Traumatic reticulo pericarditis Flashcards

1
Q

Aetiology and pathogenesis

A
  • Sharp linear metallic FB ingested
    ◦ Typically wires from tyres used to weigh down silage cover
    ◦ Associated with clamp silage feeding
  • FB penetrates reticular wall -> into pericardial sac (through diaphragm)
    ◦ Not always cranial direction!!!
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1
Q

Effects on heart function

A
  • Cardiac tamponade
  • Reduced cardiac output
    ◦ Forward failure
  • Progresses to CHF
    ◦ Backward cardiac failure
    ◦ C/s related to this (e.g. oedema)
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2
Q

History

A
  • Typically non-specific
    ◦ Milk drop
    ◦ Non-specific illness 1-2 weeks prior to exam that appeared to resolve before recurring
    ◦ Inappetence
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3
Q

Presenting signs - early stages

A
  • Pain
    ◦ Abducted elbows
    ◦ Withers positive
    ◦ Arched back
    ◦ Firm palpation
  • Rubbing/friction sounds on auscultation
  • Tachycardia
  • Pyrexia +/- associated BRD signs
  • In the early (acute) stages, heart sounds can change daily
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4
Q

Presenting signs - later stages

A
  • Muffled heart sounds (bilateral)
    ◦ Regular rhythm
    ◦ Splashing, squeaking, rubbing sounds
  • Difficulty palpating apex beat
  • +/- pyrexia
  • Signs related to congestive heart failure
    ◦ Jugular distension, ventral oedema, tachycardia, dyspnoea, injected scleral vessels
  • In the later (chronic) stages, heart sounds tend to be consistent
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5
Q

How would you diagnose?

A

Ultrasound

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6
Q

Treatment

A
  • Early stages (i.e. before fibrinous septic pericarditis has developed)
    ◦ Magnet
    ◦ Broad-spec antibiotics (e.g. amoxicillin, oxytetracycline)
  • Late stages (septic pericarditis has developed)
    ◦ Prognosis = poor to hopeless
    ◦ PTS on welfare grounds
    ◦ Pericardiotomy/percardiostomy and drainage are reported
    ‣ Poor outcomes
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7
Q

Prevention

A
  • Due diligence regarding silage (and other feed) management
  • Magnets
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