vestib - BPPV Flashcards
what are 8 sx of BPPV and what is the most common one
dizziness
vertigo**
imbalance
“fuzzy”
n/v
nystagmus - horizontal, torsional
inc postural sway
generalized motion sensitivity
what is the typical duration of sx
brief - few seconds
what are provocative factors and commonly associated situations
head mvmts
- rolling over
- bending over/forward
- looking up
transfers, gardening, showering, dentist, hair salon, tying shoes
what are possible PMH / etiologies
head injury
MVA
advanced age (crystals loosened)
hx of prior BPPV
idiopathic
what are 2 important considerations about the sx seen of BPPV
self-imposed activity limitation common
- avoidance of certain mvmts
heightened anxiety/fear - unpredictable
what is the mechanism of BPPV and the 2 main types
otoconia (aka crystals) become dislodged from gelatinous matrix that overlies otolithic membrane (utricular portion) and travels into SCC
- canalathiasis = otoconia float freely in canal
- cupulolithiasis = otoconia can affix themselves to cupula
how does cupulolithiasis impact the presentation of BPPV
when otoconia attach to cupula, makes it heavy and pulls on it
-> causes deflection to last longer than supposed to
= sx will last longer duration than in canalithiasis
what is the most common cause of BPPV and what are 3 other possible causes
idiopathic (58%)
post-traumatic (18.2%)
vestib neuritis (8.6%)
VBI (2.6%)
(other)
what is the most common canal involved with BPPV
posterior
what is the least likely canal to be involved in BPPV
anterior
what specific presentation of lateral canal BPPV is most common
geotropic
what is pure vertical nystagmus often indicative of
CNS
posterior canal BPPV nystagmus
upward & torsional
anterior canal BPPV nystagmus
downward & torsional
lateral canal BPPV nystagmus
sx occur during head rotation to both sides
- geotropic = toward ground = canalathiasis
- apogeotropic = toward ceiling = cupulo-
how do you determine R vs L canal involvement w geotropic vs apogeotropic lateral canal BPPV
intensity/speed of beatign
geotropic
- side of inc intensity = affected side
apogeotropic
- side of dec intensity = affected side
canalathiasis vs cupulolithiasis: onset, duration, latency of sx
ONSET:
- can: delayed 1-40sec (takes ~25sec for otoconium to traverse 1/4 of canal)
- cup: immediate (bc wt of cupula)
DURATION:
- can: short, <60sec (crystal settles)
- cup: longer, >60sec (cont wt on cupula), lasts while person in provoking position
LATENCY:
- can: crescendo and decrescendo
- cup: no latency (immediate onset)
what should be performed prior to exam for BPPV and why
cervical spine screen
- AROM (if ext or rot limited, need modifications to positional provocation tests)
- VBI (concerned ab CNS path)
what is the significance of hx taking during a BPPV exam
high sensitivity (88) and specificity (92)
what are the 4 main steps to a PT exam for BPPV
- hx
- oculomotor exam - r/o CNS vs PNS
- cervical spine screen - AROM, VBI
- positional provocation tests - dx
- dix hallpike test - PCs and ACs
- sidelying test - modified DH
- supine roll test - HCs