: scalloped outline
Interdental papilla: firm, knife-edge, fills space.
Bleeding: nil
Sulcus: Probing depths between 1-3mm.
Histiologically: 
JE: JE attached along CEJ and part of the enamel
Gingival fibres: intact
Alveolar bone: crest intact
PDL fibres: intact
Cementum: normal
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3
Q

How does a. Clinical gingival health on an intact periodontium health appear clinically?

A

Gingival health: Defined as <10% bleeding sites with <3mm probing depth.
Intact periodontium: characterised by absence of BOP, erythema and edema, symptoms, CAL and bone loss. Physiological bone levels range from 1.0-3.0mm apical to the CEJ.

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4
Q

How does b. Clinical gingival health on an reduced periodontium health appear clinically?

A

Gingival health: defined as <10% bleeding sites with <3mm probing depths.
Reduced periodontium: characterized by an absence of BOP edema and pt symptoms, in the presence of reduced CAL and bone levels

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5
Q

How does b. clinical gingival health on a reduced periodontium appear clinically?

A

Successfully treated and stable periodontitis pts remain at in ↑ risk of recurrent progression of periodontitis.
Stable periodontitis: stability is characterized by successful to through control of local and systematic risk factors, resulting in minimal (<10%) BOP, no probing depths of 4mm or greater than BOP, optimal improvement in other clinical parameters and lack of progressive periodontal destruction.
NON-PERIODONTITIS PT.: Refers to recession and crown lengthening

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6
Q

What does gingivitis look like clinically and histologically?

A

Colour: red - redish/blue
Gingival margin: swollen, may cover more of the crown
Interdental papillla: bulbous, swollen
Bleeding : present
Sulcus: Probing depth 1-3mm, can be greater due to swelling only.
HISTOLOGICALLY
JE: JE attached along CEJ and part of enamel, has undergone some coronal detachment.
Gingival fibres: damage is present but reversible
Alveola bone: crest intact
PDL fibres: intact
Cementum: normal

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7
Q

How does Gingivitis dental biofilm induced a. associated with dental biofilm alone appear clinically?

A

Inflammation is consistent with the amount of plaque present.
>10% BOP <3mm
Localized gingivitis 10%-30% bleeding sites
Generalized gingivitis >30% bleeding cites.

No other local or systemic factors preent that could modify the host response

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8
Q

How does dental biofilm induced gingivitis b. Mediated by systemic or local risk factors appear?

A

Predispose a pt to plaque accumulation and induced gingivitis as a result.
Can also impact upon its rate of progression.

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9
Q

What are some systemic factors that impact dental biofilm-induced gingivitis?

A

Smoking, metabolic factors, nutritional factors, pharmacological factors, elevations in sex steroid hormones (menopause) (pregnancy) , hematological conditions

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10
Q

What are some local risks factors that impact dental biofilm-induced gingivitis?

A

Local risk factors encourages plaque accumulation at a site.
Biolfilm retention factors: subginigival restoration margins.
Hyposalivation
- Reduced cleansing of teeth, reduced dental biofilm removal, enhanced gingival inflammation, incompetent lip seal, medications

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11
Q

What are some drug-induced gingival enlargement that impact dental biofilm-induced gingivitis?

A

It is plaque bacteria in conjuctions with any of the following drugs.

  • Anti epileptic drugs (phenytoin and sodium valporate).
  • Certain calcium channel blockers
  • Immunoregulating drugs (cyclsporine)
  • High does oral contraceptives

Usually occurs within the first 3 months of medication, in younger age groups and observed in the anterior dentition/papilla first

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12
Q

What are non-dental biofilm induced gingival diseases?

A

Less common than plaque induced, areas not directly caused by plaque.
Clinical course may be exacerbated by the presence of plaque and gingival inflammation.
Eg. Fungal infections, allergic reactions, Vit C deficiencies, tooth-brushing trauma

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