Vocal Folds Flashcards
5 layers covering vocal folds
Epithelium Superficial Lamina Propria Intermediate Lamina Propria Deep Lamina propria Vocalis muscle (medial thyroarytenoid
Hoarseness
abnormal voice changes, breathy, raspy, strained, weak
Dysphonia
general alteration of voice quality. Usually a laryngeal source
Dysarthria
defect in rhythm, enunciation, articulation. Usually a neurological or
muscular source
Stridor
- large airway noise from obstruction
- Inspiratory – supraglottic, extrathoracic
- Expiratory – tracheal, large bronchi intrathoracic
- Biphasic – laryngeal, immediate subglottis
Stertor
snoring sound from nose, nasopharynx, throat
Wheezing
pulmonary from smaller airways
Hoarseness-Causes
Viral laryngitis – acute Reflux - chronic Vocal abuse Allergies, PND Chronic cough Nodules Polyps Trauma Age Neurological disorders Smoking without malignancy Malignancies of thyroid, larynx, lungs Others
Vocal Fold Cysts
Etiology – Trauma, previous injury Hemorrhagic verses mucous – Recurrence Treatment – Therapy, can require surgery Reactive masses – The other side – Often resolves with therapy
Vocal Fold Polyps
Etiology
– Trauma, predisposition
Hemorrhagic verses fibrotic
– affects how likely it is to resolve on own
Treatment
– Usually requires surgical intervention for resolution
Granulomas
Contact verses vocal process Etiology – Reflux – Vocal Abuse Treatment – Correct abuses – Surgery /Botox
Reinke’s Edema
Smoker – Usual but not always Edema, Erythema Treatment – Correct cause – removal of excess material
Vocal Fold Hemorrhage
Etiology
– Trauma may be mild (cough, one scream, etc..)
Treatment
– Strict Voice Rest
Resolution
– Depends on technique and individual healing
– Usually complete
Vocal Fold Tear
Etiology – Same as hemorrhage or intubation Treatment – Same as hemorrhage Resolution
Sulcuses and Webs
Definitions – Sulcus: groove in VF – Web: anterior fusing of VF Congenital or Traumatic Can be asymptotic Treatment – Difficult surgical problems if symptomatic
Presbyphonia and Vocal Fold Bowing
Aging and the Voice – What’s normal? What’s not? Superior Laryngeal paralysis can be a cause Treatment – Increase VF bulk (therapy, surgery) Prevention – Good techniques, exercise
Laryngeal Trauma
Skeleton Injuries Arytenoid Dislocations – Etiology Intubation, trauma – Treatment Quick diagnosis critical Cricothyroid Joint Injuries
Immobile Vocal Fold
Remember Nerve Path w/u includes CT scan of skull base through aortic arch with contrast Laryngeal EMG Intubation Trauma -> dislocation Rheumatoid Arthritis Relapsing Polychondritis Most reliable method of differentiating between paralysis and fixation is laryngeal electromyography (LEMG). Normal implies fixation Abnormal implies paralysis
Laryngopharyngeal Reflux
Hoarseness Chronic cough Foreign Body sensation (globus) Tracheal Stenosis Chronic ear disease? Chronic sinusitis?
Symptoms of reflux include
bad breath or bitter taste in a.m.
a.m. hoarseness or after meals
sensation of a lump in the throat (globus)
sensation of post-nasal drip but no nasal issues
heartburn not always present
Precancerous and Cancerous Lesions
Smoker/ Drinker, but not always Early treatment important Stage and type important Treatment – Conservative Surgery – Radiation
Papillomas
Adult verses Juvenile onset Etiology – Viral Treatment – Lifelong disease Transformation to cancer occurs in some
Vocal Cord Dysfunction (VCD)
Diagnosed by fiberoptic laryngoscopy
Bronchoprovocation studies can exacerbate VCD.
VCD may worsen with bronchoprovocation, but will not change FEV1 or PC20
Acute treatment is anxiolytics, helium-oxygen mixture
Long term treatment is speech therapy, underlying triggers
Avoid over treating asthma