Unit 3 - Descending tracts/ UMN/ LMN Flashcards

1
Q

What is a descending tract?

A

neural pathway by which motor signals are sent from the brain to LMN (via UMN)

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2
Q

what is the difference between UMN and LMN?

A

upper: axons remain CNS and cell body in cortex/ brainstem
lower: cell body in ventral horn and axon supplies muscle

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3
Q

what are the two types of descending tracts? give origin

A

pyramidal: voluntary control of body and face
- arise from cortex
extrapyramidal: involuntary and autonomic control of all musculature
- arise in brainstem

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4
Q

what is the difference between the two types of pyramidal tracts?

A
  • corticospinal: supplies musculature of all body

- corticobulbar: supplies head and neck

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5
Q

describe the pathway of the corticospinal tract

A

neurons from primary cortex, premotor cortex, supplementary motor area and somatosensory area converge

  • descend through internal capsule to cris cerebri, pons to medulla
  • tract splits in two at medulla into lateral and anterior tract
  • lateral decussates and descends spinal cord to ventral horns to supply body
  • anterior remains ipsilateral and descends cord and terminates in ventral horn of cervical/ upper thoracic levels
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6
Q

describe the pathway of the corticobulbar tract

A

neurons from the primary motor cortex converge and descend through the internal capsule to brainstem (cris cerebri, pons, medulla)

  • terminate in motor nuclei of cranial nerves and synapse with LMN
  • innervate bilaterally
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7
Q

what are the two exceptions to the corticobulbar tract?

A

facial/ hypoglossal nerve - contralateral innervation

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8
Q

what are the four extrapyramidal tracts?

A

vestibulospinal
reticulospinal
rubrospinal
tectospinal

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9
Q

what is the type of innervation and function of the extrapyramidal tracts?

A

vestibulospinal - ipsilateral - balance/posture - axial muscles/ extensors
reticulospinal - ipsilateral - balance/ posture - axial muscles /proximal limb
rubrospinal - contralateral - single finger movements
tectospinal - contralateral - head movement

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10
Q

what would be the effect of a lesion to extrapyramidal tract?

A

dyskinesia - abnormality of voluntary movement

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11
Q

what would damage to corticospinal tract cause?

A

damage = UMN Lesion

  • hypertonia (increased tone)
  • hyperreflexia (increased muscle stretch reflex)
  • clonus (involuntary rhythmic contraction of antagonist muscles)
  • babinski’s sign (when hard object scraped on bottom of foot toes go into extension - extensor plantar response)
  • muscle weakness
  • spastic paralysis - continued contractions due to dissipation of UMN signal
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12
Q

what would damage to corticobulbar tract cause?

A

mild muscle weakness

  • facial nerve - spastic paralysis of contralateral lower quadrant of face
  • hypoglossal nerve - tongue deviation contralaterally
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13
Q

what is a motor unit?

A

1 LMN and skeletal muscle cells it contacts/ controls

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14
Q

smaller muscles require what type of motor units?

A

smaller motor units - rapid, precise control

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15
Q

what is tone?

A

muscle’s resistance to passive stretch during resting state

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16
Q

what are signs of LMN lesions?

A

muscle weakness

  • hypotonia
  • hyporeflexia
  • fasciculations - involuntary twitches of skeletal muscle
  • atrophy