Unti 28 Peds Cardiac Disorders Flashcards

1
Q

In fetal circulation, how does the heart get oxygenated?

A

By the placenta

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2
Q

Regarding fetal circulation, what does the Foramen Ovale do?

A

Pumps blood from the Right to Left Atrium

Closes usually half an hour after birth due to pressure changes

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3
Q

Regarding fetal circulation, what does the Ductus arteriosus do?

A

Shunts blood from the pulmonary artery to the descending aorta

Could take up to 4 days to close

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4
Q

Describe Congenital Heart Defects and Acquired Heart Defects.

A

Congenital:

  • Present at birth
  • Family Hx/genetics
  • Down syndrome carries higher risk
  • High risk maternal factors such as Age > 40, diabetes, substance abuse, and rubella during pregnancy

Acquired:

  • Technically heart failure
  • Cardiomyopathy
  • Infection
  • Toxins
  • Hypertension/hyperlipedemia
  • Kawasaki Disease
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5
Q

What are the classifications of Congenital Heart Defects?

A

[Increased pulmonary blood flow]
-abnormal structure pushes more blood towards lungs, less blood to body. Heart works harder

[Decreased pulmonary blood flow]
-Abnormal structure pushes less blood to lungs, deoxygenated blood to body

[Obstructive]
-Narrowing stricture, heart works harder

[Mixed]

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6
Q

Briefly describe Increased Pulmonary Blood Flow.

A
  • Left to right shunt where oxygenated blood re-enters the pulmonary circulation.
  • R. Ventricle strain, dilation, hypertrophy because it is working harder
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7
Q

Name the defects with Increased pulmonary blood flow, state what each means, and what happens regarding all of them.

A

VSD (ventriculoseptal defect): abnormal opening in the ventricular septum (slight murmur)

ASD (atrioseptal defect): abnormal opening in the atrial septum (slight murmur)

PDA (patent ductus ateriosus): ductus ateriosus fails to close (you’ll hear machine like murmur)

In all of these:

  • Blood is recirculated through the LUNGS
  • Less blood is available to rest of the body
  • Heart works harder
  • May correct themselves, if not usually 1 Sx with excellent prognosis

If defects not treated, or worsens, then will result in heart failure and/or pulmonary hypertension

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8
Q

What is the most common congenital heart defect?

A

VSD - ventriculoseptal defect

classified as an increased pulmonary blood flow congenital heart defect

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9
Q

What is the obstructive congenital heart defect? Describe it. What will nurses look for? How is it fixed?

A

Coarctation of the aorta

  • It is the narrowing of the aorta
  • Decreased blood flow below defect
  • Increased pressure above defect
  • Femoral pulses weak or absene
  • Radial pulses bounding
  • Upper extremity hypertension

can be fixed with patch to aorta

Nurses will assess pulses, BP upper and lower

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10
Q

Describe the defects with Decreased Pulmonary blood flow and what you will see.

A

They are right-to-left shunts meaning deoxygenated blood enters systemic circulation.

  • Results in “blue babies”
  • Decreased pulmoecnary blood flow
  • Central cyanosis
  • Low O2 stats
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11
Q

What is the treatment for defects with decreased blood pulmonary blood flow?

A

Treatment often requires emergency treatment

Sx in several stages

Prognosis varies depending on extent of defect

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12
Q

Name the most common complex lesion and explain it.

A

[Tetralogy of Fallot] is the most common complex lesion and it has to due with FOUR problems of deoxygenated blood entering the oxygenated side at the same time.

FOUR heart defects

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13
Q

What is the medical management of Tetralogy of Fallot?

A
  • Keep Ductus Arteriosus patent
    with prostiglandins
  • Vasodilators, diuretics, digoxin, ACEI’s
  • Activity/Rest balance to prevent fatigue
  • Surgical management required
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14
Q

What are signs and symptoms of Tetralogy of Fallot?

A
  • Hyper-cyanotic/blue called TET spells
  • Choking spells with periods of dyspnea

-RELIEF with squatting or placing infant in knee to chest position to increase blood flow to lungs (specific to TOF)

  • Clubbing (from chronic hypoxemia)
  • Polycythemia (increased RBC from chronic hypoxemia)
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15
Q

What is the newborn cardiac screening?

A

Using pulse ox:
Pre and post ductal screening meaning:
the RIGHT HAND and whichever foot.

  • Baby should be 25-48 hrs old
  • O2 stat needs to be >95%
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16
Q

Other testing for congenital heart defects include what?

A
  • Echocardiogram (shows function of heart)
  • Electrocardiogram
  • Chest x-ray
  • Cardiac MRI
  • Cardiac Cath
  • Stress test (older kids)
17
Q

What are the general manifestations of congenital heart disease in newborns?

A
  • May be none!
  • Intercostal retractions, difficulty breathing, tachypnea >80min, crackles, wheezes
  • Central cyanosis
  • Tachycardia >160/min
  • Uncontrollable crying
  • Altered LOC
  • Seizure, cardiac arrest
18
Q

What are the two definition of heart failure?

A
  1. Heart pumps well, but volume of blood is insufficient or structure does not work
  2. Weak heart muscle not pumping enough blood
19
Q

Where does the backup of fluid end up in right sided heart failure and left sided heart failure?

A

Right sided heart failure = fluid into the liver, veins, extremities

Left sided = fluid into the lungs

20
Q

What are the signs and symptoms of heart failure?

A
  • Fatigue
  • Change in skin temperature
  • Jugular vein distention
  • Tachypnea
  • Poor appetite
  • Fluid retention weight gain
  • Edema in hands, face, legs, ankles, abdomen
  • Cough and congestion in lungs
  • SOB (lungs fill with fluid)
21
Q

Why is their fluid retention with heart failure?

A

Body tells itself to keep fluid because of low oxygenation (RAAS system)

22
Q

What are the respiratory symptoms of heart failure?

A
  • Backup of fluid into the lungs
  • Cough and congestion into the lungs
  • Clubbing
  • Polycythemia
  • Tachypnea
23
Q

What is the treatment for heart failure in children?

A
  • For congenital heart defects or valve disease = Sx or transplant
  • Medications
  • O2 therapy
  • Occasionally fluid restriction
  • NGT feedings (can use breastmilk or HI calorie formula)
  • Pacemaker
  • LVADs
24
Q

What are nursing interventions for heart failure?

A
  • Position for comfort and increased oxygenation
  • Medications
  • Rest and activity as tolerated
  • Anticipate needs to minimize stress (parents can help here)
  • Avoid extreme temp changes
  • Strict I and Os
  • Daily weights
  • Standard precautions to prevent infections (infections will increase work for the heart)
  • Teach good handwashing
  • Limit visitors with infection
25
Q

What is the best way to monitor fluid balance/retention?

A

daily weights

26
Q

What medications are used for heart failure?

A

Digoxin -slows and strengthens heart beat

Potassium -electrolyte to replace loss from diuretics

furosemide (Lasix) - diuretic to reduce preload by decreasing reabsorption of sodium

propran’olol’ [beta-blockers] -lowers HR and worload
(BLOCKS sympathetic stimulations)
NOT GIVEN WITH LOW HR

enala’pril’ [ACEI’s] to decrease afterload; increases vasodilation
STOP IF PT COUGHING = contraindication

spironalactone -aldosterone antagonist, potassium diuretic which interrupts RAAS

27
Q

What is the use of Digoxin, what does it do, and how is it administered/rules to administration?

A

-Used for arrhythmias and heart failure
-Increases strength and efficiency of the heart
-Controls rate and rhythm of the heartbeat
(available as tablet, liquid, IV)

[The rules]
Give q12h: 1 hr BEFORE or 2 hrs AFTER feeding
DO NOT mix with foods or fluids
HOLD when checking apical pulse for 1 minute if less than 90-110 in small child or <70 in older child
Brush teeth or rinse mouth if giving liquid digoxin because it stains teeth
DO NOT give second dose if child vomits
DO NOT give if PT has hypokalemia
TWO NURSES to check dose prior to administration

28
Q

What are some parent teaching points about Digoxin?

A
  • Write down dose and time after giving
  • Refill prescription before previous is consumed
  • If dose is missed and more than 4 hrs pass skip dose, if 2 doses missed, call HCP
  • DO NOT increase or double doses
  • Keep in safe place
  • Do not give any medicine without approval of HCP
29
Q

What are signs of Digoxin toxicity?

A
  • Bradycardia*
  • Dysrhythmias*
  • Anorexia
  • N/V
  • CNS disturbances (drowsiness, confusion, headache, visual disturbances)
30
Q

What is an acquired heart disease? Describe it.

A

Kawasaki Disease:

  • Acute systemic vasculitis of unknown cause.
  • Arteries, veins, and capillaries are EXTENSIVELY INFLAMMED
  • May progress to coronary artery aneurysms, etc. death from MI (rare)
31
Q

What are the symptoms of Kawasaki Disease?

A

Fever 5+ days

Conjunctival inflammation*

Change in oral mucosa

Change in extremities (edema, redness, peeling skin)

Rash

32
Q

What is the management of Kawasaki Disease, both medication and nursing care?

A

Medication:

  • Hi dose IV gamma globulin
  • ASPIRIN large doses to decrease inflammation q6hrs
  • warfarin if aneurysm exists

Nursing care:

  • I and Os
  • Daily weight
  • Relieve symptoms
  • Assess for CHF
  • Quiet environment
  • Discharge teaching
33
Q

What is clubbing?

A

-Abnormal enlargement of distal phalanges assoacited with chronic hypoxia