Veterinary Parasitology Flashcards

1
Q

What is the morphology of ancylostoma caninum

A

It is reddish Grey in color depending on whether it has fed, it also has a hook like posture
FeMales are 15mm- 20mm
Males are 12mm

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2
Q

What is the life cycle of ancylostoma caninum

A

They have a direct life cycle
The egg hatch and develop to L3 within 5 days.
Infection is by skin pentetration and ingestion
Paratenic hosts can be important
In percutaneous infection, larva migrate from bloodstream to the lungs where they hatch to l4 in the bronchi and trachea
They are swallowed and passed to small intestine where final moulting occurs.
If infection is by ingestion, the larvae may either penetrate the buccal
mucosa and undergo the pulmonary migration described above
❑ Or pass directly to the intestine where the adult worms bury their buccal
capsules into the mucosa
They have a prepatent period of 14-21 days.
The worms are prolific egg layers and may pass millions of eggs in faceces daily for several weeks

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3
Q

The pathogenesis of Ancylostoma caninum

A

Hemorrhagic anemia
The disease is seen in dogs under 1 year old, infected by the transmammary route, they are susceptible due to their low iron reserves

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4
Q

The clinical signs of ancylostoma caninum

A

Respiratory embarrassment, anemia, lassitude, respiratory embarrassment

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5
Q

Prepatent period of Ancylostoma caninum

A

14-21 days

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6
Q

Ancylostoma caninum and Ancylostoma braziliense predilection site

A

Small intestine

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7
Q

Ancylostoma braziliense gross morphology

A

Smaller than Ancylostoma caninum
Males measure 7.5mm in length
Female 10mm in length

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8
Q

Route of infection of ancylostoma braziliense

A

Percutaneous route and oral route not transmammary

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9
Q

Route of infection of ancylostoma braziliense

A

Percutaneous route and oral route not transmammary

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10
Q

Life cycle of ancylostoma braziliense

A

Similar in many respects to A. caninum, using both oral and percutaneous routesof infection, but transmammary transmission has not been demonstrated.

❑ Rodents can act as paratenic hosts.
❑ The prepatent period is about 2 weeks in the dog and cat.

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11
Q

Main importance of Ancylostoma braziliense

A

They are the primarily cause of cutaneous larva migrants CLM

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12
Q

What is the pathogenesis of Ancylostoma Braziliense

A
  1. Hypoalbuminaemia through an intestinal leak of plasma
  2. It’s the primary cause of Creeping larva migrans
    3 CLM is characterized by turtuous erythematous inflammatory tracts caused by infective larva penetrating the skin and wondering in the dermis
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13
Q

Ancylostoma tubaeforme has what host

A

Cat

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14
Q

Common name of ancylostoma tubaeforme

A

Feline hook worm

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15
Q

What are the host for ancylostoma ceylanicum

A

Cat, dog,wild felida and man

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16
Q

Uncinaria stenocephala gross morphology

A

It is a small worm up to 1cm long
7.5mm for male
10 for female

17
Q

What is difference between the life cycle of Uncinaria stenocephala and Ancylostoma caninum

A

There is NO PULMONARY MIGRATION

18
Q

What is difference between the life cycle of Uncinaria stenocephala and Ancylostoma caninum

A

There is NO PULMONARY MIGRATION

19
Q

Life cycle of Uncinaria stenocephala

A

Infection with infective L3 by oral ingestion without pulmonary migration.
Infective larvae can penetrate the skin, infection rarely matures.
No transmammary or intrauterine transmission
Carnivores may become infected via consumption of Paratenic host such as mice. Prepatent period is 15days

20
Q

Clinical signs of Uncinaria stenocephala

A

Anemia, lethargy, anorexia,, interdigital dermatitis,diarrhea ALAID

21
Q

Diagnosis of Uncinaria stenocephala

A

In areas where A. caninum is absent, the clinical signs of the patent infection,
together with the demonstration of strongyle eggs in the faeces, is indicative of
uncinariosis.

22
Q

Second diagnosis of Uncinaria stenocephala

A

Where ancylostoma is endemic as well, they require larva culture, although treatment is similar

23
Q

Second diagnosis of Uncinaria stenocephala

A

Where ancylostoma is endemic as well, they require larva culture, although treatment is similar

24
Q

Control of Uncinaria stenocephala

A

Regular anthelmintic therapy and hygiene
The combination of ivermectin and pyrantel panoate give high efficacy
The pedal dermatitis respond poorly to symptomatic infection but regress in the absence of reinfection