Unit 3 - Descending tracts/ UMN/ LMN Flashcards
What is a descending tract?
neural pathway by which motor signals are sent from the brain to LMN (via UMN)
what is the difference between UMN and LMN?
upper: axons remain CNS and cell body in cortex/ brainstem
lower: cell body in ventral horn and axon supplies muscle
what are the two types of descending tracts? give origin
pyramidal: voluntary control of body and face
- arise from cortex
extrapyramidal: involuntary and autonomic control of all musculature
- arise in brainstem
what is the difference between the two types of pyramidal tracts?
- corticospinal: supplies musculature of all body
- corticobulbar: supplies head and neck
describe the pathway of the corticospinal tract
neurons from primary cortex, premotor cortex, supplementary motor area and somatosensory area converge
- descend through internal capsule to cris cerebri, pons to medulla
- tract splits in two at medulla into lateral and anterior tract
- lateral decussates and descends spinal cord to ventral horns to supply body
- anterior remains ipsilateral and descends cord and terminates in ventral horn of cervical/ upper thoracic levels
describe the pathway of the corticobulbar tract
neurons from the primary motor cortex converge and descend through the internal capsule to brainstem (cris cerebri, pons, medulla)
- terminate in motor nuclei of cranial nerves and synapse with LMN
- innervate bilaterally
what are the two exceptions to the corticobulbar tract?
facial/ hypoglossal nerve - contralateral innervation
what are the four extrapyramidal tracts?
vestibulospinal
reticulospinal
rubrospinal
tectospinal
what is the type of innervation and function of the extrapyramidal tracts?
vestibulospinal - ipsilateral - balance/posture - axial muscles/ extensors
reticulospinal - ipsilateral - balance/ posture - axial muscles /proximal limb
rubrospinal - contralateral - single finger movements
tectospinal - contralateral - head movement
what would be the effect of a lesion to extrapyramidal tract?
dyskinesia - abnormality of voluntary movement
what would damage to corticospinal tract cause?
damage = UMN Lesion
- hypertonia (increased tone)
- hyperreflexia (increased muscle stretch reflex)
- clonus (involuntary rhythmic contraction of antagonist muscles)
- babinski’s sign (when hard object scraped on bottom of foot toes go into extension - extensor plantar response)
- muscle weakness
- spastic paralysis - continued contractions due to dissipation of UMN signal
what would damage to corticobulbar tract cause?
mild muscle weakness
- facial nerve - spastic paralysis of contralateral lower quadrant of face
- hypoglossal nerve - tongue deviation contralaterally
what is a motor unit?
1 LMN and skeletal muscle cells it contacts/ controls
smaller muscles require what type of motor units?
smaller motor units - rapid, precise control
what is tone?
muscle’s resistance to passive stretch during resting state