TBI - 3a TBI Acute Management Flashcards
what are 6 negative influences on prognosis
coma >1wk
PTA >4wks
inc ICP, sz
DAI, hypoxic/anoxic injuries
delayed access to med care
unchanged GCS/RLA 4wks
what are 4 positive influences on prognosis
support of fam/friends
early improvement in cog
access to specialized rehab
ed/vocational opportunities
why is the support of family and friends a positive influence on prognosis
salient perspective for motivating pts
why are many TBIs medically sedated acutely
give time to dec ICP and to stabilize them hemodynamically and metabolically
what should the ICP be acutely
0-15mmHg
how is the cerebral perfusion pressure (CPP) calculated
MAP - ICP
what are the norms for CPP
60-90mmHg
what can an ICP >20mmHg mean
red flag
- can lead to herniation and bleeding
what are common surgeries for an acute TBI (4)
decompression
drainage/ventricular shunt
remove foreign object
repair MS/integ issues
what are common meds for acute TBIs
sedation
mannitol
barbituates
anti-sz meds
what is a main goal of acute med management for a TBI
cardiopulm stabilization
what are the main things to screen in an ICU assessment (8)
use the CRS-R as a guide
evidence of posturing
EO or EC
track auditory/visual stim
vocalize
active movmt- purposeful?
tactile/painful stim
VS change w external stim
tone
what are s/sx of autonomic dysfunction
**HTN
**tachycardia
hyperthermia
diaphoresis
inc spasticity
dystonia
**ext posturing
pupil dilation
vomiting
what are 2 other terms synonymous w autonomic dysfunction
storming
sympathetic storming
how can autonomic dysfunction present
recurrent and episodic
what is autonomic dysfunction and what is it caused by
exaggerated response to noxious external stim
- ie bed sore, catheter, positioning
what is the immediate response to autonomic storming
let nurse know
- can be managed w meds (IV)
try to identify and remove external stim
family ed on what to avoid
what are ex of primitive reflexes that may return
ATNR
STNR
what sz activity can be seen
partial - simple vs complex
generalize
- absent vs tonic clonic
what are changes in ms tone that can be seen
hypotonicity
spasticity
rigidity
- decorticate
- decerebrate
decorticate vs decerebrate posturing: presentation and cause
decorticate
- flex UEs, ext LEs
- lesion above upper brainstem
decerebrate
- ext of UEs and LEs
- lesion in brainstem below superior colliculus
what are 6 NM/sensory clinical manifestations of acute TBIs
motor control
sensation
proprioception/kinesthesia
coordination
motor planning
ms atrophy