vestib - objective exam Flashcards

1
Q

what are the 5 main components of the vestib objective exam

A
  1. oculomotor exam
  2. head thrust / head impulse test
  3. head shaking induced nystagmus test
  4. dynamic visual acuity test (DVA)
  5. positional testing of SCC
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2
Q

what are 8 things to look at in the oculomotor exam

A

ocular alignment
EOM
smooth pursuits
saccades
vergence
GEN
spontaneous nystagmus
VORc

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3
Q

what are 4 types of misalignments seen in resting eye position

A

esotropia
exotropia
hypotropia
hypertropia

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4
Q

ocular alignment: normal vs abnormal (and likely cause)

A

normal: position symmetrical
abnormal: misalignment
- spontaneous nystagmus (peripheral or central lesion)

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5
Q

what is the function of normal EOM

A

eyes move in concert
-> brain takes input from each and puts it together to form a single image

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6
Q

what is the test to screen EOM

A

test 6 cardinal positions in H pattern holding tip of finger 18-24in from eyes

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7
Q

EOM: normal vs abnormal (and likely cause)

A

normal: able to follow target thru full range

abnormal: dec mobility in eye
- CN III, IV, VI lesion
- potential lesion of parietal/occipital lobe, pons, cerebellum

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8
Q

what does smooth pursuits measure

A

person’s ability to accurately track a visual target in a smooth controlled manner
- looking for coordination

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9
Q

what is the task for smooth pursuits

A

single point target held parallel to ground 18-24in from eyes
- person follows single point target as slowly moves horizontally and vertically

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10
Q

smooth pursuits: normal vs abnormal (and likely cause)

A

normal: accurately track visual target in smooth controlled manner w both eyes
- eyes remained fixated on moving object

abnormal: eye mvmt not controlled
- consistent saccadic intrusions
- GEN (closer to end range)

(+) central dysfunction

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11
Q

what are saccades

A

normal rapid, ballistic mvmts of eyes that abruptly change point of fixation
- amplitude of mvmt changes depending on task

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12
Q

how are saccades tested/assessed

A

person repeatedly change gaze fixation from one target to another
- assessed both horizontally and vertically

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13
Q

saccades: normal vs abnormal (and likely cause)

A

normal: eyes should be conjugate, have normal amplitude and brisk velocity

abnormal: consistently overshoots (hypermetric) or undershoots (hypometric) w multiple small saccades to get to target

*(+) central path

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14
Q

what is vergence and how is it tested

A

simultaneous mvmt of both eyes in opposite directions to obtain or maintain single binocular vision

person focuses on tip of finger while slowly moves toward pt nose

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15
Q

vergence: normal vs abnormal (and likely cause)

A

normal: eyes should converge symmetrically and pupils should constrict
- NPC <4’’

abnormal: eye mvmt asymmetrical
- NPC >4’’

*(+) indicate central path
- some people show ocular deviation at baseline, 6% of gen pop has convergence insufficiency

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16
Q

what is spontaneous nystagmus

A

nystagmus at rest w fixed gaze

17
Q

why are frenzel lenses a good tool to utilize when testing for spontaneous nystagmus

A

help to block out light
- makes it so pt can’t fixate on object to suppress nystagmus -> CNS can suppress peripheral causing spontaneous nystagmus by fixating on target

also makes eyeballs larger to more accurately identify mvmt

18
Q

what is spontaneous nystagmus a key clinical sign in

A

pts w acute vestibular syndrome (AVS)

19
Q

what are causes of spontaneous nystagmus and what are ways to diff dx

A

acute unilateral peripheral hypofunction
- dec w gaze fixation
- resolves w/i 3-7days

central lesion (brainstem, cerebellar abnormality)
- doesn’t dec w gaze fixation
- doesn’t resolve w/i days and is chronic in nature

20
Q

what is rebound nystagmus

A

keeps going back and forth w/o a slow or fast beat phase

21
Q

how is GEN/holding nystagmus assessed

A

watch pt in primary gaze and then in lateral gaze (usually 30deg to each side)
- assess direction and # of beats

22
Q

GEN: normal vs abnormal

A

normal: <1-2 beats in end range
- present in >50% of gen pop w normal vision
- GEN normal in people >65yo bc eye ms lose ability to tonically hold end range

abnormal: sustained GEN > 2 beats

23
Q

GEN of a central vs peripheral origin

A

central: dominant characteristic is direction changing nystagmus, pure vertical/torsional
- could be rebound

peripheral: consistently beats away form affected side (toward intact side)
- non-direction changing
- can be torsional
- may be suppressed w gaze fixation

24
Q

what is VOR cancellation and how is this assessed

A

in order to shift direction of gaze along w head motion, VOR needs to be overridden/cancelled

assess person’s ability to maintain fixation on target while head and target are moving

25
Q

VORc: normal vs abnormal (and likely cause)

A

normal: can maintain visual fixation w/o c/o blurriness

abnormal: saccadic eye mvmts/intrusions (eyes move off target, eyes jump back to target)
- person c/o blurriness

*(+) central dysfunction

26
Q

what are tests for the VOR and what do abnormal results of these tests usually indicate

A

head thrust/impulse test
head shaking induced nystagmus
dynamic visual acuity

unilateral vestib hypofunction
- aka peripheral dysfunction

27
Q

indication of CNS vs PNS involvement in abnormal VOR findings

A

CNS = vertical nystagmus

PNS / vestib hypofunction
- horizontal nystagmus
- beat away from affected side

28
Q

dynamic visual acuity: normal vs abnormal (and likely cause)

A

normal: 2-line or less difference b/w static and dynamic visual acuity

abnormal:
UVH >3 line loss
BVH > 5 line loss

29
Q

how is the head thrust/impulse test helpful for diff dx

A

helpful to r/o stroke d/t high specificity/sensitivity to detect vestib hypofunction

30
Q

what are 2 important considerations in performing a head thrust / impulse test

A

head flex forward 20-30deg to level horizontal canals

high acceleration thrusts to kick in VOR

31
Q

head thrust: normal vs abnormal (and likely cause)

A

normal: vision fixation maintained

abnormal: corrective saccades after head rotation inability to maintain visual fixation

(+) peripheral hypofunction on side head turned toward during testing
- (+) thrust R, corrective L saccade = R UVH
- (+) thrust R and L = BVH

32
Q

what is an important consideration if you want to perform a head shaking induced nystagmus test

A

only performed w fixation blocked (ie Frenzel lenses)

33
Q

procedure for head shaking induced nystagmus test

A

head flex down 30deg, eyes closed, head rapidly oscillated by PT 20xs
- cued to open eyes just prior to stopping head motion and observe for nystagmus

34
Q

head shaking induced nystagmus: normal vs abnormal (and likely cause)

A

normal: none or 1-2 beats

abnormal: >/= 3 beats
- existing nystagmus accentuated
- usually beats to intact side

35
Q

what do the presentations of head shaking induced nystagmus indicate

A

beats L = R UVH
beats R = L UVH
vertical nystagmus = central vestib