Traumatic reticulo pericarditis Flashcards
1
Q
Aetiology and pathogenesis
A
- Sharp linear metallic FB ingested
◦ Typically wires from tyres used to weigh down silage cover
◦ Associated with clamp silage feeding - FB penetrates reticular wall -> into pericardial sac (through diaphragm)
◦ Not always cranial direction!!!
1
Q
Effects on heart function
A
- Cardiac tamponade
- Reduced cardiac output
◦ Forward failure - Progresses to CHF
◦ Backward cardiac failure
◦ C/s related to this (e.g. oedema)
2
Q
History
A
- Typically non-specific
◦ Milk drop
◦ Non-specific illness 1-2 weeks prior to exam that appeared to resolve before recurring
◦ Inappetence
3
Q
Presenting signs - early stages
A
- Pain
◦ Abducted elbows
◦ Withers positive
◦ Arched back
◦ Firm palpation - Rubbing/friction sounds on auscultation
- Tachycardia
- Pyrexia +/- associated BRD signs
- In the early (acute) stages, heart sounds can change daily
4
Q
Presenting signs - later stages
A
- Muffled heart sounds (bilateral)
◦ Regular rhythm
◦ Splashing, squeaking, rubbing sounds - Difficulty palpating apex beat
- +/- pyrexia
- Signs related to congestive heart failure
◦ Jugular distension, ventral oedema, tachycardia, dyspnoea, injected scleral vessels - In the later (chronic) stages, heart sounds tend to be consistent
5
Q
How would you diagnose?
A
Ultrasound
6
Q
Treatment
A
- Early stages (i.e. before fibrinous septic pericarditis has developed)
◦ Magnet
◦ Broad-spec antibiotics (e.g. amoxicillin, oxytetracycline) - Late stages (septic pericarditis has developed)
◦ Prognosis = poor to hopeless
◦ PTS on welfare grounds
◦ Pericardiotomy/percardiostomy and drainage are reported
‣ Poor outcomes
7
Q
Prevention
A
- Due diligence regarding silage (and other feed) management
- Magnets